MT I Flashcards

1
Q

Definition of a lame horse:

A

Structural or functional disorder in one or more limbs and related structures.

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2
Q

Anatomical landmarks/location of lameness

A
  • Hoof, navicular bone
  • Tendons, ligaments
  • Tendon sheat, bursae
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3
Q

Explain the reciprocal apparatus

A

Stifle is always moving together with the hock. So if you flex the stifle the hock will be flexed as well, as the fetlock & the digits work together.

Peroneus Tertius/fibularis tertius on the front attached from the extensor fossa of femur and insert in 3rd tarsal bone

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4
Q

What are the Phases of the strides

A

1. Supporting phase
- Landing
- Loading
- Stance
1. Breakover Phase
- Heel lift
- Toe pivot
1. Swinging phase

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5
Q

Which phase of the stride is this?

A

Break over phase
* Extension: Fetlock
* Hyperextension: DIPJ (Distal interphalangeal joint)

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6
Q

Which phase of the stride is this?

A

Stance phase
Hyperextension: Fetlock
Flexion: DIPJ

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7
Q

Which phase of the stride is this?

A

Breake over phase

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8
Q

Which phase of the stride is this?

A

**Stance phase
**

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9
Q

Which phase of the stride is this?

A

Swinging phase
- Flexion (Caudal)
- Extension (Cranial)

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10
Q

What is shown on the picture

A

Abnormal Joint Hyperextension

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11
Q

What is shown on the picture

A

Constant DIPJ Hyperextesion

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12
Q

Explain the different Interference forms at the trot

A

A. Front limb to front limb
B. Ipsilateral front to hind
C. Pacer (Diagonal limbs)
D. Ipsilateral hind to front

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13
Q

Explain the different feet movement from a skyline view

A

Normal – padding – winging – plaiting

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14
Q

What could be potential causes for Lameness

A
  • Trauma
  • Congenital (Ex. Navicular disease)
  • Acquired
  • Infection
  • Metabolic disturbances
  • Circulatory disorders (Aortoiliac Thrombosis)
  • Nervous system
  • Pain
  • Mechanical
  • Paralytic disorders
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15
Q

Lameness due to infection
* If foal is lame (No injury) Nr 1 reason is?
* Treatment?

A

* Nr 1 reason is –> Joint Infection
* * Hematogen origin, swollen joint, fetlock – stifle. Effusion of infected site, think first of all joint.
* If infection do not give steroids but AB intraarticular.

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16
Q

Lameness due to metabolic disorders
* Example
* What to do?

A
  • Laminitis
  • Endotoxemia

What to do with this:
* Make **abaxial perineural block **
* Check pulsation – if laminitis hard pulsation

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17
Q

Lameness due to circulatory disorders
- Example
- Signs?
- How to Diagnose?

A

* Aortoiliac Thrombosis

  • Beginning light lameness on left – sweat and colic symptoms.
  • Worsening during training, shows great pain
  • Saphenous vein (Dors.med. aspect of heart) Less arterial blood supply, less backflow so the veins are empty.
  • **One leg is warm, while the other is cold **

Diagnose with Ultrasonography.

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18
Q

Lameness due to the nervous system
* Example
*

A
  • Sweeney:
  • Suprascapular Neuropathy. When horse walk to you, horse flexing shoulder against abaxial direction.
    * Atrophy of supraspinatus and infraspinatus muscle (Colateral ligament)
  • Can have a hit from anything and damage to the peripheral nerve but able to regenerate itself.
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19
Q

What are the different degree of lameness

A

**1 – 5 **or 1 – 10 (Europe) 0 - 5 (AAEP)

* Grade I: Very mild
* Grade II: Mild
* Grade III: Moderate
* Grade IV: Severe
* Grade V: Non – Weight bearing

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20
Q

How to distinguish the Character of the lameness

A
  1. Unchanging (Warming up)
  2. Changing =
    * Improving during training/examination (must probably be Chronic Osteoarthritis)
    * Intermittent (Few steps, very lame and walk again)
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21
Q

How to classify the lamenes

A

Classification of lameness
* Supporting limb lameness
* Swinging limb lameness
* Compensatory lameness
* Untypical lameness
* Special lameness

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22
Q

How to recognize supporting limb lameness
- Phase of the stide
- Head and neck movement
- Where to localize the pain
- What happens to the fetlock?
- What happens to the cranial phase of the stride in relation to supporting or swinging phase lameness?

A

Cranial phase is longer
* More pain during contraction
* Will quickly shift weight to the other side.

Head and neck movement:
* Head and neck **elevation on the affected side.*

The problem is usually lower

Worse in inside circle

IMPORTAINT:
Lameness in when viewing from front:
1. Head and neck elevated
2. Hyperextension in fetlock during stans
3. **Cranial phase of the stride **
* Longer when supporting lameness
* Shorter when swinging phase lameness

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23
Q

How to recognize swinging limb lameness

A
  • Cranial phase is shortened
  • It is evident during motion
  • Usually the problem is higher
  • Worse in outside circle
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24
Q

How to recognize compensatory lameness?

A
  • Uneven distribution of weight on another limb
  • Lame FL –> Other FL
  • Navicular diesase –> Sole bruise
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25
Q

How to recognize Untypical lameness

A
  • When more than one limb is effected
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26
Q

How to recognize special lameness
Example
Diagnosis
Picture

A
  • Ex:** rupture of peroneus tertius**
  • Upward fixation of the patella
    * DDFT rupture –> Take sonography
    *** Chronic navicularis syndrom **

Picture: Achilles tendon to loose –> Stifle is in full extension : The whole reciprocal apparatus is destroyed. Fibularis tertius and peroneus tertius ruptured. Yearling – Preform x – ray from stifle

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27
Q

Patella fixation
* Type of abnormality
* What to do surgically
* How to lock the patella
* How to improve without surgery

A
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28
Q

Fibrotic Myopathy

A
  • Last movement of swinging phase –> Dropping the leg back again, due to semimembranosus and semitendinosus myopathy
  • Can see a groove, do not forget to palpate.
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29
Q

Steps of the lameness examination

A
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30
Q

Anamnesis in lameness examination

A
  • How long has the horse been lame?
  • What is the cause of the lameness?
  • How did it happen?
  • Has been rested or exercised?
  • Previous treatment, what was the result?
  • When was the horse shod?
    Can get aseptic inflammation around the chorion –> test the nails
    Hoof abcess.
  • Is he still in work?
  • Does he warm out of the lameness?
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31
Q

What can be seen

A

Osteoarthrithis in distal Interphalangeal joint.

  • Swelling around coronary bone = ring bone
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32
Q

What can be seen

A

Ancleosis: Joint fuse and become one structure.

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33
Q

What can be seen

A

Bone sparring on hock

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34
Q

LAMENESS
Visual examination
At a distance and close examination

A

Distance:
- Conformation
- Body condition
- Positure
- Atrophy, asymmetry

Close:
- Hoof
- Swelling, distension

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35
Q

LAMENESS
Visual examination
What are you able to preform in the practice

A
  • Anamnesis
  • Visual examination
  • Palpation
  • Provocation test
  • Diagnostic anaesthesia
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36
Q

Visual examination

A
  1. At a distance (All directions)
  2. At rest
  3. At exercise
  4. What are you able to preform in the practice
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37
Q

Lameness
Supplementary diagnostic aids

A
  • RTG
  • Arthroscopy
  • MRI
  • UltraSound
  • Synovia analysis
  • CT
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38
Q

What is shown on the picture

A

Provocating tests
(flexion)

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39
Q

What is the purpose of diagnostic analgesia

A
  • Find site of pain causing lameness
  • Confirm suspected site of pain
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40
Q

Diagnostic analgesia
What is the reason of failure?

A
  • Bad technique
  • Variation of peripheral nerve anatomy
  • Local anaesthetic diffuses proximally
  • 70 – 80% positivity is a positive result
  • Deep bony pain is difficult to anaesthetize
  • You cannot block out all intraarticular pain.
  • Mechanical lameness.
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41
Q

Diagnostic analgesia
What local anaesthetics should we use?

A

Less irritant:
* Mepivacaine,
* Prilocaine,
* Bupivacaine

More irritant:
* Lidocaine

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42
Q

Diagnostic analgesia
Effect of duration

A

Fast acting (2h DOA)
* Mepivacaine
* Prilocaine

Slower acting (4h DOA)
* Bupivacaine

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43
Q

Diagnostic analgesia and restrain

A
  • Depends on horse
  • Minimal restraint is less stressful
  • Good handler is essential
  • Position yourself safely

Physical:
o Twitch

Sedation
o Small doses
o Xyalzine (0.1 – 0.2 mg/BWT)
o Detomidine (0.005mg/Bwt) –> Safer, affect result in higher doses.

Leg position (On ground, held by clinician, held by assistant)

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44
Q

Diagnostic analgesia
Patient preparation
- Perineural analgesia

A

o Clean procedure (Clip if hairy)
o** Antiseptic scrub** until clean
(Povidone iodine, chlorhexidine)
o Alcohol with swab then spray.

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45
Q

Diagnostic analgesia
Patient preparation
- Intrasynovial analgesia

A

o Aseptic procedure

o Clip

o 5 minute antiseptic scrub

o Alcohol wash

o Sterile gloves

o Fresh bottle anaesthetic

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46
Q

Diagnostic analgesic procedure
1. Size needle
2. Quantity of analgesia
3. Time for evaluation
4. Intra synovial analgesia
5. Post block

A

1.** 18 – 25 gauge needles** (Use fine needles when possible) –> Length depends on size
2. Quantity of anaesthetic depends on site
3. Evaluate
o 5–10minutes
o 20 – 30 minutes
o 60 minutes

4. Intrasynovial analgesia
o Usually acts more quickly
o** Shorter duration**

** 5. Post block
**o Sterile wrap (5 . 10 min)

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47
Q

Diagnostic analgesia
– fore limb strategy

A

NO clinical suspicion as to site of pain
- Block from** distal to proximal**
- Use regional blocks
- Differentiate with intrasynovial blocks later if necessary.

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48
Q

Which Perineural analgesia blocks can be done on the – FORELIMB

A
  • Deep digital nerve block (TPA 1)
  • (Prox palm digital block (TPA2)
  • Abax.sesamoid bloc (MPA)
  • Four point block
  • High palmar block
  • (Subcarpal block)
  • Lat. Palmar block
  • Ulnar block
  • N. Medianus et N.musculocutaneus block.
49
Q

Perineural analgesia blocks – FORELIMB
Deep digital nerve anaesthesia
1. Size needle
2. Quantity
3. Anatomical landmark
4. Structure anaesthetised

A
  • 23G 1.5 needle
  • 1.5 ml anaesthetic
  • At level of collateral cartilage

Structures anaesthetised:
o Palmar foot
o Toe
o DIP joint
o +/- distal DDFT lesions
o Part of P2 and joint

50
Q

Perineural analgesia blocks – FORELIMB
Positive low or prox palm digit analgesia
Differentiate structures with:

A
  • DIP
  • Navicular bursa block
  • PIP block
51
Q

Perineural analgesia blocks – FORELIMB
3. Abaxial Sesamoid Block
1. Anatomical landmark
2. Size of needle
3. Quantity
4. Used in case of
5. Structures anaesthetised

Anatomical land mark 2.
Size of needle
Quantity

A
  1. At level of prox. Sesamoid bone:
  2. **22–23G ** —> 1.5cm needle
  3. 3ml local anaesthetic
  4. Use this block if laminitis. Nervovascular bundle surrounded by tubular facsia –> Do not have a stable type anaesthesi. Set the block parallel to the nerve not straight in it.
  5. Structures anaesthetised
    * As for PDB (Proximal pastern)
    * Sometimes include –> Fetlock joint (MCP) & Sesamoid bone locally.
    * Block** distal DDFT lesions**

At level of distal aspect of prox sesamoid bone
* **26G **–> 1.5 cm needle
* 1.5ml of local anaesthetics.

52
Q

Interarticular analgesia blocks – FORELIMB
4. Distal interphalangeal joint analgesia
Procedure
Size needle
Quantity
Anaesthetised structures

A
  • Aseptic procedure
  • 19 G - 3cm needle
  • 6ml anaesthetic
  • DIP joint, dorsal sole (toe)
    -** 10 ml anaestetics –> Block heel as well**
53
Q

Interarticular analgesia blocks – FORELIMB
5. Navicular bursa analgesia
Procedure
Size needle
Quantity
Anaesthetised structures

A
  • Aseptic procedure
  • Radiographic control
  • 19 G 7cm needle
  • 3.5ml anaesthetic
  • 0.5ml iohexol contrast solution
  • Structures anaesthetised
    o Navicular bursa
    o Dorsal sole (toe)
    o Navicular bone
    o Navicular ligaments (Not heel)
    o 30 minutes: DIP joint
54
Q

Perineural analgesia – FORELIMB
6. Proximal interphalangeal joint (PIPJ)

A

Doral approach
- 21 G 2.5cm needle
- 5ml anaesthetic

Palmar approach
- 21 G 2.5cm needle
- 5ml anaesthetics

Structures anaesthetised
- Pip joint

55
Q

Perineural analgesia – FORELIMB
7. 4 point block (N. digit.palm.,nn. Metacarpales)

A
  • 23 G 1.5 cm needle
  • 1.5 – 3ml anaesthetic
  • Palmar nerves (lat.med)
  • Palmar metacarpal nerves
56
Q

Perineural analgesia – FORELIMB
8. Positive 4 point block

A

Differentiate structures with:
o MCP analgesia
o Digital sheath analgesia

57
Q

Perineural analgesia – FORELIMB
9. Metacarpophalangeal joint anesthesia

A

Structures anaesthetised
- MCP joint
- Subchondral bone pain –> slow to respond
- 30min may anaesthetize;
- Dist. Suspensory branches + sesamoids
- Dorsal and palmar approaches
o 19–22G 3-4cm needle
o 10ml anaesthetic

58
Q

Perineural analgesia – FORELIMB
9. Metacarpophalangeal joint anesthesia
Dorsal approach

A

o Easier
o Articular cartilage easily traumatised

59
Q

Perineural analgesia – FORELIMB
9. Metacarpophalangeal joint anesthesia
Palmar approach

A
  • Between suspensory ligament and MC3
  • Sometimes difficult to be sure of centesis
60
Q

Perineural analgesia – FORELIMB
10. Digital flexor tendon sheath (DFTS) analgesia

A

Structures anaesthetizes
- Digital sheath
- Local structures with time
- Annular ligament
- Often only a partial improvement
- Proximal and Palmar approach

61
Q

Perineural analgesia – FORELIMB
10. Digital flexor tendon sheath (DFTS) analgesia
Proximal approach

A
  • 20G 2.5cm needle
  • 10 – 15ml anaesthetic
62
Q

Perineural analgesia – FORELIMB
10. Digital flexor tendon sheath (DFTS) analgesia
Distal Palmar approach

A
  • 20G 2.5cm needle
  • 10 – 15ml anaesthetic
63
Q

Perineural analgesia – FORELIMB
11. High palmar block

A
  • 20G 3cm needle
  • 5ml in each side
  • Palmar nerves
64
Q

Perineural analgesia – FORELIMB
12. Subcarpal block

A
  • Supf + DDF tendon anesth.
  • Structures anaesthetised
    o Whole metacarpal region
    o 65% chance of penetrating carpometacarpal (CMC) joint
    o Dorsal branches must be anaesthetised
    to block the skin dorsally
65
Q

Perineural analgesia – FORELIMB
13. Lateral palmar analgesia

A
  • Spesific for suspensory ligament lesions
  • 22G 1.5cm needle
  • 5ml anaesthetic
  • less chance of blocking the CMC joint
66
Q

Perineural analgesia – FORELIMB
14. Suspensory ligament origin infiltration

A
  • Spesific for suspensory ligament lesions
  • 19G 5cm needle
  • 10ml anaesthetics
  • From lateral
  • Include palmar metacarpal nerves
67
Q

Perineural analgesia – FORELIMB
**15. Postive subcarpal analgesia
**

A
  • Perform middle carpal joint analgesia
  • Can diffuse around palmar nerves
    And block metacarpal structures
68
Q

Perineural analgesia – FORELIMB
16. Middle carpal and antebrachiocarpal analgesia

A
  • Dorsal pouches
  • Medial or lateral
  • 19G 3cm needle
  • 5ml anaesthetic
  • Check in 5 – 20 minutes
  • Remember: Subcarpal analgesia and middle carpal joint analgesia may block the same structures
  • Clinical findings may help to differentiate
69
Q

Perineural analgesia – FORELIMB
17. N. Ulnaris analgesia

A
  • 18G 4cm needle
  • 10 – 15ml anaesthetic
  • 10cm prox from accessory carpal bone
70
Q

Perineural analgesia – FORELIMB
18. N. Medianus analgesia

A
  • 5cm below elbow joint, medial side
  • Caudomedial surface of radius
  • Just cranial from m.flex. carpi radialis
  • 10ml local anaesthetic
  • Arteria and vein located caudally form it
  • False positive response because of elbow joint
71
Q

Perineural analgesia – FORELIMB
N. Musculocutaneous anaesthesia

A
  • Branch for skin
  • Seldom necessary
  • 4x3ml
    o V. Cephalica cran.caud .
    o V. Cephalica. Access.cran. caud
72
Q

Perineural analgesia – FORELIMB
20. Elbow anaesthesia

A
  • Cranial + caudal pouch
  • 19G 9cm needle
  • 25ml anaesthetic
  • NB radial nerve

Cran approach: Signs of radial paresis
Method: Infront of collateral ligament 2/3 distance between humerus epicondyle and tub. Radii in cranial direction

Caud approach: Method: Infront of olecranon caud form epicondyle long needle, may need skin local anaesthesia

73
Q

Perineural analgesia – FORELIMB
21. Shoulder joint anaesthesia

A
  • 19G 9cm needle
  • 25ml anaesthetic
  • Wait 30 minutes
  • Inbetween tub majus pars cran et caud humeri
  • Infront of infraspinatus insertion
74
Q

Perineural analgesia – FORELIMB
22. Bicipital bursa anaesthesia

A
  • 19G 9cm needle
  • 20ml local anaesthetic
  • Puncture under ultrasonographic control adviced
  • 4cm distal and 6-7 cm caudal from tub.majus pars cranialis humeri
75
Q

Diagnostic analgesia – HINDLIMB
Indication and Procedure

A
  • Fractious horses
  • Low doses of detomidine and butorphanol don’t seem to affect lameness.
  • Aspesis for intra – articular blocks
  • Bind tail out of the way
76
Q

Diagnostic analgesia – HINDLIMB
Strategy

A
  • No obvious site of lameness (Sequential block from distal)
  • Conformation of clinical suspicions (Specific blocks)
77
Q

Nerve blocks of the hindlimb

A
  1. Plantar digital block
  2. Pastern ring block
  3. Low plantar six point
  4. Subtarsal (Deep branch of lateral plantar nerve, LPN)
  5. Intra – articular Tarsometatarsal and Centrodistal
  6. Intra – articular Tarsocrural joint
  7. Tarsal sheath
  8. Tibial and Peroneal
  9. Intra – articular Femoropatellar, medial and lateral Femorotibial
  10. Intra – articular coxofemoral
  11. Sacroilliac joint
78
Q

Nerve blocks hindlimb
1. Plantar digital block

A
  • Same as in forelimb
79
Q

Nerve blocks hindlimb
2. Pastern ring block

A
  • Include dorsal branches
  • And dorsal metatarsal nerves
  • Blocks pastern and foot
80
Q

Nerve blocks hindlimb
3. Low plantar six point

A
  • Plantar digital nerve
  • Plantar metatarsal nerve
    o Important for subchondral bone of distal MCIII
  • Dorsal metatarsal nerve
    o Deep peroneal nerve, N. Saphenous + supf peroneus nerve
  • Blocks all tissues distal to block
  • Positive (return later to block)
    o Fetlock
    o Digital sheath.
81
Q

Nerve blocks hindlimb
4. Subtarsal (high plantar)

A
  • Now less frequent used,
  • Instead Deep branch of lateral plantar nerve (LPN)
  • Plantar and plantar metatarsal
  • 5ml deep and supf 20G 4cm needle
  • Blocks suspensory ligament
  • Complete ring block to anaesthetise whole metatarsus
82
Q

Nerve blocks hindlimb
5. Intra – articular Tarsometatarsal and Centrodistal

A
  • Usually communicate
  • Do TMT first plantolateral
  • 21G,4cm needle
  • 5–10ml
  • Usually communicate
  • Do TMT first from plantarolateral
83
Q

Nerve blocks hindlimb
6. Intra – articular Tarsocrural joint

A
  • 19G 4cm needle
  • 10 – 15ml
  • Dorsomedial: Axial to saphenous vein
  • Communicates w/ prox. Intertarsal joint.
84
Q

Nerve blocks hindlimb
7. Tarsal sheath

A
  • 19G, 4cm needle
  • 15 – 20ml
    o Easier when distended
    o Plantar to TC joint capsule
85
Q

Nerve blocks hindlimb
8. Tibial and Peroneal

A
  • Superficial and deep peroneal
    • Hands breadth above point of hock
    • Between long and lat dig ext muscles
  • o 15ml 4cm deep
    o 5ml under skin
  • TIBIAL
    o 20ml 1cm deep
    o From medial side

TIBIAL and PERONEAL
o May have medial and plantar superficial sensation
o Should eliminate sensation in foot and suspensory ligament o Blocks all tarsal structures
o May not achieve 100% resolution of lameness

86
Q

Nerve blocks hindlimb
9. Intra – articular Femoropatellar, medial and lateral Femorotibial

A
  • 19G 6cm needle for all blocks
  • 20 – 35ml anaesthetic
  • Usually communicate –> Do all three together
  • 50% improvement is probably significant
  • May not block bone cysts, patellar and collateral ligament injuries.
87
Q

Nerve blocks hindlimb
**9. Intra – articular Femoropatellar, medial and lateral Femorotibial*

- Femoropatellar joint

A

o Either side of middle patellar ligament
o Difficult to retrive synovial fluid

88
Q

Nerve blocks hindlimb
**9. Intra – articular Femoropatellar, medial and lateral Femorotibial*

- Medial femorotibial

A

o Pouch cranial to medial collateral ligament o Fluid should be retrived

89
Q

Nerve blocks hindlimb
9. Intra – articular Femoropatellar, medial and lateral Femorotibial

Lateral femorotibial joint

A

o In extensor notch cranial or caudal to long digital extensor tendon

90
Q

Nerve blocks hindlimb
**9. Intra – articular Femoropatellar, medial and lateral Femorotibial*

A
91
Q

Nerve blocks hindlimb
10. Intra – articular coxofemoral

A
  • 19G 20 cm needle
  • 30ml anaestethic (Rarely preformed and difficult)
92
Q

Nerve blocks hindlimb
11. Sacroilliac joint

A
  • Be aware! Sciatic nerve ventral to the S/I joint
  • Cranial gluteal nerve caudal margin ileal wing
  • Medial approach preferred to avoid nerve
  • Cranial to tuber sacrale
  • Caudal to L6 dorsal spine
  • From contralaterally
  • Probably peri articular
  • Usually block bilaterally
  • 20 – 25cm needle
  • 30 ml anaesthetic
  • Can use ultrasound guidance
93
Q

DISEASES OF TENDONS LIGAMENTS, TENDON SHEATH AND BURSAE

Tendons vs Ligaments

A

Tendon
o Connects muscle to bone
o Its purpose is to move a joint

Ligament
o Connects bone to bone
o Stabilizing role

94
Q

TENDONS COMPOSITION

A

Energy storing - acts as a spring
o SDFT

Positional
o Digital extensor tendons

Somewhat different internal characteristics

95
Q

Characteristics of an energy storing tendon

A
  • Support the hyperextended metacropophalangeal joint during weight-bearing
  • Release energy when the limb is protracted
  • Horse bounces up and down on springs
96
Q

Blood supply to the tendon

A

Blood supply:
o From muscle at musculotendonous junction
o From the bone at insertion site
o From paratenon (in tendon sheath)
o From mesotendon (in tendon sheath)

97
Q

Mechanical properties of the Tendon
Stress-strain curve

A

o 1 - toe (elimination of crimp)
o 2 - linear phase
o 3 - yield point (beyond it irreversible damage)
o 4 - rupture

98
Q

Mechanical properties of the Tendon
- Normal strain in diff gates
- Ultimate tensile strength, where does it rupture?

A

Normal strain
o Walk 2-4%
o Trot 4-6%
o Gallop 16,6% has been recorded in vivo

SDFT ultimate tensile strength (rupturing in mid-metacarpal region)
o 12kn or 12 tons!

99
Q

Type of injury

A

Traumatic injury of DDFT

100
Q

Tendons
Occurence of injury
Traumatic injury

A
  • Traumatic: any type of horse
101
Q

Tendons
Occurence of injury
Strain-induced predilection sites
- Racing thoroughbred
- Elite show jumpers
- Elite eventers
- Dressage horses
- Injuries within tendon sheats

A

o Racing thoroughbred:
- SDFT in forelimb (mid-MC region)
o Elite-show jumpers:
- Forelimb SDFT and DDFT injuries
o Elite eventers:
- Forelimb SDFT injuries
o Dressage horses:
- Hindlimb suspensory ligament injuries

Injuries within tendon sheaths:
- very poor healing capacity, so poor prognosis!!

102
Q

Type of injury

A

SDFT injury

103
Q

Tendons
Traumatic injuries
DDFT
Prognosis

A

Complete rupture, loss of function
- Deep digital flexor tendon (DDFT)
o Toe flips up (see photo above) - always the sign!
o If in the metacarpal/metatarsal region and outside the DFTS - can heal satisfactorily if immobilized
o If ruptured near the insertion/within the DFTS -very poor to hopeless prognosis

104
Q

Tendons
Traumatic injuries
SDFT
Prognosis

A

Superficial digital flexor tendon (SDFT)
o **Reasonable prognosis if immobilized **
- Cast or Robert-Jones bandage
- Often transected together with DDFT

105
Q

Type of injury

A

Suspensory ligament

106
Q

Tendons
Traumatic injuries
Suspensory ligament
Prognosis

A

Suspensory ligament, often in racehorses
* Prox. sesamoid bone drops
* Fetlock drops
- Graveprognosis
- Degenerative/traumatic/catastrophic

107
Q

Tendons
Traumatic injuries
Other traumatic injuries

A
  • Overreach injuries
    o Pastern
    o (Metacarpalregion)
  • Other physical trauma
  • o From fence etc.

Boots/bandages won ́t prevent tendinitis/tendinopathy but can protect the limb from physical injuries

108
Q

NON-TRAUMATIC TENDON AND LIGAMENT INJURIES

A
  • Tendinitis/tendinopathy
  • Desmitis/desmopthy
109
Q

NON-TRAUMATIC TENDON AND LIGAMENT INJURIES
Pathogenesis

A
  • Overstain injuries (sudden overload)
    o Most ligament and some DDFT injuries
  • Degenerative process
  • Often bilateral
  • Often asymptomatic initially
  • Associated with age and exercise
110
Q

REPAIR OF TENDON AND LIGAMENT INJURIES

A

NO REGENERATION!!
- Intratendinous haemorrhage (immediately after injury!)
- Inflammatory reaction
o Designed to remove damaged tendon tissue
o But excessive and causes further damage (because of the cytokines are in such high amounts)
- Reparative phase
o Starts within a few days
o Angiogenesis in the tendons
o Scar tissue formation - higher ratio of collagen type III/I
- Remodeling phase (will never have the same tendon back)
o Gradual, incomplete replacement of collagen type III to type I

111
Q

Tendon and ligament injuries
PRINCIPLES OF DIAGNOSIS

A
  1. Clinical examination
    - Visible signs
    - Palpation (focal heat, pain)
  2. Gait assessment
  3. Diagnostic analgesia
  4. Ultrasonographic examination to look at the soft tissue
  5. (MRI, if it is not clear in ultrasound, or if it is in the hoof capsule)
112
Q

Site of injury

A

SDFT

113
Q

Superficial digital flexor tendon
Sites of injury
Occurence in?

A

Most common site:
- Mid-metacarpal region
o Common injury in racehorses and jumpers
- Within the DFTS and the carpal sheath (proximally)
o Less common
- In the pastern region
o Often traumatic, overreach

114
Q

What should you be aware off in case of SDFT injuries?

A

Beware of ultrasound too quickly
o Might not see the extent of the injury
o The hemorrhage can mask or cover the injury
o The injury can an grow a lot in the first weeks
o True extent of the injury may not be apparent
o Repeat ultrasound a week later

115
Q

Which injurie?

A

Injurie of SDFT due to over reach, injurie in the pastern region

116
Q

PRINCIPLES OF TREATMENT
SDFT injurie

A

Acute
Subacute
Chronic

117
Q

PRINCIPLES OF TREATMENT
SDFT injurie
ACUTE PHASE
Aim
Therapy
Medication
Surgery?

A
  • Minimize inflammation and limit the action of proteolytic enzymes
  • Physical therapy: rest, cold (cooling machine, gel) , immobilization (put in boxrest)
  • Systemic short-acting corticosteroids in first 24-48 hours
  • (NEVER into a tendon, can lead to mineralization in the tendon which is irreversible!!)
  • Never intralesional steroids: calcification!
  • NSAID ́s: some controversy
  • (Surgical treatment - tendon splitting, desmotomy of AL-SDFT)
118
Q

PRINCIPLES OF TREATMENT
SDFT injurie
Subacute (fibroplastic) phase
Aim
Montoring
Therapy

A
  • Progressive mobilization (important that they move, but not too much)
  • Ultrasonographic monitoring
  • Regenerative therapies
    o Mesenchymal stem cell therapy
    (Autologous blood, stem cells from sternum)
    –>Stem cells differentiate into tenocytes, regenerate matrix o PRP (protein rich plasma)
    –> Autologous blood (stem cells from sternum), centrifugation, gravity filtration
    –> Soup of growth factors - stimulate cell proliferation and matrix synthesis
119
Q

PRINCIPLES OF TREATMENT
SDFT injurie
Chronic (remodeling) phase

A
  • Not really much to do except controlled exercise
  • Regular ultrasonographic monitoring
  • To prevent re-injury
  • Takes time - at least 6-12 months before returning to full work (NO therapy can shorten this)