Nonfluent Aphasias Flashcards
Nonfluent Aphasias
may be referred to as: expressive aphasia, motor aphasia, or anterior aphasia
Major characteristics of nonfluent aphasias
- major symptoms characteristic of nonfluent syndromes of aphasia (generalities)
1. decreased rate of speech
2. decreased phrase length
3. decreased prosody
4. decreased initiation of speech
5. decreased talking in general
6. increased effort
Four types of nonfluent aphasias
broca’s aphasia
global aphasia
transcortical motor aphasia
mixed aphasia
Broca’s Aphasia
- first described in 1861
- associated w/damage to the cerebral language areas surrounding the sylvian fissure but not extending to Wernicke’s area
Neuroanatomical bases of Broca’s Aphasia
- posterior-inferior (third) frontal gyrus of the left hemisphere is known as Broca’s area (Brodmann’s areas 44 and part of 45)
- area known as the anterior language cortex
- middle cerebral artery blood supply
- lower part of the premotor cortex
General Characteristics of Broca’s Aphasia
- more easily recognized than Wernicke’s pts
- typically present w/contralateral hemiplegia or hemiparesis
- weakness of [r] side facial muscles
- most motor problems improve over time
- pts are often very depressed; catastrophic reactions refusing to cooperate or continue testing
Language Characteristics of Broca’s
- nonfluent and effortful speech
- agrammatic speech
- impaired repetition of words/sentences
- impaired naming
- questionable auditory comprehension
- oral reading
- writing problems
Broca’s Aphasia:
- Lesion site
- Verbal expression
- Auditory comprehension
- Naming
- posterior-inferior frontal gyrus of the left hemisphere
- severely impaired; agrammatic; telegraphic; dysprosodic; possible AOS; minimal paraphasias
- better than expression; some degree of deficiency in some pts; essentially intact for most
- impaired (anomia)
Broca’s Aphasia:
- Repetition
- Oral Reading
- Reading Comprehension
- Writing
- impaired; especially for grammatical features
- impaired; similar to oral expression
- impaired to some extent
- impaired
Transcortical Motor Aphasia (TCM)
- extrasylvian aphasic syndrome; lies outside of the perisylvian language zones
- nonfluent aphasia w/good repetition skills
TCM Neuroanatomical bases
- anterior superior frontal lobe
- usually above or below Broca’s area
- lesions often impact association pathways
- impacts supplemental motor area
- supplied by the anterior cerebral artery
General Characteristics of TCM
- similar to Broca’s aphasia
- motor disorders: rigidity of UE, akinesia, bradykinesia
- hemiparesis
- pts may demonstrate apathy or behavioral withdrawal
- exhibit little to no interest in using language
TCM Language Characteristics
- muteness, echolalic, reduced spontaneous speech
- agrammatic speech, paraphasic
- impaired naming w/intact repetition
- intact serial speech (relatively)
- intact knowledge of grammar/meaningfulness
- limited naming; may use motor prompts
- better comprehension than production
- essentially mute, motor prompts may help them
TCM Aphasia
- Lesion site
- Vertical expression
- Auditory comprehension
- Naming
- deep portions of left frontal lobe below or above Broca’s area
- impaired; initially mute; paraphasic; agrammatic; telegraphic; limited word fluency; pt will have no apraxia of speech
- intact for simple; subtle problems w/complex material
- mildly impaired; better for confrontational naming
TCM Aphasia
- Repetition
- Oral Reading
- Reading comprehension
- Writing
- intact; may demonstrate echolalic and perseverative speech
- Impaired
- good except for syntactically complex material
- impaired
Mixed “Transcortical” Aphasia
- rare nonfluent aphasia type
- combine TMA and TSA
- language impairment is severe and extensive
- pts retain repetition skills
- has been labeled isolation aphasia
Neuroanatomical Bases of Mixed Transcortical
- caused by various conditions that decrease blood flow throughout the cerebral arteries
- hypoxia of various origins
- cardiac arrest
- cerebral edema
- multiple embolic strokes
- supplied by the middle cerebral artery, and the anterior/posterior cerebral arteries
- Broca’s, Wernicke’s and the arcuate fasciculus are spared
General characteristics of Mixed Transcortical Aphasia
- varied clinical picture
- bilateral UMN paralysis
- severe spastic quadriparesis (weakness of all 4 limbs)
- visual field deficits (typical problem is right hemianopia)
- weakness in hip/shoulder muscles
- severe brain damage
Language Characteristics of Mixed Transcortical Aphasia
- extremely limited spontaneous verbal expression, echolalic
- severely impaired fluency
- severely impaired auditory comprehension
- marked naming difficulty
- unimpaired automatic speech
- normal articulation
- severe reading deficits
- severe writing impairments
Mixed Transcortical Aphasia
- Lesion site
- Verbal Expression
- Auditory Comprehension
- Naming
- watershed area; pg 113
- often severely impaired; agrammatic w/paraphasias
- often severely impaired
- Impaired
Mixed Transcortical Aphasia
- Repetition
- Oral Reading
- Reading Comprehension
- Writing
- Additional Information
- good but parrot-like; nonfunctional repetition
- often severely impaired
- often severely impaired
- impaired
- Broca’s aphasia with decreased auditory comprehension
Global Aphasia
- may account for 30-55% of pts w/aphasia
- most severe form of aphasia; has a generalized effect on communication skills
- impacts all modes of communication and spares no particular skill
- possible for global aphasia to evolve into another type of aphasia
- be careful of prognosis
Neuroanatomical Bases of Global Aphasia
- lesion likely involve the entire perisylvian region affecting both Broca’s and Wernick’e areas
- subcortical areas may even be impacted
- more common sites are impacted by the middle cerebral artery
pg 148 (text; has good picture…figure 5-3)
- widespread destruction of the left fronto-temporo-parietal regions
General Characteristics of Global Aphasia
- presence of strong neurological symptoms; weakeness, paralysis, sensory loss
- apraxia including both verbal and oral types
- hemi-neglect: left neglect is common in right hemisphere damage