Midterm - Radiology Flashcards

2
Q

three compartments for thoracic neoplasms

A

lung, pleura, mediastinum

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3
Q

use a CT to determine the?

A

extent of disease LOCALLY

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4
Q

use a PET-CT to determine the?

A

whole body stage of a disease (are there mets??)

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5
Q

when to use an MRI?

A

suspect soft tissue tumor (pancoast, mesothelioma)

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6
Q

three primary cancers of the lung

A

adeno, squamous, BAC (ACIS/MIC)

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7
Q

most common neoplasm of the pleura

A

mets, fibrous tumor (mesothelioma are RARE)

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8
Q

four T’s of the anterior mediastinal mass

A

thymoma, thyroid cancer, teratoma, terrible lymphoma

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9
Q

primary lung cancer presenting as mediastinal mass

A

small cell

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10
Q

which lung cancer presents centrally?

A

squamous cell

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11
Q

if you see well defined heart margins on a CXR, then the mass is?

A

posterior, often lower lobe

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12
Q

lung cancers are most often in what lobe?

A

upper lobes

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13
Q

BAC has what characteristic CT finding?

A

ground glass opacity

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14
Q

what does BAC look like on a PET-CT?

A

“warm” because it is slow growing (not as bright as other cancers)

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15
Q

collapsed lobes are most frequently from cancers originating in what part of the lung?

A

central masses

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16
Q

“snow storm” on CXR suggests?

A

stage 4 metastatic cancer, probably adenocarcinoma

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17
Q

cavitary lesions suggest metastases from what type of cancer?

A

likely head/neck if smoker, cervical if female non-smoker

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18
Q

what looks like a ball under a rug, causing sharp defined borders on CXR?

A

fibrous tumor of pleura

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19
Q

most common metastasis to pleura?

A

lung cancer

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20
Q

if the trachea is displaced and there is a dense soft tissue mass in the upper anterior mediastinum, it is probably?

A

thyroid cancer

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21
Q

the most common anterior mediastinal neoplasms are?

A

thymoma and lymphoma

22
Q

what is first-line imaging for lung dz?

A

chest film (PA and lateral)

23
Q

what are the benefits of CXR?

A

inexpensive, low radiation, lots of info

24
Q

PA are preferred because AP films cause?

A

magnification of the heart due to distance from the film cassette

25
Q

what three tubes go through the diaphragm?

A

IVC, esophagus, aorta

26
Q

which side of the diaphragm is higher and why?

A

right side, due to liver

27
Q

what forms the far right border of the heart on a CXR?

A

right atrium

28
Q

what lobe is directly next to the RA?

A

right middle lobe

29
Q

how many ribs should you see on a CXR?

A

about 10 (count the posterior ones)

30
Q

radiographic findings of emphysema

A

too few markings in upper lobes, narrow trachea PA/wide lateral view, big A-P diameter, flat diaphragm, wide CVA, CT shows black dots due to cetrilobular destruction of alveoli

31
Q

radiographic findings of bronchiectasis

A

big A-P diameter, wide CVA, diffuse markings throughout lung –> can see all of the vessels; CT shows dilated air spaces with thick walls

32
Q

three locations of interstitial lung disease

A

random, centrilobular, peribronchovascular/perilymphatic

33
Q

random pathology is due to?

A

hematogeous source (mets or infection)

34
Q

centrilobular pathology is due to?

A

airways dz (usually) or vasculitis of artery

35
Q

perilymphatic pathology is due to?

A

lymphatic sources (sarcoid, lung cancer metastasizing)

36
Q

potential etiologies of interstitial lung disease

A

smoking, autoimmune, drug toxicity, aspiration, abnormal proliferation

37
Q

chronic inflammation of the interstitium leading to fibrosis is seen is what three interstitial lung diseases?

A

UIP, NSIP (fibrosing type), DIP

38
Q

what is the best way to image ILD?

A

high res CT

39
Q

what is the most imporant thing when diagnosing an interstitial lung disease?

A

determine if it is or is not UIP

40
Q

pathology of UIP

A

spatial and temporal heterogeneity with dense fibrosis and fibroblastic foci; honeycombing

41
Q

important CT findings for UIP diagnosis

A

bilateral basilar subpleural reticular opacities that progress to honeycombing, traction bronchiectasis, septal thickening

42
Q

NSIP pathology

A

cellular or fibrosing subtype, spacial and temporal homogeneity, inflammation of alveolar septal walls

43
Q

NSIP CT findings

A

ground glass opacity, bronchiectasis, subpleural sparing, microcystic honeycombing, basilar predominant

44
Q

RB-ILD

A

centrilobular ground glass nodules, homog or upper lobe predominant, pigmented macrophage accum.,

45
Q

DIP pathology

A

homogenous thickening of alveolar septa, accum of pigmented macrophages, caused by smoking

46
Q

DIP CT findings

A

periph ground glass opacities and nodules, emphysema and cysts, basilar

47
Q

COP/BOOP pathology

A

mild chronic interstitial inflammation and organized fibrosis in distal airways, homogenous with preservation of lung architecture

48
Q

COP/BOOP CT findings

A

patchy peripheral ground glass or dense opacities, air bronchograms, atoll sign, none or basal gradient

49
Q

AIP etiology and pathology

A

ideopathic, probably sepsis or resp insult –> DAD (ciritcally ill)

50
Q

AIP CT findings

A

ground glass opacities and consolidation, bronchial dilatation, Basal gradient, never do well