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Clinical Disaplines > Pediatric Neurology > Flashcards

Pediatric Neurology Flashcards

1
Q

a. Fasciculation’s absent

A

UMN

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2
Q

gait unsteady until age

A

6

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3
Q

babinski goes down after

A

18 months

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4
Q

most reflexes are integrated by what month

A

6 moths

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5
Q

what is the most concerning HA pattern

A

chronic, progressive

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6
Q

chronic and progressive HA think what

A

increased ICP

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7
Q

increased ICP HA is worse when what

A

they lie flat

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8
Q
  1. Frontal, bitemporal or unilateral pounding/throbbing for 2-72h
A

migraines

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9
Q

what is the first treatment for migranes

A

eliminate triggers

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10
Q

what is the pharm treatment for migranes

A

NSAID,
acetaminophen,
Triptans

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11
Q

AKA idiopathic intracranial hypertension

A

Pseudotumor Cerebri

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12
Q

a. Increased ICP without space occupying lesion

A

Pseudotumor Cerebri

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13
Q

a. Most common in children of childbearing age can happen in kids > 11

A

Pseudotumor Cerebri

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14
Q

a. Papilledema on PE

A

Pseudotumor Cerebri

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15
Q

, vision loss often chief complainant

A

Pseudotumor Cerebri

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16
Q

what is the MC type of CP

A

1.spastic

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17
Q

4 types of CP

A

spastic
athetoid
ataxic
atonic

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18
Q

a. Neural tube disorder (NTD) = defective closure of the caudal neural tube early in gestation (about week 4)

A
  1. Spina Bifida
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19
Q
  1. Spina Bifida

types 3

A

i. Myelomeningocele
i. Meningocele
i. Spina bifida occulta

20
Q

how do you prevent spina bifida

A

folic acid

21
Q

DX of spina bifida 2

A
  1. alpha-fetoprotein (AFP) level (16-18 weeks

2. US 20 weeks

22
Q

i. Hypopleasia

associated with what chiari

A

4

23
Q

i. Herniation below the formen magnum

A

3

24
Q

i. Hydrocephalus

ii. Myelomeningocele

A

2

25
Q

i. Sx related to ventricular distention and increased ICP due to obstruction of CSF flow

A

Hydrocephalus

26
Q

Headache, vomiting, altered mental status, visual changes, ocular nerve palsies, and focal neurologic findings

A

Hydrocephalus

27
Q

a. Progressive degeneration of the anterior horn cells progressive weakness of lower motor neurons
b. Genetic (autosomal recessive)
c. Presents with progressive proximal weakness , decreased spontaneous mvmt, “floppiness”
d. Normal mental, social and language skills. Sensation is preserved.
e. Decreased facial expression and drooling
f. Dx: EMG, muscle bx, DNA testing

A

SMA

28
Q

Campylobacter jejuni

A

GBS

29
Q

i. Ascending weakness (symmetric)
ii. May have autonomic nerve dysfunction
iii. Children- refusal to walk and leg pain are most common presenting symptoms

A

GBS

30
Q

i. LP- elevated CSF protein without increased WBC

A

GBS

31
Q

treatment for GBS

A

IVIG

plasmaphoresis

32
Q

a. Gower’s sign
b. Proximal muscles affected first, then distal; lower first, then upper
- calf hypertrophy

A
  1. Duchenne Muscular Dystrophy
33
Q

muscle that is X liked recessive

A
  1. Duchenne Muscular Dystrophy
34
Q
  1. Diagnosis
    a. Serum CK
    b. biopsy
    c. genetic testing
A

DMD

35
Q

a. more common, autosomal dominant (chromosome 17)

A

NF: 1

36
Q
  • also autosomal dominant (chromosome 22)
A

NF 2

37
Q

i. Referred to as peripheral type

ii. More skin and brain abnormalities

A

NF: 1

38
Q

i. Manifests as bilateral vestibular schwannomas (VIII nerve tumors)

A

NF: 2

39
Q

Café-au-lait spots are NOT part of NF type 2

A

TRUTH

40
Q

Most common neurologic disorder of infants & young children

A

Febrile Seizure

41
Q

what age can you have febrile seizure

A

6 - 5 years

42
Q

a. Viral infections commonly associated (HHV-6, influenza)

A
  1. Febrile Seizure
43
Q

a. Low-risk for long term neurologic complications

A
  1. Febrile Seizure
44
Q

a. IV benzos

A

for Sz lasting longer than 5 minutes

45
Q

a. Brain & skin have same embryonic origin

A

Truth

Clinical Disaplines (7 decks)

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