paediatric ITU Flashcards

1
Q

pads

A

consider attention span / co-operation
patient therapist interaction is difference vs adults
communication syle
consider family /carer

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2
Q

physiological difference

between adult and child

A

increased compliance in chest wall
decreased compliance in lungs
meaning decreased FRC and increased work of breathing

decreased % of T1 muscle fibres in diaphragm
diaphragm more prone to fatigue –> less able to withstand respiratory distress

increased metabolic rate ov O2 consumption
–> hypoxaemia develops more rapidly

preferential ventilation of upper lung when positioned in side lying

ensure SaO2 maintenance when repositioning

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3
Q

anatomical difference

A

decrease d of airways
increase ariway resistance
increase respiratory difficulties with any inflammation of airways

preferential nasal breathers
NG tubes
narrow d and increase WOB
ensure nasal passages are cleared of secretions

larger tonsils and tongue
increase airway obstruction

floppy cartilage
predisposes to airway collapse

more horizontal ribs and more cylindrical
lack of bucket handle and pump handle movement, unable to increase lung volumes

poorly developed intercostal muscles
nearly solely reliant on diaphragm for respiration

immature cilia
increased accumulation of secretions, mucus plugging

decreased alveolar surface area
decreased space for gas exchange

increased heart size in infants

less room for lung expansion

poor collateral ventilation in infancy

increased risk of atelectasis during RTI

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4
Q

collateral ventilation

A

Adults have a developed respiratory system therefore more collateral ventilation
Functional in paediatrics:
Pores of Kohn: 1-2 years (intra aveolar)
Canals of Lambert: 4-6 years (Bronchiol-aveolar)
Channels of Martin: Develop in pathology (Interbronchial)

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5
Q

ITU assessment

A
NB Respiratory +/- neurological if appropriate depending on PC
Subjective Assessment
Objective Assessment
Problem List
Treatment Plan
Goals
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6
Q

subjective assessment in ITU

A

Presenting complaint
History of presenting complaint
Past medical history
Respiratory (normal chest status)
Orthopaedic (spinal deformities)
Neurological (reduced cough, risk of aspiration)
Bone health (osteopenia, osteoporosis)
Seek information from parents/carers if present
Information from staff nurse/ medical staff /ICIP including update on medical /nursing status
Tolerance of interventions / handling over last 12 – 24 hours

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7
Q

subjective assessment

birth history

A

Birth history
Particularly relevant for neonates and children with post natal problems
Method of delivery (immediate delivery, trauma)
Gestation and weight (lung development)
? SCBU (length of time, ventilation, oxygen requirements)
Chronic lung disease

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8
Q
subjective assessment 
developmental history 
family history 
medications 
other considerations
A
Developmental History
Normal level of cognition and functioning
Local service, specialised equipment used
Family history
Parents and siblings fit and well?
Smokers, pets at home?
Recent travel?
Genetics
Parental responsibility
Child protections issues
medications 
at home 
nebulisers 
patch - hyoscine 
order of medications
Pain relief, sedation due?
How does the child respond to handling?
Apnoeas? Bradycardias?
When was the last feed?
Can the child cooperate?
What is the parents’ understanding?
Consent for respiratory treatment
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9
Q

objective assessment

A
observation 
Environment in ITU (Emergency bell, trolley, MIE set, suction) 
Your patient:
Breathing pattern
Colour (pink, pale, cyanosed)
Level of alertness
Are they puffy periorbitally, peripherally
Position   
Lines: ETT, arterial line, catheter

ventilation
is the patient intubated and ventilated
nasally vs oral
what ventilation mode is the patient on

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10
Q

ventilation modes

vitals

A
Main modes in ICU:
SIMV(PC)+PS
PRVC
CPAP+PS (weaning mode)
HFOV
NIV (CPAP+BiPAP)

Aim TV’s 6-8mls/kg
4-6mls/kg in adults

baseline vitals from subjective exam if known respiratory pathology 
neuro GCS
temp HR
RR BP SaO2 CRT
PEWS
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11
Q

objective assessment
auscultation
palpation
expansion

A
auscultation 
air entry 
added sounds: creps
wheeze 
stridor if extubated 

palpation
any palpable remits

expansion

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12
Q

signs of respiratory distress

/WOB

A
tachypnoea 
nasal flare 
tracheal tug 
recession - intercostal, subcostal, substernal 
mild moderate severe 
grunting 
paradoxical breathing 
preparation and tense 
sternal retraction 
intercostal retractions
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13
Q

cough

A

if known respiratory condition may have an objective PCF measurement
if cough is strong or weak effective, dry or wet

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14
Q

chest x-ray considerations

A
position of ETT if intubated 
exposure and rotation of x-ray 
chest shape 
bones 
lung fields 
NG tube placement 
compare to previous if appropriate
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15
Q

blood

A

white cell count
Hb
platelets
CRP

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16
Q

ABGs

A

resp acid decrease pH increase pCO2 normal HCO3

resp alk. increase pH decrease pCO2 normal HCO3

metabolic acidosis
decrease pH normal PCO2
decrease HCO3

metabolic alk
increase pH
normal PCO2
increase HCO3

17
Q

medications

A
sedations 
cardiac support 
antibiotics 
nebulised therapies 
anti convulsants 
diuretics 

fluid output
24 hour, length of stay

18
Q

microbiology

A

Nasopharyngeal aspirate (tests for viral conditions e.g bronchiolitis, influenza, coronovirus etc)
Sputum sample
Broncheoaveolar lavage
*Check previous sputum samples

19
Q

other investigations

feeds

A

Other investigations:
CT thorax, DEXA, sleep studies

Feeds:
Oral, NG, NJ, PEG
Continuous vs bolus
Feeds must be stopped for >30mins prior to physiotherapy

20
Q

indications for physio

A

chest xray changes
lobar collapse / atelectasis

retained pulmonary secretions

aspiration pneumonia

absent / ineffective cough

deteriorating ABGs
respiratory infection

background pathology
CF
NM
neurological disease

post op
prolonged immobility

21
Q

aims of physio

A

Minimise the adverse effects of critical illness and intubation on the respiratory system

  • Reduction of secretion retention
  • Maintain alveolar expansion
  • Improvement of V/Q matching
  • Reduce airway resistance
  • Optimise oxygenation
  • Preservation of respiratory and peripheral muscle strength

Comellini et al 2019

22
Q

physiology of secretion clearance

A

The mucociliary escalator consists of mucus producing goblet cells and the ciliated epithelium
Cilia beat in co-ordinated, efficient movements

23
Q

mucus clearance in ITU

A

Patients admitted to ICU suffer from severe impairment of airway clearance capacity
Muscular weakness and sedation decrease ability to cough – retained secretions
If I+V mucociliary transport stops at the end of the ETT
Mucus movement in the airways occurs through 2 primary mechanisms:
Expulsive airflow (i.e coughing) however if ETT present holds glottis open
Two phase gas liquid interactions

24
Q

expulsive flow

A

normal cough mechanism
deep inspiration of 80-90% of total lung capacity
glottic closure with a pause
effective contraction of expiratory muscles to generate rapid expiratory flows

25
Q

two phase gas liquid interaction

A

The transfer of kinetic airflow energy onto liquid airway surface exerts a shear force moving sputum along the airway mucus layer in the direction of proximal airflow

Airflow remains laminar when flow rates are low

As flow velocity increases the mucus layer becomes unstable and forms ripples that move in the direction of the air flow

26
Q

treatment techniques

A
reposition 
manual techniques 
MHI 
saline instillation 
suction 
cough assist 
early mobility
27
Q

repositioning

A

Positioning to optimise ventilation or postural drainage
Children preferentially ventilate the uppermost lung
Head down tip avoided in babies as risk of GOR and aspiration

  • Used alongside other techniques, can be of benefit to advise nursing staff positioning over 24 hour period
  • Positioning specific programmes at bedside
28
Q

manual techniques

A

Percussions:
Rhythmical cupped hand or palm cup over chest wall

Contraindications:
Undrained pneumothorax
Raised intracranial pressure
Rib fractures
Osteopenia/Osteoporosis
Platelets of <40
Pulmonary haemorrhage

expiratory vibrations
compressive vibratory pressure on chest wall during expiration

CI: as per percussions

29
Q

Manual hyperinflation

performing MHI

A

Prevent atelectasis and recruit areas of collapsed lung
Improve lung compliance and gas exchange
Increases movement of pulmonary secretions toward central airways
Prevent airway mucus plugging

Use manometer

2 breaths at PIP/PEEP on ventilator, 3rd breath 10% above PIP

Followed by end inspiratory hold with quick release to increase expiratory air flow

30
Q

manual hyperinflation with expiratory vibrations

A

For effective treatment to mobilise secretions effectively:
- Expiratory flow during MHI must be greater then inspiratory flow

MHI with expiratory vibrations significantly increases expiratory flow to facilitate expulsive airflow and mobilisation of secretions
- More effective than just MHI alone

Expiratory vibrations should be applied at early expiration (during quick release phase) to achieve greatest expiratory flow, therefore mobilising the most secretions

MHI with expiratory vibrations:
80% increase in expiratory flow

31
Q

MHI - CI / precautions

A
Contraindications/precautions for MHI:
Cardiovascular instability
Raised intracranial pressure
Labile BP
Undrained pneumothorax
High ventilatory pressures
Recent pneumonectomy
Bullae
Recent lung surgery
Haemoptysis
Clamped chest drain
32
Q

suctioning
aims
CI/ PRECAUTIONS

A

To clear secretions thus maintaining a patent airway
To improve ventilation and oxygenation
To reduce the work of breathing
Contraindications/Precautions for suctioning:
- Unexplained haemoptysis
- Laryngospasm (stridor)
- Bronchospasm
- Basal skull fractures (no nasal suction)
- Recent oesophageal or tracheal surgeries
- Severe hypoxia
- Raised intracranial pressure
- Acute hypo or hypertension

33
Q

oropharyngeal suctioning

nasopharyngeal suctioning

A

Oropharyngeal suctioning: Measure corner of mouth to ear lobe and add approx 2 cms

Nasopharyngeal suctioning: Measure tip of nose to ear lobe to thryoid cartilage

34
Q

suction

A

tracheostomy patients
Ensure appropriate depth of suction
High risk of airway trauma and granulation tissue

35
Q

suction

A
Beta-2 agonists (Salbutomol, atrovent)
Effects:
Bronchodilates airways
Reduces inflammation
Improve mucocillary clearance
Mucolytics (0.9%, 3%, 7% saline)
Effects: 
- Rehydrate secretions
- Increase airway surface liquid
- Improve mucocilliary clearance 
- Stimulate cough
Beta-2 agonist nebuliser
                 ↓  (Wait 15 mins)

              Mucolytic

                  ↓

    Chest physiotherapy
36
Q

cough assist

cough assist precautions

A

Appliance of a positive pressure to the airway, followed by a rapid shift to negative pressures

Rapid shift increases expiratory flow aim ↑ peak cough flow

Cough assist in ICU:
Can reduce reintubation rates and ICU length of stay
Shorten treatment time in ICU without any difference in secretion clearance

As per positive pressure MHI

  • Cardiovascular instability
  • High intracranial pressure
  • Undrained pneumothorax
  • High ventilatory pressures
  • Recent pneumonectomy
  • Bullae
  • Recent lung surgery
  • Haemoptysis
  • Clamped chest drain
37
Q

early mobility in ITU

benefits

CI

A

PICU mortality rates have ↓, however the proportion of children surviving their critical illness with significant functional morbidity is rising
Prolonged immobility is an important predictor of poor functional outcome and the development of critical illness acquired morbidities
There is growing evidence that demonstrates the benefits of early implementation of rehabilitation and mobilisation

Reduces length of stay in ICU and overall length of stay
Secretion mobilisation and clearance
Increased TV, RR, MV
Improves levels of consciousness
Assists fluid distribution
Improves venous return
Reduces length ICU delerium
Improves sleep 
CV instability
Spinal precautsios
Unstable fractures
Open abdomen
Open chest
ECMO
<24 hrs admission to PICU