ORPTH ADONS DSE OF PULP N PERIAP TISS Flashcards
1
Q
- The most common cause of odontalgia is
(a) Dental caries
(b) Pulpitis
(c) Root fracture
(d) Periodontitis
A
- (b) Pulp is a soft connective tissue like any other connective
tissue throughout the body and reacts to bacterial or
other stimuli by an inflammatory response which causes
toothache or odontalgia.
2
Q
- The phenomenon by which bacteria circulating in blood
accumulate at the site of pulpal inflammation is called as
(a) Chemotaxis
(b) Retrograde pulpitis
(c) Anachoretic pulpitis
(d) Aerodontalgia
A
- (c) Anachoresis is a phenomenon by which blood-borne
bacteria, dyes, pigments, etc. are attracted to the site of
inflammation.
3
Q
- Most accepted explanation for anachoretic pulpitis is
(a) Increased capillary permeability
(b) Increased vascular pressure
(c) Presence of large number of dilated capillaries
(d) Lack of collateral blood supply
A
- (a) Current evidence indicates increased capillary permeability
to be the most probable cause for the phenomenon
of anachoresis. The capillary size increases due to the liberation of cytokines by the inflammatory cells mediating
the response.
4
Q
- Pulpitis aperta and pulpitis clausa refer to types of pulpitis
classified on the basis of
(a) Severity of inflammation
(b) Extent of inflammation
(c) Location of inflammation
(d) Presence/absence of direct communication with oral cavity
A
- (d) Pulpitis aperta and clausa refer to open and closed pulpitis
respectively. Open pulpitis refers to pulpitis in which direct
communication exists between the inflamed pulp and oral
cavity while it is absent in case of closed pulpitis
5
Q
- The more accepted terminology for pulp hyperemia is
(a) Focal irreversible pulpitis
(b) Focal reversible pulpitis
(c) Subtotal pulpitis
(d) Pulpitis clausa
A
- (b) Vascular dilatation can occur pathologically due to dentinal
as well as pulpal irritation and also artefactually during
tooth extraction. Hence the term pulp hyperemia needs to
be avoided.
6
Q
- Focal reversible pulpitis is most commonly seen in all of the
following cases except
(a) Large metallic restorations
(b) Shallow carious lesions
(c) Deep carious lesions
(d) Restorations with defective margins
A
- (b) Focal reversible pulpitis is a mild form of pulpitis localized
primarily to the pulpal ends of irritated dentinal tubules
and is mostly seen in deep carious lesions, beneath large
uninsulated restorations and restorations with defective
margins.
7
Q
- Pain which increases in intensity as the patient lies down is
characteristic of
(a) Focal reversible pulpitis
(b) Acute pulpitis
(c) Chronic hyperplastic pulpitis
(d) Chronic pulpitis
A
- (b) As intrapulpal abscess formation involves more of the
pulp tissue, the pain becomes more acute and is liable to
increase when the patient lies down. This may be due to
the pressure on pulpal nerves by the intrapulpal abscesses.
Heat application can exacerbate the pain.
8
Q
- Microabscess formation within inflamed pulp is characteristic
of
(a) Acute pulpitis
(b) Focal reversible pulpitis
(c) Chronic pulpitis
(d) Pulp hyperemia
A
- (a) Rise in intrapulpal pressure associated with inflammatory
exudate leads to collapse of venous part of circulation in that
area which leads to anoxia, which in turn, leads to localized
destruction and formation of small abscess called micro or
pulp abscess.
9
Q
- Sensitivity to electric pulp vitality tester in acute pulpitis is lost
in later stages because of
(a) Necrosis of pulp
(b) Reduction in inflammatory exudate
(c) Increase in the size of capillaries locally
(d) Decreased secretion of prostaglandins
A
- (a) Early stages of acute pulpitis is characterized by stabbing or
lancinating pain and high sensitivity to electric pulp vitality
tester. But when more of pulp is involved and necrosis sets
in this sensitivity is lost.
10
Q
- Pulp reacts to electric pulp vitality tester at higher levels in chronic
pulpitis due to
(a) Slow advance of pulp inflammation
(b) Lack of edema fluid collection within inflamed pulp
(c) Degeneration of nerves in affected pulp
(d) Deposition of collagen around inflamed area
A
- (c) Due to degeneration of the small nonmyelinated nerve
fibers, the pulp in chronic pulpitis exhibits little or no pain
and reacts to electric pulp vitality tests at higher voltages
even in advanced cases.
11
Q
- Which amongst the following characteristics is not associated
with chronic hyperplastic pulpitis?
(a) Open carious lesion
(b) Occurs in children and young adults
(c) Occurs in people with high tissue resistance
(d) Occurs around margins of a restoration
A
- (d) Pulp polyp or chronic hyperplastic pulpitis is characterized
by excessive and exuberant pulp proliferation. It is seen
almost exclusively in children and young adults with large,
open carious lesions.
12
Q
- Which teeth are most commonly involved by chronic hyperplastic
pulpitis?
(a) Deciduous anteriors and permanent canines
(b) Deciduous molars and permanent 1st molars
(c) Deciduous canines and permanent incisors
(d) Exclusively deciduous molars
A
- (b) These teeth are primarily involved because of their excellent
blood supply and large root opening.
13
Q
- All of the following except _______ occur as a sequel to pulpitis.
(a) Lateral periodontal cyst
(b) Periapical granuloma
(c) Apical periodontal cyst
(d) Periapical abscess
A
- (a) Lateral periodontal cyst is a developmental variety of
odontogenic cyst and does not arise due to inflammatory
changes within pulp
14
Q
- All of the following except _______ are common radiological
features associated with a periapical granuloma.
(a) Thickening of PDL around root apex
(b) Well-defined radiopacity
(c) Root resorption of involved tooth
(d) Well-defined radiolucency with sclerotic borders
A
- (b) Periapical granuloma is characterized radiologically by a
well-defined radiolucent lesion associated with the root of
a pulpally involved tooth
15
Q
- Giant cell hyaline angiopathy and Rushton bodies are associated
with which of the following lesions?
(a) Ludwig angina
(b) Odontogenic keratocyst
(c) Pulp polyp
(d) Periapical granuloma
A
- (a) Giant cell hyaline angiopathy consists of inflammatory cell
infiltration, collections of foreign body type giant cells and
ring-like eosinophilic material called Rushton bodies.