paediatric ITU Flashcards
pads
consider attention span / co-operation
patient therapist interaction is difference vs adults
communication syle
consider family /carer
physiological difference
between adult and child
increased compliance in chest wall
decreased compliance in lungs
meaning decreased FRC and increased work of breathing
decreased % of T1 muscle fibres in diaphragm
diaphragm more prone to fatigue –> less able to withstand respiratory distress
increased metabolic rate ov O2 consumption
–> hypoxaemia develops more rapidly
preferential ventilation of upper lung when positioned in side lying
ensure SaO2 maintenance when repositioning
anatomical difference
decrease d of airways
increase ariway resistance
increase respiratory difficulties with any inflammation of airways
preferential nasal breathers
NG tubes
narrow d and increase WOB
ensure nasal passages are cleared of secretions
larger tonsils and tongue
increase airway obstruction
floppy cartilage
predisposes to airway collapse
more horizontal ribs and more cylindrical
lack of bucket handle and pump handle movement, unable to increase lung volumes
poorly developed intercostal muscles
nearly solely reliant on diaphragm for respiration
immature cilia
increased accumulation of secretions, mucus plugging
decreased alveolar surface area
decreased space for gas exchange
increased heart size in infants
less room for lung expansion
poor collateral ventilation in infancy
increased risk of atelectasis during RTI
collateral ventilation
Adults have a developed respiratory system therefore more collateral ventilation
Functional in paediatrics:
Pores of Kohn: 1-2 years (intra aveolar)
Canals of Lambert: 4-6 years (Bronchiol-aveolar)
Channels of Martin: Develop in pathology (Interbronchial)
ITU assessment
NB Respiratory +/- neurological if appropriate depending on PC Subjective Assessment Objective Assessment Problem List Treatment Plan Goals
subjective assessment in ITU
Presenting complaint
History of presenting complaint
Past medical history
Respiratory (normal chest status)
Orthopaedic (spinal deformities)
Neurological (reduced cough, risk of aspiration)
Bone health (osteopenia, osteoporosis)
Seek information from parents/carers if present
Information from staff nurse/ medical staff /ICIP including update on medical /nursing status
Tolerance of interventions / handling over last 12 – 24 hours
subjective assessment
birth history
Birth history
Particularly relevant for neonates and children with post natal problems
Method of delivery (immediate delivery, trauma)
Gestation and weight (lung development)
? SCBU (length of time, ventilation, oxygen requirements)
Chronic lung disease
subjective assessment developmental history family history medications other considerations
Developmental History Normal level of cognition and functioning Local service, specialised equipment used Family history Parents and siblings fit and well? Smokers, pets at home? Recent travel? Genetics Parental responsibility Child protections issues medications at home nebulisers patch - hyoscine order of medications Pain relief, sedation due? How does the child respond to handling? Apnoeas? Bradycardias? When was the last feed? Can the child cooperate? What is the parents’ understanding? Consent for respiratory treatment
objective assessment
observation Environment in ITU (Emergency bell, trolley, MIE set, suction) Your patient: Breathing pattern Colour (pink, pale, cyanosed) Level of alertness Are they puffy periorbitally, peripherally Position Lines: ETT, arterial line, catheter
ventilation
is the patient intubated and ventilated
nasally vs oral
what ventilation mode is the patient on
ventilation modes
vitals
Main modes in ICU: SIMV(PC)+PS PRVC CPAP+PS (weaning mode) HFOV NIV (CPAP+BiPAP)
Aim TV’s 6-8mls/kg
4-6mls/kg in adults
baseline vitals from subjective exam if known respiratory pathology neuro GCS temp HR RR BP SaO2 CRT PEWS
objective assessment
auscultation
palpation
expansion
auscultation air entry added sounds: creps wheeze stridor if extubated
palpation
any palpable remits
expansion
signs of respiratory distress
/WOB
tachypnoea nasal flare tracheal tug recession - intercostal, subcostal, substernal mild moderate severe grunting paradoxical breathing preparation and tense sternal retraction intercostal retractions
cough
if known respiratory condition may have an objective PCF measurement
if cough is strong or weak effective, dry or wet
chest x-ray considerations
position of ETT if intubated exposure and rotation of x-ray chest shape bones lung fields NG tube placement compare to previous if appropriate
blood
white cell count
Hb
platelets
CRP
ABGs
resp acid decrease pH increase pCO2 normal HCO3
resp alk. increase pH decrease pCO2 normal HCO3
metabolic acidosis
decrease pH normal PCO2
decrease HCO3
metabolic alk
increase pH
normal PCO2
increase HCO3
medications
sedations cardiac support antibiotics nebulised therapies anti convulsants diuretics
fluid output
24 hour, length of stay
microbiology
Nasopharyngeal aspirate (tests for viral conditions e.g bronchiolitis, influenza, coronovirus etc)
Sputum sample
Broncheoaveolar lavage
*Check previous sputum samples
other investigations
feeds
Other investigations:
CT thorax, DEXA, sleep studies
Feeds:
Oral, NG, NJ, PEG
Continuous vs bolus
Feeds must be stopped for >30mins prior to physiotherapy
indications for physio
chest xray changes
lobar collapse / atelectasis
retained pulmonary secretions
aspiration pneumonia
absent / ineffective cough
deteriorating ABGs
respiratory infection
background pathology
CF
NM
neurological disease
post op
prolonged immobility
aims of physio
Minimise the adverse effects of critical illness and intubation on the respiratory system
- Reduction of secretion retention
- Maintain alveolar expansion
- Improvement of V/Q matching
- Reduce airway resistance
- Optimise oxygenation
- Preservation of respiratory and peripheral muscle strength
Comellini et al 2019
physiology of secretion clearance
The mucociliary escalator consists of mucus producing goblet cells and the ciliated epithelium
Cilia beat in co-ordinated, efficient movements
mucus clearance in ITU
Patients admitted to ICU suffer from severe impairment of airway clearance capacity
Muscular weakness and sedation decrease ability to cough – retained secretions
If I+V mucociliary transport stops at the end of the ETT
Mucus movement in the airways occurs through 2 primary mechanisms:
Expulsive airflow (i.e coughing) however if ETT present holds glottis open
Two phase gas liquid interactions
expulsive flow
normal cough mechanism
deep inspiration of 80-90% of total lung capacity
glottic closure with a pause
effective contraction of expiratory muscles to generate rapid expiratory flows