Part 3 Flashcards
Somatopsychic
how subluxation can influence the emotions and other central states
somatopsychic hypothesis
pain overloads the ascending pathways and overexcited the reticular activating system, which affects sleep (insomnia, anxiety, depression)
cerebral dysfunction syndrome
reduced blood flow to specific regions of the brain, causes “hibernation” or a state of suspended electrical activity in those regions, this effects higher functions
symptoms of cerebral dysfunction syndrome
dizziness/giddiness lethargy/fatigue insomnia depression nervousness anxiety disorientation personality alteration hyperkinesia "whining child syndrome" headache memory problems learning disability changes in viscual acuity auditory difficulty poor concentration clumsiness mixing up words loss of libido
psychogenic (psychosomatic)
emotional state causes the subluxation
possible mechanisms of psychogenic
postural changes associated with non-verbal communication (body language)
diaphragm hypertonicity as a result of acute/chronic alarm state can result in stress in the spine
axoplasmic aberration hypothesis
neurotrophic support of body tissues can be adversely affected by VSC due to the disruption of normal axoplasmic transport mechanisms
what did Korr says about axoplasmic aberration?
axoplasmic transport can be affected by compression without damage to nerve conduction
things that are transported intracellularly
proteins, phospholipids, enzymes, glycoproteins, neurotransmitters, mitochondria
vertebrobasilar arterial insufficiency hypothesis
cervical subluxation, especially in the presence of spondylosis, can cause compression of deflexion of the vertebral artery, results in cerebral ischemia and neurologic dysfunction
what did Haldeman say about VBAI?
potentially more cranial blood flow can diminish through vasoconstriction by autonomic influence
what did Ferezy say about VBAI?
careful, biomechanically-correct cervical adjusting may normalize forces applied to the vertebral artery, preventing more VBAI than it causes
5Ds and 3Ns of stroke
diploplia nausea dizziness numbness drop attacks nystagmus dysarthria dysphagia
intraneural microcirculation ischemia
effect of mechanical compression, torsion or traction that may be narrowing small blood vessels supplying nerves themselves, causing neuroischemia and altering nerve physiology
neurapraxia
manifested clinically as paresthesias
venous and lymphatics stasis
small scale mechanical stresses may be sufficient to cause localized venous congestion and or lymphatic stasis with resulting edema or accumulation/metabolites and byproducts of inflammation
altered cerebrospinal fluid flow
imporper circulation of CSF due to spinal dysfunction can cause CNS dysfunction
axoplasmic flow aberration
VSC can disrupt normal axoplasmic flow without interrupting nromal nerve flow
vertebrobasilar artery insufficiency
VSC in cervicals can decrease blood flow, leading to cerebral ischemia and neurologic dysfunction
cord compression/myelopathy
compression of the cervical neural canal can lead to neural injury and dysfunction
neurodystrophic/neuroimmune hypothesis
subluxation can lead to lowered tissue resistance and compomise the body’s ability to adapt to microorganism
somatopsychic hypothesis
how subluxation can influence the emotions and other central states
psychogenic hypothesis
emotional state causes the subluxation
altered CSF flow
improper CSF flow because of subluxation cause cause CNS dysfunction
