Neck P! with Movement and Coordination Impairment (WAD) Flashcards

1
Q

What is the proposed underlying cause of Whiplash Associated Disorder (WAD)? (7)

A
  • Trauma often associated with a motor vehicle accident
  • Central and Peripheral Nervous system sensitization are common (Nociplastic pain)
  • Motor impairment
  • Muscle strain and ligamentous sprain
  • Fatty infiltration of the neck/extensors
  • PTSD
  • Concussion is possible
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2
Q

Differential diagnosis for WAD should include other impairment-based diagnoses. What are those different diagnosis?

A
  • Neck pain with mobility deficit
  • Neck pain with headache
  • Neck pain with radiating pain
  • Rotator cuff related shoulder
  • Thoracic and/or rib pain with mobility deficit
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3
Q

What system, structure, pain mechanism (phenotype), and phase of healing are unique to WAD patients?

A

System
- Neuromuscular

Structure
- Ligaments, muscles, central/peripheral nervous system

Pain Mechanism
- Nociceptive (WAD I - WAD B){Minor injury},
- Nociplastic (WAD IIC),
- Nociplastic/Neuropathic (WADIII)

Phase of healing
- Muscle strain 2-4 weeks, ligament sprain 10-12 weeks

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4
Q

What are common subjective reports of patients with motor coordination impairments (WAD)?
(10) Get big picture

A
  • History of trauma often involving MVA
  • Neck pain with possible somatic referred pain in UE
  • High pain and disability score
  • Variable agg/ease factors and 24 hr pain behavior based on dominate pain mechanisms and Quebec classification
  • Various nonspecific concussive S/S
  • Dizziness/nausea
  • Headache, concentration or memory difficulties
  • Confusion
  • Hypersensitivity to mechanical, thermal, acoustic, odor, light stimuli
  • Heightened affective distress
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5
Q

What is the Pain Phenotype/Mechanism of pain for Movement Coordination impairments (WAD)

A

Nociceptive Pain

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6
Q

What are common findings for patients with movement coordination impairments (WAD) based on the Quebec Classification? {WAD 0-1}

A

WAD 0
- No symptoms of neck pain
- No physical signs

WAD I
- Neck symptoms of pain, stiffness, or tenderness
- No physical signs

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7
Q

What are common findings for patients with movement coordination impairments (WAD) based on the Quebec Classification? {WAD II}

A

WAD II
Neck Symptoms
- Pain, stiffness, or tenderness
Musculoskeletal signs
- Decreased ROM
- Point tenderness

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8
Q

What are common findings for patients with movement coordination impairments (WAD) based on the Quebec Classification? {WAD III}

A

WAD III
Neck Symptoms
- Pain, stiffness, or tenderness

Musculoskeletal signs
- Decreased ROM
- Point tenderness

*Neurological
- Altered DTRs
- Weakness and sensory deficits

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9
Q

What are common findings for patients with movement coordination impairments (WAD) based on the Quebec Classification? {WAD IV}

A

WAD IV
Fracture or Dislocation

Neck Symptoms
- Pain, stiffness, or tenderness

Musculoskeletal signs
- Decreased ROM
- Point tenderness

*Neurological
- Altered DTRs
- Weakness and sensory deficits

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10
Q

What are common findings for patients with movement coordination impairments (WAD) based on the Sterling Subclassification? {WAD II A}

A

WAD II A {Flexor Endurance is Poor}
Neck Pain

Motor Impairment
- Decreased ROM
- Altered recruitment patterns (CCFT)

Sensory Impairment
- Local Cervical Mechanical Hyperalgesia

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11
Q

What are common findings for patients with movement coordination impairments (WAD) based on the Sterling Subclassification? {WAD II B}

A

WAD II B
Neck Pain

Motor Impairment
- Decreased ROM
- Altered recruitment patterns (CCFT)

Sensory Impairment
- Local Cervical Mechanical Hyperalgesia

Psychological Impairment
- Elevated psychological distress

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12
Q

What are common findings for patients with movement coordination impairments (WAD) based on the Sterling Subclassification? {WAD II C}

A

WAD II C
Neck Pain

Motor Impairment
- Decreased ROM
- Altered recruitment patterns (CCFT)
- Increased joint positioning errors

Sensory Impairment
- Local Cervical Mechanical Hyperalgesia

Generalized sensory hypersensitivity (Mechanical, thermal, bilateral upper lomb neurodynamics testing

Sympathetic nervous system disturbances

Psychological Impairment
- Elevated psychological distress
- Elevated levels of acute post-traumatic stress

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13
Q

In the Objective Examination, what you looking for when doing AROM and PIVM with the patient?

A

We are looking at the patients ability to move and for Pain Provocation

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14
Q

In the Objective Examination, What will you typically find with AROM with WAD?

A
  • ROM limitations and symptoms provocation will depend on individual patient presentation
  • Neck P! with mid-range motion worsens with end-range positions
  • May see altered muscle recruitment and aberrant motions
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15
Q

In the Objective Examination, What will you typically find with PIVM (Passive Intervertebral Motion) with WAD?

A
  • Spasm and/or local and/or referred pain reproduction at the involved segments
  • Possible hyper-or hypomobility throughout the cerviothoracic spine
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16
Q

When doing Special Test and Measures for Pt. with WAD, What test will you conduct for Advanced Sensory Examination (WAD IIA-WAD IIC and WADIII), and what may you find?

A
  • Pressure Algometry: Reduced pressure threshold (local and regional)
  • Ice-Pain Test: Cold Hyperalgesia (Local and Regional)
  • ULND 1 (Median N.): + for gross neurodynamics mechanosensitivity
17
Q

When doing Special Test and Measures for Pt. with WAD, what may you find when you are doing Palpations on the Patient?

A
  • Point tenderness may include myofascial trigger points; Increased muscle tone of superficial cervical muscles
18
Q

In the Objective Examination for WAD, what special test and measures are you using for muscle performance examination?

A
  • Craniocervical Flexion Test (CCFT)
  • Deep Neck Flexor Endurance Test
  • Deep Neck Extensor Endurance Test
  • Cervical joint position sense test
  • Parascapular muscle recruitment and endurance testing
19
Q

What is the Prognosis for WAD?

A
  • Risk stratification
    –Mild disability/post-traumatic stress(45% of pt.)
    –Initially moderate improving to mild (40% of pt.)
    –Chronic severe problems (15% of pt.)
  • Recovery happens most rapidly in the 6-12 weeks post-injury
  • 50% will fully recover within 1 year
  • Considerable slowing to no recovery after 12 months
  • No significant recovery between 12 and 24 months
20
Q

When determining the Prognosis for WAD, what is the clinical prediction rule for this diagnosis?

A
  • Pt. older age (>40)
  • Initially higher levels of disability (NDI >40%)
  • Hyperarousal symptoms (>6 on hyperarousal subscale)
    –Posttraumatic Diagnostic Scale

Patients with this are at a higher risk for long-term disability

21
Q

What factors may impact the Prognosis of WAD?

A
  • High pain intensity (NPRA >/= 6/10)
  • High self-reported disability (NDI >/= 30%)
  • High Pain catastrophizing (PCS >/= 20)
  • High PTSD Symptoms (IES >/= 33)
  • Cold hyperalgesia (extremely sensitive to cold)

Strong association with long-term disability

22
Q

How would a patient present with WAD in the Acute stage of condition? What is the intervention goal?

A
  • Severity and Irritability are often high
  • Pain at rest or with initial to mid-range spinal movement; before tissue resistance
  • Pain control is often the intervention goal at this stage
23
Q

How would a patient present with WAD in the Subacute stage of condition? What is the intervention goal?

A
  • Severity and Irritability are often moderate
  • Pain experienced with mid-ranged motions that often worsen with end-range spinal movement; At tissue resistance
  • Movement control is often the intervention goal at this stage
24
Q

How would a patient present with WAS in the Chronic stage of condition? What is the intervention goal?

A
  • Severity and Irritability are often low
  • Pain that worsens with sustained end-range spinal movements or positioned; overpressure into tissue resistance
  • Functional optimization is often the intervention goal at this stage
25
Q

What are the interventions recommended for WAD in the Acute stage of condition?

A

Education:
- Return to normal non-provocative pre-accident activities as soon as possible
- Minimize collar use
- Perform postural and pain-free mobility exercises to decrease pain and increase ROM
- Reassurance that the recovery is expected to occur with the first 2-3 months
- Monitor for acceptable progress and initiate more intense rehab for those with delayed recovery

Exercise:
- Comprehensive exercise program (strength, endurance, and/or coordination)

26
Q

What are the interventions recommended for WAD in the Subacute stage of condition?

A

Education:
- Advice and counseling

Exercise:
- Active cervical ROM and isometric low-load strengthening
- Stretching, strengthening, endurance, neuromuscular re-education exercises
- Include postural, coordination, and stabilization elements

Manual Therapy:
- Cervical Mobilizations/Manipulations

Biophysical Agents
- Ice, heat, TENS

27
Q

What are the interventions recommended for WAD in the Chronic stage of condition?

A

Education:
- Advice focusing on reassurance, encouragement, prognosis, and pain management

Exercise:
Individualized Progressive Exercise
- Low-load cervioscapulothoracic strengthening, endurance, flexibility
- Functional training using cognitive behavioral principles
- Vestibular rehabilitation, eye-head-neck coordination, neuromuscular coordination elements

Biophysical agents
- TENS

28
Q

When should we consider interprofessional or Intraprofessional referral and what are other treatment options?

A

Imaging:
- Canadian C-Spine rule
- 5-view X-ray
- CT scan

Medial Intervention:
- NSAIDs
- Muscle relaxers
- SSRIs (selective serotonin re-uptake inhibitors) / SNRIs (Serotonin Norepinephrine re-uptake inhibitors)
- Antiepileptics

Psychosocial
- Counseling