Opthalmology Flashcards

1
Q

What is cataract

A

any opacity in the lens

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2
Q

cataract risk factors

A

age
db
smoking
alcohol

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3
Q

Presentation of cataract

A

blurred vision

unilateral - unnoticed but depth perception affected

bilateral - gradual painless loss of vision ±glare with bright lights, ±monocular diplopia, haloes, faded colours

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4
Q

cataracts management

A

mydriatic drops
sunglasses
surgery - phacoemulsification

day case with local anaesthetic

  • incision -> phacoemulsion-> artifical lens
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5
Q

Types of uveitis

A

anterior - iritis

posterior - choroiditis

intermediate - vitrous

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6
Q

What is subconjunctival haemorrage, who is it common in?

A

harmless pool of blood behind conjunctiva from small bleed

common in elderly women

ask about anti-coags

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7
Q

Presentation of conjunctivitis

A

itchy, burning, watering eyes

sticky discharge causes eyes to stick

conjunctiva red and inflamed

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8
Q

conjunctivitis management

A

symptomatic - artificial tears and antihistamines (emedastine)
topical abx used in bacterial to reduce length and transmission risk

Abx if ?sexual, contact lens, immunocomp
(chloramphenicol or fusidic acid)

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9
Q

Investigations for red eye

A

Gonioscopy - evalutaed internal drainage system ?glaucoma

Slit lamp - see leucocytes in anterior chamber if iritis

?culture if suspected sexual cause or recurrent unresponsive conjunctivitis

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10
Q

Iritis presentation

A

pain, blurred vision, photophobia, non sticky discharge, red eye

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11
Q

iritis management

A

refer

prednisolone drops to reduce inflam

cyclopentolate - keeps pupils dilated to prevent adhesions between lens and iris and also to relieve spasms of ciliary body

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12
Q

Acute closed angle glaucoma presentation

A

onset hours/days

  • blurred vision, haloes
  • painful red eye
  • generally unwell
  • N&V
  • headache
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13
Q

Management of acute closed angle glaucoma

A

B Blockers triad (supress aqueous production)
-timolol, pilocarpine, IV acetazolamide

Monitor IOP

peripheral iridectomy once IOP under control - removal of piece of iris

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14
Q

Episcleritis Presentation

A

Inflammatory nodule

Blue sclera below cone shaped wedge of engored vessels

dull acbe in eyes, tender

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15
Q

Episcleritis managemtn

A

Symptomatic relief - artificial tears and topical/systemic NSAIDs

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16
Q

Scleritis presentation

A

constant severe dull ache that bores into eye

painful ocular movements

?headache and photophobia

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17
Q

scleritis management

A

referral

anterior: NSAIDs ± oral pred
posterior: more aggressive

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18
Q

Ketatitis causes

A

bacterial: pseduomonas aeruginsoa, staph
fungal: aspergillus, candida
viral: HSV

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19
Q

Keratitis presentation

A

PAIN, photophobia, decreased visual acuity

discharge

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20
Q

Keratitis management

A

treat causative organism

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21
Q

How would a patient with open angle glaucoma present?

A

asymptomatic until visual fields badly impaired

loss of vision peripherally -> tunnel vision

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22
Q

Why is there a need for screening high risk groups for open angle glaucoma?

A

doesnt present until visual fields are badly impaired

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23
Q

What are high risk groups for open angle glaucoma

A

+ve FH

Diabetic

thyroid eye disease

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24
Q

How would you confirm a diagnosis of open angle glaucoma?

A

Tonometry: pressure >21

Central corneal thickness measurement

gonioscopy: ant chambre assessment

Visual fields

Optic nerve

25
Q

Medical management of OAG

A

1) prostaglandin analogues: increase uveoscleral outflow
2) B blockers: decrease production of aqueous
3) Alpha adrenergic agonists: decrease production of aqueous and increase uveoscleral outflow
4) carbonic anhydrase inhibs: decrease aqueous production
5) miotics: decrease resistance to aqueous outflow

26
Q

Examples and SE of prostaglandin analogues

A

latanoprost, travoprost

  • Eyelash growth
  • iris colour change
  • Red eye
27
Q

Examples and SE of B blockers for use in open angle glaucoma

A

Timolol, betaxolol

  • dry eyes
  • corneal anaesthesia
  • *asthmatics *HF
28
Q

Exampes and side effects of alpha adrenergic agonists

A

Dorzolaminde, brimonidine

  • fatigue
  • dyspepsia
29
Q

side effects and examples of miotics

A

pilocarpine

  • decreased acuity
  • brow ache (ciliary muscle spasm)
30
Q

Surgical option for open angle glaucoma

A

trabeculectomy

31
Q

Synonyms for squint

A

strabismus, tropia

32
Q

types of squint

A

esotropia - inward (convergence)

exotropia - outward (divergent)

hypertropia - upwards

hypotropia - downwards

33
Q

How do you diagnose a squint

A

cover test - movement of uncovered (bad) eye to correct fixation when good eye is covered

corneal reflection - reflection from bright light falls asymmetrically on cornea

34
Q

Management of squint

A

glasses

patch/drops(atropine) in good eye to encourage use of bad eye

Operation: resection and recession of rectus muscles

35
Q

What would the eye look like in a 3rd nerve palsy?

A

down and out

36
Q

what woudl the eye look like in a 4th nerve palsy?

A

up

pt might tilt head

37
Q

what would the eye look like in a 6th nerve palsy?

A

diplopia in horizontal plane

medial deviation

eye cant move laterally from horizontal plane

38
Q

What is amblyopia

A

reduction in visual acuity due to problem in focussing in early childhood - brain isnt stimulated to develop correctly

39
Q

list 3 causes of amyblyopia

A

*squint

refractive defects

congential cataracts

40
Q

how would you diagnose amblyopia?

A

snellen chart

lack of red reflex

41
Q

How would you manage amblyopia?

A

strabismus: patch and drops

refractive error: glasses

congential cataracts: surgery

42
Q

Causes of CRAO

A

atherosclerosis

embolism

inflammation

thrombophilic disorders

43
Q

How would a patient with CRAO present?

A

sudden painless unilateral visual loss

44
Q

What would you expect to find on examination of a patient with suspected CRAO

A

pale retina with cherry red spot

afferent pupil defect

?carotid auscultation for bruits, murmers, radial pulse for AF

45
Q

How would you manage a patient with CRAO

A

occular massge with in 100mins of onset

surgical removal of aqueous

acetazolamide (intraocular hypotensive)

46
Q

What long term management would you advise for patient with CRAO

A

opthalmolmic follow up

low vision aid clinic

carotid endarterectomy

DVLA notification

47
Q

How would optic neuritis present?

A

1) unilateral reduced vision
2) eye pain
3) impared colour vision

48
Q

What would you expect to find on examination of a patient with optic neuritis?

A
  • reduced vision
  • decreased pupillary light reaction in affected eye
  • abnormal sensitivity and colour vision
  • papillitis
  • scotoma
49
Q

How would you manage optic neuritis

A

corticosteroids in acute phase

methylprednisolone speeds up visual recovery in acute phase

50
Q

what is drusen?

A

lipid deposit under retina - yellow appearance

51
Q

what are the types of macular degeneration?

A

wet - choroidal neovascularization

dry- geographic atrophy

52
Q

how would wet macula degen present?

A

blurred vision

metamorphopsia

poor night vision

central vision loss

53
Q

what would u find on examination of pt with wet macular degen

A

macular drusen

XS vessels (fluorescein angiography)

haemorrhage

Amsler grid shows waving lines

54
Q

How woudl u manage wet macular degen

A

anti-VEGF injections

55
Q

how would dry macular degen present?

A

gradual visual loss - difficulty reading

central scotoma

56
Q

what would you expect to find OE in dry macular degen

A

macular drusen

retinal pigment epithelium clumping or atrophy

57
Q

how would you manage dry macular degen

A

zinc, vit A/C/E

58
Q
A