Musculoskeletal Flashcards
What are the risk factors for osteoporosis?
Genetics, thin white and asian women. Age, postmenopausal women as they lose estrogen. Nutrition. Physical exercise. Lifestyle choices. Meds, esp steroids. Comorbidity.
What test is used to diagnose osteoporosis?
Dual x-ray absorptiometry (DEXA) scan
S/s of osteoporosis?
Bone pain or tenderness, fractures with little or no trauma, loss of height over time, neck or lower back pain due to fractures, stooped posture
s/s of osteomyelitis?
Fever, chills, tachycardia, malaise, pain, edema, erythema, ulceration, drainage
Diagnosis of osteomyelitis?
Radiologic confirmation, bone scans, leukocytosis, elevated erythrocyte sedimentation rate (ESR), wound/blood cultures\
Management of osteomyelitis?
Prevention, pain control, antibiotics, supportive measures, hyperbaric treatment, surgical debridement, wound care, glycemic control
A fracture that occursthrought an area of disease bone; can occur without trauma or fall.
A fracture that results from repeated loading of bone and muscle
Pathologic fracture
Stress fracture
s/s of osteomyelitis? Diagnosis?
Pain, loss of function, deformity, shortening, crepitus, localized edema and ecchymosis.
Diagnosis has no special lab tests, but confirmation via radiology.
5 P’s of a neurovascular check? Same as 5 P’s of what?
Pain, pallor, pulselessness, paresthesia, paralysis
Same as the 5 P’s of acute compression syndrome
What are the advantages of an external fixator?
Facilitate the alignment of fracture fragments, permits early mobilization, permits wound care, permits ROM to unaffected joints.
Open ones allow for direct visualization of the fracture
Disadvantages of an external fixator?
Potential for infection at pin site is high. Increases incidence of osteomyelitis. Altered body image.
What are factors that inhibit fracture healing?
Infection, malignancy, bone disease, age, corticosteroids, NSAID’s, smoking, diabetes
Complications of amputations?
Phantom limb pain, hemorrhage, infection, flexion contractures, skin breakdown
s/s of FES, fat embolism syndrome?
Hypoexmia, usually seen first. Neurologic compromise, usually develops after respiratory s/s. Petechial rash, develops after respiratory and neurologic s/s. Fever, fat in the urine.
Nursing care for FES?
Steroids, methylprednisone. Position every 2 hours. Pain management. Monitor respiratory, cardio, and neuro. Fracture immobilization. Care manipulation. Strict I and O, pt may need . Frequent vitals.
Autoimmune, connective tissue disease that is characterized by periods of remissions and exacerbations. Pathophysiology?
Rheumatoid arthritis. Autoimmune reaction begins in synovial tissue. Results in breakdown of collagen. Edema, proliferation of synovial membrane, panes formation (destroys cartilage, erodes bone), loss of articular surface and motion, muscle weakness.
Medical management of ACS?
Contact the surgeon immediately, it’s a surgical emergency. Open the splint. Bivalve the cast. Prepare for surgery. Keep the extremity at heart level, not above. Fasciotomy.