MSK - Lower Body Flashcards
Bones, muscles & stuff...
Stages of Healing
- Inflammation: immediately following injury to 3-5 days, signs of inflammation, POLICE
- Repair: day 5 to 4-6 weeks, essential to begin moving the injured joint through ROM, formation of scar tissue
- Remodelling: 6 weeks - 6 months, functonal actitivites, maturation of myofibers. organization of scar tissue
Contusion
- Muscle injury caused by sudden external force
- Results in bleeding in deep muscle regions
- Recovery is dependent on grade: grade 1 = 2-3 wks, grade 2 = 4-6 wks, grade 3 = 8 wks
- Treatment: POLICE, no heat, no massage, no alcohol, no running, put muscle on stretch to prevent healing in shortened range, gentle pain free ROM
- Complications: compartment syndrome (check capillary refill), myositis ossificans (formation of bone within muscles, suspect if haven’t improved in 2-3 weeks)
Strains and Tears
- Acute or chronic (excessive lengthening vs overuse)
- Majority occur at biarticular muscles at the muscle tendon junction
- Mainly occur during eccentric loading or high intensity
- Grade One: microscopic tearing, pain and tightness, no weakness, relative rest to protect
- Grade Two: partial, macroscopic tearing, pain and sturctural change (laxity and decreased strength)
- Grade Three: complete tear, painless and weak, may see lump
Strains and Tears: Treatment in Recovery Stages
- Acute: POLICE, crutches if LE
- Repair: modalities (US, IFC) DTF, strength and stretching
- Remodeling: strength (increase in velocity) + stretching (static and dynamic)
Laceration
- Cutting of muscle fibres, require surgical repair with sutures
- Acute phase: optimize gait so scar tissue can arrange properly
- Repair: gradual ROM and strength
- Promote remodelling by gradually increasing load and velocity
Mechanical Low Back Pain: MOI, S/S, testing, treatment approach, education, precautions
MOI: Uniatera pain with no referral below the knee, often centralized to back, may be caused by muscles (strains) or ligament sprains, facet joint or SIJ. May be caused by traumatic injury, postural pain, lumbar sprain/strain.
Risk factors: Increase when pregnant, obsessive, increased driving or sitting time, anxiety and/or depression, prior LBP
S/S: Refers into buttock and thighs, AM stiffness or pain is common, pain with initiation of movements, pain worse at the end of day
Testing: AP
Treatment approach: Exercise, postual control, manual therapy, soft tissue release, NO traction or TENS
Education: Lifting mechanics, stay physically active
Precautions: AP
Degenerative Disc Disease (Spinal Stenosis): MOI, S/S, testing, treatment approach, education, precautions
MOI: Narrowing of spinal canal. Caused by degeneration of vertebral disc or lumbar facet. Associated with aging. Age of onset in 60’s - often insidious.
S/S: Bilateral numbness and tingling, pain in bilateral legs, flexed position decreases pain (sitting and bending), extension increases pain (walking and standing), pain decreases as morning progresses, repetitive movements cause pain, chronic pain with acute episodes
Testing: Bicycle test, shopping cart sign
Treatment approach: lumbar flexion exercises, ergonomic advice, stabilization of core, traction, improve ROM, aerobic exercise, core stability, aquatic exercise
Education: easing positions, options for exercise that allow flexion (cycling)
Precautions: AP
Lumbar Spine Disc Herniation/Radiculopathy: MOI, S/S, testing, treatment approach, education, precautions
MOI: Nucleus pulposus is displaced and protrudes beyond the annulus fibrosis, most common direction is posterior-lateral
S/S: Pain worsens in weight bearing activities, pain with cough/sneeze, no pain in reclined position, can impinge of surrounding nerve roots (radic). Most common L4-L5 and L5-S1, pain can be localized to low back + with herniation can radiate into lower leg.
Testing: Lumbar spine scan (myotomes, dermatomes, reflexes), SLR (+ if pain from 35-75 degrees), slump test
Treatment approach: extension exercises, traction, core stabilization
Education: avoid flexion, posture
Precautions: avoid prolonged flexion
Greater Trochanter Hip Bursitis: MOI, S/S, testing, treatment approach, education, precautions
MOI: Inflammation of bursa on greater trochanter. Can come from falls, contact blows to lateral hip or repetitive contractions glute med and IT band (running, walking, over training). Infections (TB).
Risk factors: W > M, affects all images, obesity
S/S: pain with palpation of GT bursa, pain with abduction and/or ER, prolonged standing pain, may see Trendelenburg sign.
Testing: FABER, Obers, pain with resisted abduction.
Treatment approach: myofasical release of ITB/TFL, flute/ITB stretching, clamshells, leg lifts.
Education: Activity modification
Precautions: 3 different times (inflammation, hemorrhagic from blow, septic from infection)
Piriformis Syndrome: MOI, S/S, testing, treatment approach, education, precautions
MOI: Peripheral neuritis of the branches of sciatic nerve caused by abnormal piriformis. More common in women with larger Q angle.
Two types: primary (anatomical cause, piriformis lays over sciatic nerve and some have nerve come thru muscle), secondary (macro trauma of buttock, muscle spasm, swelling, nerve compression)
S/S: painful buttock or hip pain, referral sacrum, gluteal and down posterior thigh, irritated with long sitting, walking, squating and hip adduction. No + neuro signs.
Testing: TOP over piriformis, resisted lateral rotation
Treatment approach: gluteal stretching, core strengthening, education.
Education: avoid extended periods of sitting, take micro breaks, remove items in back pocket.
Precautions: Ensure referral pain is not due to spinal pathology.
Plantar Fascitis: MOI, S/S, testing, treatment approach, education, precautions
MOI: Overuse caused by medial calcaneal tuberosity, can come from over training/sudden increase in load
Risk factors: excessive or repetitive weight bearing work, increase in training, obesity, flat feet OR high arches, decreased DF, hyperpronation, leg length discrepancy, Windlass mechanism
S/S: Pain over plantar fascitis and calcaneal tuberosity, pain first thing in the morning (first few steps), antalgic gait, 30% patients present bilaterally, decreased DF
Testing: active and passive - full ROM, resisted isometrics, sensation & reflexes - WNL
Treatment approach: Activity modification, Strasberg sock, low dye taping, stretching & strengthening (increase calf length and stretch, intrinisic foot muscle), mobilization (increase DF), biomechanics (pronator and supinator)
Education: Activity modification, POLICE
Precautions: PF may be accompanied by heel spurs
AP for differental diagnosis
Medial Tibial Stress Syndrome: MOI, S/S, testing, treatment approach, education, precautions
MOI: Associated with running or any vigourous. Happens when muscular and periosteum become over worked.
Risk factors: Increase in unaccustomed activity or trianing load, flat feet or high arches, obese, improver foot wear.
S/S: Pain on medial aspect of tibia, 2nd third of posterior medial border of tibia, pain during or after exercise, worse at the beginning of exercise.
Testing: Tenderness on palpation of medial border + pain with resisted gastroc complex
Treatment approach: Low dye taping, ice massage, compression socks, stretching and strengthening (increase calf length and strength + intrinsic foot muscles)
Education: Prevention and gradually increasing training load
Precautions: Rule out stress fracture.
Ankle Sprain: MOI, S/S, testing, treatment approach, education, precautions
MOI: Lateral, Syndesmosis, Medial
S/S: Grade 1-3. Common grade 2/3 presentation –> local pain, edema, increased temperature, ecchymosis, hyper mobility or instability, loss of motion and/or function.
Testing: Anterior drawer, posterior drawer, Kleigers, etc.
Treatment approach: AP
Education: Preventitive rehab - common re-occuring injury
Precautions: Suspected # - Ottawa Ankle Rules
Lateral Ankle Sprain: MOI
Inversion and PF
Anterior talofibular ligament (ATFL) and calcaneofibular ligament (CFL) normally injured
MOI, S/S, testing, treatment approach, education, precautions
MOI:
S/S:
Testing:
Treatment approach:
Education:
Precautions:
Achilles Tendinopathy: MOI, S/S, testing, treatment approach, education, precautions
MOI: Overuse of Achilles tendon/plantar flexors + increase activity. Insertional is more common than non-insertional.
Risk factors: pronation, reduced DF, reduced ipsilateral plantar flexion strength, tight calf, age, diabetes, high cholesterol, glute med weakness
S/S: Localized pain and perceived stiffness in Achilles tendon following periods of inactivity, decreased dorsiflexion and subtalar joint ROM, decreased PF strength due to pain and weakness, pain worse in the morning or during warm up.
Testing: Strength and ROM testing, (hop test, London Hosiptal test)
Treatment approach: Eccentric loading (heel drops), footwear/orthodics, stretching/strength, mobilization, taping, heel lifts, deep transverse friction
Education: Monitor exercise, activity modifcation
Precautions: Rule out Achilles tendon rupture
Meniscal Tear: MOI, S/S, testing, treatment approach, education, precautions
MOI: Contact or non-contact injury. Repetitive or forceful rotation of the knee that is combined with flexion and varus/valgus stress.
Risk factors: age, increased knee laxity of ACL surgery
S/S: Joint line pain, loss of flexion more that 10 degrees, loss of extension more than 5 degrees, swelling, crepitus, positive special test, reported “locking”, swelling at time of injury
Testing: ROM, strength, McMurray’s, Apley’s
Treatment approach: Injury to 4 weeks –> restore normal knee extension, decrease swelling, safe use of protection equipment. 4 weels to 3 months –> single leg stand control, normalize gait, good control and no pain with functional movements. 3+ months –> good control and no pain with sport and work specific movements.
Education: Same as above.
Precautions: Post surgical percautions
Common differential diagnosis: Plica syndrome - fluid that failed to reabsorb during development
Cruciate Ligament Sprian: ACL: MOI, S/S, testing, treatment approach, education, precautions
MOI: Tibia translates anteriorly on the fixed femur. Plant and twist when changing direction, suddenly stopping or direct impact on the knee. More common in females over males.
S/S: Pain, feeling of instability, giving way of the knee, loss of ROM, swelling, hemathrosis in the knee - refer back to doctor
Testing: Lachman’s (most common), Anterior Drawer
Treatment approach: Closed chain functional strengthening of quads, hamstrings, important to restore extension, joint mobs start as soon as pain free
Education: time line, unhappy triad (ACL, MCL, medial meniscus)
Precautions:
Syndesomosis Ankle Sprain: MOI
ER of the ankle is most common OR excessive DF and ER of the leg on a foot when its planted (slamming on brake)
Anterior, posterior and transverse tibiofibular ligaments and interosseous membrane support
Medial Ankle Sprain: MOI
Gradual degeneration of medial ligaments OR valgus force to leg creating eversion OR awkward fall resulting in excessive pronation or eversion of foot
Fan shaped deltiod ligament: anterior and posterior timbotalar, tibionavicular and tibiocalcaneal ligaments.
Collateral Ligament Sprain: MCL: MOI, S/S, testing, treatment approach, education, precautions
MOI: Valgus force (towards center of body) with or without external rotation. Can be contact or non-contact.
S/S: Joint line tenderness, localized knee pain, stiffness, brusing and redness. Protective spasm, ligament laxity.
Testing: Valgus stress test at 0 and 30 degrees.
Treatment approach: AP
Education: Possible outcome measures: LEFS, balance tests, vertical jump, SL squat
Precautions: Differential - Pes Ansetine Bursitis.
IT Band Friction Syndrome: MOI, S/S, testing, treatment approach, education, precautions
MOI: Inflammation of the IT band from excessive knee flexion and extension (most affected at 30 degrees of knee flexion). TFL is contractile portion of IT band. Inserts on lateral epicondyle on tibia and Gerdy’s tubercle. Provides lateral stability of hip and anterior-laterally supports knee is stance phase.
Risk factors: weak hip abductors and tight TFL, internal knee rotation during stance phase, abnormal neuromuscular control, rapid training increases, downhill running
S/S: Deep achy pain along lateral aspect of knee, fluid accumulation around lateral epicondyle
Testing: Nobles compression, Ober’s
Treatment approach:
Education: POLICE in acute, hip abduction exercises, stretching lateral structures, muscle and postural re-education.
Precautions: 2x more common in women than men. Cyclists and runners.
MOI, S/S, testing, treatment approach, education, precautions
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Patella femoral pain syndrome: MOI, S/S, testing, treatment approach, education, precautions
MOI: “Runners knee”. Caused by overuse, overloading, improper alignment. Women more than men.
External Risk Factors: vigorous physical activity (repeated stress). Sudden change in PA. Improper technique. Changes in footwear.
Internal Risk Factors: Poor patellar tracking (Q angle, excessive knee valgus or vacuum, weak or imbalanced quads)
S/S: Tight quads, weak hip abductors, medial tibial torsion. Anterior knee pain, pain with climbing or descending stairs, stepping up or down, prolonged sitting, squating
Testing: Thomas test, hip abductors, hamstrings, knee extensors, DF/PF, MMT VMO & glute med. SL squat, step down. Clarke’s sign (patellar grind).
Treatment approach: Strengthening weak muscle groups.
Education: Activity avoidance. Taping.
Precautions: Avoid high impact activities (running, jumping and open chain leg extension exercises)
Osgood Schlatters Disease: MOI, S/S, testing, treatment approach, education, precautions
MOI: Stress on developing tibial tuberosity at patellar tendon insertion. Commonly bilateral. Boy 12-15, girls 8-12. Repetitive use of quads. Runners and gymnasts.
S/S: Inflammation at tibial tuberosity - warmth, red, swelling, pain. Pain with squatting, stairs and jumping.
Testing: TOP tibial tuberosity. History of adolescent who is active.
Treatment approach: Activity modification, encourage non-weight bearing exercises. Stretch quads, educate on pain management. Knee padding, taping or bracing.
Education: As above.
Precautions: Avoid high intensity exercises to strengthen quads as it would increase stress on tibial tuberosity.
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Serious Spinal Red Flags
- Presentaion younger than 20 or onset older than 50
- Violent trauma, such as fall from height or car
- Constant, progressive, non-mechanical
- Thoracic pain
- Hx of carcinoma, systemic steroids, drug abuse, HIV
- Systemically unwell weight loss
- Persisting severe restriction of flexion in lumnar spine
- Structural deformity
Cauda Equina
- Difficulty with urination
- Loss of anal sphincter tone or fecal incontinence
- Saddle anesthesia
- Widespread (more than one nerve root) or progressive motor weakness
- Gait disturbances
Flexion Exercises for Low Back Pain for DDD/spinal stenosis
- PPT
- Single knee to chest
- Double knee to chest
- Forward bend in sitting and standing
- Partial or full sit ups
Extension Exercise for Low Back Pain for disc protrusion.
- Prone lying
- Prone on elbows
- Prone press up onto hands
- Standing extension
- Bridge
- Prone leg extension
Lumbar Stabilization Exercises: TA
- Heel slides
- Hip flexion
- Knee fall outs
- Cat cow
- Belly button to spine
- Bridging
Myositis Ossificans (Or Heterotopic or Ectopic Bone Formation)
- Part of hematoma is replaced with bone
- Can be recognized on plain film radiographs
- Contra-indication in acute stage: heat and massage
- Work on PROM, AROM and isometrics