Ophtho Flashcards

1
Q

4 borders of the orbit

A

sup: frontal sinus
medial: ethmoid bone (lamina papyracea “paper thin”)
inferior: maxillary sinus
laterally: zygomatic bone

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2
Q

eye exam

A

-adnexa
-acuity
-visual fields
-pupils
-EOM
- inspect globe
-retinal exam with fundoscope
- slit lamp –> ant chamber
- fluorescein
- IOP

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3
Q

test for corneal laceration?

A

Seidel’s sign

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4
Q

normal IOP? contraindication to checking IOP with tonopen?

A

10-20 mm Hg
-suspected globe rupture

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5
Q

to examine cornea, slit lamp should be?

A

bright, thin, 45 degree angle (allows assessment of anterior chamber)

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6
Q

common triggers for preseptal cellulitis?
most common age?
most common bugs?

A

URTI, sinusitis, chalazion, hordeolum, insect bites and trauma. mostly in ppl <10
staph, strep

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7
Q

triggers for orbital cellulitis?

A

-paranasal sinusitis (esp ethmoid)
-trauma, intraorbital fb, postop, spread for preseptal infection

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8
Q

presentation of orbital cellulitis?

A

URI, facial pressure, fever, pain with EOM, photophobia, abn pupillary response, decrease visual acuity,

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9
Q

rare but highly m&m complication of orbital cellulitis?

+ other ones?

A

cavernous sinus thrombosis, presents with HA and deficit of CN 3, 4, 6

meningitis, encephalitis, epidural abcess, brain abcess.

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10
Q

dx and tx of orbital cellulitis?

A

dx: clinical + CT can also make dx
tx: IV ABX, ophtho consult, lateral canthotomy if inc IOP

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11
Q

abx for orbital cellulitis secondary to sinusitis?

A

ctx and flagyl

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12
Q

what is stye/hordeolum?
how is it managed

A

acute bacterial infection from blockage of glands. think “pig stye”, “whoredeolum”, therefore dirty compared with chalazion

usually loacted at lid margin/las line

tx: warm compress, erythro ointment

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13
Q

what is chalazion?
how is it managed

A

acute or chronic inflammation, no infection.
warm compress
ophtho if not resolving or if vision affected

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14
Q

blepharitis, most common cause and tx

A

overgrowth of s epidermidis
tx: lid hygiene (wipes) abx oint if severe

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15
Q

3 rule outs in bacterial conjunctivitis

A

corneal ulcer (look before fluorescein) then, abraision and herpetic lesions (with fluorescein)

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16
Q

how long for viral conjunctivitis to resolve?

A

1-3 weeks, very contagious

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17
Q

triggers for sub conjunctival hemm?
how long to resolve?

A

trauma, valsalva, HTN, spontaneous
about 2 weeks

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18
Q

Herpes simplex vs herpes zoster fluorescein finding?

A

Dendritic lesion, compared to HZO which has pseudodendrite (poorly staining mucous plaque), no actual erosion which true dendrite has

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19
Q

herpes zoster ophatlmicus treatment

A

Oral antivirals

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20
Q

corneal ulcer etiologies

A

Exposure kerstitis (eg Bell’s palsy)
Trauma
Direct microbial invasion
Contact lenses that
Ocular surgery

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21
Q

corneal ulcer signs/symptoms

A

Redness swelling if lids and conjunctiva
Discharge
Ocular pain
Fb sensation
Photophobia
Blurred vision
You see a corneal opacification with slit lamp and flare and cells in ant chamber

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22
Q

corneal ulcer mgmt

A

Emergent ophtho consult
Start cipro drops; usually after cultures taken by ophtho so just call them

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23
Q

What is Hutchinson sign?

A

Shingles lesions at top of nose, predicts corneal involvement

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24
Q

Mgmt HSV keratocomjunctivitis

A

Topical antivirals: topical acyclovir, ganciclovir or trifluridine
Can also add oral antivirals
Defs add oral if there are lesions on the adnexa

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25
Q

What nerve is involved in HZO

A

V1 (ophthalmic nerve)

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26
Q

Some organisms that cause corneal ulcer

A
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27
Q

What is ultraviolet keratitis

A

Damage to cornea from ultraviolet light. A.k.a. snow blindness or welders arc. Can also be from tanning beds. Symptoms of pain developed 6 to 12 hours later. Can have photophobia. Resolve on its own and 24 to 36 hours.

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28
Q

Anterior uveitis is synonymous with?

A

Iritis

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29
Q

Causes of anterior uveitis

A

Systemic inflammation: ulcerstive colitis, ankylosing spondylitis, bechets, sarcoid

Infections: tuberculosis, Lyme disease, HSV, syphilis

Malignancy: leukemia, lymphoma, melanoma

Trauma, foreign body

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30
Q

Signs and symptoms of anterior uveitis

A

Red eye, photophobia, decreased visual acuity.

Findings of associated systemic disease.

Perilimbal flush, a.k.a. ciliary flush, usually no discharge

Cells and flare

consensual photophobia is highly suggestive.

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31
Q

3 causes, in order, of endopthalmitis (infection of vitreous or aqueduct humour)

A

Post eye surgery
Penetrating trauma
Hematogenous spread

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32
Q

Signs and symptoms endopthalmitis

A

Pain, redness, discharge

Hypopion, photophobia, ciliary flush

Can have adnexal swelling

RF of surgery or fb paramount

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33
Q

Symptoms of vitreous hem/detachment

A

Painless vision loss, flashers floaters, haziness
Usually send to ophtho to r/o retinal detachment after doing full eye exam

34
Q

And for corneal abrasion, contact wearer vs non-contact wearer

A

Non: erythro ointment
Wearer cipro ointment, ofloxacin ointment

35
Q

Similar to corneal abrasion and laceration there is…..

A

Conjunctival. Similar exam, usually less symptoms. Mgmt is the same

36
Q

Why abx in corneal abrasion

A

Prevent iritis, they are a must!

37
Q

Relief of pain with topical anesthetics is almost diagnostic for?

A

Corneal abrasion
Very painful, pain can be delayed by a few hours after injury,

38
Q

Best analgesics for corneal abrasion

A

Simple oral analgesics, nsaids best

39
Q

Exam finding ls for corneal laceration

A

Misshapen iris, hyphema, Seidel test shallow anterior chamber

40
Q

If clinical suspicion for penetrating eye injury or severe blunt trauma do?

A

Consult ophtho and get ct of orbit. Even if ct normal need to consult

41
Q

corneal FB removal?

A

anesthetize eyes (somtimes freezing both helps):
NS rinse
moist cotton applicator
25 g needle

42
Q

when does rust ring appear

A

when metal fb present for more than a few hours

43
Q

when to be worried about globe rupture from fb?

A

hyphema, seidel sign or seen to be deeply embedded on slit lamp

44
Q

is it necessary to remove rust ring in the ED?

A

no, can refer to ophtho, but should still try and get fb out

45
Q

criteria for ophtho to see lid lacerations

A

lid margin, 6-8 mm from medial canthus, involving lacrimal duct, inner surface of lid, ptosis or involving tarsal plate and most full thickness

so basically all of them….?

46
Q

in full thickness lacerations what needs to be ruled out?

A

globe rupture/corneal laceration

47
Q

how to know if canalicular system is cut

A

put fluorescein in eye and if it comes out the wound, its cut

48
Q

which type of lid lacerations are the ones repaired in ED and how

A

partial thickness not meeting criteria:

lid margin, 6-8 mm from medial canthus, involving lacrimal duct, inner surface of lid, ptosis or involving tarsal plate and most full thickness

use 6 or 7-0 soft suture. can try and hold ends down with subsequent suture

49
Q

If lid margin lac cannot be repaired by optho, describe how to do it

A

Note not necessary if less than 1mm

50
Q

how to get eyelids open in blunt eye trauma

A

eye speculum or can use paperclips bent in proper way

51
Q

if restricted EOM in blunt ocular trauma think?

A

orbital blowout fracture, need to get CT facial bones/orbits

52
Q

things to check in blunt ocular trauma

A

acuity, glove integrity, IOP, EOM

53
Q

two most common spots for orbital blow out fracture

A

inferior wall (maxillary sinus)
medial wall (lamina papyracea aka ethmoid)

54
Q

how to manage suspected or confirmed golobe rupture?

A

HOB 45 degrees, cover eye with paper cup, broad spectrum IV abx, immediate ophtho consult

55
Q

signs of globe rupture? none are overly sens or specific

A

large sub con hemm, chemosis (raised sub con hemm) ,seidel’s sign, shallow anterior chamber, hyphema, tear drop shaped pupil, decreased acuity

56
Q

imaging to detect occult globe rupture?

A

CT of eye

57
Q

what is post septal hemorrhage

A

aka orbitla hematoma
secoanr to trauma, causes orbitla compartment syndrome

58
Q

clinical features of post septal hemorrhage?

A

eye pain, proptosis, impaired EOM, elevated IOP, decrease acuity

can also get oribital CT scan if unsure

59
Q

indications for lateral canthotomy and cantholysis

A

retrobulbar hemm with IOP > 40, if IOP < 40 but dx is suspected get a CT

or retrobulbar hemm with RAPD, ophthalomoplegia, cherry red macula, optic nerve head pallor or severe pain in affected eye

60
Q

walk through steps of lateral canthotomy and cantholysis

A

go!

61
Q

retinal ischemic time in orbital compartment syndrome

A

90-120 mins

62
Q

only contraindication to lateral canthotomy and cantholysis

A

globe rupture

63
Q

mgmt in chemical ocular injury

A

-Morgan lens and wash with 1-2 L of NS, then examine (washout before testing visual acuity)
-can check pH first
-irrigate until pH is between 7 and 7.4, re-check 30 mins after irrigation and continue to irrigate if pH is out of range

64
Q

how to get super glue out of eye

A

apply tons of erythro ointment, which loosens it, remove what comes off easily. tell pt to keep applying erythro ointment and arrange ophtho f/u

65
Q

treatment for acute angle closure glaucoma?

A

TAPAM

timolol drops 1 drop bid
acetazolamide 500 po/iv
pilocarpine drops
analgesics (opiods lower pressure)
mannitol if refractory (ie IOOP > 40 after 1 hour)

check re check IOP hourly

66
Q

basic pathophys of acute angle closure

A

lens or iris blocks trabecular meshwork stopping aqueous humor from exiting eye

67
Q

what usually triggers acute angle closure glaucoma?

A

anything that dilates pupil (anticholinergics, sympathomimetic, emotional stress, dim lighting)

68
Q

signs and symptoms of acute angle closure glaucoma?

A

symp:
HA, decreased vision, eye pain, n/v

signs: raised IOP, fixed, mid dilated pupil, hzy cornea, conjunctival injection, eye is “rock hard”

69
Q

is optic neuritis painful

A

usually mild to mod pain, but can be painless

70
Q

timeline of optic neuritis?

A

vision loss over days or hours, usually unilateral can be profound or just bluriness

71
Q

CRAO:
what is it?
epidemiology
s/s
mgmt

A

-central retinal artery occlusion - ischemic embolic stroke of eye (1st branch off ICA is ophthalmic artery)
-quite rare, age > 50, same rf as stroke
-sudden painless vision loss (monocular); pale retina (pale bc no flow) with cherry red spot
-ophthalmic emergency, consult ophtho stat, irreversible vision loss after 4 hours

72
Q

CVAO:
what is it?
epidemiology
s/s
mgmt

A

central retinal vein occlusion - thrombus of vein
- same rf as stroke
-“blood and thunder” ie diffuse retinal hemorrhage, presents as rapid painless monocular vision loss
-no consensus treatment, consult ophtho

73
Q

important initial distinction for “flashers and floaters”

A

monocular vs binocular
bi: intracranial
mono: ocular

74
Q

mgmt for ?retinal vs vitreous detachment

A

ophtho consult for dilated exam

75
Q

symptoms/signs of retinal detach

A

flasher/floaters, curtain over field of vision, visual field deficit, decreased central or peripheral visual acuity

76
Q

age at which to consider GCA?

A

> 50

77
Q

symptoms GCA

A

HA, jaw claudication, fever, myalgias, decreased vision, temporal artery tenderness

78
Q

blood test to r/o GCA

A

CRP or ESR

79
Q

if you suspect GCA what do you do

A

high dose steroids, arrange biopsy with ophtho

80
Q

What is a pcomm artery aneurysm until proven otherwise?

A

Acute cranial nerve 3 palsy (down and out) with ipsilateral pupillary dilatation

81
Q

what is horner’s syndrome triad? what causes it?

A

ptosis, miosis, anhidrosis
causes: stroke, tumor, ICA dissection, HZV, trauma

if neck pain and horners = carotid dissection