Ortho Flashcards

1
Q

Common ortho congenital defects?

A
  • clubfoot (most common)
  • developmental dysplasia of the hip (DDH)
  • legg-calve-perthes disease (hip osteonecrosis)
  • spine deformities (scoliosis and kyphosis)
  • muscular dystrophy
  • limb defects
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2
Q

What is clubfoot? How common? Tx?

A
  • Talipes equinovarus
  • 1/1000 births, 50% bilateral
  • anatomic changes: talus plantar flexed, heel cord tight, fore foot adducted/supinated
    tx:
  • most can be tx conservatively
  • Ponseti method: casting +percutaneous heel cord lengthening (gold std)
  • severe, long-standing deformity may require several surgeries
  • serial casting, bracing
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3
Q

What is DDH? Incidence? Presentation?

A
  • loss of normal femoral head-acetabular relationship/stability: 2.7-17/1000 live birhts
  • caused by physiological and mechanical factors: ligamentous laxity, hormonal and familial factors, breech position and congenital deformities
  • wide range of presentations:
  • hip that is reduced but is unstable and can be dislocated
  • dislocation can be reduced
  • fixed dislocation that can’t be reduced
  • bony deformities that reqr surgery
  • all cases may not be detectable at birth: late cases are not always cases of missed dx
  • routine sonography isn’t cost effective
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4
Q

What is Barlow and ortolani test?

A
  • barlow: hip is reduced but can be dislocated
  • ortolani: hip is dislocated but can be reduced
    (clunk positive sign while being reduced)
  • lack of full abduction: hip is out and it can’t be reduced
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5
Q

Tx of DDH?

A
  • depends on extent of defomrity and if hip can be reduced
  • if hip can be reduced, harness or pillow first 6 months of life, confirm reduction with u/s after 3 weeks, effective 90% of time
  • if hip won’t stay in, reduce under anesthesia, hold with spica cast: confirm reduction with U/S after 3 weeks, femoral head isn’t visible on x-ray for 4-6 months
  • if it still won’t stay in: need surgery, femoral and/or acetabular osteotomy
  • if femoral head can be held in normal relationship with socket: pt will develop a normal hip
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6
Q

Hip abduction devices?

A
  • pavlik harness (most common)
  • Frejka pillow
  • boch harness
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7
Q

What is Legg-Calve-Perthes Disease?

A
  • males 3-11
  • loss of blood supply to femoral head:
    head can collapse and subluxation of femoral head, eventually revascularizes, but may not occur until fixed deformity present
  • variable hip/knee sxs: limping and pain
  • limited internal rotation and abduction of hip
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8
Q

Tx of Legg-Calve-Perthes disease?

A
- reduce pressure on femoral head:
relative rest
braces, crutches
traction
adductor muscle release
- correct resulting deformity: femoral and/or acetabular osteotomy (later on in life)
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9
Q

What is scoliosis, diff types?

A
  • lateral curvature of spine of more than 10 degrees by cobb method (become concerned when over 25 degrees)
  • types:
    idiopathic
    congenital
    secondary
    neuromuscular
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10
Q

Idiopathic scoliosis? Forms?

A
  • infantile (birth -3 yrs): 1%
  • juvenile (4-9): 12-21%
  • adolescent (10 yrs - end of growth): 80-90%
  • forms:
    lumbar, thoracic, thoracolumbar
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11
Q

Adolescent idiopathic scoliosis?

A
  • lateral curvature of spine with rotation in child older than 11 without any obvious cause
  • most common type
  • typically right thoracic curve
  • frequency: 1.9-3%
  • family hx: 30%
  • more severe forms more common in females
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12
Q

Dx adolescent idiopathic scoliosis?

A
  • adam’s forward bend test
  • radiographic exam: AP and lat full length of spine while standing
  • MRI: useful if neuro deficits, neck stiffness or HA
  • usually R scapulae becomes more prominent
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13
Q

What is kyphosis? More common in who?

A
  • increased thoracic curvature in saggital plane
  • postural: usually seen in girls, gentler, more pliable curve
  • corrects with time/bracing
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14
Q

What is Scheuermann’s disease?

A
  • more severe: in boys more, but not that common
  • osteochondrosis of the spine
  • ring apophyses don’t develop normally, resulting in wedged vertebra
  • sharper, more rigid curve
  • may need surgical correction
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15
Q

What is muscular dystrophy? Signs?

A
  • progressive weakness and wasting of muscles
  • onset: 3-5, genetic: primarily males
  • clumsiness, frequent falls, **difficulty climbing stairs, running, riding bike, waddling gait, breathing muscles become affected, life threatening infections common
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16
Q

Dx and tx of muscular dystrophy?

A
  • dx: bx, EMG

- tx: PT/OT, bracing, surgery (scoliosis)

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17
Q

Tx of limb defects?

A
  • live with deformity

- referral to ortho specialist for reconstruction surgery

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18
Q

Normal musculoskeletal variants?

A
  • metatarsus adductus
  • axial rotation
  • idiopathic toe walking
  • pes cavus (flat foot)
  • angular variations:
    genu varus (bow legged)
    genu valgus (knock-knees)
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19
Q

What is metatarsus adductus?

A
  • excessive amt of adduction of metatarsals relative to long axis of foot
  • **most common congenital foot deformity
  • female more than males affected
  • left more than right
  • most likely cause: intrauterine restriction
  • 85-90% resolve spontaneously by 1
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20
Q

Management of metataruss adductus?

A
  • flexible metatarsus adductus: stretching 5x at each diaper change
  • flexible MA beyond 8 mo: referral for biweekly casting, correction usually achieved in 3-4 casts
  • extreme adduction of great toe: surgical release of abductor hallucis done b/t 6-18 months of age
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21
Q

Toe in axial rotation causes?

A
  • internal femoral torsion (too much hip anteversion) - results from W sitting
  • internal tibial torsion (most common cause): results from intrauterine positioning
  • metatarsus adductus
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22
Q

Toe-out axial rotation causes?

A
  • external femoral torsion (too much hip retroversion): results from intrauterine positioning
  • external rotation contracture
  • external tibial torsion
  • flat foot
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23
Q

Tx of axial rotation at diff ages? When is surgery considered?

A
  • infant: good sleeping positions (back or side)
  • toddler: good sitting habits (avoid sitting in positions with exaggerated lower limb deformation)
  • nocturnal bar (dennis browne bar) for internal tibial torsion present after 18 months
  • weekly corrective casting for 4-5 weeks if no better by age 4

surgery if:
-failure to correct spontaneously with growth, gross asymmetric deformity, sx evolving congenital or neuromuscular conditions

24
Q

What is idiopathic toe walking? Assoc with?

A
  • walk with toe-toe gait pattern in absence of any known cause
  • dx of exclusion
  • prevalence not well described
  • may have good ankle ROM or more fixed contractures
  • etiology unclear
  • typically seen in kids less than 4
  • often assoc with subtle neuro abnormalities such as speech and language delay
25
Q

What is tx if contracture is present in idiopathic toe walking?

A
  • PT/OT (stretching, strengthening, and gait training)
  • orthotics (night and day)
  • serial casting (if past age 4)
  • surgical heel cord lengthening if pt fails conservative tx and are older than 4-5
26
Q

What is flat foot? Tx?

A
  • immature foot - normal foot variant (most toddlers have flat feet)
  • low arch heel valgus
  • arch starts to form around age 4
  • no need for formal tx
  • can try wedge but orthotics not necessary and are costly (esp in growing child)
27
Q

What is genu varum? Etiologies?

A
  • bow legs
  • physiologic bowing: usually corrects by age 2
  • metabolic bowing: vit D deficiency (rickets)
  • blounts disease: damage to epiphysis - may need bracing or surgery
28
Q

What is genu valgum?

A
  • knock knees
  • most correct spontaneously
  • braces and modified shoes not effective
  • after 11-13 may need surgery for marked deformity
  • likely to have patellar instability later on if not fixed early on
29
Q

Common ped ortho injuries?

A
  • slipped capital femoral epiphysis (SCFE)
  • radial head dislocation (nursemaid elbow)
  • ped fractures
  • osteochondrosis
  • patellofemoral arthralgia (PFA) - anterior knee pain
  • spondylolysis
30
Q

SCFE - Most common in who and increased with what?
Unstable and stable?
What movements are restricted?

A
  • most common from 9 yrs to end of growth
  • more prevalent in males
  • increased frequency with endocrine disorders; hypothyroid, renal disease, growth and sex hormone imbalance
  • obesity increases likelihood ( places more stress across femoral epiphysis)
  • unstable: sudden, severe pain with limp
  • stable: limp with groin pain, variable medial knee and or anterior thigh pain
  • 36% will later involve opp side
  • restricted internal rotation, abduction and flexion
31
Q

Dx and tx of SCFE?

A
  • X-rays: AP, frog leg lateral
  • mild slips: subtle changes on frog leg view only
  • complications: avascular necrosis (AVN), chondrolysis, osteoarthritis
  • immediate referral: surgical pin or screw placement
32
Q

What is radial head dislocation? Tx?

A
  • Nursemaid’s elbow
  • subluxation or dislocation injury from sudden pull of child’s arm
  • elbow becomes locked in slight flexion with forearm pronation
    dx:
  • patient guards elbow and refuses to use arm
  • may be swollen and tender with palpation of radial head
  • xray often normal
    tx:
    gentle supination of hand while flexing elbow with thumb placing gentle pressure over radial head, may use sling for a few days as needed for comfort
33
Q

Common ped fractures?

A
  • growth plate (epiphyseal) fractures (high %, pt of least resistance of bone)
  • supracondylar humerus fracture
  • wrist/forerm fractures
  • clavicle fractures
34
Q

Why do kids have such a good ability to heal fractures?

A
  • kids have ability to remodel bone unlike adults
  • this ability diminishes with age and closure of growth plates
  • can accept a surprising amt of displacement and plastic deformity
35
Q

Growth plate (physeal) fractures complications?

A
  • many childhood fractures involve the physis
  • 20% of all skeletal injuries in children
  • can disrupt bone growth
  • injury near but not at physis can stim bone to grow more
36
Q

Salter harris classification of growth plate fractures?

A

SALTR

  • type 1: slip
  • type 2: above
  • type 3: lower
  • type 4: through
  • type 5: rammed
37
Q

Supracondylar humerus fractures - when do thes occur? may be assoc with what kind of fracture? What other type of injury are you worried about?

A
  • most common fracture around elbow in kids: 60% of elbow fractures
  • occurs from fall on outstretched hand (trampolines!): ligamentous laxity and hyperextension of elbow impt mechanical factors
  • may be assoc with distal radial or forearm fractures so check wrist and forearm as well!
  • worried about nerve injury - incidence is high, b/t 7-16%: median, radial and ulnar nerve
  • anterior interosseous nerve injury most comonly injured nerve
  • do neurovascular exam - pulse, cap refill!!
  • carefully document pre-manipulation exam: post manipulation neuro deficits can alter decision making
38
Q

Tx of supracondylar humerus fracture?

A
  • long arm cast for stable/non-displaced fractures
  • percutaneous fixation with K wires for unstable fractures
  • look at rotation!
39
Q

Common wrist/forearm fractures?

A
    1. torus (buckle) fractures: usually nondisplaced - strong periosteum, subtlem may be best seen on lateral
    1. greenstick fractures: disruption of only one cortex
    1. complete (transverse) fractures: break through both cortices
40
Q

Tx of torus fractures?

A
  • no reduction needed: if older than 48 hrs, ok to cast at first visit
  • otherwise splint and cast at 5-7 days
  • short arm cast for 4 weeks
  • repeat x-rays at weeks to show evidence of healing
  • splint add 2 weeks after cast removal
41
Q

Tx of greenstick fractures?

A
  • short arm cast if non-displaced
    if displaced more than 15 degrees:
  • reduce and immobilize in long arm splint, 4 weeks in cast after swelling improves, 2 weeks in splint after cast is removed
  • usually do long cast, don’t want to allow arm to supinate or pronate
42
Q

Tx of complete fractures?

A
  • non-displaced fractures =short arm cast for 2-6 weeks
  • displaced fractures= reduce ASAP and cast
  • older the child, longer the immbolization
  • if x-rays are normal initially but there is tenderness over growth plate: immobilize for 2 weeks, bring child back in 2 weeks to re-examine and re-xray
  • if no callus - fracture unlikely
43
Q

Distal radius fractures? Why so common, result from what? What should you check?

A
  • peak injury time coorelates with peak growth time
  • bone is more porous
  • most injuries result from FOOSH
  • check sensation: median and ulnar nerve: nerve injury more likely to occur with significant angulation of fragment or with sig swelling
  • examine elbow (supracondylar) and wrist (scaphoid)
  • bleeding and swelling can cause acute carpal tunnel syndrome
44
Q

Clavicle fractures - how common? Most occur when? Tx?

A
  • most common ped fracture
  • in children, 90% of middle third (good blood supply)
  • almost half of all clavicle fractures occur under age 7
  • younger than 10, most are non-displaced, older than 10, most are displaced
  • stable injuries, tx with sling or figure of 8 splint for 2 weeks
45
Q

What is osteochondrosis? Causes?

A
  • apophyseal injury
  • pain at tendonous insertions at secondary ossification centers (apophyses)
  • causes: increase in activity level, increase in mass, and puberty
  • common examples: osgood schlatter (patellar tendon-tibial tuberosity), and sever disease (achilles-calcaneus)
46
Q

Osgood schlatter - general features? Tx?

A
  • inflammation where patellar tendon inserts on tibia
  • leaves a lump - prominent, tender tibial tubercle
  • clinical dx
  • x-rays may show aphophysitis (widening of growth plate)
  • majority of the time: pt outgrow it (closure of physis)
    tx:
  • conservative (same as PFA)
  • eccentric strengthening
  • iontophoresis/steroid (US)
  • brace/pressure band
  • excision of detached/fragmented bone fragment (risk: weakening tendon insertion)
47
Q

Tx of osteochondrosis?

A
  • conservative: rest, NSAIDs, ice, PT, casting/boot (sever’s disease - prevents pulling of achilles)
  • surgical: indicated for avulsion of apophysis greater than 1 cm
48
Q

What is patella-femoral arthralgia (PFA)? CLinical dx?

A
  • chondromalacia patellae: more common in girls (4:1)
  • means sore kneecap, common, frustrating but not serious
  • inflammation of articular surface of patella, vague, diffuse anterior discomfort
  • clinical dx:
    overuse more than acute injury
  • anterior knee discomfort, pain with stairs, can’t sit with bent knee
  • increased Q angle, tender undersurface of patella, tight hamstrings, weak quadriceps
  • if you have valgus knees (tibial or femoral torsion): at risk for developing this
49
Q

Tx of PFA?

A
  • responds to good management: PT -
    exercise programs, setting realistic goals, may need to involve parents
  • conservative:
    relative rest
    PT (quad strengthening)
    ice and NSAIDs
    patellar stabilization brace (if assoc instability)
    -surgical: rarely indicated
    where underlying anatomy is abnormal may need lateral release and/or tibial tubercle osteotomy
50
Q

What is spondylolysis? Dx? tx?

A
  • stress fracture of pars
  • repetitive hyperextension of back: gymnastics, football, wrestling, rowing
  • progressive low back pain with activity: radicular sxs are rare, may progress to slip (spondylolisthesis)
  • Dx: xray, CT, bone scan or MRI: look for neuro sxs
  • Tx: rest, brace, rare cases: fusion
51
Q

What is acute septic arthritis and why is it an emergency? most common jt affected? most common organism?

A
  • pyogenic bacteria invade a synovial jt
  • ped incidence has close correlation with osteomyelitis!!
  • most common jt: hip* and elbow
  • most common organism: staph aureus (cover for MRSA)
  • emergency!!!
52
Q

Presentation of acute septic arthritis in infants?

A
  • may develop with few clinical manifestations
  • tenderness
  • increased warmth over jt in question
  • pseudoparalysis: pain with movement of jt
  • painful restriction
  • fever and WBC misleadingly slight (jt will be red hot!!!)
53
Q

Presentation of acute septic arthritis in older kids?

A
  • severe pain: pain with passive motion, won’t move jt themselves
  • protective muscle spasm (guarding)
  • marked tenderness
  • fever (not all the time)
  • elevated WBC
  • elevated ESR/CRP
54
Q

Eval of acute septic arthritis? Tx?

A
  • C&S blood,urine
  • xray
  • US: very useful for guiding injection, esp in hip jts
  • immediate needle aspiration: inspect aspirate (cloudy), C&S, gram stain, crystals (rule out gout)
  • refer immediately for surgical washout (I&D)
  • empiric IV abx:
    3rd gen cephalosporin and penicillinase resistant synthetic PCN
  • MRSA should be covered with either clindamycin or vanco
55
Q

Open fractures - how serious are these? Easy to see?

A
  • fracture with overlying skin compromise - may be subtle

- reqrs IMMEDIATE referral to ortho: must be washed out w/in 24 hrs