Neurological Diagnosis 3 Flashcards
swan neck deformity
hyperflexion of the DIP and hyperextenson of PIP
seen with RA
boutonneire deformity
hyperextension of DIP and hyperflexion of PIP
seen with RA
de quervain’s tenosynovitis
inflammation of extensor pollicis brevis and abductor pollicis longus tendons on the side of the wrist at the base of the thumb. can be brought on by simple strain injury. treated by bracing the thumb and wrist
+finkelstein’s test
syringomyelia
pathological longitudinal cyst of the central canal of the spinal cord. fluid filling cavities expand in adult years (congenital weakening usually at the C5-6 area
loss of sense of pain and temperature over the shoulders and back in a cape like distribution
multiple sclerosis
demyelination of CNS producing demyelinization of the spinal cord and later in the brain. plaques are present (MRI). both motor and sensory tracts are affected. most commonly seen in females age 20-40.
periods of exacerbation and remission
heat exacerbates the the symptoms
multiple sclerosis S/S
diplopia, scotomas, transient blindness, optic neuritis, pain, vertigo, and UMNL in the legs causing distal weakness.
+Lhermitte’s test
Charcot’s triad: scanning speech, intention tremors, nystagmus
myasthenia gravis
autoimmune disease in which the body makes antibodies against acetylcholine receptors. myoneuronal junction dysfunction. affects the motor cranial nerves first and then the proximal muscles
myasthenia gravis S/S
ptosis, diplopia, dysarthria, fatigue of muscles (especially following exercise)
muscle weakness varies during the day being worse at the end of the day
females age 20-40. diagnosed with ACHr, acetylcholine receptor test and treated with cholinesterase inhibiting drugs
lou Gehrig’s disease
ALS
affects the corticospinal tract and anterior horn. usually begins in hands/feet and life expectancy is short seen in males >40
S/S of lou gehrig’s disease
fasciculation are present as well as spasticity and increased DTRs. LMNL in the arms and UMNL in the legs
parkinson’s
a chronic progressive condition associated with loss of dopamine in the substantia nigra causing basal ganglionic dysfunction. gradual onset over age 50
S/S parkinson’s
resting tremors, mask like face, festinating gait, cogwheel/lead pipe rigidity, forward stooped posture, bradykinesia, soft voice, constipation
guillian-barre syndrome
inflammatory, demyelinating polyneuropathy of the PNS linked to recent immunizations or seen after recent illness.
S/S gullian-barre syndrome
ascending paralysis and sensory symptoms
generally can recover
charcot-marie-tooth
muscle weakness, bilateral foot drop, decreased muscle size, decreased sensation, tremor, hand contracture
propulsion/festinating/shuffling
parkinson’s, forward leanig posture with small, shuffling steps, decreased arm movement
steppage
anterior compartment syndrome, foot drop, L4 lesion/paresis of tibialis anterior
trendelenburg
weak gluteus medius causing lurching and drastic pelvic tilting on affected side