Orthopedics Flashcards
where do bone metastases arise from?
THE PUBLIK
Thyroid
H?
E oesophageal
Prostate (!) Urinary bladder Breast (!) Lung (!) Intestines Kidneys
(!) indicated most common
features of a benign bone lesion
a geographic border (well demarcated)
narrow zone of transition
single layered periosteal reaction
e.g. non-ossifying fibroma (common in children and adolescents), osteochondroma (owergrowth near growth plate, common in ages 10-30) and endochondroma (ages 10-30)
features of malignant bone lesions
a poorly demarcated border
wide zone of transition
multi-layered periosteal reaction (types –> codman triangle, onion-peel, sunburst)
e.g. osteomyelitis, Ewing’s sarcoma (5-15 y/o), osteosarcoma (10-30 y/o - second commonest primary bone tumour) and multiple myeloma (>40 y/o - the commonest primary bone tumour - spine, skull, ribs and pelvis)
NOF O/E
abducted, externally rotated and shortened
NOF location and risk of AVN
displaced intracapsular # - high risk of AVN (total hip replacement/ hemi-arthroplasty)
undisplaced intracapsular # - moderate risk of AVN (pin)
intratrochanteric or subtrochanteric extracapsular # - low risk of AVN (dynamic hip screw)
mortality risk after a #NOF
10% in 1 month
33% in 1 year
what is Cauda Equina syndrome
compression of the nerves below the level of L1
can be due to malignancy, a slipped disc, vertebral #, trauma, infection
symptoms - bilateral sciatica pain, numbness around anus, painless urinary retention leading to overflow incontinence and bowel retention
must check sensation, muscle tone, muscle weakness and the rectal cough reflex
–> MRI to confirm and surgery within 48hrs
Salter-Harris classification of epiphyseal injuries
SALT CRUSH
1 - Straight across (sheer injury of growth plate)
2 - Above (above GP with metaphysis #)
3 - Lower (# of epiphysis
4 - Through (# through epiphyseal GP and metaphysis)
5 - CRUSH (crush injury to GP)
median nerve
sensory: lateral palm, thumb and lateral 2.5 fingers
motor: LOAF hand muscles
movement thumb abduction against resistance
injury: carpal tunnel
palsy: thenar eminence wasting, hand of benediction (can’t make a fist), weak thumb abduction (ape hand - can’t oppose)
tests: Tinel/Phalen +ve
ulnar nerve
sensory: medial hand and medial 1,5 fingers
motor: small muscle of the
movement: finger abduction against resistance
injury: elbow (Golfer’s elbow - medial)
palsy: hypothenar eminence and 1st web wasting, claw hand (can’t straighten), weak finger abduction
tests: +ve Froment’s sign
radial nerve
sensory: lateral dorsum of hand
motor: extensors - fingers, wrist and elbow
movement: wrist/ finger extension against resistance
injury: humeral shaft #
palsy: Saturday night palsy. wrist drop, weak inger and wrist extension
where in the finger does a Mallet finger deformity affect?
the DIP joint (bent forward)
can be due to an avulsion injury or due to the extensor tendon rupture
where in the finger does a Boutonniere deformity affect?
the PIP joint (will not straighten and the DIP is bent back)can be due to trauma or RA
causes of Carpal Tunnel syndrome?
MEDIAN TRAP
Myxoedema E - oedema premenstrual Diabetes Idiopathic Acromegaly Neoplasm
Trauma
RA
Amyloidosis
Pregnancy
Compartment Syndrome
> 20mmHg pressure is abnormal but >40mmHg is abnormal
6Ps Pain (excessive for injury present) Paraesthesia Paresis (muscle weakness) Poikilothermia (impaired temperature, very cold) Pallor Pulseless
–> emergency fasciotomy
what classification is used to classify open fractures?
the Gustilo and Anderson classification
how would you manage an open fracture?
ABCDE Ax, analgesia and tetanus booster control bleeding - fluid/ blood replacement assess soft tissue and NV status cover wound with sterile saline gauze splint # and reassess NV status surgical debridement and stabilisation
Weber’s classification of distal fibular #s
A - below syndesmosis (usually stable - reduce and cast)
B - at level of syndesmosis (variable stability - may require open reduction and internal fixation ORIF)
C - above level of syndesmosis (unstable - ORIF required)
Osgood Schlatter’s disease
inflammation of the area just below the knee where the patellar tendon attaches to the tibia
most often occurs during growth spurts (10-14)
conservative treatment - ice, analgesics and NSAIDs
Perthes Disease
rare childhood (4-10) condition of the hip causing AVN ~15% of cases are bilateral
stages:
initial/ necrosis: limb and change of gait (several months)
fragmentation: woven bone replaces dead bone (1-2 years)
re-ossification: stronger bone develops
healed:
symptoms - limp/ change of gait, pain in hip/groin, muscle spasm, if untreated can lead to early onset arthritis
treatment –> observation and limited activity, NSAIDSs, physio, casting and bracing
septic arthritis
50% of cases are in the knee, the most common organism is Staph Aureus
bacterial seeding via bacteraemia, direct inoculation or contiguous spread
causes irreversible cartilage destruction
symptoms - pain, fever, erythema, effusion, warm and tender, inability to weight bear and unable to do PROM
extremity is FABER (flexed, abducted and externally rotated)
–> IV Ax. .operative irrigation and drainage
tendonopathies
disease of the tendon, pain on palpation and on movement
spinal stenosis
narrowing of the spinal canal or neural foramina producing root ischaemia and neurogenic claudication
DDx - cauda equina syndrome
osteoarthris
degenerative joint disorder, progressive loss of hyaline cartilage and new bone formation at the joint surface
risk factors: age, female, obesity and developmental dysplasia of the hip
–> analgesics, intracapsular injections and total hip replacement
main signs on X-ray (LOSS): loss of joint space, osteophytes, subchondral cysts and subchondral sclerosis