PBL 4: Weeds and Herbicides: Plant diseases/pesticides Flashcards

1
Q

Weeds

A

Compete with crops and can transmit diseases and pests. Can result in more than a 30% loss of yield on good land:
1. Annual weeds
2. Biennial weeds
3. Perennial Weeds

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2
Q

Annual weeds

A

plants that complete their life cycle in one growing season.
-Thrive on frequently disturbed sites (i.e. corn+soybean production).
-Often have high levels of seed production
-Tillage+herbicide are effective at killing annual plants but often develop from soil seed bank.

Summer annual weeds: germinate+grow spring/summer

Winter annuals: germinate fall/vegetative in winter, germinate in spring.

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3
Q

Biennial weeds

A

Plants that require two growing seasons to complete their life cycles.
-Are vegetative in the first year, flower in the second year.

-Intolerant of tillage in the first/second year

-Mowing in the second year before they flower can eliminate seed production.

-Persist in the permanent pastures, waterways, and roadside areas.

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4
Q

Perennial weeds

A

Plants that live for 3+ years. They have some specialised reproductive structures that allow for:
-overwintering
-long-term persistence (crowns rhizomes, stolons)

-Once established, difficult to control except by herbicides.

-Best adapted to survive under a diversity of cropping systems.

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5
Q

Grasses and Broadleaves

A

Monocot weeds: grasses (seed remains underground, only leaves emerge)

Dicots: Broadleaves, may have compound leaves. Most seeds energy from below ground during germination and are easier to kill compared to grass.

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6
Q

Impacts of Weeds

A

-Reduce agricultural productivity and profitability
-Reduces yield: competing with crops
-increase production costs during their control
-Some are allelopatheic
-Some are parasitic
-May contain toxins that are harmful to livestock

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7
Q

Allelopathy

A

the adverse effect of one plant on another due tot production of a chemical inhibitor.
-Living or decaying plant tissue may excrete the chemicals

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8
Q

Grain contamination

A

Mixing weed seed with grain can result in economic loss if separation is not possible at harvest or later during processing.

-may decrease its value for livestock feeding or human use: wild oat contamination of wheat, barley, wild garlic, etc.

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9
Q

Unique weed traits: Seed production

A

Many weeds produce small seeds and very high numbers of seed.

-provides an opportunity for weed populations to rapidly increase.
-ensures the survival of species

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10
Q

Unique weed traits: Seed dormancy

A

Weed seeds can have a long dormancy period and a staggered rate of seed germination.
-enables weed seed to persist and remain viable in soil for long periods.

Seed bank: total dormant+nondormant seed accumulated in the soil, provides a reserve of seed.

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11
Q

Unique weed traits: Seed dispersal

A

-Dispersed by numerous means: wind, water, animals, humans, appendages

-e.g.: purple loosestrife, aquatic weed that attaches to feet of ducks/geese

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12
Q

Unique weed traits: negative (asexual) reproductive:

A

-enables spreading the weed from the original plant without using seed.

-Rhizomes
-creeping roots
-stolons

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13
Q

Biotypes

A

Populations of weeds that develop special traits like herbicide resistance.

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14
Q

Herbicides

A

-primary method of weed control in conventional agriculture.
-used for killing or injuring plants: modern herbicides are synthetic chemical compounds with specific mechanisms for killing plants.

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15
Q

Herbicide Advantages

A

-Production costs per acre reduced vs mechanical weed control/hand weeding.

-Herbicides provide greater flexibility in timing of weed control than mechanical approaches.

-Selectively can remove weeds from turf+forages without damaging the crop.

-Reduce/eliminate the need for mechanical tillage

-Can persist in soil to provide long-term weed control

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16
Q

Herbicide Disadvantages

A

-Can potentially kill/injure non target plants
-Contamination of water supplies
-Can lead to development of resistant weed species
-Human health.

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17
Q

Glyphosate

A

A nonselective herbicide, most widely used herbicide.
-Chemical fallowing
-Resistance built from GMO (Roundup Ready crops).
-Low-cost herbicide.
-when used properly, has minimal environmental impact.

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18
Q

LD50

A

Lethal dose: amount ingested where 50% of test mice die. Acute toxicity.

Acute toxicity: instant result
Chronic toxicity (more dangerous for humans: accumulation of long-term result

Routes of entry:
Exposure to: mouth, nose, eyes, skin

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19
Q

Modes of Action

A

Biochemical mechanism that herbicides use to kill plants. Can be selective or or nonselective related to the weeds they affect:

  1. Growth regulators
  2. Amino acid synthesis inhibitors
  3. Lipid synthesis inhibitors
  4. Nitrogen metabolism inhibitors
  5. Seedling growth inhibitors
  6. Photosynthesis inhibitors
  7. Cell membrane disruptors
  8. Pigment inhibitors
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20
Q

Modes of Action: Growth regulators

A

Disrupt plant growth by interfering with hormones+protein synthesis. Mostly taken up by the foliage but can be absorbed by the roots. Translocated in the plant’s vascular system

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21
Q

Modes of Action: Amino Acid Synthesis inhibitors (ANNUALS)

A

disable enzymes reponsible for specific amino acid synthesis: therefore proteins important for plant growth are not formed.

-instead, taken up by the foliage and translocated in plants.

22
Q

Modes of Action: Lipid synthesis Inhibitors

A

prevent the formation of fatty acids (key roles in cell membranes and also energy storage)

23
Q

Modes of Action: Nitrogen metabolism inhibitors

A

impede enzyme that helps produce glutamine, which is required for nitrogen metabolism.

24
Q

Modes of Action: Seedling growth inhibitors

A

Affect growth of roots+shoots of germinating seedlings. Active at multiple sites in a plant and affect mitosis+photosynthesis, etc. Must be applied before germination

25
Q

Modes of Action: Photosynthesis inhibitors

A

prevent plants from conducting photosynthesis and producing energy by binding to the chloroplasts

26
Q

Cell membrane disruptors

A

destroy plant foliage by directly rupturing cell membranes: quick-acting contact herbicides that require sunlight for activation

27
Q

Pigment inhibitors

A

prevent plants from forming the chlorophyll that is essential for photosynthesis. Plants turn white and often become translucent

28
Q

Application of herbicides: Preplant herbicides

A

-Applied to the soil before planting; target germinating weed seeds

29
Q

Application of herbicides: Preemergence herbicides

A

Applied to the soil after the crop has been planted but before the crop/weeds have emerged. Need to be leached into the soil by rainfall/irrigation

30
Q

Application of herbicides: Postemergence herbicides

A

Applied after the crop/weeds have emerged. Most often depend on foliar contact for activity. Most widely-used types (glyphosate)

31
Q

Application of herbicides: Postharvest herbicides

A

Applied after harvest but before tillage, foliar contact is necessary.

32
Q

Types of herbicides

A

Contact herbicides: Annual weeds
Systemic herbicides: translocation, perennial weeds.

33
Q

Herbicide resistance

A

tolerance of herbicides by weed species that were once susceptible. Occurs because of repeated exposure to a specific herbicide.

Avoiding:
-Rotate herbicides over years and to use herbicides with different modes of action
-Use alternative control strategies (crop rotation/tillage)

34
Q

Biological weed control

A

employs biological agents such as diseases, insects, animals to reduce/eliminate populations of weed species.

-Consume foliage
-Destroy roots+crowns
-Prevent flowering+reproduction.

-Natural, nonpolluting, target

35
Q

Mechanical weed control

A

use of tillage/mowing implements drawn by horses or tractors for weed control
-Tillage: uproots and dismembers mature plants, burying/stimulating germinating/dormant seeds

-Mowing: removes aboveground shoot, leaves, and flowers of plants.

-Covercrops
-Intercropping: allelopathy

36
Q

Plant disease

A

a progressive deviation from a plant’s normal development, appearance, function.

3 factors must be present:
1. a susceptible host
2. pathogen (disease-causing agent)
3. favourable environment for infection.

37
Q

Biotic plant diseases

A

caused by living organisms (fungi, bacteria, mycoplasma-like, viruses, nematodes, parasitic flowering plants)

38
Q

Abiotic plant diseases

A

caused by nonliving factors (air pollution, nutrient imbalances, water excesses/deprivation, extreme temperatures, soil acidity/alkalinity, herbicides)

39
Q

Pests

A

Agents of disease/damage

40
Q

IPM Program: 5 Basic methods that can be applied for pest management

A
  1. Cultural
  2. Biological
  3. Mechanical
  4. Genetic
  5. Chemical
41
Q

Cultural pest management

A

attempts to manipulate the environment in which plants are grown, includes:

-Crop rotation: prevents buildup of soil-borne pathogens. Must be diverse enough.

-Irrigation+fertilisation: well hydrated crops with no nutrient deficiencies are more resistance to pathogens

-Sanitation: Equipment, tools, soil residues.

42
Q

Biological pest management

A

uses one living organism to control another by predation, parasitism, or competition to prevent overpopulation of pests.

-Beneficial insects: prey on plants without harming the crop plant

-Bacteria: Bt gene can be injected or applied topically to kill various insects.

-Green manures: crops grown to be plowed under to provide an environment that will increase soil populations of organisms that are antagonistic to plant pathogens

-Plant chemicals: (i.e. nicotine from tobacco, pyrethrum from chrysanthemum), or phytoalexins.

43
Q

Phytoallexins

A

Other plants produce toxins called phytoalexins when they are damaged/injured, which are poisonous to other pests.

44
Q

Mechanical pest management

A

attempts to manipulate the environment around the crop by using mechanical equipment or some other physical means to make it less favourable

-Handpicking: labour-intensive

-Tillage

45
Q

Genetic pest management

A

Uses plant breeding and genetic engineering to develop plants that are more resistant to important plant pests.

-A process that can take years to complete (Bt Corn)

46
Q

Chemical Pest Management

A

-disinfectants (sterilisation), fungicides, nematicides (nematode populations), antibiotics, insecticides (modes of action).

-smart: identify pest life cycle to apply when most vulnerable

-may cause environmental, animal and health risks

47
Q

Insects

A

-six legs, exoskeleton, 3 main sections, outer-shell coverings (wings+antennae)

No metamorphosis: insects emerge from eggs as miniature adults

Incomplete metamorphosis: emerge from eggs and gradually develop into adult

Complete metamorphosis: separate life cycle stages: egg, larva, pupa, adult (larva+adult=danger)

48
Q

Chewing insects

A

Have mandibles for chewing plant tissues while feeding

49
Q

Piercing/sucking insects

A

Have a piercing mouthpart called a labrum that it uses to puncture plant tissue and suck the plant fluids.

-Can cause additional damage because they can inject toxins/transmit bacteria (aphids)

50
Q

Aphids

A

Extremely common pests in both field and greenhouse situations

-reproduce many times in one year and can overwinter as eggs/adults.

-ladybird beetles are natural predators. ants eat aphid excretion.

51
Q

Stages of IPM

A
  1. Prevention and Suppression: combination of tactics into preventive strategies
  2. Monitoring: Observation, Forecast, Diagnostic
  3. Decision-making: Use of threshold+multiple criteria

4-7. Intervention: Non-chemical methods, least side effects, reduced pesticide use, anti-resistance strategies

  1. Evaluation: Assessment of entire process, adoption of new standards.