Musculoskeletal Flashcards

1
Q

What are the risk factors for osteoporosis?

A

Genetics, thin white and asian women. Age, postmenopausal women as they lose estrogen. Nutrition. Physical exercise. Lifestyle choices. Meds, esp steroids. Comorbidity.

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2
Q

What test is used to diagnose osteoporosis?

A

Dual x-ray absorptiometry (DEXA) scan

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3
Q

S/s of osteoporosis?

A

Bone pain or tenderness, fractures with little or no trauma, loss of height over time, neck or lower back pain due to fractures, stooped posture

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4
Q

s/s of osteomyelitis?

A

Fever, chills, tachycardia, malaise, pain, edema, erythema, ulceration, drainage

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5
Q

Diagnosis of osteomyelitis?

A

Radiologic confirmation, bone scans, leukocytosis, elevated erythrocyte sedimentation rate (ESR), wound/blood cultures\

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6
Q

Management of osteomyelitis?

A

Prevention, pain control, antibiotics, supportive measures, hyperbaric treatment, surgical debridement, wound care, glycemic control

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7
Q

A fracture that occursthrought an area of disease bone; can occur without trauma or fall.
A fracture that results from repeated loading of bone and muscle

A

Pathologic fracture

Stress fracture

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8
Q

s/s of osteomyelitis? Diagnosis?

A

Pain, loss of function, deformity, shortening, crepitus, localized edema and ecchymosis.
Diagnosis has no special lab tests, but confirmation via radiology.

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9
Q

5 P’s of a neurovascular check? Same as 5 P’s of what?

A

Pain, pallor, pulselessness, paresthesia, paralysis

Same as the 5 P’s of acute compression syndrome

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10
Q

What are the advantages of an external fixator?

A

Facilitate the alignment of fracture fragments, permits early mobilization, permits wound care, permits ROM to unaffected joints.
Open ones allow for direct visualization of the fracture

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11
Q

Disadvantages of an external fixator?

A

Potential for infection at pin site is high. Increases incidence of osteomyelitis. Altered body image.

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12
Q

What are factors that inhibit fracture healing?

A

Infection, malignancy, bone disease, age, corticosteroids, NSAID’s, smoking, diabetes

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13
Q

Complications of amputations?

A

Phantom limb pain, hemorrhage, infection, flexion contractures, skin breakdown

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14
Q

s/s of FES, fat embolism syndrome?

A

Hypoexmia, usually seen first. Neurologic compromise, usually develops after respiratory s/s. Petechial rash, develops after respiratory and neurologic s/s. Fever, fat in the urine.

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15
Q

Nursing care for FES?

A

Steroids, methylprednisone. Position every 2 hours. Pain management. Monitor respiratory, cardio, and neuro. Fracture immobilization. Care manipulation. Strict I and O, pt may need . Frequent vitals.

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16
Q

Autoimmune, connective tissue disease that is characterized by periods of remissions and exacerbations. Pathophysiology?

A

Rheumatoid arthritis. Autoimmune reaction begins in synovial tissue. Results in breakdown of collagen. Edema, proliferation of synovial membrane, panes formation (destroys cartilage, erodes bone), loss of articular surface and motion, muscle weakness.

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17
Q

Medical management of ACS?

A

Contact the surgeon immediately, it’s a surgical emergency. Open the splint. Bivalve the cast. Prepare for surgery. Keep the extremity at heart level, not above. Fasciotomy.

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18
Q

S/s of RA, rheumatoid arthritis?

A

Symmetric join pain, early morning stiffness, joint swelling/warmth, erythema, loss of function, spongy joints, ulnar deviation (swan-neck), rheumatoid nodules, fever, weight loss, fatigue, anemia, enlarged lymph nodes

19
Q

Diagnosis of RA?

A

History and physical exam. Labs, increased ESR and C-reactive protein. Mild leukocytosis (rheumatoid factor, anti-nuclear antibody, ANA). X-rays. Aspiration of joint.

20
Q

Non-inflammatory, degenerative disorder of the joints. Primary and secondary?

A

Osteoarthritis, degeneratie joint disease. Primary is related to advanced age, while secondary more about damage to cartilage including trauma or long-term mechanical stressors.

21
Q

Risk factors for osteoarthritis?

A

Age, female, obesity, athletes, previous injury, muscle weakness, genetics

22
Q

s/s of OA?

A

Pain, joint stiffness, decreased function, enlarged joint, decreased ROM, edema, muscle atrophy, crepitus

23
Q

Treatment for OA?

A

Salicylates, NSAIDs, acetaminophen, intra-articular injections, opined analgesics, glucosamine and chondroitin, rest, weight loss, ambulatory devices, braces, splints, physical therapy
Surgical is total knee replacement (TNR), total hip replacement (THR)

24
Q

Nursing care of TNR after surgery?

A

Compression bandage, wound suction device, no hyper-flexion of the knee, physical therapy, ROM exercises, patient-controlled analgesia, Ice, SCDs/TED hose, neurovascular check, continuous passive motion

25
Q

How to avoid hip dislocation after a THR surgery?

A

Prevent hip adduction, avoid flexing the hip more than 90 degrees, don’t turn the affected leg inward, elevated toilet seat, no crossing of legs

26
Q

s/s of hip dislocation?

A

Increased pain at the surgical site, swelling, immobilization, acute groin pain at the affected hip, report of hearing a popping sensation, internal/external rotation affected, shortening of the affected extremity, restricted ability to move

27
Q

What are the types of hearing loss?

A

Sensorineural hearing loss

Conductive hearing loss, something physically blocking transmission of sound waves

28
Q

What is the safest way to remove ear wax?

A

Through gentle irrigation of the ear with warm water. Avoid inserting objects into the ear canal.

29
Q

Occurs due to defect in the cochlea, the eighth cranial nerve or brain. Often permanent hearing loss.

A

Sensorineural hearing loss, often permanent. From constant exposure to loud noises. Ringing in the ears. Ototoxic meds, meniere’s disease, diabetes mellitus

30
Q

Accumulation of med within inner ear that leads to cellular damage, impairs hearing, and impairs balance.

A

Ototoxicity. If pt is on an ototoxic med and they report tinnitus, hold the drug. Notify HCP.

31
Q

Risk of ototoxicity is high in?

A

High trough levels of the drug, impaired renal function, the elderly, prolonged use of the meds

32
Q

Ototoxic meds?

A

Antibiotics, diuretics, NSAIDs, antineoplastic meds

33
Q

Disorder of the vestibular system that can lead to a disturbed sense of balance. Clinical manifestations?

A

Vertigo. Nausea, vomiting, feeling of falling, hearing loss, tinnitus.

34
Q

Treatment of vertigo?

A

Avoid sudden head movement, hydration, uncluttered environment, assistive devices to ambulate prn, avoid driving during episodes

35
Q

Vertigo meds?

A

Dimenhydinate (dramamine)
diazepam (valium)
meclizine (antivert)

36
Q

Etiology of tinnitus?

A

Presbycusis (aging), otosclerosis, meniere’s disease, drugs, exposure to loud noise, inner ear problems

37
Q

Treatment of tinnitus?

A

Identify and eliminate the cause. If no cause can be found mask the sounds. Referral to support groups.

38
Q

Progressive and debilitating, periods of remission and exacerbations. Cause is unknown.

A

Meniere’s disease.

Associated with infections, allergic reactions, fluid imbalances, stress, ages 20-50, affects white men more

39
Q

Clinical manifestations of meniere’s disease?

A

Severe, intense, debilitating vertigo. Tinnitus. Fluctuating sensorineural hearing loss.

40
Q

How does meniere’s disease work?

A

Backed-up fluid leads to swelling and pressure in the endolymphatic scale. Distorts balance and sound information in the canals. Distorted information travels to the brain.

41
Q

Treatment for meniere’s disease?

A

Goal is to preserve hearing. Low sodium diet, spreads food out during the day, avoid caffeine and alcohol, no MSG. Move head slowly. Quitting smoking.

42
Q

Medication for meniere’s disease?

Surgical treatment?

A

Diuretics, antihistamines, antiemetic, diazepam

Endolymphatic decompression with drainage and shunt.

43
Q

Care of a patient after ear surgery?

A

Safety, avoid increasing pressure in the ear. Keep the ear dry. Avoid moving head too rapidly, report excessive draining, may experience some changes in hearing related to packing and swelling