Module 1 Flashcards
Arm IV location
Antecubital fossa (enti kYOO be dl fo sa)
Cephalic (dorsal) / basilic (medial) veins
Counter indications for AC IV
- Infection/tissue damage
- proximal IV infiltration
- thrombosis
- fistula
- recent surgery
- Conula no longer in vein
- Fluid spilling outside of vein
- Extravasation
- Infiltration
Pulling fluids where they don’t exist (like when checking conulation)
Aspiration / to aspirate
Not sterile but clean of pathogens
Aseptic
IO access full name
Intraosseous (intra O si es, also O-C-S)
IO locations
- Humoral head/Proximal humorous = greater tubercle (TU ber kl)
- Proximal tibial / tibial tuberosity (TU be ra si ti). Distal tibial for peds
Contraindications for EZ-IO Access
- Fracture
- Previous orthopedic procedures near
- Infection at the site
- Inability to locate landmarks/ excessive tissue
The inability to feel pain
Analgesia (anal ji zi ya)
Analgesic (anal ji zik) = takes away pain
Adrenergic receoptors
- Alpha 1 - vasoconstriction
- Beta 1 - increase heart rate, contraction force, myocardial conduction
- Beta 2 - Bronchodilation
Continuously measure and adjust the drug dosage
Titrate (tai trate)
A molecule or ion that binds to a receptor to send signals between cells.
Ligand (LI gend)
Most significant modulator of plasma oncotic pressure and transport ligands
Albumin
Water and plasma proteins pressure
Osmotic pressure (osmosis) = water
Oncotic pressure = plasma proteins, notably albumin, that causes a pull on fluid back into the capillary
Moving solutes - potassium, magnesium, organic phosphates, and protein (albumin) from higher concentration to lower.
Diffusion
Three types of osmosis
Isotonic = equal tension between the cell and the solution it is in
Hypertonic = higher concentration solution outside the cell; water is leaving the cell in an attempt to dilute the hypertonic solution and the cell shrivel
Hypotonic = lower concentration solution outside the cell; water enter the cell to dilute the cell and the cell grows and can even explode (“hypotonic leads to hippo cell :-)”)
Term for Active Transport
ATP Adenosine Triphosphate (a DE nasin trai FAs fate)
Resistant to treatment or cure
Refractory
Enzymes that metabolize drugs in the liver
Cytochromes (sai ta krom, “sight a chrome”)
pH lower than 7.35 is…
Acidosis … body buffers with bicarbonate
pH greater than 7.45 is…
Alkalosis … body buffers with carbonic acid (H2CO3)
A common excitatory neurotransmitter
Acetylcholine (ACh) (asido KO lin)
When we are breathing too slow
CO2 (an acid) stays in the lungs = Respiratory acidosis (hypo)
When we breath too fast
Respiratory alkalosis (hyper) - we get rid of too much CO2
Too much base in the metabolic process.
- Metabolic alkalosis - increase in bicarbonate HCO3, low on acid H+
- Renal correction - kidneys keeps the H+ in the body to increase acidity
Too much acid in the metabolic process.
- Metabolic acidosis - we don’t have sufficient levels of bicarbonate (HC03), just a lot of H+
- Renal correction - Kidneys work to filter out Hydrogen (H+) and keep more (and even produce new) bicarbonate (HCO3)
