Midterm 1 Flashcards by A B

Q

Benazepril (Lotensin)

A

ACE inhibitor

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Q

Captopril

A

ACE inhibitor

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Q

Enalapril

A

ACE inhibitor

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Q

Fosinopril

A

ACE inhibitor

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Q

Lisinopril

A

ACE inhibitor

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Q

Moexipril

A

ACE inhibitor

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Q

Perindopril

A

ACE inhibitor

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Q

Quinapril (Accupril)

A

ACE inhibitor

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Q

Ramipril

A

ACE inhibitor

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Q

Trandolapril

A

ACE inhibitor

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Q

Azilsartan

A

Angiotension-Receptor Blockers (ARBs)

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Q

Candesartan

A

Angiotension-Receptor Blockers (ARBs)

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Q

Eprosartan

A

Angiotension-Receptor Blockers (ARBs)

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Q

Irbesartan

A

Angiotension-Receptor Blockers (ARBs)

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Q

Losartan

A

Angiotension-Receptor Blockers (ARBs)

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Q

Olmesartan

A

Angiotension-Receptor Blockers (ARBs)

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Q

Telmisartan

A

Angiotension-Receptor Blockers (ARBs)

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Q

Valsartan

A

Angiotension-Receptor Blockers (ARBs)

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Q

Aliskiren

A

Renin Inhibitor

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Q

Atenolol

A

Beta Blocker- beta 1 selective

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Q

Bisoprolol

A

Beta Blocker- beta 1 selective

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Q

Carvedilol

A

Beta Blocker

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Q

Labetalol

A

Beta Blocker

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Q

Metoprolol

A

Beta Blocker- beta 1 selective

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Q

Nadolol

A

Beta Blocker

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Q

Nebivolol

A

Beta Blocker- beta 1 selective

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Q

Propranolol

A

Beta Blocker

Q

Chlorthalidone

A

Thiazide Diuretics

Q

Hydrochlorothiazide (HCTZ)

A

Thiazide Diuretics

Q

Metolazone

A

Thiazide Diuretics

Q

Bumetanide

A

Loop Diuretics

Q

Furosemide (Lasix)

A

Loop Diuretics

Q

Torsemide

A

Loop Diuretics

Q

Amiloride

A

“Potassium-Sparing” Diuretics

Q

Eplerenone (Inspra)

A

“Potassium-Sparing” Diuretics

Q

Spironolactone (Aldactone)

A

“Potassium-Sparing” Diuretics

Q

Triamterene

A

“Potassium-Sparing” Diuretics

Q

Acetazolamide (Diamox)

A

Carbonic Anhydrase Inhibitors

Q

Methazolamide

A

Carbonic Anhydrase Inhibitors

Q

Mannitol (Osmitroll)

A

Osmotic Diuretics

Q

Amlodipine (Norvasc)

A

Vasodilators- Calcium Channel Blockers

Q

Diltiazem

A

Vasodilators- Calcium Channel Blockers

Q

Felodipine

A

Vasodilators- Calcium Channel Blockers

Q

Isradipine

A

Vasodilators- Calcium Channel Blockers

Q

Nifedipine (Procardia)

A

Vasodilators- Calcium Channel Blockers

Q

Verapamil

A

Vasodilators- Calcium Channel Blockers

Q

Hydralazine

A

Vasodilator

Q

Minoxidil

A

Vasodilator

Q

Fenoldopam

A

Parenteral Agents

Q

Nitroprusside (Nitropress)

A

Parenteral Agents

Q

Clonidine

A

Centrally Acting Alpha-2 Agonists

Q

Methyldopa

A

Centrally Acting Alpha-2 Agonists

Q

Doxazosin (Cardura)

A

Alpha Blockers

Q

Prazosin (Minipress)

A

Alpha Blockers

Q

Terazosin (Hytrin)

A

Alpha Blockers

Q

What is the MOA for ACE inhibitors?

A

prevent conversion of angiotension I to angiotension II

Q

What is the MOA for Angiotension II Receptor Blockers?

A

antagonist at AT1 receptor

Q

What is the MOA for Renin inhibitors?

A

directly inhibits renin (acts earlier than ACE inhibitors or ARBs)

Q

What is the MoA for β-Adrenoceptor (Beta) Blocking Agents?

A

Lower BP mainly by decreasing cardiac output

Oxygen demand is lowered due to decreased heart rate and force of contraction
Also decrease sympathetic outflow from the CNS and inhibit the release of renin

Q

What is the MOA for Thiazide Diuretics?

A

Inhibits Na/Cl co-transporter

-Increase concentration of Na and Cl in the tubular fluid

Q

What is the MOA for Loop Diuretics?

A

Inhibit co-transport of Na/K/2Cl

-Reabsorption is decreased

Q

What is the MOA for Aldosterone antagonists?

A

Blocks aldosterone receptors
Prevents production of proteins that stimulate Na/K exchange sites of the collecting tubules
-Prevents Na reabsorption and K/H secretion

Q

What is the MOA for Sodium Blocking channels?

A

Block sodium transport channels
Results in decreased sodium/potassium exchange
Does NOT depend on the presence of aldosterone

Q

What is the MOA for Carbonic Anhydrase Inhibitor?

A

Carbonic anhydrase catalyzes reaction to form bicarbonate

- CAIs decrease the kidney’s ability to exchange Na for H

Q

What is the MOA for Calcium Channel Blockers?

A

Block inward movement of calcium
Less calcium available to activate contraction
Produces reduction in vascular tone
Produces dilation of coronary and peripheral arteries
Decreases force of contraction and reduces cardiac workload