objectives-1 Flashcards

1
Q

for paralysis of face, does LMN and UMN affect ipsilateral or contralateral face?

A

LMN- ipsi

UMN- contra

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2
Q

bulbar palsy

A

weakness in chewing, speaking, or swallowing (aka bulbar functions)

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3
Q

UMN pseudobulbar vs LMN (CN 7, 9, 10, 12) bulbar palsy

A

UMN: hyperactive jaw jerk (masseter reflex)

LMN: atrophy and fasciculations of muscles of the face, jaws, palate or tongue

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4
Q

tabetic gait

A

foot slapping

from syphilis or severe polyneuropathy

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5
Q

pseudotumor cerebri presentation

A

throbbing HA, +/- N/V, diplopia.

Patients are often obese

papilledema on exam

LP will have normal CSF @ high opening pressure

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6
Q

what causes should you think when an older vs younger patient presents with trigeminal neuralgia?

A

younger: MS
older: Tortuous or kinked BV compressing trigeminal

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7
Q

treatment of trigmenial neuralgia

A

carbamazapine

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8
Q

bulbous neuroma

A

following aberrant re-growth of a damaged

nerve, often exquisitely painful.

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9
Q

where is lesion with Ataxic respiration (variable breaths at an irregular interval)?

A

medulla

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10
Q

where is lesion with large fixed pupils

A

tectal (dorsal) midbrain lesion involving the parasympathetic fibers there

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11
Q

where is lesion with UNILATERAL BLOWN PUPIL, unresponsive consensually or directly?

A

ipsilateral CN III compression from swollen temporal lobe (uncal herniation)

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12
Q

where is lesion with small fixed pupils

A

pontine lesion involving sympathetic fibers

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13
Q

Oculocephalic reflex:

A

eyes roll in opposite direction of head turn with intact brainstem.

Remain in fixed position despite rolling if brainstem not intact

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14
Q

Oculovestibular (cold caloric) reflex:

A

eyes should move slowly toward the cold (irrigated) ear

i. If not present, brainstem compromised

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15
Q

how is sleep apnea diagnosed?

A

sleep study

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16
Q

cataplexy

A

periodic loss of muscle tone often provoked by emotional triggers

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17
Q

narcolepsy features

A

REM naps suddenly

cataplexy

sleep paralysis (REM hypotonia)

hynapgogic/pompic hallucinations

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18
Q

diagnosing narcolepsy

A

via multiple sleep latency tests, where the abnormally early onset of REM is
recorded as the patient is allowed to fall asleep several times but not in a state of sleep deprivation

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19
Q

syncope symptoms

A

dimming vision, tachycardia, diaphoresis, and “light-headedfeeling”.

can recall after episode; return to baseline

20
Q

seizure prodrome symptoms

A

nausea or dizziness,

but patient cannot remember events

+/- urinary incontinence, post-ictal confusion/lethargy/irritation

21
Q

seizure signs

A

tongue bite mark, rapid RR, bowel/bladder incontinence. Can have elevated WBC

22
Q

simple partial seizure of motor cortex

A

jerking, rhythmic movements

23
Q

simple partial seizure of temporal lobe

A

deja vu, amnesia, dysphoric/euphoric feelings, abnormal taste/smell

24
Q

simple partial seizure of parietal lobe

A

paresthesia, dysesthesia, numbness, vertigo

25
simple partial seizure of occipital lobe
visual hallucinations
26
complex partial seizures
LOC Usually involves medial temporal lobe=staring into space, deja vu, emotional changes, automatisms
27
generalized absence seizure
LOC several seconds of staring/freezing in place, post-ictal phase, pediatric. 3 hz spike and wave, sux to have absence seizures
28
generalized tonic clonic contraction
seizure=LOC, first tonic contraction forcing air out (cry) and arching of back, then clonus with violent jerking, salivation/drooling, tongue biting, post-ictal phase
29
tremor causes
resting: parkinsons postural: essential kinetic: essential or cerebellar
30
dystonia causes - focal vs gen
Focal=localized (ie Torticollis) Generalized=diffuse, disabling (seen in hereditary disorders)
31
dystonia description
continual, sustained, painful contractions leading to spasms, turning of limbs
32
myoclonus associations
toximetabolic encephalopathy, Cruetzfeldt-Jacob Dz, Wilson’s Dz
33
Asterixis description
flapping tremor that “looks like foot tapping”
34
asterixis cause if bilateral vs unilateral
bilateral: Hepatic/metabolic encephalopathy uni: Structural or ischemic lesion
35
TIAs last how long
usually 15 min, within 24 hrs
36
where is path for amaurosis fugax
carotids via ophthalmic artery
37
MCA infarct characterized by what symptoms
transient hemiparesis, aphasias
38
PCA infarct characterized by what symptoms
transient homonymous hemianopsia
39
Basilar/SCA/AICA characterized by what symptoms
Dizziness, dysphagia, dysarthria, diplopia, cerebellar ataxia
40
Vertebral/PICA characterized by what symptoms
cerebellar ataxia, +/- medullary syndrome
41
workup for potential stroke
MRI/MRA brain, carotid doppler, TTE/TEE, check glucose and BP control, if no yield: - vasculitis autoimmune serum panel - LP for CSF analysis
42
treatment for atherosclerotic stroke
Start antiplatelet (ASA, or ASA+ clopidigrel common) and statin 1. Surgical approach: carotid endartectomy, possibly ballooning/stenting 2. Prevention: control DM, HTN, HLD, smoking cessation
43
treatment for embolic stroke
start anticoagulation (heparin, warfarin, Factor X inhibitors, etc)
44
pathophys mechanisms for intracranial hemorrhage
i. Deep hemorrhage-from small vessel, (lenticulostriate) rupture due to HTN (remember cocaine) ii. Superficial hemorrhage-from head trauma, possibly CT disorders iii. Both can be compounded if patient is on anticoagulation
45
workup of intracranial hemorrhage
CT non contrast looking for bright blood!!!, others CTA, MRI/MRA, CBC/CMP, check INR
46
treatment for intracranial hemorrhage
Manage underlying bleeding disorder if any, FFP and coag factors in some cases, Neurosurg for coil/clip placement