periodontology Flashcards

1
Q

in absence of disease how does the width of attached gingivae change with age

A

increases

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2
Q

staining used to differentiate keratinised gingivae

A

schillers iodone solution

keratinised = orange, other = purple/blue

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3
Q

symptoms and signs of primary herpetic gingivostomatits

A

fever, pyrexia, headaches, malaise, dysphagia, regional lymphadenopathy
aggressive marginal gingivitis, fluid filled vesicles with grey membranous covering - burst to form yellow/grey ulcer
heal without scarring 14 days
on gingiva, tongue, hard palate, buccal mucosa

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4
Q

what is plasma cell gingivitis

A

contact hypersensitivity reaction

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5
Q

clinical features of plasma cell gingivitis

A

fiery-red gingiva, extreme pain

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6
Q

3 disorders causing desquamative gingivitis

A

MMP
lichen play
pemphigus vulgaris

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7
Q

clinical features of desquamative gingivitis

A

fiery-red

keratinised gingiva

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8
Q

causes of necrotising gingivitis

A

depressed immune system - AIDS, malnutrition
smoking
stress
poor OH/preexisiting PD

fusiform-spirochaetal bacteria

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9
Q

clinical features of ANUG

A

punched out ulcers, yellow/grey pseudomembranous slough
interdental papillae effected first
painful
halitosis

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10
Q

what is calculus

A

plaque which has become mineralised by calcium + phosphate ions in saliva

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11
Q

5 plaque-retentive factors

A
overhanging restoration
defective crown margin
bridge pontics
partial denture
orthodontic appliances
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12
Q

potential spread of perio/endo lesions to make a periodical-endo lesion

A
apical foramina
lateral/furcation accessory canals
exposed dentinal tubules
root defects by caries
fractures
perforations during operative procedures
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13
Q

prevalence of gingival hyperplasia with different drugs

A

phenytoin 50%
ciclosporin 30%
calcium channel blockers 20%

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14
Q

condition causing ‘strawberry’ appearance of gingiva + hyperplastic gingivitis with petechiae

A

wegeners granulomatosis

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15
Q

what is an epulide

A

localised hyperplastic lesions on gingiva

trauma or chronic irritation - granulation tissue

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16
Q

how does occlusal interference affect the periodontium

A

may increase tooth mobility due to compensating widening of PMS
does not cause PD but may make it worse

17
Q

effects of traumatic occlusal forces

A
pain
fracture
faceting
attrition
bruxism
TMD
tooth mobility
18
Q

syndromes and medical conditions causing PD

A

downs syndrome
papillon-lefevre
Ehlers-danlos
hypophosphatasia

19
Q

3 factors contributing to reduced probing depths after RSD

A

shrinkage of soft tissue as inflammation resolves
increased resistance to probe penetration as inflammation resolves
formation of LJE at base

20
Q

indications for crown lengthening

A

increase ferrule
access for subginigval crown finish lines/impressions
sub gingival caries
root fractures/resorption in cervical third of tooth root
aesthetic improvement of anterior teeth

21
Q

3 surgerys used to improve recession

A

free gingival graft
pedicle sliding graft
sub epithelial connective tissue graft

22
Q

microorganism most strongly associated with molar incorrectly pattern periodontitis

A

actinomycetemcomitans

often heridary + its show neutrophil deficiencies

23
Q

how does GTR work

A

membrane placed, blood clot forms below and acts as scaffold for migrating cells that produce new periodontal ligament, cementum + bone
gingival epithelium excluded