ORTHO Flashcards

1
Q

What is jumpers knee?

A

Patellar tendonitis

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2
Q

What occurs in Patellar tendinitis ?

A

microtears at tendon insertion at distal pole

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3
Q

Where does patella tendon insert?

A

Inserts into the tibial tuberosity

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4
Q

What are clinical signs of patella tendinitis?

A

pain in anterior knee
thickening and swelling
tender to palpation on tibial tuberosity

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5
Q

treatment options for patella tendinitis?

A

RICE, NSAIDS, Strapping, Brace

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6
Q

what is Osgood Schlaters injury?

A

Inflammation of the patella tendon (ligament) at the insertion point on the tibial tuberosity

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7
Q

What age and gender is osgood schlaters injury most common in ?

A

more common in boys aged 10-15 years of age

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8
Q

What is mechanism for osgood chlaters injury?

A

repeated tensil stress on the tendon leads to minor avulsion and inflammation at the tibial tuberosity head.

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9
Q

What is clinical presentation and clinical examiniation sign for osgood schlaters injury ?

A

pain on anterior knee.
exacerbated by kneeling or by jumping.

Examination will show:

  1. tender lump over the tibial tuberosity
  2. pain on resisted knee extension
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10
Q

What is treatment options for osgood schlaters injury?

A

treatment is symptomatic reflief as:

Benign, self limited condition

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11
Q

When will osgood schlaters injury most likely resolve?

A

resolves at growth hiatus

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12
Q

Anterior Knee Pain Causes

A
Patellofemoral Syndrome (young females) 
OSgood Schlatters (young males) 
Chrondromalacia Patellae
Lateral Patellar Compression Syndrome 
ITB syndrome 
Patellar Instability (dislocation/sublaxation) 
Patellar tenditnitis 
Pre-oatellar bursitis 
Plica
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13
Q

Knee Dislocation: What is this associated with in terms of injury?

A

EMERGENCY: rare but serious: indicates severe injurysuch as high speed MVA with associated tears of multiple ligaments

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14
Q

What is seen physical examination?

A

Knee large effusion, swelling, pain and instability and ischaemic limb (due to popliteal artery injury) and potentially peroneal nerve injury

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15
Q

What is treatment of knee dislocation?

A
  1. Non operative URGENT closed reduction
  2. Operative ( vascular repair and ligament repair/reconstruction)
  3. Additional 6 weeks immobilisation
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16
Q

What are the acute complications of knee dislocation?

A

Common peroneal nerve injury
Popliteal artery occlusion
Compartment syndrome

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17
Q

What are 3 long term complications of a knee dislocation ?

A

Chronic stiffness (#1)
Chronic knee instability
post-traumatic arthritis

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18
Q

What is important about knee dislocation and the popliteal artery?

A

Popliteal artery occlusion can occur within the first 12 hours = important to check regularly on patients

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19
Q

What is used to diagnose knee dislocation ?

A

XRAY: AP/Lateral
CT Angiogram: Evaulate for arterial injury
Ankle Brachial Index: <0.9 indicates abnormalities
MRI: ligament injury, meniscus

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20
Q

MCL injuries: Mechanism they occur

A

Valgus force to knee ( outside to in) common in football

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21
Q

Haemarthrosis: WHy might it not be present in severe injuries?

A

Due to capsule disruption

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22
Q

MCL injuries: What is seen on examination

A

Tenderness at medial epicondyle and along tendon (inserts at around 4-6cm below tibial plateua, deep to pes aneurius)
Laxity/pain in valgus

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23
Q

What is MCL associated with injury wise?

A

ACL and medial meniscus (unhappy triad)

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24
Q

IN the case of multi-ligamntary injuried knee: treatment involves?

A

surgery

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25
Q

Treatment MCL:

A

Mostly conservative: ROM and strengthening and hinge knee brace.
Surgery is uncommon

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26
Q

Slipped capital femoral epiphysis:

What is this ?
what type salter harris

A

femoral head stays in acetabulum but slips inferiorly and the metaphysis slips nateirorlya nd superiorly = causes hip pain and a limp

Type 1 Salter Harris

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27
Q

Slipped capital femoral epiphysis: Is this bilateral or unilateral usually?

A

50% cases bilateral

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28
Q

Slipped capital femoral epiphysis: what occurs to the femur and metaphysis ?

A

Femur head remains in the acetabulum whilst the metaphysis slipps out anteriorly and superiorly

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29
Q

Slipped capital femoral epiphysis: what is this most common?

A

MOST common adolescent hip disorder

commonly occurs in puberty growth spurt

30
Q

Slipped capital femoral epiphysis: when is peak incidence

A

pubertal growth spurt

31
Q

Slipped capital femoral epiphysis: risk factors

A

obesity (main)
Male
hypothyroidism (risk for bilateral as lack of thyroid hormone influences bone age and growth hormone)

32
Q

What is clinical presentation for SCFE?
What movements restricted?

Where tender?

A

Dull hip pain/referred knee pain with painful limp

restricted internal rotation and abduction (pts tend to hold in passive external rotation)

tenderness of joint capsule

33
Q

SCFE: inability to ambulate/weight bear is classified as ?

A

unstable SCFE

34
Q

What are 2 diagnostic methods for SCFE ?

A

XRAY - AP and and frog leg lateral

TSH to rule out hypothyroid

35
Q

What will XRAY demonstrated for SCFE?

A

posterior and inferior displacement of the femoral head.

36
Q

What is treatment for SCFE and why is fast treatment inmportant ?

A

Immediate surgical screw fixation (reduces risk of AVN)

No weight bearing allowed

37
Q

What are three copmlications of SCFE ?

A

AVN
chondrolysis (resulting in loss of articular cartilage, narrowing of joint space)
premature hip osteoarthritis

38
Q

osteomyelitis pathophysiology

A

Most commonly due to S aureus via inoculation, haematogenous spread or contiguous spread from adjacent tissue

39
Q

osteomyelitis where is it found child vs adults

A

child = long bones

adult: vertebrae

40
Q

osteomyelitis risk factors

A

IV drug uses
Immunocompromised
Diabetes
Trauma

41
Q

Signs and symptoms osteomyelitis

A

signs: pain fever
signs: redness, oedema, abscess + draining sinus traact

42
Q

osteomyelitis diagnosis:

Imaging:
Definitive test
Bloods

A

Imaging: MRI gold standrad, X-ray shows periosteal elevation

Definitive: Bone Biopsy/aspirate culture

Labs: WBCS, CRP, CBC and blood culture

43
Q

Treatment osteomyelitis

A

Flucoxaccilin + vancomycin
Surgical: Irrigation and Debridement
+ hardware removal if present

44
Q

Septic Arthritis: Pathophysiology:

adults
hardware
newborn

A

adults: S aureus
CONS: hardware
N gonorrhoea newborn or sexuallya ctive

45
Q

clincal presenation Septic arthritis

A

localised joint pain
(erythem,a warmth, swelling)
may be systemic
inabability to wirght bear

46
Q

SA Joint aspirate: will show?

A
yellow
cloudy fluid 
increased WBC
increased protein
decreased glucose
positive gram stain
47
Q

SA treatment:

A

empiric flucox + vancomycin then guided by resutlts
non operative: theraputic joint aspirate
operative: arthoscopic/open irrigation and drainage

48
Q

outcomes of SA

A

10 - 15% mortality

rapid joint destruction

49
Q

Compartments syndrome clinical presentation

A

6 p’s

Pain (out of proportion with injury)
Pain on passive stretch (sensitive tests) 
Pulseness
Parasethesia 
Pallor
Paralysis
50
Q

treatment: Compartments

A

urgent fasciotomy +/- necrotic tissue debridement

51
Q

complicaitons compartments

A

Volkman’s contracture (ischaemia causing necrosis then secondary fibrosis and calcification)

Rhabdomylosis: muscle breakdown - myoglobinuria

52
Q

myotome vs dermatome

A

dermatome: single nerve skin supply
myotome: group of muscles supplied by single spinal nerve

53
Q

UPPER LIMB MYOTOMES:

Elbow

A
C5-C6 = elbow flexion
C7-C8= elbow extension
54
Q

Shoulder

A

c5 shoulder abduction

c678 adduction

55
Q

Wrist

A

c6 extension

c7 flexion

56
Q

finger

A

c7 finger extension
c8 flexion
t1 abduction/adduction

57
Q

finger

A

c7 finger extension
c8 flexion (little finger)
t1 abduction/adduction

58
Q

Ulnar nerve roots

59
Q

Median Nerve roots

60
Q

Radial Nerve roots

61
Q

fat embolism cause

A

piece of fat that can become lodged in vessels:

caused by long bone or pelvic fractures

62
Q

Presentation fat embolism

A

respiratory distress (ARDS due to inflammatory damage to lungs)
neurological dec LOC
anaemia and thrombocytopenia

63
Q

Subacromial impingement

A

compression (between head of humurs and underside of acromiom) of rotator cuff tendons (esp. supraspinatus) and subacromial bursa

64
Q

subacromial impingement Causes spectrum of:

A

bursititis
tendonitis
tearing of Rotator cuff

65
Q

subacromial impingement : clinical presentaiton

A

insidious onset pain wekaness and on active movement (painful arc of 60-120 degrees)

66
Q

subacromial impingement non operative treatment

A

physio, NSAIDS and steroid injections

67
Q

subacromial impingement operative

A

arthroscopy or open surgical repair either acromioplasty (shaving off acromiom) or repairing tendons (rotator cuff repair)

68
Q

supraspinatus movement

A

abduction

suprascapular nerve

69
Q

infra spinatus movement

A

external rotation

subscaprular nerve

70
Q

Teres minor movement

A

external rotation

axillary nerve

71
Q

subscapularis movement

A

internal rotation and adduction

subscapular nerve