MOD 1 Cardiovascular Meds Flashcards
Decrease the amount of water in the bloodstream - decreasing blood pressure
Diuretics
Diuretics
- Blocks angiotensin that constricts blood vessels
- help to decrease salt and water in the body
- decrease heart workload and increase blood flow and oxygen to the heart and organs
- given in combination with diuretics to treat HTN
- Side Effects: hypotension, proteinuria, hyperkalemia, persistent dry cough, angioedema - severe
Captopril, Enalapril (Vasotec)
Angiotensin - Converting Enzyme Inhibitors - ACE
Diuretics
- Blocks effects of angiotensin (vasoconstriction, sodium, and water retention)
- Few side effects
- NOT TO BE GIVEN TO PATIENTS WITH LIVER FAILURE
Losartan (Cozaar), Valsartan (Diovan)
Angiotensin II Receptor Antagonists (Blockers)
Diuretics
- Blocks calcium from entering muscles of the heart and arteries - causes a decrease in the contraction of the
heart - dilates arteries - Used to treat dysrhythmias and HTN
- Used to treat migraines and Raynaud’s disease
Dilitazam (cardizem), Verapamil (Calan), Norvasc
Calcium Channel Blocker
Diuretics
- Block receptors in the arteries and smooth muscle - relaxes blood vessels and increases blood
- Not typically used as a first-line antihypertensive
- Clonidine (Catapress)
Alpha - Blockers
Diuretics
- Works directly to relax blood vessel walls
- Used to treat HTN in emergencies
- SIDE EFFECTS: dizziness, headache, orthostatic hypotension, nausea, vomiting, pancreatitis, hyperglycemia
- Hydralazine, minoxidil (Loniten), Nipride
Direct Vasodilators
Antidysrhymic Drugs
- Increase cardiac blood flow
- Decrease heart rate
- Increase heart contraction
- Cardioversion to NSR
- Maintain NSR after cardioversion
- Amniodarone (Cordarone)
Potassium Channel Blockers - V-Tach, V-Fib, convert from A-Fib to SR
Antidysrhymic Drugs
- Slow conduction through the SA and AV nodes - decreasing heart rate
- Artery dilation
- Blood pressure is lowered
- Blood flow and oxygen to the heart is increased
- Diltiazem (Cardizem), Verapamil
Calcium Channel Blockers - SVT
Antidysrhymic Drugs
- heart rate and cardiac output are increased
Atropine - Bradydysrhymias
Antidysrhymic Drugs
- Heart rate is decreased, causing the heart to regain an organized rhythm
Digoxin - A-Fib
Antidysrhymic Drugs
- Helps to slow ventricular contraction
- Given Rapid IV push - stop the heart to “reset.”
- Must be given while pt is on close RN and Tele Supervision - code cart should be nearby
Adenosine - SVT
Antianginal Drugs
- Cause generalized vascular and coronary vasodilation
- Increase blood flow through vessels
- Reduces myocardial ischemia but can cause hypotension
Nitrates
Antianginal Drugs
- Block effects of epinephrine
- Decrease heart rate, which then lowers blood pressure
Metoprolol, Labetalol, Atenolol
Beta Blockers
- Heart Failure
- Preload - Fluid volume status - anything that affects the blood in getting back to the heart (how much volume is in the body)
- Afterload - How hard is it to get fluid out of the ventricle - pulmonary vascular resistance - pressure in the aorta
- Ejection fraction - % of blood pumped with each contraction
Cardiac Glycosides
- Work to increase the force of each heartbeat
- Slows the overall heart rate to allow the heart to fill with blood and make each beat more effective.
- Apical heart rate must be checked daily before dose. Hold medication if the heart rate is less than 60
- The antidote for dig toxicity is DIGOXIN IMMUNE FAB (Digibind)
Digoxin (Lanoxin)
Cardiac Glycosides