Ophthalmology Flashcards
Episcleritis sx
Red eye
classically not painful
Feels sore or gritty
The redness often appears in a focal or segmental pattern
Herpes zoster ophthalmicus tx
oral antiviral treatment for 7-10 days
ideally started within 72 hours
Acute angel closure glaucoma SX
fixed dilated pupil with conjunctival injection
Ocular pain, decreased visual acuity, worse with mydriasis(dark rooms), haloes around lights
AACG mx
initial medical treatment emergency treatment.
combination of eye drops, for example:
a direct parasympathomimetic - pilocarpine,
a beta-blocker (e.g. timolol,
an alpha-2 agonist apraclonidine,
intravenous acetazolamide
reduces aqueous secretions
some guidelines also recommend the use of topical steroids to reduce inflammation
Definitive management
laser peripheral iridotomy
Herpes zoster ophthalmicus
blistering rash in the distribution of the ophthalmic division of the trigeminal nerve. It is caused by reactivation of latent varicella-zoster virus
Hutchinson’s sign: rash on the tip or side of the nose. Indicates nasociliary involvement and is a strong risk factor for ocular involvement
Sx of Orbital cellulitis
Redness and swelling around the eye
Severe ocular pain
Visual disturbance
Proptosis
Ophthalmoplegia/pain with eye movements
Eyelid oedema and ptosis
Differentiating orbital from preseptal cellulitis
reduced visual acuity, proptosis, ophthalmoplegia/pain with eye movements are NOT consistent with preseptal cellulitis
Mx of orbital cellulitis
admission to hospital for IV antibiotics
Retinitis pigmentosa
night blindness + tunnel vision
fundoscopy: black bone spicule-shaped pigmentation
Anterior uveitis sx
painful red eye with photophobia
uveitis: small, fixed oval pupil, ciliary flush
Pain worse when using the eye, such as when reading or moving the eye.
hypopyon- white cells in ant chamber
Analgesia for corneal ulcer
Oral
Topical delays healing
Diabetic maculopathy
hard exudates and other ‘background’ changes on macula
check visual acuity
Non-proliferative diabetic retinopathy
Mild NPDR
1 or more microaneurysm
Moderate NPDR
microaneurysms
blot haemorrhages
hard exudates
cotton wool spots (‘soft exudates’ - represent areas of retinal infarction), venous beading/looping and intraretinal microvascular abnormalities (IRMA) less severe than in severe NPDR
Severe NPDR
blot haemorrhages and microaneurysms in 4 quadrants
venous beading in at least 2 quadrants
IRMA in at least 1 quadrant
Proliferative diabetic retinopathy features
retinal neovascularisation - may lead to vitrous haemorrhage
fibrous tissue forming anterior to retinal disc
more common in Type I DM, 50% blind in 5 years
Scleritis vs episcleritis
The main method of differentiating the two presentations is the use of phenylephrine or neosynephrine eye drops. These drops will cause blanching (go away) of the blood vessels in episcleritis, but not in scleritis.
Scleritis is often painful
Epi- is not
Eyelid conditions
blepharitis: inflammation of the eyelid margins typically leading to a red eye
stye: infection of the glands of the eyelids
firm painless lump in the eyelid.
hordeolum externum- external
hordeolum internum- internal
chalazion (Meibomian cyst)
entropion: in-turning of the eyelids
ectropion: out-turning of the eyelids
Central retinal vein occlusion vs central artery occlusion
Vein - sudden painless vision loss, optic disc swelling, and multiple flame-shaped and blot haemorrhages
Artery
sudden painless vision loss and a pale retina with a cherry-red spot at the fovea on examination.
Age-related macular degeneration
dry macular degeneration
90% of cases
also known as atrophic
characterised by drusen - yellow round spots
wet macular degeneration
10% of cases
also known as exudative or neovascular macular degeneration
characterised by choroidal neovascularisation
leakage of serous fluid and blood can subsequently result in a rapid loss of vision
central scotoma
Mx of allergic conjunctivitis.
Non pharma- avoid allergen, cold compress
Then topical or systemic antihistamines
Endophthalmitis sx and mx
Red eye, pain and visual loss following intraocular surgery
urgent ophthalmic review
Argyll-Robertson pupil
Bilaterally small pupils that accommodate but don’t react to bright light. Causes include neurosyphilis and diabetes mellitus
Horner syndrome
Miosis (pupillary constriction), ptosis (droopy eyelid), apparent enophthalmos (inset eyeball), with or without anhidrosis (decreased sweating)
Marcus-Gunn pupil
Relative afferent pupillary defect, seen during the swinging light examination of pupil response.
Adie pupil
Tonically dilated pupil, slowly reactive to light with more definite accommodation response
Caused by damage to parasympathetic innervation of the eye due to viral or bacterial infection. Commonly seen in females, accompanied by absent knee or ankle jerks.
Herpes simplex keratitis mx
topical aciclovir
Central retinal artery occlusion mx
If temporal arteritis - IV steroids
Intraarterial thrombolysis may be attempted but currently, trials show mixed results.
cherry red spot appearance
CRAO and temporal arteritis
Subconjunctival haemorrhage
Flat, red patch on the conjunctiva. Normal vision, not painful
Self limiting
If the cause is traumatic consider a referral to the ophthalmologist to ensure no other damage has been caused to the eye