MNT liver Flashcards

1
Q

How much blood is circulated in the liver per min

A

1500 ml

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2
Q

how does blood exit the liver

A

via the hepatic veins into the interior vena cava

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3
Q

what makes poop brown

A

bile

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4
Q

where is bile made

A

liver

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5
Q

where is bile stored and concentrated

A

gallbladder

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6
Q

steatoreaha

A

fatty greasy diharea

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7
Q

how much liver function is needed to sustain life

A

10-20% cannot survive totally wihtout it

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8
Q

functions of the liver

A

activation/storage of vits and minerals
converts vit D to active form
formation/excreation of bile
metabolism of steriods
conversion of amonia to urea (affects MNT)
synthesis of clotting factors(bleeding affects MNT)

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9
Q

thorugh all liver diseases what can happen

A

10-20% liver function

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10
Q

Viral Hepitis

A

won’t impact us unless it gets to ESLD

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11
Q

Steatosis

A

fat in the liver or fatty liver

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12
Q

How much wt loss can improve steatosis

A

3-5% wt loss

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13
Q

how much wt loss is needed to reverse NASH

A

10%

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14
Q

meds for NAFLD

A

Vit e if no TD2
coffee unsweetend =protective

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15
Q

ALD

A

most common liver disease
40% of deaths from cirrhosis atributed to ETOH

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16
Q

ALD charaterized by

A

fatty liver hepatitis or cirrhosis

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17
Q

Alchoholic cirrhosis may lead to

A

GI bleed hepatic encephalopathy, portal HT ascites

18
Q

Necrosis

A

death of cells

19
Q

Alcoholic liver micronutrient deficiency

A

thiamin is most common

20
Q

Bottom line for Hepatitis

A

liver disease is a spectrum of severity
MNT is for complications

21
Q

Cirrhosis

A

irreversible fibrotic changes ( scarring) of the liver with inflamtion blood wont flow thorugh as well

22
Q

Portal HTN

A

increased pressure in portal vein

23
Q

varices

A

englarged blood vessels in esophagus
we care if feeding tube has to be placed and nick can cause bleed due to lack of cltting factors being made

24
Q

increased prothrimbin time

A

increased clotting time

25
MNT for Portal HTN and Esoph Varices
if acute GI bleed no EN PN if NPO 5-7 days No PEG either dt the acites
26
Ascites
accumulation of fluid in the abdominal cavity
27
Medical treatment for ascites
paracentesis diuretics
28
MNT for ascites
NA restriction 2 g ( not lower to avoid decrease oral intake) adequate protein
29
MOnitor for ascites
wt to know energy needs electrolytes (NA and pottasium) abdominal girth know where the wt is
30
Paracentesis
remove 5 liters of fluid , does pull off electrolytes and protein need to get the dry wt after the fluid is removed for energy calulations
31
Hyponatremia
low sodium dt decreased water excretion sodium losses with paracentesis
32
Hyponatremia MNT
fluid restriction 1-1.5 l day low sodium 2 g /d
33
Hepatic Encephalopathy
impaired mentiaiton or thinking neuromuscualr diturbances and altered conciousness
34
Hepatic encephalopathy possible mechanism
high ammonia impaired urea synthesis
35
Sources of ammonia
GI tract protein metabolism blood from GI bleed
36
HE MEds
lactulose and antibiotics both cause diharea to get rid of the ammonia in the body
37
MNT encephalopathy
avoid unnecessary protein restriction fiber increases aids in excretion of N compunds intake of .25 g/kg of oral BCAA
38
DIseased liver isn't able to
maintain blood glucose levels or keep bs from tanking in periods of fasting
39
MNT for ESLD
recomend carb consistent intake consitent meal intake (3-6) day including bedtime snack conating carb and protein nocturnal EN for malnurished pts
40
milk thistle
may help liver butn ot in liver disease
41
VIt def in ESLD
vit A D E K
42
Liver transplant
established treatment for ESLD malnutrion is common at time of transplant