hyperinflation and suctioning Flashcards

1
Q

hyperinflation

A

giving a patient an augmented tidal volume via manual bag or ventilator to a patient with an artificial airway
manual vs ventilator delivered

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2
Q

ventilated critically ill patient

A
increase sputum
tracheal membrane irritation 
impaired mucociliary clearance 
decreased cough+ ciliary clearance 
loss of compliance 
loss of sigh breaths 
progresive atelectasis
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3
Q

indications

A
prevention and treatment of atelectasis 
mobilisation of retained secretions 
assess and increase lung compliance 
stimulate FET 
improve gas exchange 
improve oxygenation pre/post suction
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4
Q

precautions /CI

A
CI 
undrained PT
head injury ICP >25mmhg
bronchopleural fistula 
lung abscess 
bullae
Precautions 
unstable CV system 
high PEEP 
nitric Oxide dependency 
high peak airway pressure 
raised ICP and head injuries 
severe bronchospasm /wheeze
high fiO2 requirements 
acute pulmonary oedema 
haemoptysis 
pressure control ventilation 
recent lung surgery 
lung abscess
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5
Q

MHI technique

A

patient disconnected from ventilator and given an altered breathing pattern
slow inflation flow rate
larger than tidal volume breath
end-inspiratory hold to utilise collateral ventilation channels
quick release to enhance expiratory flow rate
interspersed with tidal volume breaths

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6
Q

equipment

A
2L bag for adults 
mapleson C-circuit 
O2 flow 15 Ipm
peak insp pressure 30-40cm 
not over 40 as risk of barotrauma
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7
Q

how does it aid secretion clearance

A

increase TV increases elastic recoil and hence increase exp flow rates
quick release produces a fast expiratory flow mimicking a cough
uses CV channels
bronchiole=alveolar channels of Lambert
idntraalveolar pores of Kohn
can recruit air distal to a mucus plug which can help propel it into more proximal airways

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8
Q

how does it aid lung recruitment

MHI

A

Slow, sustained inspiration
Low inspiratory flow
 P = Flow  Resistance
Allows better distribution of gas to slow filling lung units
Inspiratory Hold utilises collateral ventilation
Increased tidal volumes
Interdependence: Expanding force one alveoli exerts on neighbouring alveoli (greater stretch 2° greater pressure)

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9
Q

how does it improve gas exchange MHI

A

increase TV + longer inspiratory time = increase gas exchange

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10
Q

MHI - how does it increase compliance

A

alveolar recruitment
increase FRC
increased compliance
(lungs more stretchable)

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11
Q

treatment regime

A

based on tolerance response and efficacy of modality

6 sets of 6 hyperinflation breaths with 30s TV breaths between each set

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12
Q

latent effects

A

Monitor SaO2, BP, HR etc
Document values pre- and post- treatment
Notify nursing staff before leaving the area if manual hyperinflation is performed
Check prior to next treatment for latent effects and stability

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13
Q

VHI define

A

Definition: VHI involves altering the ventilator settings to gradually increase tidal volume (Imle & Klemic 1989)

Proposed to produce the same effects as MHI whilst controlling airway pressures and maintaining PEEP (Brown 1987)

changing pressure or volume or insp time
can add in inspiratory hold

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14
Q

VHI method

A

gradually increase TV by 50% but peak airway pressures should be limited to 40cmH2O

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15
Q

MHI VHI

A
MHI 
disconnection 
P reg by valve 
mimic FET 
assess compliance 
VHI 
no disconnection/ PEEP maintained 
V/P controlled by vent 
no FET 
unable to assess compliance 
possibly less secretion removal 
easier to control Airway pressures
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16
Q

positive effect of MHI

A
re-expansion of acute atelectasis 
increased oxygenation 
enhanced removal of secretions 
reduced inspiratory resistance 
increased lung compliance
17
Q

MHI - key points

A
ALWAYS use a manometer within the circuit
Assess patient, monitors continually
Larger tidal volumes generated can:
Mobilise secretions
Help resolve atelectasis
Improve lung compliance
Improve gas exchange
18
Q

suctioning

A

define:
mechanical aspiration of pulmonary secretions from a patient with an artificial airway in place
involves patient prep, suctioning events and follow up care.

19
Q

indications for suctioning

A
audible secretions on auscultation 
visible secretions in airway
increase work of breathing 
need to maintain latency and integrity of airways 
deterioration of ABGs/ obs
20
Q

precautions for suctioning

A
CV instability 
suspected/ confirmed increase in ICP 
bronchospasm 
base of skull fracture 
recent upper GI/lung surgery 
bronchopleural fistula 
disordered coagulation/haemoptysis 
dependence on high level ventilatory support
21
Q

hazards include

A
Hypoxia/hypoxemia
Tissue trauma to the trache and/or bronchial mucosa
Cardiac arrest
Respiratory arrest
Cardiac Dysrythmias
Pulmonary atelectasis
InfectionBronchospasm/ Bronchoconstriction
Pulmonary haemorrhage
Elevated Intracranial pressure
Hypertension
Hypotension
22
Q

types of suctioning

A

Endotracheal
Trachesotomy
Nasopharyngeal
Orophayngeal

23
Q

Airway

A

nasopharyngeal
guedel airway
trace
ETT - closed or open

24
Q

Assessment of respiratory status

A
breathing pattern 
can they clear their secretions?
are they too proximal?
if they can't why not?
RR HR BP temp 
CXR
auscultation
 humidification 
recent events 
colour and tenacity of secretions
25
Q

equipment required

A
apron gloves 
PPE 
portable machine 
catheters
sterile gloves 
oxygenation equipment 
(pre and post oxygenate patients)
SaO2 monitor
26
Q

preparation of patient

A

explain to paitent
hyper oxygenation
100% oxygen for >30 seconds prior to suctioning
patient monitored using pulse oximeter

27
Q

suction event

A
sterile technique 
continuous suction technique 
<15 seconds per suction event 
fast in slow out 
suction pressure mx safe limits 100-150mmHg
28
Q

Follow up care

A

pt should be monitored for adverse reactions
hyperoxygnated for 1 min 100%
breathing control
position of ease
RR and O2 sats return to at lease pre suction levels

29
Q

Assessment of outcome

A

removal of pulmonary secretions
improvement of breathing sounds
decreased work of breathing
improvements of ABGs or SaO2