Non surgical + TFO Flashcards

1
Q

How long may sensitivity after ScRD last

A

2 weeks.

Salt water rinse, apply sensodyne sensitivity relief at cej

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

How Long does it take for long je to form after ScRD

A

7-14 days Caton and Zander 1979

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

When does maturation of connective tissue complete after ScRD

A

Week 4-8 Biagini et al 1988

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Teeth with constant bop during recalls had __ times higher risk for future tooth loss compared to teeth without BOP

A

46 schatzle et al 2004

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Average clinical improvement baseline <3mm

A
  1. 03mm Pd reduction
    - 0.34mm CA gain

Cobb 2002

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Average clinical improvement baseline <4-6mm

A
  1. 29mm Pd reduction
  2. 55mm ca gain

Cobb 2002

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Average clinical improvement baseline <6mm

A
  1. 16mm Pd reduction
  2. 19mm ca gain

Cobb 2002

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Treatment outcome statistics for pocket and patient

A

35% of baseline pathological pockets did not reach endpoint success Wennstrom et al 2005

39% of patients reached end point success of probing depths ≤5mm van dear Weijden et al 2019

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Can you leave calculus behind

A

Clinically acceptable levels of gingival wound healing can occur despite microscopic aggregates of residual calculus Nyman et al 1986

Reduction of gram negative bacterial load is more crucial, periodontal healing possible on calculus as long as subgingival plaque removed from root surfaces

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

How does tfo result in mobility

A

Jiggling forces, multidirectional heavy loading, gradually increase in width of PDL on both sides of the teet. Pdl space increase, active bone resorption, increase mobility

Bone resorption induced by tfo is reversible (in a healthy periodontium)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

If occlusal forces are too great and PDL unable to adapt to occlusal forces

A

Inflammation is pushed down into zone of co destruction, move down PDL space, connective tissue attachment loss.

There is bone resorption due to occlusal forces and bone resorption due to periodontal inflammation

Angular bone defect, widened PDL space. Occlusal adjustments —> no improvement in attachment level because lost by bacteria

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

How does tfo accelerate progression of periodontitis

A

Glickman: trauma causes bacteria to enter zone of co destruction, further ingress into pdl space

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

TFO does or does not aggravate gingivitis associated with plaque in a healthy periodontium

A

Does not

Does not CAUSE periodontitis, only accelerate progression

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

When does tfo cause intrabony defect/further connective tissue loss and bone loss

A

If there is subg plaque in addition to heavy occlusal loading

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

What to check when diagnosing TFO

A

MI and lateral excursion

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Important signs and symptoms to diagnose tfo + key radiographic feature

A

Increased tooth mobility, fremitus

Pathogenic tooth migration

Tenderness of muscles of mastication/TMD dysfunction

Craze and crack lines

Widened PDL space

17
Q

Can diagnosis of TFO be made from hypermobility alone

A

Ramfjord and ash 1981
Increasing tooth mobility found over series of repeated measurements but cannot be made from hypermobility alone

Need other signs eg widened PDL space

18
Q

Management of TFO

A

Occlusal adjustments

Selective grinding (not equilibration, just adjust tooth with heavy occlusal contact)

Removable/fixed appliances eg bite splint

Ortho treatment to adjust occlusion

19
Q

Occlusal equilibration vs selective grinding

A

Equilibration: to reshape occluding surfaces to create harmonious contact relationship

Selective grinding: alter occlusal forms to decrease/redirect occlusal forces

20
Q

How thick is shi stock

A

0.005 inch

21
Q

ScRP result in __ reduction in HbA1c

A

0.4%, equivalent to action of 1 oral medication Hngebretsson and Kocher 2013 (Falcao and Bullon 2019 found this controversial)

Chapple et al 2013 reduced severity of perio disease —> reduced pro inflammatory TNFa —> increased glucose uptake by cells

22
Q

Effectiveness of OH with no ScRD

A

25% reduction in bleeding

Can resolve signs of inflammation associated with gingivitis Badersten et al 1984

Westfelt et al 1998: 8% of such sites had further attachment loss, 5% unchanged at end of study

Brushing alone has limited effect on subgingival microflora and infiltrate in deeper pockets

23
Q

Efficacy of poor OH with ScRD

A

Pockets return to baseline at end fo two months due to reestablishemnt of microbiological profile

24
Q

Efficacy of good oh with scrd van dear weijden et al 2002

A

Significant reduction in % of deep pockets >6mm from 11% to -.3% after.3 years

25
Q

Critical probing depth to benefit from surgery

A

Only probing depth 5.5mm and above would benefit from benefit from additional surgical therapy Heinz Meyfield et al 2002

5.4mm critical probing depth

26
Q

Surgical intervention should only be considered after ___

A

Non surgical perio therapy and good plaque control

Surgery in plaque infested dentition —> recurrence of disease, ineffective to prevent recurrence of periodontitis Nyman et al 1977

27
Q

Critical probing depth Lindhe et al 1982 for nspt and spt

A

Nspt: loss of attachment when clinical probing depth ≤2.9mm

Spt: loss of attachment when clinical probing depth ≤4.2mm. Above this value there is clinical attachment gain