Peds Cardiac Flashcards
Changes that occur with baby’s first breath of air
Arterioles dilate significantly, high pressure system becomes low pressure system, shunts close
What are the two types of cardiac defects?
Congenital (CHD) and acquired
Characteristics of acquired cardiac defects
Disease or infection-related, autoimmune response, environmental factors, familial tendencies
Most common cause of congenital heart defect
Complex interaction between genetics and environmental factors
Prenatal risk factors for congenital heart defect
Chronic disease, alcohol/illicit drugs, infection, IUGR or macrosomia
Prenatal chronic diseases that increase risk of heart disease in infants
Autoimmune, lupus, diabetes
Prenatal infections that increase risk for fetal cardiac anomalies
Rubella, toxoplasmosis
Genetic risk factors for congenital heart defect
Marfan, hypertrophic cardiomyopathy, frequent loss or SIDS, Down syndrome or Turner syndrome
Causes of CHF
Volume overload, pressure overload, decreased contractility, high cardiac demands
HF characterized by elevated CVP and venous engorgement
Right-sided
HF characterized by increased pressure in pulmonary veins and LA, increased pulmonary pressures due to lungs congested with blood, and pulmonary edema
Left-sided
Cardiac and circulatory manifestations of CHF
Tachycardia during rest (compensatory mechanism), gallop rhythm, weak pulses, slow cap refill, low BP
Integumentary manifestations of CHF
Diaphoresis, mottled skin, cold extremities
Respiratory manifestation of CHF
Poor exercise tolerance (“unable to keep up with peers”)
Neurological manifestations of CHF
Fatigue and irritability
S/S of left sided heart failure (pulmonary congestion)
Tachypnea, dyspnea, hypoxemia, bronchial edema and coughing
S/S of right-sided heart failure (systemic venous congestion)
Hepatomegaly, weight gain, gravity dependent swelling, visually distended veins
CHF management: improving cardiac function
Digoxin, ACE inhibitors, vasodilation
CHF management: removing accumulated fluid
Lasix
CHF management: decreasing cardiac demands
Neutral-thermic (newborns), treat infections, semi-fowlers, sedation/rest
CHF management: improving oxygenation
O2 supplementation
Pediatric weights that are concerning
Major changes in weight percentiles, 5% or lower, 95th or higher
Non-specific CHF manifestations
Poor feeding, respiratory difficulty (tachypnea/tachycardia), FTT, developmental delays, non-cardiac congenital defects
Cardiac-specific manifestations of CHF
Clubbing of fingers, circumoral cyanosis, hypercyanotic/tet spells, sweating, polycythemia
Hypercyanotic spells or “tet” spells are most often associated with
Tetralogy of Fallot
Interventions for tet spells
Stay calm, knee-chest position, morphine
Screening performed on newborns 4 hours after life that is highly effective at detecting congenital heart defects
Critical Congenital Heart Defect Screening (CCHD)
CCHD is highly effective at detecting what 7 congenital heart defects?
Hypoplastic left heart syndrome, pulmonary atresia, tetralogy of fallot, total anomalous pulmonary venous return, transposition of the great arteries, tricuspid atresia, and truncus arteriosis
Describe CCHD screening
Non-invasive and quick, pulse ox placed on foot and hand comparing oxygenation on different parts of body to help catch defects early
An infant would pass the CCHD screening of pulse ox is greater than ___% in RH or F AND a difference of ___% or less between RH and F
95; 3
An infant would fail CCHD screening if pulse ox is ___% or lower in RH or foot
89
If an infant’s pulse ox is between ___ and ___% in RH or F OR a difference of ___% or higher between RH and F, then repeat the CCHD screen in 1 hour
90-94; 4
What should be done If an infant reads the same pulse ox after repeating CCHD screening?
Repeat screen in 1 hour again; if no change, the infant fails the CCHD screen
Diagnostic that assesses the electrical conduction within the heart
ECG
Diagnostic that looks for signs of HF by assessing changes in size and position of organs
Chest X-Ray
Diagnostic that assesses/measures valves, chambers, and EF; may be ordered if CHF screening is failed
Echocardiography
Nursing considerations for CHF
Patients at risk for neurologic complications, developmental delays (cognitive and motor), thrombotic events, and bacterial endocarditis
Examples of increased pulmonary blood flow defects
Arterial septal defect, ventricular septal defect, and patent ductus arteriosis
Characteristics of atrial septal defect
Blood flow from left atria (high pressure) to right side (low pressure), right atrial/ventricular enlargement BUT HF is rare, pulmonary vasculature changes only occur after decades
S/S of atrial septal defect
Asymptomatic, HF after 3 or 4 decades, murmur, risk for atrial dysrhythmia
Increased pulmonary blood flow defects result from an abnormal connection between the two sides of the heart being either the _________ or the _____ _______
Septum; great vessels
Increased pulmonary blood flow defects lead to _________ blood volume on the right side of the heart, _________ pulmonary blood flow, _________ systemic blood flow, and symptoms of _____.
Increased; increased; decreased, HF