Pediatrics Flashcards
Routine management of the newborn
physical exam
apgar
eye care
dz prevention and screeing
Routine management of the newborn
right after birth
mouth and nose are suctioned
clamping and cutting of umpbilical cord
dried wrapeed in clean towels and placed under a warmer
gentle rubbing or sitmulating hte heels helps to stimulate crying and breathing
Routine management of the newborn
intubation and ABG analysis are indicated if
resp distress
Routine management of the newborn
nasogastric tube
indicated when GI decompression isneeded
Routine management of the newborn
antibiotics
indicated for sepsis
late preterm neonate
34-37 weeks
Term neonate
38 weeks or more
Normal vital signs in a newborn
RR - 40-60
HR - 120-160
vital signs in a newborn are always
higher, babies are faster
APGAR Score: Newborn Assessment
overview
measurement for the need and effectiveness of resuscitation, it does not predict mortality
APGAR Score: Newborn Assessment
one minute score
evaluates conditions during labor and delivery
APGAR Score: Newborn Assessment
five minute score
evaluates the response to resuscitative efforts
a low apgar score is not associated with
cerebral palsy
APGAR Score: Newborn Assessment
what it stands for
appearance pulse grimace activity respiration
APGAR Score: Newborn Assessment
appearance
skin color/complexion
0 - blue all over
1 - normal except extremities
2 - normal all over
APGAR Score: Newborn Assessment
pulse
pulse rate
0 - <60bpm or asystole
1 - >60 bpm but <100bpm
2 - >100bpm
APGAR Score: Newborn Assessment
grimace
reflex irritability
0 - no response
1 - grimae/feeble cry
2 - sneeze/cough
APGAR Score: Newborn Assessment
activity
Muscle tone
0 - none
1 - some flexion
2 - active movement
APGAR Score: Newborn Assessment
respiration
breathing
0 - absent
1 - weak or irregular
2 - strong
Newborn Eyecare
conjunctivitis day 1
mostl iley cause is chemical irritation (silver nitrate, this is not an allergy)
Newborn Eyecare
conjunctivitis days 2-7
most likely neisseria gonorrhoeae (gram neg diplo, prevent w/ ointments, treattwith ceftriaxone)
Newborn Eyecare
conjunctivitis day more than 7 days after deliver
most likely due to chlamydia trachomatis (not effectively prevented by prophylaxis, treate with oral erythromycin)
Newborn Eyecare
conjunctivitis 3 weeks or more after delivery
mostl likely due to herpes infection (treate with systemic acyclovir and topical vidarabine)
Newborn Eyecare
conjunctivitis treatment
2 types of ab drops to prevent ophtamoa neonatuorum which is attributed to neisseria gonorrhoeae or chlamydia trachomatis:
erythroycin or tetracycline ointments
silver nitrate solution
Vitamin K Deficient Bleeding
Definition
as the neonates colonic flora has not adequately colonized, E coli is not present in suffcient quantities to make neough vitamin k to produce clottting factors II, VII, IX, and X and proteins C and S. W/o tese factors the newborn is omrel ikely to have bleeding fromt he GI tract, belly button, and urinar tract
Vitamin K Deficient Bleeding
prophylactic treatment
to prevent Vitamin K Deficient Bleeding (formerly know as hemorrhagic disease of the newborn), a single im dose of vit k is recommended and has been shown to decrease the incidence of Vitamin K Deficient Bleeding
Screening Tests
all neonates must be screened for wht prior to discharge?
PKU congenital adrenal hyperplasia (CAH) biotinidase beta thalassemia galactosemia hypothyroidism homocysteinuria cystic fibrosis
Most commonly tested disorders in newborns
g6pd deficiency pku galactosemia congenital adrenal hyperplacia congenital hypothyroidism hearing test cystic fibrosis
G6PD deficiency
x linked recessive disease, characterized by hemolytic crises.
treatment involves reducing oxidative stress and specialized diets
PKU
autosomal recessive genetic disorder characterized by a deficiency in the enzyme phenylalanine hydroxylase (PAH) that leads to mental retardation.
Treatement is with a special diet low in phylalanine for at least the first 16 years of the patient’s life
Galactosemia
rare genetic disorder that precludes normal metabolism of galactose.
Treatment is to cut out all lactose-containing products
Congenital Adrenal hyperplasia
any of several autosomal recessive diseases resulting in errors of steroidogenesis.
treatment is to replace mineralocorticoids and glucocorticoid deficiencies and possible genital reconstructive surgery.
Congenital hypothyroidism
a condition affecting 1 in 4000 infants that can result in cretinism
Hearing test
excludes congenital sensory-neural hearing loss. necessary for early detection to maintain speech patterns and assess the need for cochlear implantation
Cystic Fibrosis
autosomal disorder causing abnoramlly thick mucus.
Cystic Fibrosis
Best initialtest:
sweat chloride
Cystic Fibrosis
Most accurate test:
genetic analysis of the CFTR gene
Cystic Fibrosis
Classic findings on the USMLE
Combination of an elevated sweat hloride, presence of mutationsin CFTR gener, and/or abnormal functioning in at least one organ system
Hepatitis B Vaccination
Every child gets a hepatitis B vaccination, but only those with HBsAG-Positive mothers should receive hepatitis B IG (HBIG) in addition to the vaccine
Transient polycythemia of the newborn
hypoxia during delivery stimulates erythropoeitin and acaues an increase in ciriculating red blood cells. the newborn’s first breathwill increase O2 and cause a drop in erythropoeitin,which in turn will ead to normalization of hb
splenomegaly is a normal finding in newborns
Transient Tachypnea of the newborn
compression of the rib cage by passin throuh the omther’s vaginal canal helpt so remove fluid in the lungs. Newborns who are delivered bia cesarean birth may have excess fluid in the lungs and therefore by hypoxic. if tachypnea lasts more than 4 hours, it is considered sepsis and must be evaluated with blood and urine culutres.lumbar puncture with csf analysis and culture is done when the newborn displays neurological signs such as irritability,lethargy, temp irregularity, and feeding problems.
transient hyperbilirubinemia
over 60% of all newborn infants are jaundiced. This is due to the infant’s spleen removing excess red blood cells that carry Hgb F. This excess breakdown or RBCs leads to a physiological release of hb and in turn a rise in bilirubin.
Deliver-Associated injury in the newborn
Subconjunctival hemorrhage
minute hemorrhages may be present in the eyes of the infant due to a rapid rise in intrathoracic pressure as the chest is compressed while passing through the canal. no treatment is indicated
Deliver-Associated injury in the newborn
skull fractures
Linear: most common
Depressed: can cause further cortical damage w/o surgical intervention
Basilar: most fatal
Deliver-Associated injury in the newborn
scalp injuries
Caput succedaneum is a swelling of thesoft tissues of the scalp that does notcross suture lines. cephalohematoma is a subperiostealhemorrhage that does not cross suture lines. diagnosis is made clinically and improvement occurs gradually without treatment over a few weeks to months
Deliver-Associated injury in the newborn
Brachial Palsy
Etiology
brachial plexus injuries are secondary to births with traction in the event of shoulder dystocia. Brachial palsy is most commonly seen in macrosomic infants of diabetic mothers and has 2 major forms:
duchenne-erb paralysis: C5-C6
klumpke paralysis: c7-c8+/-T1
Deliver-Associated injury in the newborn
duchenne-erb paralysis: C5-C6
waiter tip appearance; secondary to shoulder dystocia
the infant is unabelt o uabduct the shoulder or externally rotate and supinate the arm
diagnosis is made clincially and immobilization is the best treatment
Deliver-Associated injury in the newborn
klumpke paralysis: c7-c8+/-T1
claw hand due to a lack of grasp reflex
paralyzed hand with horner syndrome (ptosis, miosis, and anhydrosis)
diagnosis is made clincially and immobilization is the best treatment
Deliver-Associated injury in the newborn
clavicular fracture
this is the most common newborn fracture as a result of hsoulder dystocia. xray is the best diagnostic test, and the fracture is treated with immobilization, splinting, and physical therapy
Deliver-Associated injury in the newborn
facial nerve palsy
facial nervye palsy is paralysis of structures innervated by the facial nerve, caused by trauma secondary to forcep use in deliver. diagnosis is made clinically and improvement occurs gradually over a few weeks to months. however, if no recovery is seen, then surgical nerve repair is necessary.
Deliver-Associated injury in the newborn
amnioticfluid abnormalities and associated manifestations
in amniotic fluid, 80% is a filtrateof the mothers plasma
the baby produces the remianing 20% by swallowing, absorbing, filtering, and urinating
Deliver-Associated injury in the newborn
amnioticfluid abnormalities and associated manifestations
polyhydramnios
too much fluid secondary to fetus not swallowing
causes:
neuro, werdnighoffman (infant unable to swallow)
GI, intestinal atresia
Deliver-Associated injury in the newborn
amnioticfluid abnormalities and associated manifestations
oligohydramnios
too little fluid bc fetus cannot urinate
causes:
prune-belly: lack of abdominalm uscles so unablet o bear down and urinate. treat with serial foley catheter placements but carries high risk of UTI
renal agenesis: incompatible with life, associated with potter syndrome
flat facies due to high atmospheric pressure causing compressio of the fetus that is normally buffered by the amniotic fluid
When does shoulder dystocia occur?
after delivery of the head when the babys anterior shoulder gets stuck behind the mother’s pubic bone
Abnormal abdominalf findings
diaphragmatic hernia
omphalocele
umbilical hernia
gastroschisis
wilms tumor
neuroblastoma
diaphragmatic hernia
a hole in the diaphragm that allows the abdominal contents to move into the thorax
bowel sound int he chest can be heard
air fluid levels are seen on chest xray
two types:
- morgagni: defect is restrosternal or parasternal
- bochdalek: defect is posterolateral
bochdalek
most common diaphragmatic hernia
commonly occur on the left side
Omphalocele
defect in which intestines and organs from beyond the abdominal wall with a sac covering. Results from failure of the GI sac to retract at 10-12 weeks gestation
screening is conducted by maternal alpha fetoprotein levels and us. surgical reintroduction of contents in needed. highly associated with edwards syndrome (tri 18)
elevated afp levels indicate
neural tube defects and abdominalw all defects
most common cause of elevated afp is
incorrect dating
Umbilical hernia
there is a congenital wall weakness of the rectus abdominis muscle which allows for protrusion of vessels and bowel. it is highly associated with congenital hypothyroidism. ninety percent close spontaneously by age 3. after the age of 4, surgical intervention is indicated to prevent bowel strangulation and subsequent necrosis
Gastroschisis
a wall defect lateral to midline with intestines and organs forming byoned the abdominal wall with no sac covering. multiple intesintal atresias can occur. treatment calls for immediates surgical itnervetnion with gradual introduction of bowel and silo formation. overly agressive surgical reintroduction of the bowel will ead to third psacing and bowel infarction.
Wilms Tumor
A large palpable abdominal mass is felt. it is caused by hemihypertrophy of one kidney due to its increased vascular demands. aniridia is highly associated wtih this malignancy and is usually the clinicians most valuable clue. an affected child will show signs of consitpation and complain of abdominal pain that is accompanied by nausea and vomiting
wilms tumor
diagnostic tests
best initial: abdominal us
most accurate: contrast-enhanced CT
Wilms tumor
treatment
total nephrectomy with chemotherapy and radiation may be indicated based upon staging. bilateral kidney inolvement indicated partial nephrectomy
most common abdominal mass in children
wilms tumor
Wagr
Wilms tumor
aniridia
genitourinary malformations
retardation
results from a deletion on chromosome 11
Neuroblastoma
adrneal medullar tumor similar o a pheochormocytoma but with fewer cardiac manifestation. the percentageof cases presenting with metastases ranges from 50% to 60%
Neuroblastoma
highly tested findings
Hypsarrhythmia (on EEG) and opsomyoclonus are hallmark findings.the prefix opso- refersto the presenceof intermittent jerky eye movements (dancing eyes) and the root myoclonus refers to myoclonic jerks andcerebellar ataxia (dancing feet).
increased vanillyl mandelic acid (VMA) and meanephrines on urin collection are diagnostic.
most common cancers in infancy and the most common extracranial slid malignancy
neuroblastoma
Abnormal genitourinary findings
hydrocele
varicocele
cryptorchidism
hypospadias
epispadias
development achievements
- sucking reflex - baby will automatically suck on a nipplelike object
- grasping reflex
- babinski reflex - toe extension
- rooting reflex - if you touch the baby’s cheek, the baby will turn to that side.
- moro reflex - arms spread symmetrically when the baby is scared
- stepping reflex - walking like maneuvers when toes touch the ground
- superman reflex - when held facing the floor, arms go out
Hydrocele
hydrocele is a painless, swollen gluid filled sac along the spermatic cords within the scrotum that transilluminates upon inspection
- remnant of tunica vaginalis
- usually will resolve within 6 months
- must differentiatefrom inguinal hernia
Varicocele
varicocele isa varicose wein in the scrotal veinscausing swelling of pampiniform plexus and increased pressure. the most common compalint is a dull ache and heaviness in the scrotum
Varicocele
testing
best initial testis pe coinciding with bag of worms sensation
most accurate test is us of the scrotal sac showing dilatation of the vessels of the pampiniform plexus >2mm. (always us the other testicle, it is a b/l disease and so if present on one side it is probably indolent on the other side)
varicocele
treatment
indicated for delayed growth of the testes or in the those with evidence of testicular atrophy
Cryptorchidism
is the absence on one testicle in the scrotum, and is usually found within the inguinal canal
- 90%of cases can be felt in the inguinal canal
- orchipexy is indicated to bring the testicle down into the scortum after the age of 1 to avoid sterility
Cryptorchidism is associated with
an increased risk for malignancy regardless of surgical intervention
Hypospadias
the opening of the urethra is found ont he ventral surgace of the penis
- high association with cryptorchidism and inguinal hernias
- needssurgical correction
- circumcision is ci due to difficulties in surgical correction of the hypospadias
epispadias
the opening of the urethra is found on the dorsal surface
- high association with urinary incontinence
- must evaluate for concomitant bladder exstrophy
- needs surgical correction
Cyanotic lesions
tetrology of fallot
transposition of the great vessels
hypoplastic left heart syndrome
truncus arteriosus
total anomalous pulmonary venous return
squatting and exercise intolerance are pathognomonic for
tetralogy of fallot
most common cyanotic heart defect in kids
tetralogy of fallot
Tetralogy of Fallot
definition
overriding aorta
pulmonary stenosis
Right ventricular hypertrophy
ventricular septal defect (VSD)
Tetralogy of Fallot
etiology
cause is thorugh to be due to genetic facors and enfironemenal factors. is is associated iwth Chromosome 22 deletions
Tetralogy of Fallot
presentation
Cyanosis of the lips and extremities
holosystolic murmur best heard at the lower left sternal border
squatting after exertive exercises
-causes and increased preload and increased systemic vascular resistance. this decreases the right to left shunting, leading to increased pulmonary blood oxygen saturation.
Tetralogy of Fallot
diagnostic tests
cxr showing boot shaped heart
decreased pulmonary vascular marking
Tetralogy of Fallot
treatment
surgery is the only definitive therapy
VSD is common in
Down (tri21)
edwards (tri 18)
patau (tri 13)
3 holosystolic murmurs
mitral regurg
tricuspid regurg
VSD
most common congenital heart defect in Down Syndrome
endocardial cushion defect of atrioventricular canal
Transposition of the great vessels
this condition is characterized by an aorta that origniates fromt he right ventricel and pulmonay artery that comes fromt he left ventricle.no oxygenation of blood can occur without a patent ductus arteriosus (PDA), ASD or VSD.
Transposition of the great vessels
presentation/diagnostic tests
Early and severe cyanosis is seen. A single S2 isheard. CXR will show an egg on a string
Transposition of the great vessels
treatment
neonates must have an open ductus arteriosus (PDA). they require prostaglandin E1 to keep the ductus open, and NSAIDS (especially indomethacin) are ci bc they will cause closure of the ductus.
2 seperate surgeris are necessary; however, each surgery carries a 50% mortality rate. Therefore, only 1 in 4 will survive the surgeries.
Tetralogy of Fallot is the most common cyanotic lesion in children after
the neonatal period
Transposition of the great vessels is the most common cyanotic lesion during
the neonatal period
pulsus alternans
sign of left ventricular systolid dysfunction
pulsus bigeminus
sign of hypertrophic obstructive cardioyopathy (HOCM)
pulsus bisferians
in aortic regurgitation
pulsus tardus et parvus
aortic stenosis
pulsus paradoxus
cardiac tamponade and tension pneumothorax
irregularly irregular
atria fibrillation
Hypoplastic Left Heart Syndrome
this is a syndrome consisting of left ventricualr hypoplasia, mitral valve atresia, and aortic valve lesions
Hypoplastic Left Heart Syndrome
presentation
absent pulses with a single S2
increased right ventricular impulse
gray rather than bluish cyanosis
Hypoplastic Left Heart Syndrome
diasnotic tests
cxr will show a globular shaped hehart with pulnonary edema.
echo is the most accurate diagnostic test
Hypoplastic Left Heart Syndrome
treatment
only therapy is 3 seperate surgeries or a heart transplant. each surgery carries and extremely high mortality
Truncus Arteriosus
occurs when a single trunk emerges from both right and left ventricles and gives rise to all major circulations.
Truncus Arteriosus
presentation
symptoms occur whtin the first few days of life and are characterized by
- severe sypnea
- early and frequent respiratory infections
- single s2 is heard as there in only one semilunar valvee and a systolic ejction murmur is heard bc these valve leaflets are usually abnormal in functionality.
- peripheral pulses are bounding
Truncus Arteriosus
diagnostic tests
cxr will show cardiomegaly with increasedpulmonary markings
Truncus Arteriosus
treatment
the omst severe sequela of this condition is
Total Anomalous Pulmonary Venous Return
congenital condition in which there is no venous return between pulmonary veins and the left atrium, oxygenated blood instead returns to the superior vena cava. there are 2 forms: with or wihtout obstruction of the venous return. obstruction refers to the anlge at which the veins enter the sinus.
TAPVR with obstuction
signs/symptoms
early in life with respiratory distress and severe cyanosis
TAPVR with obstuction
diagnostic tests
xcr shows pulmonary edema
echo is definitive
TAPVR with obstuction
treatment
surgery is the definitive choice for treatment
TAPVR w/o obstuction
signs/symptoms
age 1-2 years with right heart failure and tachypnea
TAPVR w/o obstuction
diagnostic tests
cxr shows snowman or figure 8 sign
mosta ccurate test is echo (diagnosed by echo and not cxr)
TAPVR w/o obstuction
treatment
surgical intervention to restore proper blood flow
a child who was healthy but now presents with a holosystolic murmur and ftt
vsd with right ventricular hypertrophy
Summary of cyanotic heart defects
tof
r to l shunt
vsd
surgery
Summary of cyanotic heart defects
tGV
r to l shunt
pda dependent
surgery
Summary of cyanotic heart defects
hypoplastic left heart syndrome
r to l shunt
pda dependent
surgery
Summary of cyanotic heart defects
truncus arteriosus
r to l shunt
vsd
surgery
Summary of cyanotic heart defects
tapvr
r to l shunt
surgery
Ventricular septal defect
most common congenital heart lesion
Ventricular septal defect
presentation
dyspnea with respiratory distress
high-pitched holosystolic murmur over lower left sternal border
loud pulmonic s3
Ventricular septal defect
diagnostic tests
cxr shows increased vascular markings
echocardiogram is diagnostic and cardiac catheterization is definitive
Ventricular septal defect
treatment
smaller lesions usually close in the first 1 to 2 years while larger or more symptomatic lesions require surgical intervention. diuretics and digoxin can be used for more conservative treatment. if left untreated complications can lead to chf, endocarditis, and plumonary htn
pansystolic=
holosystolic=throughout systole
Atrial Septal Defect
a hole in the septum between both atria that is twice as common in women as in men
Atrial Septal Defect
3 main types
- primum defect: concomitatnt mitral valve abnormalities
- secundum defect: most common and located in the center of the atrial septum
- sinus venosus defect: least common
Atrial Septal Defect
presentation
usually asyptomatic except for a fixed wide splitting of s2
Atrial Septal Defect
diagnostic tests
the most definitive test is cardiac catheterization. However, echo is less invasive and can be just as effective
cxr shows increased vascular markings and cardiomegaly
Atrial Septal Defect
treatment
vast majority close spontaneously
surgery or transcatheter closure is indicated for all symptomatic pts
dysrhytmias and possible paradoxical emobli from dvts later in life
PDA
defined as the failure of spontaneous closure of the ductus. it usually closes when PO2 rises above 50 mmhg. low po2 can be caused by pulmonary compromise due to prematurity. areas of high altitude have an increased occurrence of PDA due to low levels of atmospheric oxygen
When is pda a normal finding
first 12 hours of life, after 24 it is considered pathologic
PDA
Presentation
machinery like murmur
wide pulse pressure
bounding pulses
PDA
most common complication later in the childs life
high occurrence of respiratory infections and infective endocarditis
mitral elsions radiate to teh
axilla
tricuspid and pulmonary lesions radiate to the
back
aorticl lesions radiate to the
neck
PDA
best initial test
echo
PDA
most accurate test
cardiac catheterization
PDA
ekg
may show lvh secondary to high systemic resistance
PDA
treatment
give indomehtacin (nsaid inhibits prostaglandins) to close the pda unless it is needed to live in convurrent conditions like tof
give prostaglandins to
pop open a pda
giv indomethacin
inhibit popping open of pda
cxr
pear hsaped heart
pericardial effusion
cxr
boot-shaped heart
tof
cxr
jug handle appearance heart
primary pulmonary artery htn
cxr
3 like appearance or rib notching
coarctation of the aorta
Long QT syndrome
hearing loss, syncope, normal vitals and exam, fam hx of sudden cardiac death
coarctation of the aorta
congenital narrowing of the aorta in the area of the ductus arteriosus. it has a grequent assocation iwth turner syndrome
coarctation of the aorta
presentation
severe CHF and resp distress withint he first few months of life
diffpresuresa ndpulses betweenthe upper and lower extremities
reduced pulses in the lower extremities and htn in the upper extremities due to narrowing
coarctation of the aorta
diagnostic tests
rib nothcing and 3 sign are seen on cxr
cardiac catheterization is the omst accurate test
coarctation of the aorta
treatment
primary treatment is surgical resection of the narrowed segment and then balloon dilation if recurrent stenosis occurs
if the exam question mentions short gir with webbed neck shierld chest streak gonads horseshoe kidneys or shortened fourth metacarpal think
turner and coarctation of the aorta
Pathologic Jaundic of the newborn
hyperbilirubinemia is considered pathological when
it appears on the first day of life
bilirubin rises more than 5 mg/dl/day
bilirbuin rises above 19.5 mg/dl in a term child
direct bilirubin rises above 2 mg/dl at any time
hyperbilirubinemia persists after the second week of life
Pathologic Jaundic of the newborn
most serious complication
the deposition of bilirubin the basal ganglia called kernicterus.
kernicterus presents with hypotonia, seizures, choreoathetosis, and hearing loss
Pathologic Jaundic of the newborn
diagnostic tests
direct and indirect bilirubin levels
check blood type of infant and mother for ABO and Rh incompatibility
analyze peripheral blood smear and retic count for hemolysis
Pathologic Jaundic of the newborn
treatment
phototherapy with blue-green light helps break down bilirubin to excretable components. consider exhcange transfusion if bilirubin rises to 20-25 mg/dl
Esophageal Atresia
in esophageal atresia, the esophagus ends blindly. in nearly 90% or cases it communicates with the trachea through a fistula known as a tracheoesophageal fistula (TEF)
Esophageal Atresia
presentation
the child willtypically exhibit vomiting with first feeding or choking/coughing and cyanosis due to the TEF. there will be a hx of possible polyhdramnios
Esophageal Atresia
diagnostic tests
a gastric air bubble and esophageal air bubble can be seen on cxr
coiling of the ng tube seen on cxr and an inability to pass it into the stomach are diagnostic
ct or esophagram can also be used
Esophageal Atresia
treatment
surgical repair must be done in 2 steps to correct the anomaly
antibiotic coerage for anaerobes must also be considered due to high risk of lung abscess formation secondary to aspiration
fluid resuscitation before surgery must be done to prevent dehydration of the infant
Esophageal Atresia
a
ea with distal tef (80-90% of cases)
Esophageal Atresia
b
isolated ea w/o tef
Esophageal Atresia
c
ea with both proxiam and distal tefs
Esophageal Atresia
d
h type tef
string sign
pyloric stenosis
doughnut sign
intussuusceptions
birds beak
achalasia
steeple sign
croup
Pyloric Stenosis
a hypertrophic pyliric sphincter prevents proper passage of GI contents from the stomach intot he duodenum. ost comon cuase is idiopathic
Pyloric Stenosis
presentation
hypertrophy of the pylorus is not commonly found at birth but rather becomes more pronounced by the first month of life. and an present as alst as 6 months old
auscultation will reveal a succussion splash, which is the sound of stomach contents slapping into thepylorus like waves on a beach.
nonbiilous projectile vomiting is the hallmark feature. metabolic imbalance demonstrates a hypcholoremichypokamiec metabolic alkalosis due to the vast loss of hydrogen ionsi n the vomitus. the potassium loss also worsens from aldosterone realease in response to hypovolemia. aldosterone increases urinary excretion of potassium
olive sign, which delineates aplapabe mass the size on an olive felt in the epigastric region, is highly associated with this condition
what sign is frequently tested on USMLE
olive sign