Brainscape's Knowledge GenomeTM

Browse over 1 million classes created by top students, professors, publishers, and experts.


See full index

Philosophy III (Rachael) > Part 1 > Flashcards

Part 1 Flashcards

1
Q

Kinesiopathology

A

Bones of spine have lost their normal motion and position. Sets in motion the 4 other components

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Myopathology

A

Muscles supporting the spine can weaken, atrophy or become too tight and go into spasm. Scar tissue can limit motion and require multiple adjustments

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Histopathology

A

Changes in blood or lymph can cause inflammation and swelling. Discs bulge, herniate, tear or degenerate. Ligaments can be permanently damaged

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Neuropathology

A

Improper spinal function can compress, stretch or irritate nerve tissue (spinal nerve or cord)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Pathophysiology

A

Bone spurs or other abnormal bony growths in attempt to fuse long term nerve dysfunction

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

What is the 5 component model to subluxation theory?

A

Kinesiopathology, myopathology, histopathology, neuropathology, pathophysiology

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

What are the part to the 3D Kent model of subluxation theory?

A

Dyskinesia, dysponesis, dysautonomia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Dyskinesia

A

Abnormality or impairment in voluntary motion

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Dysponesia

A

Reversible physiopathologiv state consisting of unnoticed, misdirected neurophysiologic reactions to various agents

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Dysautonomia

A

Neurophysiologic state reflecting abnorma, balance between tone of the sympathetic and parasympathetic divisions of the ANS

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

What is themost widely accepted definition of subluxation?

A

ACC

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Dysponesia is associated with what theory?

A

Safety pin cycle theory

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

What is the safety pin cycle theory?

A

Unnoticed errors in afferent transmission result in covert errors in output, which can lead to disease

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Abnormal vertebral movement can be broken down into what categories?

A 
Spinal fixation (hypomobility)
Hypermobility due to instability, compensation or trauma
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Nervecompressio n theory

A

Impingement occurs when a nerve’s surroundings become confined, or narrowed due to vertebral misalignment or other mechanical factors which can cause a nerve block or nerve irriataion

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Nerve block

A

Nerve signalz are stopped

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Nerve irritation

A

Send more signals that they should

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Mechanoreceptor deafferentation theory

A

This concept suggests that when there is spinal fixation or loss of moveme t, importang nerve signals dont reach the CNS, this can cause balance and equilibrium problems and can affect the part of the brain that influences immunity and personality

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

What are the three parts of the facilitation theory?

A

Somatosomatic, somatovisceral, viscerosomatic

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

Facilitation theory

A

The subkuxation complex is a problem of hyperexcitability that disrupts normal reflexes in spinal cord

21
Q

Carver

A

Basic compensatory scoliosis

22
Q

Jirout

A

hypomobility in a motion that leads to hypermobility other places

23
Q

Kapanji

A

Biomechanics (change in central axis of motion)

24
Q

Menelle

A

Facets, loss of end joint play

25
Q

Suh

A

Positional dyskonesia

26
Q

Korr

A

Fixation due to muscles inhibiting muscles, fascilitation

27
Q

Seamen

A

3D model

somatic dysafferentation

28
Q

Cyriax

A

Shift fragment of IVD

29
Q

Lewit

A

Meniscoid entrapment/interarticu,ar phenomena

30
Q

Grostic

A

Subluxations of cervical spine, particularly C1, can cause the stabilizing attachmentz of the dentate lig. To distort the cord by traction, leading to neural dysfunction

31
Q

Carrick

A

Chiros dont take pressure off nerves, they put pressure on mechanorecpetors

32
Q

Murphy

A

Nerve interference
Mechanical joint problem–> chronically stressed tissues–> afferent discharge–>efferent reflex–>efferent discharge–>collagen breakdown–>DJD–>radiculopathy and myelopathy

33
Q

Kent

A

3 component model of subluxation

34
Q

Phase 1 of 4 phase model

A

0-20 years
Motility abberations
Bony architecture intact
Initial soft tissue changes

35
Q

Response of phase 1 of 4

A

Return to normal with care in weeks to months, depending on chronicity

36
Q

Phase 2 of 4 phase mode,

A

20-40 years
Phase 1 cha ges plus fibroids
Early osseous chsnges on x ray
Disc, bony outline changes

37
Q

Response to care for phase 2 of 4

A

Significant improvement, but long term care over a few years required to halt degeneration, reversal of some chanfes may be possible

38
Q

Phase 3 of 4 model

A

40-60 years
Major degenerative and regenerative changes
Beginnings of bony fusion in ant or posg motion units

39
Q

Response in phase 3 of 4

A

Slow or retard progression of subluxation degeneration, lifetime care

40
Q

Phase 4 of 4 phase model

A

60+years

All changes of previous phases plus total fusion of motion units

41
Q

Response in phase 4 of 4

A

None, but care is given to segments not in this phase to retard progress of degeneration in those segments

42
Q

What region of the spine is least likely to have cord compresion?

A

Lumbosacral

43
Q

What are the conceptual models of degeneration?

A

Inflammation/immobilization

IVF encroachment/DRG irritation/neudologic model

44
Q

describe phase 1 of the 3 phase model (sandoz)

A

segmental dysfunctional
kinesiopathology
pre-radiologic

45
Q

describe phase 2 of the 3 phase model

sandoz

A

instability
cartilage, ligament histopathology
“true” subluxation (misalignment)

46
Q

describe phase 3 of the 3 phase model

sandoz

A

stabilization
significant DJD
ankylosis

47
Q

somatic dysafferentation

A

increase in nociceptive afferent impulses from somatic components of motion segments in combination with diminished proprioceptive impulses, primarily from mechanoreceptors

48
Q

Suh

A

positional dyskonesia

Philosophy III (Rachael) (4 decks)

Brainscape helps you reach your goals faster, through stronger study habits.
© 2024 Bold Learning Solutions. Terms and Conditions