multi d final Flashcards
TIA (transient ischemic attack)
less than 24 hours
strong indicator of pending CVA (15% in 90 days)
how to manage TIA
observation
treatment of risk factors
anticoagulation
carotid endarterectomy
CVA
4th leading cause of death in US
sudden onset of neuro deficits within 24 hours
risk factors of CVA
older age
african american or hispanic
male
HTN
CAD
hyperlipidemia
elevated LDL levels
hyper-coagulable state
DMII
obesity
tobacco use
alcohol abuse
sedentary lifestyle
prevalence of strokes
12% of deaths globally
lifetime stroke risk is 25% for those >25 years of age
hemorrhagic stroke
blood leaks into brain tissue
ischemic stroke
clot stops blood supply to an area of brain
present with predetermined syndromes
can predict what vasculature will be affected
ICH (intracerebral hemorrhage)
10-15% of all strokes
from rupture of cerebral vessels
result of high BP
primary symptoms of ICH
spontaneous rupture of small vessels damaged by chronic hypertension
secondary symptoms of ICH
bleeding of cerebrovascular abnormalities, tumors, or impaired coagulation
ICH outcomes
associated with higher risk of fatality
damages brain cells
may increase pressure on brain or spasms in vessels
death prevalence in ICH
nearly half of all pts with primary ICH die within the first month after the acute event
lacunar stroke
25% of ischemic strokes
creates deep cavities in brain tissue
occlusion of vessels from the circle of willis
middle cerebral artery infarction presentation
contralateral hemiparesis
facial paralysis
sensory loss in face and UE
general info about MCAI
90% of all strokes
largest of the brain’s arteries
supplies most of outer frontal, parietal and temporal lobes
MCA syndrome
presents with neglect and poor motivation
MCA syndrome - Gerstmann syndrome
L/R disorientation
acalculia
agraphia
finger agnosia
anterior cerebral artery infarction
involves medial cerebral cortex
compromises motor and sensory of LE
ACAI left sided lesions
transcortical motor aphasia
ACAI right sided lesions
confusional state and motor hemineglect
superficial posterior cerebral artery infarction
visual and somatosensory deficits
larger PCAI
hemisensory loss
hemiparesis due to involvement of thalamus
where does the PCA supply?
occipital lobe
inferior temporal lobe
thalamus
vertebrobasilar infarction (VBI) presentation
ataxia
vertigo
headache
vomiting
oropharyngeal dysfunction
visual-field deficits
abnormal oculomotor findings
VBI - mid basilar artery
locked in syndrome
ipsilateral CN7 palsy
decreases level of consciousness
hemiparesis and hemitaxia
oculomotor deficits
arm posturing
VBI - intracranial vertebral artery
wallenburg syndrome
dizziness
diploplia
VBI - distal basilar artery
comatose state
defective vertical gaze
hemiparesis and hemitaxia
diplolia
speech difficulties
altered mental state
small, poorly reactive pupils
weakness
ataxia
VBI - origin of extracranial vertebral artery
dizziness
blurred vision
ataxia
vomiting
cerebellar infarction
loss of coordination
ataxia
nausea
vomiting
headache
dysarthria
vertigo
BEFAST stroke signs
balance
eyes
face
arms
speech
time
beyond BEFAST
numbness
sudden confusion
trouble seeing
trouble walking
severe headache