NeuroMSK Flashcards
Contusion:
- Rx:
- complications
Rx: 1st 48hrs:
- PRICE, no harm (heat, alcohol, running, massage)
- put mm on as much stretch as possible (prevent healing in short ROM)
- Crutches if necessary
- pain free ROM/ stretch
- progress exercises after acute phase
Complications:
- compartment syndrome: check capillary refill + sensation, mm strength
- myositis ossificans: suspect if no improvement in 2-3 weeks
Strains and tears:
- where and when?
- grades
- Rx phases
Majority in bi-articular mm at mm tendon junction during eccentric loading
Ax: A/PROM, strength, length, stability, special tests
Grade:
1= micro tearing, pain/ no weakness
2= macro tearing, pain + structural changes (laxity, dec strength)
3= complete: painless + weak, may see lump
Rx:
- Acute/inflammation: PRICE
- Repair: Strength, stretching, modalities, DTF
- Remodelling: Strength (inc load and velocity), stretch, stability
- RTP: symmetrical mm length, strength, power, core control, sport specific RTP program.
DOMS
- pathology
- Rx
Possibly due to local nerve endings response to altered pH, swelling, inflammation)
Rx: light activity, massage, avoid anti-inflammatories,
Pelvic floor dysfunction:
- nerve
- how to ax
- Pudendal nerve: S2,3,4 off sacral plexus
Chronic pelvic pain:
- causes?
- Rx goals
Causes: msk, neuro, gynaecological, urogenital
Rx: - decrease PF resting tone - Increase PF proprioception - increase PF motor control - decrease pain sensitization -
Pelvic girdle pain:
- pregnancy and pathology
- s/s
- signs
50% pregnancies = weakness/laxity of PFM during pregnancy and childbirth
S/s:
- pelvic, groin, thigh, abdomen pain with sustained posture and transitional movements
Signs:
- Posture: locked knees, Lumbar lordosis, thoracic kyphosis, FHP
- Asymmetry: standing and supine
- gait: shuffling, waddling, leg drag
Pelvic girdle pain:
- Ax
- Rx
Ax:
- ASLR with force and form closure
- hip quadrant (IR/ER)
- SI joint stability (P4, ganslens, FABER, long dorsal lig palpation
- TOP spinous processes, Piriformis
- resisted hip ADD/ abd
Rx:
- Education
- Posture: standing: soft knee, equal weight, sitting: knees at hip heigh or lower, sleeping: pillows to maintain neutral
- Manual Therapy:
- exercise: PFM (endurance w/ functional activities), maintenance = 8-12 contractions, 2x/week
- movment strategies:
Syndesmotic ankle sprain
- MOI
- ligaments affected
MOI: planted foot + IR of leg = ER of mortise OR/ hyperDF or hyperPF
Ligs affected:
commonly: ATFL, PTFL, interosseous
+ others,#, bone bruise / OA
High ankle sprain:
- Dx
- Rx
Dx:
- PROM foot ER stress test, squeeze test, fibular PA translation, squat test, heel thump, SL hop test
Rx:
1) protection phase (2 weeks): PRICE, decrease inflammation, modalities for edema, NWB with crutches
2) Management phase: joint mobs, regain ROM, strength & fxn, PWB , bilateral stance training
3) Management: increase fxn; unilateral balance + strength
4) RTP: 2x as long as regular ankle sprains. Cutting, jumping etc
Muscle injury and repair:
- phases of healing
1) Inflammation: 24hrs- 4 days after
- myofibres rupture and necrosis
- Hematomas
- inflammatory cell reaction
2) Repair: 5days- 2 weeks
- phagocytosis of necrotic fibres
- regeneration of myofibres
- formation of scar tissue
- capillary in growth
3) remodelling: 14 days +
- maturation of myofibres
- contraction and organization of scar tissue
- recovery of function
Tendon:
- composed of?
- loading response?
Composed of:
- Tenocytes (crave mechanical load) + collagen, glucosaminoglycan (Extracellular matrix)
Loading response:
- increased collagen synthesis, cellular proliferation, alignment
Tendonopathy:
- defined
- do and don’t see?
- Rx
Chronic micro trauma causing loss of collagen organization
- no inflammation
Do see: collagen disorganization, glucosaminoglycan, variable Tenocyte density, increase vasularity.
Rx: proper loading
Achilles Tendinopathy
- Risk factors
- s/s
Risk factors: age, BMI, Male, running, foot wear, pronation, decrease DF, decrease LE strength, tight/weak calf mm
s/s:
- thickened tendon, TOP
Achilles tendinopathy
- DDX
DDX:
- Achilles’ tendon partial rupture
- Sever’s disease (peds):
- ->inflamed calcaneal apophysis causing pulling on tendon at insertion. Effects growing active children. Rx = activity modification, gentle exercises
Achilles tendinopathy
- Rx:
NSAID if acute
- alter contributing factors ( pronation, mm imbalances, myofacial restrictions, core
- Progressive exercise program: eccentric loading is necessary
- orthotics, heel lifts
- stretching, manual therapy
De Quervain’s Tenosynovitis:
- defined
- mm OIF
- Tendinosis of sheath/tunnel surrounding Extensor Pillicis Brevis + abductor pollicis longus.
EPB:
- O: post lower 1/3 of radius
- I: base of proximal thumb phalanx
- F: extension + abduction of thumb
Abd PL:
- O: ulna, radius, interosseous membrane
- I: base of 1st metacarpal
- F: abduct thumb
De Quervain’s synovitis:
- s/s:
-
S/s: chronic pain, tendon thickening, worse with repetitive hand/wrist movements
Ax:
- Finkelstein test: tuck thumb in fist, ulnar wrist deviation, +ve if pain along distal radius
Rx:
- acute = offload tissue, PRICE, education
- chronic = corticosteroid injections (50% success)
Lateral elbow pain:
- mm involved OIF
- s/s
ECRB:
- O: lateral epicondyle
- I: base of 2nd metacarpal
- F: ext/ abduction of hand
- N: deep branch of radial
(10%) Common extensor tendon + ECRL:
- O: Lat supracondylar ridge
- I: 2nd metacarpal base (radial side)
- F: ext + Abd hand
- N: radial
- symp: with wrist/finger extension & gripping
S/s: gripping/ repetitive reaching and grasp, typing/ overload
Tennis elbow:
- Ax:
- DDx
Ax:
- resisted 3rd finger PIP extension (maudsley’s test)
- resisted active wirst extension + radial deviation
- passive pronation wrist flexion + elbow extension
- TOP, pain over lateral epicondyle
- No nerve s/s: check radial ULTT
DDx:
- Cspine referred (C5-7), shoulder referred, nerve entrapment, bursitis of radio-humeral), LCL sprain, proximal radio-ulna joint issue
Tennis elbow
- Rx
Acute:
- PRICE, modalities (US, TENS), ROM, offload tissue, education (avoid NSAIDS, rest)
Repair Phase:
- gentle stressing of collagen: DTFM, stretching, eccentrics, manual therapy,
Outcome measure:
- pain free grip w/ handheld dynamometer
Rotator cuff tendonopathy:
- structures at fault
- types of impingement: + risk factors
- long head of biceps + supraspinatus (impingement)
Types:
1st impingement: narrowed subacromial space (older pt)
- anatomical abnormalities, degenerative changes, mm imbalances, postural faults
2nd impingement: instability ( instability –> subluxation of humeral head –> impingement
- overhead sports cause trauma of stabilizers
- anterior capsule laxity + posterior capsule tight = anterior humeral head sublux
Rotator cuff tendinopathy:
- s/s
- ax
- Rx
S/s:
- pain with overhead activity, penetrative motion, longstanding
Ax: neers, speeds, empty can
Rx:
- correct biomechanical faults, mm imbalances
- modalities, DTFM, manual therapy, education
Patellar tendinopathy:
-
” jumpers knee”
Risk factors:
- male, jumping athlete, jump height, reduced DF, age, BMI
Rx: slow heavy loads (concentric and eccentric), mm imbalance, avoid knee valgus
Fasciitis/ myofascial restriction:
Pain syndrome s/s + Rx
TOP (trigger point)
- onset from sudden overload, over-stretching, repetitive strain,
Rx:
- IMS
- flexibility, ROM
- Soft tissue massage
- manual therapy
- modalities
Loose body:
- defined
- s/s
- Ax
Free floating piece of bone or cartilage often from OA or chip fracture
s/s
- locking/ catching
Ax:
- ROM end feel as a bony block or “springy”
Hypermobility:
- defined + features
Instability:
- defined
Excessive laxity or length of tissue
- increase ROM/ neutral zone (osteoligamentous structures provide minimal support)
Instability:
- excessive ROM of arthrokinematics or osteokinematics
- no mm control, (form vs. Force closure)
Causes:
- trauma vs non trauma ( genetic, adjacent hypo mobility, habitual movements
Rx:
- mobilize a stiff joint/ tissue ( IMS, manual, massage, stretch)
- strengthen to. Stabilize hyper-mobile segment
- movement retraining, tape and braces
Lysis vs. Listhesis of spine
- Lysis = pars #
- Listhesis = # or slip of cranial vertebrae anteriorly
Spondylosis
- defined
OA of spine = degeneration of joints
- can lead to stenosis or disc herniation
Spondylolysis:
- defined
- population at risk
- Pars interarticularis (fibrous tissue) defect = degeneration
Seen in younger ppl with hyperEXT and rotation sports
- most are assymptomatic
- if bilateral it may lead to spondylolithesis
Spondylolithesis:
- defined
- common location
- MOI
- types
- grades
Slippage of one vertebrae on another
- common L5/S1
MOI: hyper extension, in young athlete
Types:
- Spondyloytic spondylolistesis: progressive period of rapid growth, rarely progressive to adult life, younger population
- Degeneration spondylolistesis: 2nd to DJD + Z-joint subluxation = OA of joints in spine, foramina narrowing, older population
Grades: 1-4: 25% of each grade of slippage
Spondylolisthesis:
- s/s
- Rx
- when to get surgery
S/s:
- Central LBP +/- referred pain
- aggravating factors = extension
- easing factors = flexion
Rx: stability
- Flexion exercises
- inner unit exercise = DNF + TA/multifidus/ PF
- brace if appropriate
Surgery:
- increased instability or slippage
- hard neuro signs
- evidence of SC involvement
Hypomobility:
- MOI
- structures
- Contributing factors
MOI: adaptive shortening of soft tissue, inappropriate end feel
Structural:
- muscle: atrophy and weakness
- tendon: decrease tensile strength
- ligament: decrease tensile strength + increase stiffness/adhesions
- cartilage: decrease synovial fluid, H2O content
- Bones: increase resorption, decrease done mass/mineral content
Countributing factors:
- prolong immobilization, postural dysfunction,
- sedentary lifestyle/ aging
- paralysis/ tone abnormalities
- mm imbalances