Opthalmology Flashcards

1
Q

What are the symptoms of acute glaucoma?

A

Symptoms of acute glaucoma may include headache with a painful red eye and misty vision or haloes, and in some cases nausea. Dilated pupil.

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2
Q

How is glaucoma treated?

A

Let the person lie flat with their face up and head not supported by pillows, as this may relieve some of the pressure on the angle.

Beta blocker - timolol +
Azetozolamide
Pilocarpine

Flat backpack

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3
Q

Suggest the correct treatment for acute gluacoma?

A

alpha blocker: If the drugs are available, give: pilocarpine eye drops, one drop of 2% in blue eyes or 4% in brown eyes;

acetazolamide 500 mg orally to reduce production of aqueous humour (provided that there are no contraindications);

analgesia; and an

anti-emetic,

angle

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4
Q

What is definitive management for acute glaucoma?

A

Surgical procedures such as iridotomy, phacoemulsification, or iridoplasty can then be considered.

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5
Q

What are the benign eyelid disorders?

A

Meibomian cyst (chalazion). See the CKS topic on Meibomian cyst (chalazion) for more information.

Cysts of Moll — arise from blocked apocrine sweat glands on the eyelid margin. They are typically dome-shaped papules or nodules filled with clear fluid.

Cysts of Zeis — arise from blocked sebaceous glands on the eyelid margin. They are typically filled with yellow oily secretions.

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6
Q

How are styes managed?

A

Advise that a stye is usually self-limiting and rarely causes serious complications.

Apply a warm compress (for example, using a clean flannel that has been rinsed with hot water) to the closed eyelid of the affected eye for 5–10 minutes 2–4 times daily until the stye drains or resolves.

Advise the person not to attempt to puncture the stye.

Advise to avoid using eye makeup or contact lenses until the area has healed.

The NHS information leaflet Stye and the patient information leaflet Stye available on the www.patient.info website may be helpful.

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7
Q

How are meibobum cysts managed?

A

Suspect a meibomian cyst if there is a firm, painless, localized eyelid swelling that has developed slowly over several weeks. In some cases, the person may report initial discomfort which then resolved.
A meibomian cyst:
Is more commonly found on the upper eyelid.
Is usually 2–8 mm in diameter.
Is most commonly sited away from the lid margin.
Can affect one or both eyes (more than one meibomian cyst may be present).
May rupture through the skin.

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8
Q

What should always be checked with benign eyelid disorders?

A

Always rule out pre-septal and orbital cellulitus

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9
Q

What are the symptoms of blepharitis?

A

Burning, itching and/or crusting of the eyelids.
Symptoms are worse in the mornings.
Both eyes are affected.
Recurrent hordeolum.
Contact lens intolerance.

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10
Q

How is blepharitis managed?

A

warm compress (a clean cloth warmed with hot water) should be applied to closed eyelids for 5–10 minutes once or twice daily

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11
Q

What anaesthesia is used in cataracts surgery?

A

Offer sub‑Tenon’s or topical (with or without intracameral) anaesthesia for people having cataract surgery.

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12
Q

Symptoms of cataracts

A

Your vision is cloudy or blurry.
Colors look faded.
You can’t see well at night.
Lamps, sunlight, or headlights seem too bright.
You see a halo around lights.
You see double (this sometimes goes away as the cataract gets bigge

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13
Q

What are the symptoms of CRAO and CRVO and how are they managed?

A
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14
Q

What are the types of diabetic retinopathy?

A

Non-proliferative diabetic retinopathy (NPDR)
(background/preproliferative)
In the international (AAO) classification, NPDR is graded as:
* Mild
* Moderate
* Severe

Proliferative diabetic retinopathy (PDR)
NVD - new vessels on disc
NVE - New vessels everywhere

Diabetic maculopathy (DM)

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15
Q

What is Rubeosis iridis?

A

Rubeosis iridis is a medical condition of the iris of the eye in which new abnormal blood vessels (formed by neovascularization) are found on the surface of the iris.

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16
Q

What is NVD and NVE?

A

New vessels on disc and new vessels everywhere

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17
Q

What are the structures of the retina?

A
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18
Q

What are the treatments for diabetic retinopathy?

A

Diabetes control
Anti-VEGF
Laser photocoagulation

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19
Q

How is anterior uveitis managed?

A

Steroids and cycloplegics

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20
Q

What are the features of anterior uveitis?

A

Painful mitotic pupil
Perilimbal injection
Posterior synechiae
Hypopyon

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21
Q

What is Chandler’s classification for orbital cellulitis?

A
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22
Q

How should the eyes be examined in orbital cellulitis?

A
23
Q

What are the treatments for scleritis?

A

steroid eye drops
corticosteroid pills (medicine to control inflammation)
nonsteroidal anti-inflammatory drugs (NSAIDs) like aspirin or ibuprofen for pain and inflammation
oral antibiotic or antiviral drugs
drugs that weaken or modify the response of the immune system may be used with severe scleritis (immunosuppressive and immunomodulatory drugs)

24
Q

What are the symptoms of scleritis?

A

pain
tenderness of the eye
redness and swelling of the white part of the eye
blurred vision
tearing
extreme sensitivity to light

25
Q

What is amblyopia?

A

Amblyopia, also known as ‘lazy eye

26
Q

What is hyphaemia?

A
27
Q
A

Following ocular trauma, an assessment should be made for orbital compartment syndrome as this may require immediate decompression prior to imaging etc

28
Q

How are corneal abrasions investigating?

A

Fluorescein staining

29
Q

What are the features of posterior vitreous detachment?

A

Flashes of light (photopsia) - in the peripheral field of vision
Floaters, often on the temporal side of the central vision

30
Q

What are the features of retinal detachment?

A

Dense shadow that starts peripherally progresses towards the central vision
A veil or curtain over the field of vision
Straight lines appear curved
Central visual loss

31
Q

What are the features of vitreous haemorrhage?

A

Large bleeds cause sudden visual loss
Moderate bleeds may be described as numerous dark spots
Small bleeds may cause floaters

32
Q

How is herpes zoster opthalmicus treated?

A

Herpes zoster ophthalmicus requires urgent ophthalmological review and 7-10 days of oral antivirals

33
Q

What is associated with central retinal venous occlusion?

A

Retinal haemorrhages

34
Q

What do the different medications involved in gluacoma do?

A

Prostaglandin - increases uveoscleral outflow
Beta blockers - reduce aqeous production
Alpha blockers - reduce about aqeous production and increases outflow
Carbonic anhydrase - reduces aqueous production

PA increases outflow

35
Q

What is the Keith Wagner grading?

A

Narrowing
Nipping
Cotton wall spots
Paplioedema

36
Q

What are some features of vitreous haemorrhage?

A

Red tinge
Dark floaters

37
Q

How is a dendritic ulcer managed?

A

Topical antivirals

38
Q

How is herpes zoster opthalmicus managed?

A

Oral antivirals

39
Q

What is an Argyll Robertson pupil?

A
40
Q

What is a Holmes Adie pupil?

A
41
Q

T or F, in episcleritis, the injected vessels are mobile when gentle pressure is applied on the sclera

A

T

42
Q

How is dry macular degeneration managed?

A

High dose of beta-carotene, vitamins C and E, and zinc

43
Q

What are the side effects of laser photocoagulation?

A

Reduction in visual fields + loss of night vision

44
Q

How does an Argyll Robertson pupil react?

A

Quickly to accomodation
Not to light

45
Q

How does a Holmes pupil react?

A

Not to slight
Slowly to accomodation

46
Q

T or F, Cataract removal operations should never be rationed on the basis of visual acuity

A

T

47
Q

T or F, Flashes + floaters are most commonly caused by a posterior vitreous detachment

A

T

48
Q

What is the triad involved in carotid cavernous fistula?

A

Proptosis, ocular bruit and chemosis

49
Q

What happens in transient ischaemia attacks?

A

Ischaemic optical neuropathy

50
Q

How is emdopthalmitis treated?

A

Intravitreal vancomycin

51
Q

What is the Keith Warner classification?

A

Grade 1 - narrowing
Grade 2- nipping
Grade 3 - exudate and haemorrhage
Grade 4 - paplidema

52
Q

What can resolve redness in episcleritis?

A

10% phenylephrine

53
Q

What happens in horners syndrome?

A

Loss of innervation of the symparhetic chain?