Acute Lesions Flashcards
What is stillman’s cleft
Soft tissue cleft due to trauma from flossing. Mucogingival triangular shaped defect predominantly seen on buccal surface
How does trauma from flossing lead to marginal gingivitis
Create flossing cleft or stillmans cleft, increasing risk of bacteria permeating into tissues and cause cleft to expand laterally
How to manage flossing cleft
If cleft is red and feels like soft tissue, likely to be reversible as only superficial layer affected. Patient stop parafunctional habit and use chx, review in 2 weeks
If cleft is white, can see bone underneath, completely coral pink —> entire width of tissue affected, has epithelialised. Irreversible, require surgical intervention
Clinical feature of chemical trauma
Immediate erythema and oedema od oral mucosa. Subsequent formation of white slough pseudomembrane covering ulceration. Ulceration has irregular border and bleeds easily.
Clinical feature of periodontal abscess
Localised purulent infection in periodontal tissues or lesion with expressed periodontal breakdown in limited period of time with easily detectable clinical symptoms
Herrera et al Perio 2000
Gingival vs periodontal abscess
Gingival abscess at marginal and interdental gingival while periodontal abscess deeper, close to mucogingival line
Gingival abscess associated with subgingivally impacted foreign object while periodontal abscess associated with periodontal disease
Acute periodontal abscess symptoms
Sudden onset of pain
Localised tenderness
Sensitive to palpation
Sappuration on gentle pressure
Tooth mobility due to periodontal breakdown of connective tissue
Elevation of tooth hence discomfort on biting down
Chronic perio abscess symptoms
Asymptomatic
Sinus tract
Localised elevation due to granulation inflammatory tissue
Do periodontal abscesses happen singly or multiply
Most patients get single lesions, associated with local factors eg a lot of calculus
Multiple lesions associated with eg uncontrollable diabetes mellitus, untreated periodontitis patient after systemic antibiotic therapy
What is the most virulent microorganism in periodontal abscess
P gingivalis
What is a pericoronal abscess
Localised inflammation of pus within overlying ginval glad surrounding crown of incompletely erupted tooth
Diagnosis of anug
Punch out craters interdental, ulcers on interdental papilla
Rapid onset of pain
Pseudomembrane over necrotic area
Fever, discomfort
Marginal erythema
Risk factors for nug
HIV positive 1-10% of HIV patients Severe malnutrition Poor OH Stress Lowered host immune response
Histological features of necrotising ulcerative gingivitis (important)
4 zones
Innermost: spirochetes infiltration zone
Tissue components intact, filled with spirochetes
Necrotic zone
Disintegrated cells with spirochetes and fusiform bacteria
Neutrophil rich zone
High number of leukocytes and neutrophils, host immune zone
Bacterial zone
Superficial mesh degenerated epithelial cells. Leukocytes, spirochetes, fusiform present
SNNB
Management of NUG
Acute phase (2-4 days)
- use ultrasonic to remove superficial plaque and calculus
- debridement daily for 2-4 days
- no toothbrushing for 1-2 days
- chx 0.12-2% mouth rinse twice a day
Clear existing perio
Corrective eg surgery to fix soft tissue craters
Maintenance