Paediatric Oral Medicine Flashcards

(98 cards)

1
Q

What are viruses that cause oro-facial soft tissue infections

A
primary herpes
herpangina
hand food and mouth
varicella zoster
EBV
mumps
measles
rubella
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2
Q

What are bacterial ore facial soft tissue infections

A
staphylococcal
streptococcal
symphilis
TB
cat scratch disease
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3
Q

What is primary herpetic gingivostomatitis

A

acute infectious disease

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4
Q

What are fungal causes of facial soft tissue infections

A

candida

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5
Q

What is the cause of primary herpetic gingivostomatitis

A

herpes simplex virus I

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6
Q

What is the transmission of herpex simplex virus I (PHG)

A

droplet formation with 7 day incubation

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7
Q

Why is PHG uncommon in children in their first year of life

A

as they have a degree of immunity from circulating maternal antibodies

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8
Q

What is the treatment of PHG

A
bed rest
soft diet/hydration
paracetamol 
antimicrobial gel or mouthwash
acyclovir for immune compromised children
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9
Q

Describe the fluid filled vesicles seen in PHG

A

rupture to painful ragged ulcers on the gingiva, tongue, lips, buccal and palatal mucosa

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10
Q

What is the most common complication of PHG

A

dehydration

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11
Q

How long does PHG last for

A

14 days

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12
Q

How does PHG heal

A

with no scarring

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13
Q

What is the recurrent disease of PHG

A

it remains dormant in epithelial cells

recurrent disease is is herpes labials (cold sores)

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14
Q

What is recurrent infection of herpes simplex I triggered by

A

sunlight
stress
other causes of ill health

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15
Q

How are cold sores managed

A

with topical acyclovir cream

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16
Q

What two infections can coxsackie A virus cause

A

herpangina

hand foot and mouth

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17
Q

What is the difference between herpangina and PHG

A

the vesicles in the tonsillar pharyngeal region i.e further back

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18
Q

How does hand foot and mouth present

A

presents with store throat and high temp initially
then see ulceration on the gingiva/tongue/cheeks and palate
a maculopapular rash is seen on the hands and feet

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19
Q

How long does herpangina last for

A

lasts 7-10 days

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20
Q

How does hand foot and mouth last for

A

7-10 days

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21
Q

What is oral ulceration defined as

A

a localized defect in the surface oral mucosa where covering epithelium is destroyed leaving an inflamed area of exposed connective tissue

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22
Q

What are the 10 key facts to ask about when taking a history on an ulcer

A

OFN SS DELAT

onset
frequency
number
site
size
duration
exacerbating dietary factors
lesions in other areas
associated medical problems
treatment so far and if it was helpful or unhelpful
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23
Q

What are the causes of oral ulceration

A
infection
immune mediated disorders
vesiculobullous disorders
inherited or acquired immunodeficiency disorders
neoplastic/haematological
trauma
vitamin deficiencies 
recurrent apthous stomatitis
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24
Q

What are viral infections that can cause oral ulceration

A
HFM
coxsackie virus
herpes simplex
herpes zoster
CMV
EBV
HIV
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25
What are bacterial infections that cause oral ulceration
TB | symphilis
26
What are immune mediated disorders that cause oral ulceration
``` crohns behcets SLE coeliac periodic fever syndromes ```
27
Vesiculobullous disorders
bullous or mucous membrane pemphigoid pemphigus vulgaris linear IgA disease erythema multiform
28
What are neoplastic/haematological conditions that cause oral ulceration
anaemia leukamia agranulocytosis cyclic neutropenia
29
What is the most common cause of ulceration in children
recurrent apthous ulceration
30
What are the different types of trauma that can cause ulceration
physical chemical thermal
31
What is recurrent apthous stomatitis
it is this when there is no underlying cause identified
32
What are the typical apthous ulcer
round / ovoid in shape grey or yellow base have varying degree of perilesional erythema
33
What are the 3 patterns of RAU
minor major herpetiform
34
What are minor apthous ulcers
<10mm
35
What are major apthous ulcers
>10mm
36
What are herpetiform ulcers
1-2mm | can be up to 100 at a time
37
What are the aetilogical factors of recurrent ulceration
``` unclear hereditary predisposition haematological and deficiency disorders (iron deficiency) GI disease (coeliac) minor trauma in susceptible individuals stress allergic disorders (toothpaste containing SLS/foods containing benzoate or sorbete preservatives) hormonal disturbance: menstruation ```
38
What are initial investigations for recurrent oral ulceration
diet diary full blood count haematinics (folate/b12/ferritin) coeliac screen - anti transglutaminase antibodies
39
What are management of exacerbating factors of RAU
nutritional deficiencies traumatic factors avoid sharp or spicy foods allergic factors
40
What are allergic factors management for RAU
dietary exclusion | SLS free toothpaste
41
What are pharmacological management of recurrent oral ulcers
prevention of superinfection protecting healing ulcers symptomatic relief
42
What do we use to prevent superinfection of RAU
corsodyl 0.2% moutwash
43
What do we use to protect healing ulcers
``` gengigel topical gel (hyaluronate) geclair mouthwash (hyaluronate) ```
44
What do we use for symptomatic relief
difflam (0.15% benzydamina hydrochloride) | LA spray
45
What is orofacial granulomatosis
uncommon chronic inflammatory disorder
46
What is the cause of OFG
idiopathic or associated with systemic granulomatous conditions (crohns or sarcoidosis)
47
What gender is OFG more common in
males
48
What is the characteristic pathology of OFG
non caveating giant cell granulomas which then result in lymphatic obstruction
49
What may OFG be a predictor of
future crohn's disease
50
What are the clinical features of OFG/oral crohns
``` lip swelling - most common full thickness gingival swelling swelling of the non labial facial tissues peri-oral erythema cobblestone appearance of the buccal mucosa linear oral ulceration mucosal tags lip/tongue fissuring angular cheilitis ```
51
What is the etiology of OFG
largely unknown limited evidence of genetic factors numerous associated allergens are reported
52
What are the associated allergens with OFG
cinnamon compounds benzoates much higher IgE mediated atopy rates compared to the general population
53
How is OFG diagnosed
clinical | lip biopsy not essential
54
What are the investigations of OFG
``` measure growth FBC haematinics patch testing - identify triggers dietary diary - identify triggers faecal calprotectin endoscopy risky in childhood serum angiotensin converting enzyme (raised in sarcoidosis) ```
55
What is the management of OFG
can be difficult OH support symptomatic relief as per oral ulceration dietary exclusion (does not cure but reduces inflammation) manage nutritional deficiencies which may contribute to oral ulceration topical steroids topical tacrolimus short courses of oral steroids (severe or unresponsive to topical) intralesional corticosteroids surgical intervention - unresponsive long standing disfigurement
56
What is geographic tongue
idiopathic and non contageous | shiny red areas on the tongue with loss of filiform papillae are surrounded by white margins
57
Why can geographic tongue cause intense discomfort in children
as they have thinner mucosa and a more intense sense of taste they may feel discomfort with spicy food, tomato or citrus fruit / juice need to have a bland diet during flareups should become less troublesome with age
58
What are the different types of solid swellings
``` fibroepithelial polyp epulides congenital epulis HPV- associated mucous swellings neurofibromas ```
59
What is fibro-epithelial polyp
common firm pink lump (pedunculate or sessile) once established remains constant in size
60
Where are fibroepithelial polyps mostly seen
in the cheeks along the occlusal line, lips and tongue
61
What is the cause fibroepithelial polyps
minor trauma
62
What is the tx of the fibroepithelial polyp
surgical excision curative may need to wait until child is cooperative as hard to justify GA when benign
63
What are epulides
common solid swellings of oral mucosa | benign hyperplastic lesions
64
What are the 3 main types of epulides
fibrous epulis pyogenic granuloma peripheral giant cells granuloma
65
What are fibrous epulis
pedunculate or sessile mass firm consistency similar color to surrounding ginviae inflammatory cell infiltrate and fibrous tissue
66
What are pyogenic granuloma/pregnancy epulis
they are soft, deep red/purple swellings often uclerated can haemorrhage spontaneously or with mild trauma vascular proliferation supported by a delicate fibrous storm probably a reaction to chronic trauma such as calculus tend to recur
67
When should pregnancy epulis treated
regresses spontaneously after child is born | excision should be delayed until this time
68
What are peripheral giant cell granulomas
pedunculate or sessile swellings typically dark, red and ulcerated usually arises inter proximally and has an hour glass shape radiographs may reveal superficial erosion of interdental bone may recur after surgical excision
69
What makes peripheral giant cell granuloma different from other epulis
it is a focal collection of multinucleate giant cells in a rich vascular and cell stroke
70
What are congenital epulis
``` rare occur in neonates most common in anterior maxilla more common in fmelaes granular cells covered with epithelium benign simple excision is curative ```
71
What are HPV associated swellings for
verruca vulgaris | squamous cell papilloma
72
What is verruca vulgarizes
solitary or multiple intra oral lesions may be associated with skin warts most resolve spotnanesouly can be removed surgically
73
What is the cause of verruca valgarizes
caused by HPV 2 and 4
74
What is verruca vulgarisms most commonly seen
most commonly on keratinsed tissue - gingiva and palate
75
What is squamous cell papilloma
small pedunculate cauliflower like growths benign vary in color from pink to white usually solitary treatment is surgical excision
76
What are the causes of squamous cell papilloma
HPV 6 & 11
77
What are the fluid swellings
``` mucoceles ranula Johns nodules epstein perals haemangiomas vesiculobullous lesions ```
78
What are the vesiculobullous lesions
primary herpes epidermolysis bullosa erythema multiform
79
What are the 2 variants of mucocele
mucous extravasion cyst | mucous retention cyst
80
What is a mucous extraversion cyst
normal secretions rupture into adjacent tissue
81
What is a mucous retention cyst
secretions retained in an expanded duct
82
What are mucoceles
bluish soft transparent cystic swelling can affect minor or major salivary glands most common is minor glands of lower lip peak incidence is second decade most will rupture spontaneously surgery only if lesion is fixed in size as will likely damage the adjacent glands leading to recurrence surgical excision involves removal of cyst and adjacent damaged minor glands
83
What is a ranula
it is a mucocele in the floor of the mouth
84
What can ranulas arise from
minor salivary glands or ducts of sublingual / submandibular glands
85
Why is a MRI or ultrasound needed for a ranula
excludes purging ranulas | extend through the FOM into sub mental or mandibular space
86
What can ranulas occasionally be found to be
lymphangioma - benign tumor of lymphatics
87
What are Bohn's nodules
``` gingival cysts remnants of dental lamina filled with keratin occur on the alveolar rdige found in neonates usually disappear ```
88
What are Epstein pearls
``` small cystic lesions found along palatal midline thought to be trapped epithelium in palatal raphe common disappears in first few weeks ```
89
What is TMJ dysfunction syndrome characterized by
pain masticatory muscle spasm limited jaw opening
90
What questions should be asked in a TMJDS history
``` description of presenting symptoms when did discomfort begin is pain worse at any time during day what are exacerbating factors habits stress ```
91
What should an extra oral exam for TMJDS include
palpation of muscles of mastication both at rest and when teeth are clenched to assess tenderness and / or hypertrophy palpation of the TMJ at rest and when opening and closing to assess tenderness and click/creptius assessment of opening
92
When assessing opening what are we looking at
check for deviation of jaw | assess extent of opening (normal 40-50mm)
93
What does an intra oral examination of TMJDS include
assessment of any dental wear facets | signs of clenching and grinding
94
What are signs of clenching and grinding
scalloped lateral tongue surface | buccal mucosa ridges
95
What is the management of TMJDS
``` explain the condition reduction of exacerbating factors allow the over worked muscles to rest symptomatic relief if these measures are unsuccessful then referral for specialist care is indicated ```
96
How can we reduce exacerbating factors of TMJDS
management of stress - mindfulness or yoga avoid habits such as clenching, grinding, chewing gum, nail biting or leaning on the jaw a bit raising appliance may be considered if there is nocturnal grinding/clenching
97
How can we allow the over worked muscles in TMJDS to rest
avoid wide opening - stifle yawns with a closed fist | soft diet which requires little chewing
98
What is the symptomatic relief for TMJDS
use of ibuprofen which has anti-inflammatory action alternate use of hot and cold packs if these measure are unsuccessful then referral for specialist care is indicated