mTBI Flashcards
Pathophysiology of mTBI
whats the pathophysiology for a mTBI
BASICALLY - neurometabolic cascade is the thing that is messed up which disrupts the metabolic pathway that are required for good communication between neruons
worse cerebral profusion (less blood flow to brain) couple weeks after mTBI, along side ionic influx (more permeability of neruons), increased flow of CA2+, pumps increase, pumps need to be going bc ions are a mess, but we cant bc we need more energy
Prognosis for mTBI
-20% of Individuals experience symptoms longer than 1 month
-Children return to school after 2-4 days
-Adults return to work after 1-2 weeks
-Gender, history of concussion, intracranial abnormalities, etc. may affect prognosis
-Symptom burden may be predictive of recovery
-Preinjury mental health probs and post injury psychological distress (symptoms of depression and anxiety) are robust predictors of prolonged recovery
Goals of early support mTBI
-Education: what to expect, now to manage symptoms
-Return to activity guidelines: rest 24-48 hrs (strict sensory deprivation not required), then gradual symptom guided resumption of cog. and physical activities (note our change in understanding in recent yrs)
mTBI role of the SLP
-Persistent post-concussive issues
Require multidisciplinary team
Must consider pre- morbid factors
Should look at accommodations and identification of underlying issues
-Treatment from SLP perspective: guided by particular strengths and weaknesses, be sure assessments sensitive to mTBI
mTBI military specific info
•375,230 US service members sustained TBI from 2000-2016
•~75% mild (may be higher)
•Males twice as likely to sustain TBI
•Etiologies in combat theater
•Explosive devices (blast)- estimates of up to 80% of mTBI in combat zones
•Origin of the term “shell shock”
•Head impact
•Penetrating ballistic injuries
Blast injuries 4 levels
-Primary injury: direct impact from over-pressure wavel compress air filled organs, catapults body backwards
-Secondary injury: energized debris or explosive fragments impacts head/body
-Tertiary injury: body impacts wall, ground, or object
-Quarternary injury: inhalation of toxic gasses/substances
Blast injury co-morbid PTSD
intrusive thoughts
avoiding reminders
negative thoughts and feelings
arousal and reactive symptoms
Executive function deficits predictive of poorer outcome
Combined psychological interventions (CBT) PLUS cognitive rehab has shown reduction in symptoms
is it best practice to get a CT for concussion
no, not just for suspected concussion bc its radiation exposure
- need to know the why of a CT and resources
when should a head CT be considered for ages 5-18
vomiting
loss of consciousness
severe headache
dangerous MOI
GCS <15
agitation somnolence
slow response
repetitive questions
when should a head CT be indicated for ages 5-18
GCS <15
agitation somnolence
slow response
repetitive questions
when should a head CT be considered for ages 16-64
amnesia before impact > 30 or equal to
dangerous MOI
immediate referral for CT
- seizures
- double vision
- slurred speech
- worsening headache
- vomiting
- motor or sensory deficit
3 steps for clinical presentation and diagnosis for mTBI
1 establish plausible injury mechanism
2 query signs and symptoms
3 rule out confounding factors
what are premorbid factors that may impact mTBI recovery
history of concussion
other diagnosis (learning disabilities’)
medication (blood thinners)
depression
anxiety
diet
sleep
considerations for mTBI military population
return to duty
high comorbidity rates of mTBI, PTSD, and substance abuse