Personality disorders II Flashcards
prevalence of PDs
Prevalence estimates for Personality Disorders vary considerably across studies (for reviews see Coid et al., 2006; Torgersen et.al., 2001 and Weissman, 1993).
- Coid et al. (2006) used large sample
Personality disorders are relatively common and general estimates indicate that they affect around 10-13% of the general population (Weissman, 1993).
- Can be distressing to be labelled
- In the general population, schizotypal, histrionic, dependent and obsessive-compulsive personality disorders are the most common.
- Narcissistic personality disorder is the rarest – approximately 0.2% in the general population
Coid et al. (2006)
reviewed prevalence rates of PDs and report a range of 3.9% to 22.3%.
- The prevalence rates for PDs in a British population were 4.4% – this is relatively low compared with many previous studies.
- Participants with any personality disorder were more likely to be male, older, separated or divorced, unemployed or economically inactive, of lower social class, living in rented accommodation and living in an urban area – can be consequence of disorder rather than causing it
- Noted gender differences
- Correlational data
Those with cluster A disorders were three times more likely to have been in local authority care before the age of 16 years
- Foster care
- Neglect or abuse before foster care
Those with cluster B disorders were more likely to have had a criminal conviction, to have spent time in prison and have been in local authority or institutional care – poor backgrounds
Those with cluster C disorders were more likely to have received psychotropic medication and/or counselling
gender differences in personality traits
Gender differences in personality appear in childhood and are stable over time (for an overview see Paris 2007)
- Young girls more skilled at overcoming and hiding issues – female autistic children less likely to be diagnosed than males – better at developing coping skills
A meta-analysis by Feingold (1994)
Goodwin and Gotlib (2004)
While socialization theories are influential in explaining these variations, gender differences in personality are also rooted in biology.
Personality disorders can be understood as dysfunctional exaggerations of normal personality traits (e.g., Livesley, Jang, & Vernon, 1998).
The most striking difference is the higher prevalence of antisocial personality disorder in men (Black, 1999), with 80% of cases of antisocial personality being men.
The personality trait that underlies antisocial personality disorder (ASPD) has been termed impulsive aggression (Siever & Davis, 1991), a characteristic more commonly found in males.
Feingold (1994)
males were more assertive and have higher self-esteem (has been replicated in the literature), while females were higher in extraversion (less common finding), anxiety, trust and nurturance
Goodwin and Gotlib (2004)
studied the Five Factor Model of Personality in a large community sample - neuroticism, agreeableness, conscientiousness and extraversion were all higher in women, while open-ness to experience was higher in men.
costa, terracciano and McCrae (2001)
found that while gender differences can be exaggerated or reduced by cultural forces, similar gender effects on traits are seen in cultures all over the world.
- This shows that gender differences on personality, while they may be reinforced by socialization, are not entirely socially constructed – something else going on because of universal nature
Gender differences in the prevalence of PDs
vary across studies but tend to be higher for men (Coid et al., 2006):
- Men were more likely to have a disorder (5.4%) compared with women (3.4%).
- The prevalence of antisocial personality disorder was five times greater in men (1.0%) than in women (0.2%).
- Oltmanns & Emery, (2004), also provide evidence showing a strong tendency for Antisocial Personality Disorder to be more prevalent in men (4.5%) than women (0.8%).
- All personality disorder categories were more prevalent in men, apart from the schizotypal category.
possible gender biases in diagnosis
Criteria defining a disorder might derive from particular gender stereotypes and this can affect diagnosis.
Possible gender differences in manifestation of symptoms of different disorders (e.g., attention seeking – hyperfeminity/hypermasculinity).
Criteria for diagnosis of APSD includes a prior history of conduct disorder (more prevalent among boys/males). Aggressiveness and a pattern of criminal behaviour (both more common in men)
diagnosis can be controversial
Several flaws in the prevailing system of diagnosis help explain inaccuracies in estimating the overall prevalence rate for PD.
- Co-morbidity – most people with a diagnosable disorder typically have two or more disorders.
- Research suggests co-morbidity is normative with personality disorders.
- If a person meets the criteria for one personality disorder there is a 25-50% probability that they will also meet criteria for a second personality disorder (e.g., Oltmanns & Emery, 2004).
prevalence and co-morbidity
Coid et al (2006) – co-morbidity of personality disorders
- The mean number of personality disorder diagnoses among those who qualified for such a diagnosis was 1.92; of these,
- 53.5% had one disorder only
- 21.6% had two disorders
- 11.4% had three disorders
- 14.0% had between four and eight diagnoses
- Classification of personality disorder by cluster showed
- Cluster C to be the most frequent (2.6%)
- Cluster A (1.6%)
- Cluster B (1.2%) least prevalent.
diagnosis and comorbidity
Co-morbidity of personality disorders occurs in part because the categories are imprecisely defined and they have overlap.
- Many of the PDs share common features: e.g., social isolation is involved in various disorders including schizotypal, schizoid and avoidant.
- Some PDs share common features with clinical conditions also detailed in DSM-V – social phobia and avoidant PD).
Co-morbidity undermines the categorical approach and points instead to a dimensional approach in which people can be characterised according to scores on several personality dimensions.
- Problems due to categorical approach
development of PDs
The pervasive nature of personality disorders and the fact that they are resistant to change by interventions has led to the use of the terms ‘development’ and ‘management’ rather than ‘aetiology’ and ‘treatment’ (Holmes, 2010).
- Ideas around treatment side are changing
Personality disorders begin to be evident in childhood and early adolescence and persist through adulthood.
- This indicates possible genetic predispositions but traditional explanations have highlighted early disturbances of development – e.g., maternal deprivation, abuse, disruptive childhood – could occur in school as well
- Parents might be reacting to pre-existing challenging behaviours, which might be early manifestations of personality problems.
- Supported by evidence showing siblings who experienced similar parenting do not exhibit such profound personality distortions.
- Don’t want to label the child too early
Environmental, genetic and biological factors influence the development of personality disorders.
- Note of caution:
- most of the research examining personality disorders is correlational not experimental. Therefore, the direction of causality is not known.
Biological, genetic and environmental factors are highly intermingled making it extremely difficult to establish one particular cause of a disorder.
- Human personality both normal and disordered has multiple causes.
biological influences
Various biological and neuropsychological factors have been associated with personality disorders.
- E.g., particularly high or low levels of neurotransmitters dopamine and serotonin have been related to each of the three clusters of personality disorders.
- But it is not known if it’s the level of a neurotransmitter that causes the personality disorder or whether having the disorder causes the high/low level of the neurotransmitter.
- Traumatic brain injury – ‘Phineas Gage’ is an example of an abrupt personality change following neurological damage.
- Suffered severe damage to frontal lobes and developed marked anti-social traits.
- Dysfunction due to brain being injured
- Williams, Mewse et al.
biological influences - Siever and Davis (1991)
propose a neuropsychological model illustrating four psychological pathways through which neurotransmitters might influence personality disorder clusters:
- cognitive/perceptual processes (two of the remaining lectures will be focussed on these processes).
- impulsivity/aggression
- affect regulation
- anxiety/inhibition
genetic influences
Two studies that have used heritability estimates (can be problematic) to examine genetic influences on all personality disorders indicate quite substantial genetic components:
- Torgersen et al., (2001) (adult DZ and MZ twins) present heritability estimates for each of the 12 personality disorders individually as well as by cluster.
- Heritability estimates for Personality Disorders: Cluster A = 0.37, Cluster B = 0.60, Cluster C = 0.62
- Coolidge et al., 2001 (children aged 4 – 15 years, DZ and MZ twins).
- Heritability estimates for each of the twelve personality disorders ranged from 0.81 - 0.50 with an average heritability coefficient across the disorders of 0.75.
- These heritability estimates suggest that genetic influences on personality disorder show a range similar to those estimated for normal personality.
The same problems surround heritability estimates of disordered personality as those described in earlier lectures concerning heritability estimates and normal personality / intelligence.