Mental Health remaining Flashcards

1
Q

What is the issue with language in mental health conditions

A

The language that we use can actually perpetuate stigma. Especially if our language suggests certain stereotypes associated with a certain mental health condition –> makes people feel more judged?

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2
Q

What were mental health conditions referred to before

A

Abnormal psychology, however, this was changed because it suggests mental health issues is abnormal, discouraging people from speaking up

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3
Q

What is psychology

A

The scientific study of behaviour, emotion and cognition (and the potential relationship between these factors)

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4
Q

What is abnormal psychology

A

Scientific study of abnormal behaviour, emotions and cognitions

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5
Q

What is psychopathology

A

Psyche = mind, pathology = illness/disease

Study of mental illness, including classification, causes, development, treatment and outcomes

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6
Q

What is the empirical method

A

any procedure for conducting an investigation that relies upon experimentation and systematic observation rather than theoretical speculation.

It is used at different levels of mental health such as; classification/diagnosis, causation, treatment

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7
Q

Explain why empirical method might be used for describing mental health

A

To define MHC, we need to tell what is normal/healthy vs abnormal/unhealthy –> we need an empirical method of approaching this

Thus, we might try to see if there are a cluster of symptoms associated with a certain illness

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8
Q

Explain why empirical method might be used for identifying the causation of mental health

A

Causes of MHCs is complex
Involves interaction of biology, individual psychological factors, upbringing, social environment

Empirical method required to narrow down the influence of each on MHC

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9
Q

Explain why empirical method might be used for treatment of mental health

A

Critical for understanding then treating MHCs

Scientific method used in treatment through RCTs to determine if a treatment is valid (control groups, manipulations etc), placebo effect

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10
Q

Understand and explain key differences between “normal” and abnormal behaviour

A

Abnormal refers to anything whish isn’t normal –> but then what isn’t normal? (the line between the two is quite blurred)

Hard to determine as well because mental illness is subjective and varies across cultures, times etc

However, the ‘3D’s’ would help with identifying what’s considered abnormal behaviour

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11
Q

What are the 3D’s?

A

Deviates
Distress
Dysfunction

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12
Q

Explain how deviation can be used to recognise abnormal behaviour

A

Refers to deviation from statistical norm. Often the first step in identifying normality vs abnormality.

Basically looking at behaviour which isn’t the norm, and thats the first step in potentially detecting abnormal behaviour

Howeve,r important to note that sometimes a deviation from the norm could be an exception or rarit, and whats considered normal is always culturally and time bound –> we can’t use deviation as the only factor

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13
Q

Explain how distress can be used to recognise abnormal behaviour

A

This is where there is distress in oneself or others (e.g. family). Distress isn’t always necessary

However, its just when the behaviour has a negative impact on yourself and potentially others –> sign of abnormal behaviour

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14
Q

Explain how dysfunction can be used to recognise abnormal behaviour

A

if mental illness causes dysfunction in person’s ability to carry out important life goals etc, it could be classified as an illness

However, some disorders might not necessarily involve dysfunction (i.e. some people with psychopathic tendencies tned to be good CEOs - functioning well in society )

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15
Q

What is the biomedical model of mental illness

A

Dominant model in psychiatry and the underlying model of the DSM 5

Assumes that mental disroders can be diagnosed similar to physical illnsesses, and can be explained in terms of a biological disease process

Ultimately proposes that mental disorders are brain diseases and emphasises pharmacalogical treatments to target presumed biological abnormalities

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16
Q

What does biomedical model of mental illness believe is the best way to treat mental illness

A

Treat biological deficiencies (i.e. medication or electroconvulsive therapy ECT)

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17
Q

What are the criticisms/ limitations of the biomedical model

A

Need to avoid extreme reductionism (i.e. reducing the complex nature of mental health conditions into few constituents

Complexity of psychological phenomenon are impossible to explain at the neural/molecular level asw ell

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18
Q

What was the psychoanalytic model of mental illness

A

Most dominant theory in the 1st half of the 2nd century. Proposed Id (pleasure), Ego (reality satisfying both id and superego) and superego(moral self). Suggested that the Id and superego were in constant conflict, and ego tries to work out and problem solve how to meet both needs

Maladjustment/mental illness occurs when there is unresolved conflicts —> anxiety –> use of defence mechanisms –> symptoms of mental illness/ suffering

Didn’t really believe in an idea of normality

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19
Q

What did the treatment in psychoanalytic model of mental illness involve

A

Involves:

Building insight into unconscious processes
Developing awareness of unresolved conflict
Developing awareness of defence mechanisms

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20
Q

What was the significance of the psychoanalytic model of mental illness

A

Revolutionised the concept of mental illness

Made no clear dividing line between abnormal and normall

Strong influence on early stages of DSM

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21
Q

What was the criticisms/ limitations of the psychoanalytic model of mental illness

A

Lacks evidence

Not open to empirical evaluation

Unfalsifiable (unable to be proven false)

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22
Q

What was the humanistic model of mental illness

A

Significant in 60s and 70s as a reaction to negativity of psychoanalytic model (which was saying that everyone was abnormal to an extent)

Core of the model is the idea that the human being is a wholly positive figure, and the goal of the hhuman is to self actualise (involves experiencing life to the fullest, living in the present, trusting our own feeligns etc) –> every human has the potential to achieve self actualisation

Suggested that maladjustment occurred when self actualisation has been thwarted - for example the enviro imposing conditions of worth on the individual or when our experiences, emotions or needs are blocked

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23
Q

What is Maslows hierarchy of needs

A

Physiological needs –> safety needs –> love and belonging –> self esteem –> self actualsation

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24
Q

What was treatment in humanistic model of mental illness based on

A

Involves treating people with empathy and an unconditional positive regard, and trying to help them see themselves in a positive way

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25
Q

What were the critiques of the humanistic model of mental illness

A

These are important parts of therapy, but they aren’t sufficient enough (can’t just have a positive regard of them)

Difficult to research (i.e. when is self actualisation actually achievable)

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26
Q

What was the behavioural model of mental illness

A

Reaction to psychoanalysis being unfalsifiable. In comparison, this involves clasasical and operant conditioning

and suggests that maladjustment occurs as a result of our learned history/associations with negative things

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27
Q

What was the treatment with the behavioural model of mental illness

A

Involve varying treatments, in particular exposure therapy to help with extinction

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28
Q

What were the criticisms of the behavioural model of mental illness

A

Involves the overemphasis on behavioural aspects, and often excluding cognitive and emotional elements

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29
Q

What was the cognitive-behavioural model of mental illness

A

Most dominant current psychological model

Suggests that our behaviours are controlled by our cognitions, and they both work together to determine how we feel and what we do etc

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30
Q

WHat does the CB model of mental illness suggest is the root cause of mental illness

A

Negative core beliefs are the root cause of mental illness.

These are long held core beliefs or udnerstandings of the world that a person holds which influences our interaction with and interpretation of the world –> automatic thoughts coming from core beliefs. It can come from early life experiences which set blueprint for how we interpret the world

E.g. loving family = im safe, im enough , im loved

unloving family = im alone, im not worth anything

It is ultimately our automatic negative thoughts which reflect our core beliefs and might appear so quickly that we don’t notice them, leading to our mental health issues

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31
Q

What are some examples of cognitive distortions/ biases

A

All or nothing thinking

Mental filter

Overgeneralising

Disqualifying the positive

Jumping to conclusions

Magnification of catastrophies

Labelling

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32
Q

What does the CB model of mental illness suggest treatment involves

A

Psychoeducation = teaching the people about their automatic thoughts and how to catch themselves thinking those negative thoughts

Cognitive restructuring = challenging content of negative automatic thoughts

Behavioural experiments or exposure therapy can also be used

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33
Q

What were the criticisms with the CB model of mental illness

A

Too structured, which might not allow for flexibility to address unique needs

High dropout rates due to high attention needed

Oversimplifies psychological problems by focusing mainly on thought patterns and behaviours

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34
Q

Explain what psychoanalytic, humanistic, behavioural, and cognitive-behavioural believe the cause of mental illness is, and how to treat them?

A

Psychoanalytic cause = repression of unresolved conflict

Psychoanalytic treatment = insight

Humanistic cause = thwarted self actualisation

Humanistic treatment = empathy,, unconditional positive regard, self discovery

Behavioural cause = learned association

Behavioural tretment = learn new associations

CB cause = negative core beliefs, biased thinking, learned associations

CB treatment = cognitive restructuring, exposure, behavioural experiments

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35
Q

Describe what the DSM does/ what are its key features

A

It defines psychopathology, reflects the biological / medical model of mental illness and typically reflects the most agreed upon definitions / current consensus

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36
Q

How does DSM diagnose and classify mental illness

A

Uses a categorical approach to diagnosis, where mental disorders are classified into distinct categories based on specific criteira (i.e. a set of symptoms that must be present for a diagnosis to be made)

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37
Q

What are some changes which have happened in the DSM

A

Generalised anxiety disorder first introduced in DSM 3

Bing eating disorder first included in DSM 5

Asperger’s disorder removed from DSM 5

Prolonged Grief disorder removed in DSM 5

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38
Q

What are the benefits in DSM approaches to diagnosis and classification of mental illness?

A

Provides a standardised set of criteria for diagnosinng mental disorders which promotes consistency across different practitioners and settings

reflects empirical research and is often updated to reflect understandings of mental health

Supports treatment planning

Improves communication between researchers and healthcare professionals

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39
Q

What are the limitations in DSM approaches to diagnosis and classification of mental illness?

A

Labelling/stigma

Over medicalising reasonable reactions to stressful situations

Problems of validity, reliability and ambiguity

Reductionism (oversimplifying complex mental health issues)

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40
Q

What are the similarities in DSM approaches to diagnosis and classification of mental illness (with ICD - international classification of diseases)?

A

Both provide standardised criteria for diagnosing mental disorders and are widely used by clinicians worldwide

Both based on empirical research and periodically upadtes to reflect scientific knowledge

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41
Q

What are the differences in DSM approaches to diagnosis and classification of mental illness (with ICD - international classification of diseases)?

A

DSM is more detailed and focussed on psychiatric diagnosis, while ICD has a broader scope, including all diseases and health conditions

ICD uses a dimensional approach –> more flexible categories compared to DSM’s more rigid approach

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42
Q

What is anxiety

A

Anxiety is when an individual experiences intense, excessive and persistent worry and fear about everyday situations

This is typically activated in response to a perceived threat

Experience of anxiety is the same in normal anxiety and abnormal anxiety (severe, often and excessive)

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43
Q

Why does anxiety occur

A

Activation of physical, cognitive and behavioural systems

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44
Q

Explain the physical elements of anxiety (i.e. physical symptoms)

A

Increased heart rate and blood pressure

Stress hormones and diff neurotransmitters being released

Breathing speeds up to get more O2 for muscles

Saliva production reduces

Body tenses up

We sweat

Digestion slows down as blood flows away from stomach to muscles

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45
Q

Explain the cognitive elements of anxiety

A

Refer to how you pay attention, what you attend to, and your interpretations of the situation / object / person

1) perception of threat

2) Attentional shift towards threat

3) hypervigilance to source of threatening info –> difficulty concentrating on other tasks

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46
Q

Explain the behavioural elements of system (i.e. coping measures)

A

Avoidance behaviours

Restlessness and fidgeting as a way of releasing nervous energy

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47
Q

What is Yerkes Dodson Law

A

Suggests that there is an optimal arousal which allows for the best performance

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48
Q

Explain abnormal anxiety

A

Typically characterised by overestimation of threat (excessive and disproportionate)

Perceived higher probability of a negative outcome

Perceived cost of negative outcome is often increased as well

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49
Q

Differentiate between normal and abnormal anxiety

A

Normal anxiety is typically proportionate to the situation, and occurs out of an evolutionary need for survival

Abnormal anxiety is typically a lot more out of proportionate reaction/anxiety in response to a situation

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50
Q

Explain key differences between DSM 4 and DSM 5 anxiety and related disorders

A

Introduction of selective mutism

Got rid of Acute stress disorder, posttraumatic stress disorder, obsessive compuslive disorder as specific disorders. Instead, they reintroduced them under umbrella terms like ‘trauma and stressor related disorders’ or ‘obsessive compulsive and related disorders’

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51
Q

What is a panic attack

A

This describes a discrete period of intense fear or discomfort that appears abrupty and peaks usually within 10 mminutes

Classic symptoms of autonomic arousal (sweating, pounding heart, shaking, shortness of breath, nnausea, dizziness etc) –> could also be followed by a fear of dying, losing control, going mad etc

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52
Q

What is a specifier

A

Extensions to a diagnosis to further clarify a disorder or illness. They allow for a more specific diagnosis

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53
Q

What is a panic disorder? What is it characterised by?

A

This is recurrent unexpected panic attacks (unknown triggers)

At least one of the attacks was followed by 1 month (or more) of one or both of the following; persistent concern/worry about having another attack, significant maladaptive change in behaviour relative to attacks

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54
Q

What are panic related behaviourss?

A

Avoidance (of situations where panic is likely and of activities that produce panic like sensations)

Escape

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55
Q

What is the difference between panic attack and disorder

A

Panic attacks may be experienced in everyday situations, and fear focuses on negative evaluation by others and expected social embarassment

Meanwhile, panic disorders is when the attack itself becomes a problem, as indicated by fear of future attacks and altered behaviour in response to attacks (fear of fear)

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56
Q

What are some stats about panic disorder

A

~5% of Aus has had PD in lifetime, about 3-4% in any 12 month period

Most common in females

Onset in early adulthood

Comorbidity (depression, agoraphobia)

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57
Q

Describe the Clark cognitive model of panic disorder

A

1) Typically benign triggering stimulus

2) This stimulus is perceived as a threat and a sign of imminent physical or psychological catastrophe

3) Catastrophic misinterpretation

4) Anxiety response: misinterpretation —>heightened anxiety and activation of fight or flight response including release of adrenaline which intensifies the physical sensations like HR or shortness of breath

5) panic attack as these feelings are misinterpreted as a panic attack –> cycle continues

58
Q

Describe the key treatment elements for panic disorder

A

CBT: Psychoeducation, exposure and interoceptive exposure (graded exercises that induce the physical responses of a panic attack, repeated and sufficient duration, challenged beliefs about physical sensations and extinguish conditioned anxiety)

59
Q

What are the key features of specific phobia

A

A marked and consistent fear reaction to presence or anticipation of a specific object or situation

Anxiety experiences is out of proportion to the actual threat

Persistent - lasting 6 months or more

Phobic stimulus is avoided or endured with intense fear

Fear/anxiety/avoidance causes clinically significant distress or impairment in socia, occupational or other important areas of functioning

60
Q

What are some examples of subjects of phobia

A

Could include animals, blood, injections, nature etc.

61
Q

What are the potential causes of specific phobias

A

Could be due to classical conditioning (bad experience –> fear response –> phobia). However, conditioning isn’t the only cause

Some stimuli is more likely to become phobic than others (i.e. knives, guns)

Some phobias are threats to survival during evolution (i.e. phobias related to survival, such as snakes, spiders and heights are common)

62
Q

Explain the prevalence and course of specific phobia

A

COmmon in children and more intense in adults

7-9% of adults have a specific phobia

More common in females (2:1) - particularly animal, environment, situational (blood/injection/injury)

Sometimes develops after a traumatic event, observing trauma, information transmission

Can develop in childhood and adulthood as well

63
Q

What is the difference between normal fears and specific phobia as a mental health condition

A

Normal anxiety = feeling queasy while climbing a tall ladder

Phobia = avoiding climbing stairs because its tall and you’re scared of heights

Normal anxiety = worrying about taking off in a plane during a storm

Phobia = turning down a big promotion because it involves air travel

64
Q

What is Generalised Anxiety Disorder(GAD)

A

The key symptom for a diagnosis of Generalised Anxiety Disorder is excessive worrying about a variety of different outcomes (rather than about one feared outcome, as in the other anxiety disorders).

The person finds it difficult to control their worrying and experiences a number of other symptoms, such as muscle tension, irritability, or sleep problems. The worries tend to be related to everyday life, such as work, study or relationship. However, as in the case in all other anxiety disorders, the worrying is out of proportion to the actual threat involved.

65
Q

DSM 5 classification of GAD

A

Excessive anxiety and worry (apprehensive expectation), occurring more days than not for at least 6 months, about a number of events or activities (such as work or school performance).
The individual finds it difficult to control the worry.
The anxiety and worry are associated with three (or more) of the following six symptoms (with at least some symptoms having been present for more days than not for the past 6 months):
Restlessness or feeling keyed up or on edge.
Being easily fatigued.
Difficulty concentrating or mind going blank.
Irritability.
Muscle tension.
Sleep disturbance (difficulty falling or staying asleep, or restless, unsatisfying sleep).
The anxiety, worry, or physical symptoms cause clinically significant distress or impairment in social, occupational, or other important areas of functioning.
The disturbance is not attributable to the physiological effects of a substance (e.g., a drug of abuse, a medication) or another medical condition (e.g., hyperthyroidism).
The disturbance is not better explained by another mental disorder.

66
Q

Explain GAD prevalence and course

A

6.1% of people get it within their lifetime

Age of onset at around 31-39 years

Higher prevalence of females

Clinical course: most people w/ GAD dont seek help from mental health professionals, and those that do delay by over 10 years. Fluctuations in severity over time

67
Q

Explain the diagnostic features of GAD

A

Associated with 3+:

Restlessness, easily fatigued, difficulty concentrating, irritability, muscle tension, sleep disturbance

Excessive and uncontrollable worry (about wide range of events or activities)

Constant negative stream of consciousness - tihings that could go wrong, worrying about worrying, “what if?”

68
Q

What are the features and associated thought processes of GAD

A

Excessive and uncontrollable worry, which could be based on but not limited to; professional worries, finances, personal health, world events, minor matters

The focus of worry may shift fro one concern to another. Typically the intensity/duration/frequency of worry is out of proportion to actual likelihood or impact of anticipated event

Process of catastrophising can occur automatically and quickly escalates

Associated with high trait anxiety, intolerance of uncertainty, reduced ability to tolerate distress, reduced problem solving confidence

69
Q

What is obsessive compulsive disorder (OCD)?

A

Obsessive-Compulsive Disorder (OCD) is a mental health condition characterized by persistent, intrusive thoughts (obsessions) and repetitive behaviors or mental acts (compulsions) that an individual feels driven to perform. OCD can significantly impact daily life and functioning, as individuals experience intense distress over their obsessions and often perform compulsions to reduce the anxiety caused by these thoughts.

A key feature is the idea of a THOUGHT-ACTION FUSION - where they may think their thoughts are more than just thoughts and instead think that thoughts = action (i.e. thinking of killing someone means you actually kiilled them)

70
Q

What are obsessions in ocd?

A

Repeated, intrusive, irrational thoughts or impulses which cause severe anxiety or distress

71
Q

What are compulsions in ocd?

A

Reptitive/ritualised behaviours or mental acts to neutralise obsessions / reduce anxiety

72
Q

What are some common compulsions

A

Washing/cleaning

Checking

Repeating

Ordering and arranging

Mental compulsions

73
Q

What are some common obsessions

A

Contamination

Symmetry/order

Doubt/harm

Forbidden/taboo

74
Q

What is the (potential) link between obsessions and compulsions

A

Can have one without the other but both are normally connected

OCD might be connected (contamination obsessions –> cleaning compulsion)

OCD might not be connected (forbidden obsessions –> cleaning compulsion)

75
Q

What is OCD commonly associated with

A

Intolerance of uncertainty

Inflated responsibility

Thought action fusion (thinking about something is as bad as doing it) (thinking abt something makes it more likely to happen)

Medical ideation (If i dont step on the cracks, I will pass the exam)

76
Q

What are the treamtnets for anxiety disroders

A

CBT, Psychoeducation, cognitive, behavioural, and biological treatments

77
Q

Explain how CBT is used to treat anxiety disorders

A

Aims to reduce threat appraisals. Involves:

Encouraging patients to have a decreased likelihood of perceived harm, encouraging patients to decrease their perceived cost of the harm. Exposure gradual is also an essential ingredient

Likelihood + cost + exposure = effective treamtent (hopefully)

Also acceptance and treatment therapy is effective as CBT

78
Q

Explain how psychoeducation is used to treat anxity disorders

A

Person’s specific triggers, responses and impacts on their life is written down

Involves explaining to the person the idea of anxiety in general

Role of avoidance is taught (taught that anxiety can worsen with avoidance)

teach relaxation tehcniques to address fight or flight response

79
Q

Explain how cognitive techniques are used to treat anxiety disorders

A

Involves cognitive restructuring and challenging thoughts

thought diaries: to recongise automatic thoughts

Socratic questioning (am I making assumptions? COuld I misinterpret things? Am I looking at all the evidence or just what supports my thoughts? )

80
Q

Explain how behavioural techniques are used to treat anxiety disorders

A

Exposure therapy - graded exposure (very important)

Could be imaginal (making the person imagine the scenario and walking them through that experience)

In vivo (do it live)

Involves development and progression through fear hierarchy (i.e. ranking different things from 1-100, with 100 being the most scary thing, and then walking through the differnt parts of the hierarchy)

Normally coupled with relaxation techniques

These ultimately also influence cognition (exposure to feared stimuli/situation reduces judgement of probability of harm, and exposure to feared outcomes reduces judgements of cost and harm)

81
Q

Explain how biological treatments are used to treat anxiety. WHat are the issues of this?

A

Treats the symptoms and NOT the cause

Can be useful in STT

Barbituates: Quick acting - addictive, risk of OD, interacts with alc, and high relapse rate

Benzodiazepines: Quick acting - addictive, interacts with alc, high relapse rate

Antidepressants - SSRIs: Slower effects - fewer side effects, however relapse is still common

Ultimately the gold standard would be CBT + medication to treat an issue

82
Q

What are mood disorders characterised by

A

These are characterised by a disturbance in mood

They are episodic.

83
Q

What are mood disorders

A

Mood disorders are a category of mental health conditions characterized by significant disturbances in a person’s emotional state or mood, which can severely impact their daily functioning and quality of life. Mood disorders primarily involve prolonged periods of extreme sadness, elevated mood, or a combination of both. The most common mood disorders include major depressive disorder, bipolar disorder, dysthymia (persistent depressive disorder), and cyclothymic disorder.

84
Q

What is the difference between a unipolar and bipolar mood disorder

A

Unipolar is where the patient only experiences a certain extreme of a type of mood (i.e. only depressive)

Bipolar is where the patient experiences the two extremes of mood –> i.e. being both manic and depressive

85
Q

What are the key differences between DSM 4 and DSM 5 mood disorders classification

A

DSM 5 had the addition of ‘disruptive mood dysregulation disorder’ and ‘premenstrual dysphonic disorder’

In DSM 4, it categorised ‘bipolar disorders’ under mood disorders, however, in DSM 5, ‘Bipolar and related disorders’ now form a distinct category, separate from depressive disorders

DSM 5 had the new addition of persistent Depressive Disorder which combined Dysthymia and chronic major depressive disorder (?!)

86
Q

Explain sadness as a life experience

A

It is mild and temporary, and is an almost universal experience

Involves feeling blue, sad, discouraged, apathetic, lack of joy

Sadness is common after -ve experiences such as death, illness, relationship breakdown, lost/missed experiences

Feelings usually fade, and people become accustomed to the new norm

87
Q

Differentiate normal sadness from depressive disorders as a mental health condition

A

If the frequency, intensity and duration of depressive symptoms are out of proportion to person’s life situation –> depressive disorder

Responses to significant loss might include feelings of sadness, insomnia, poor appetite - resmbles depressive episode. Although these responses may be understandable or appropriate to loss, we don’t want to underdiagnose depression in people who experience loss –> clinicians use judgement based on patient history, personal and cultural context

88
Q

What is a major depressive episode

A

A major depressive episode is a period of at least two weeks during which a person experiences a pervasive and intense depressive mood or a noticeable loss of interest or pleasure in most activities. This period is marked by significant changes in emotional, cognitive, and physical functioning that differ from the person’s usual state. Major depressive episodes are central to the diagnosis of major depressive disorder (MDD) but can also occur in other conditions, such as bipolar disorder.

89
Q

What are the key symptoms of a major depressive episode

A

At least 5 or more of the following symptoms during 2 week period (need 1 and 2)

1) Depressed mood most of the day, nearly everyday

2) Markedly diminished pleasure.interest in activity

3) Significant weight loss/gain

4) Insomnia or hypersomnia nearly everyday

5) Psychomotor agitation

6) Fatigue or loss of energy nearly everyday

7) Feelings of worthlessness and excessive guilt

8) Diminished ability to concentrate

9) recurrent thoughts of death, suicide, suicide attempts

90
Q

What is a major depressive disorder

A

A diagnosed mood disorder with recurring MDEs

Single or recurrent depressive episodes? Not accounted for by other disorders?

91
Q

How is Major depressive episode different to major depressive disorder

A

Different to MDE in the idea that it is the ongoing disorder that cause episodes across individuals life. MDE just describes one episode

There could be a single or recurrent depressive episode

92
Q

Explain the prevalence and course of MDD

A

Prevalence = 5-25%

Onset is typically after puberty, peaking in 20s but could happen later on in life

Females are 2x more likely to have unipolar mood disorder vs males

Comorbidity w/ anxiety and substance taking

Course variable - remission from symtpoms, number and length of episodes

Depression has 3r highest burden of all diseases

Number one cause of non0fatal disability

93
Q

What is persistent depressive disorder

A

Depressed mood for most of the days, for at least 2 years

94
Q

Explain the symptoms of persistent depressive disorder

A

Presence of two (or more of the following)

Poor appetite or overating
Insomnia or hypersomnia
Low energy or fatigue
Low self esteem
Poor concentration or difficulty making decisions
Feelings of hopelessness

Individuals havent been without these symptoms for more than 2 months at a time

No manic or hypomanic episodes

Not better explanation by ther psychotic disorders or isn’t attributed to substane abuse or medical conditions

95
Q

Explain the prevalence and course of PDD

A

Prevalence = 1-2%

Early onset

Chronic course, might be milder than MDD but still causing distress

Lacks symptom free periods

96
Q

What are the differences between MDD and PDD (Duration, severity)

A

Duration: MDD has discrete episodes ofdepression lasting at least 2 weeks, and can experience periods of normal moods, meanwhile PDD is chronic, long term version of depression, lasting for at least 2 years with rare periods of normal mood

Severity: MDD is more severe symptoms, whereas PDD is milder but more chronic

97
Q

Outline the biological theories of depression

A

Genetic vulnerability
Neurochemistry
Neuroendocrine
Vulnerability Stress motives

98
Q

Explain genetic vulnerability as a theory of depression

A

SUggests that depression was heritable

Heritability: 35-60%. Heredity causes vulnerability to mood disorders

99
Q

Explain neurochemistry as a theory of depression

A

Low levels of nroadrenaline and/or serotonin

No good evidence for causal mechanism though

100
Q

Explain the neuroendocrine system as a theory of depression

A

Excess cortisol in response to stress

increased stress correlated to mood disorders

101
Q

Explain vulnerability stress models as a theory of depression

A

Biological vulnerability + stress –> depression

102
Q

Outline the psychological theories of depression

A

Diathesis stress models

Schema theory

Ruminative response styles

103
Q

Explain the diathesis stress model as a theory of depression

A

Cognitive vulnerability (likely from childhood –> trauma) + stress –> depression

104
Q

Explain the schema theory as a theory of depression

A

Schema = stable memory structure which guides info processing

Pre existing negative schemas could result in info processing biases (i.e. only focussing on bad things)

105
Q

Explain ruminative response styles as a theory of depression

A

Thoughts cycling over and over in our mind

106
Q

What are some biological treatments for depression

A

Drug treatments - SSRIs block reputake of serotonin, and is effective in 70-80% of the cases

Electroconvulsive therapy (ECT) - brief electrical currents to brain, and it can be effective for severe depression (80%+)

However, it is common for relapse w/ biological treatments as it is treating the symptoms, not the cause

107
Q

What is the aim of CBT in depression

A

Aim is to modify dysfunctional cognitions and related behaviours

Involves psychoeducation, behavioural activation, cognitive restructuring

108
Q

Explain the cognitive aspect of CBT in treating depression

A

Address cognitive errors

Develop realistic view

Remove lens of negative schemes

NOT positive thinking, as it doesn’t normally work - patients need to know whats going on in their mind

109
Q

Explain the behavioural aspect of CBT in treating depression

A

Behavioural experiments; testing the persons beliefs, and gathering evidence to disconfirm negative beliefs

Behavioural activation: increase reinforcing/postiive events (things to look forward to, identifying goals and values, building upward spiral of motivation + energy through mastery

110
Q

Outlne the vicious cycle of depression

A

Feel depressed –> -ve thinking about self and world –> increased lethargy –> reduction of activity and social withdrawl –> loss of pleasure and achievement –> feel depressed

cycle continues

111
Q

What are the differences between DSM4 and DSM 5 feeding and eating disorders

A

DSM 5 added the avoidant/restrictive food intake disorder, pica and rumination disorder

DSM 4 had EDNOS (eating disorder not otherwise specified) - broad category for eating dosorders that didn’t meet full criteria for anorexia or bulimia nervosa. DSM 5 replaced EDNOS w/ two more specific categories (other specific feeding or eating disorder, unspecified feeding or eating disorder)

Binge eating disorder became an official, distinct eating disorder in DSM 5, whereas DSM 4 it was in EDNOS

112
Q

What are the key/diagnostic features of anorexia nervosa (AN)

A

Restriction of energy intake –> significantly low body weight in context of age, sex, developmental trajectory and physical health

Intense fear of gaining weight or of becoming fat, or persistent behaviour that interferes w/ weight gain, even at a low weight

Disturbance in way in which one’s body weight or shape is experienced, undue influence of body weight or shape on self evaluation, or persistent lack of recognition of seriousness of current low body weight

113
Q

What is the severity of AN

A

Level of severeity depends on the BMI

Mild (BMI 17-18)
Moderate (BMI 16-17)
Severe (BMI 15-16)
Extreme (BMI <15)

Level of severeity may be increased to reflect clinical symptoms, the degrees of functional disability and need for supervision to eat

114
Q

WHat are the subtypes of AN

A

Restricting and binge-eating/purging type

115
Q

Explain AN restricting type

A

During last 3 months, individuals has not engaged in recurrent episodes of binge eating or purging –> if not it is a different diagnosis

It is the presentations in which weight loss is accomplished primarily through dieting, fasting and/or excessive exercise

116
Q

Explain AN binge-eating/purging type

A

During last 3 months, individual has engaged in recurrent episodes of binge eating or purging behaviours (i.e. self induced vomiting or misuse of laxatives, diuretics or enemas)

117
Q

What are the associated psychological problems with AN

A

Depressed mood, irritability, anger, social withdrawl, preoccupation w/ food, poor concentration

Often associated with ‘starvation syndrome’ –> tough emotional experiences

Comorbid: Mood disorder, anxiety disorder, substance use disorder, personality disorder

118
Q

What are the associated physical problems with AN

A

Low body temp, brittle hairs/nails, hair growth

Osteoporosis (brittle bones)

Metabolic disturbances, heart failure

Malnutrition, anemia, immune system suppression

Mortality rate = 5-10% over 10 year period (cardiovascular complications, suicide)

119
Q

What is the prevalence of AN

A

Affects 0.5-1.0% of females

90% of individuals w/ AN are female

Rates are increasing in Aus

Age of onset is typically mid-late adolescence - appears to be getting younger

120
Q

What is the course of AN

A

Chronic, slow recovery (up to 10 years)

Treatment seeking challenges

20% remain chronically ill

Recovery depends on the definition (right BMI? right thinking?)

About 56% go on to develop Bulimia nervosa

It also has the highest mortality rate of all psychiatric diseases

121
Q

What are the key/diagnostic features of bulimia nervosa (BN)

A

Recurrent episodes of binge eating, characterised by:

Eating in a discrete period of time an amount of food that is definitely larger than what most people would eat during a similar period of time and circumstances

Sense of a lack of control over eating during the episode

Recurrent inappropriate compensatory behaviours in order to avoid weight gain –> laxative abuse, excessive exercise, fasting

Has to occur at least once a week for 3 months

Self evaluation unduly influenced by body shape

122
Q

What are the associated psychological problems with BN

A

Comorbid mood disorders, anxiety disorders, substance abuse, personality disorders (bipolar)

123
Q

What are the associated physical problems with BN

A

Associated with binges (e.g. stomach problems)

Most physical problems are associated w/ compensatory behaviours (I.e. vomiting: stomach acid –> loss of dental enamel, scarring, ulceration of oesophagus, laxatives: loss of normal bowel function, dehydration, electrolyte imbalance)

124
Q

What was the prevalence of BN

A

Affects 1-3% of females; becoming more recognised in males

90% of individuals w/ BN are female

Males - purging is less frequent, instead they normally undertake exercise as compensatory behaviours

125
Q

What was the course of BN

A

LT outcome is better than for AN

10% still affected after 10 years

126
Q

What were the key/diagnostic features of binge eating disorder (BED)

A

Recurrent episodes of binge eating (eating in a period of time, an amoun of food that is larger than what most people would eat in a similar period and circumstance)

Marked distress regarding binge eating is present

Binge eating at least once a week for 3 months

No association w/ BN or AN

Lack of control over eating during the episode. ALso includes association w/ 3 or more of the following:

Eating much more rapidly than normal

Eating until feeling uncomfortably full

Eating large amounts of food when not feeling physically hungry

Eating alone bc of embarrassment of how much they are eating

Feeling discussed with oneself, depressed or guilty afterwards

127
Q

What is the difference between BED and BN

A

BN has active actions to try compensate for their excesive eating, wheras BED is not

128
Q

What was the prevalence of BED

A

Affects 2.5% of population (2:1 female/male ratio)

Onset is adolescence to early adulthood

129
Q

Explain the course of BED

A

Remission rates higher for BED than AN/BN

Associated w/ higher rates of obesity –> more blood pressure, more cholestrol, type 2 diabetes and heart disease

130
Q

Explain the biological theories of causation in eating disorders

A

Genetic factors; moderate heritability of AN and BN, increased depression, personality disorders and substance use in families of people w/ eating disorders

Neurotransmitter disturbances; serotonin involved in appetite regulation –> mixed findings regarding direction of causation

131
Q

Explain the psychological theories of causation in eating disorders

A

AN and BN have many features in common:

Tendency to base self worth on weight/shape

Intense fear of gaining weight

Desire to attain unrealistic levels of thinnness

Transdiagnostic model

132
Q

Explain the transdiagnostic model

A

Core low self esteem, distress tolerance, interpersonal difficulties, perfectionism

133
Q

Explain the pscyhosocial theories of causation in eating disorders

A

Family factors: High parental or sibling criticism, control and conflict, comments on eating behaviours, low parental empathy

Peer factors: Peer group w/ eating disorders, social approbal

Sociocultural factors: Cultural emphasis on thinness which emerged in the late 20th century

Ultimately, the idealisation of thinness is seen as a contributing factor to normative body dissatisfaction that females experience in western cultures.

Eating disorders are prevalent in subcultures where thin ideal is amplified

134
Q

Explain the biological treatment of eating disorders

A

Medical management (i.e. medicines)

“Re feeding”

Dietician

Inpatient/outpatient depending on severeity

135
Q

Explain the psychological treatment of eating disorders for adults

A

Stage 1 (Starting well) - personalised formulation, psychoeducation, behavioural focus (self monitor and regularly eat)

Stage 2 (Taking stock) - Joint review of progress, identify problems to address and barriers to change

Stage 3 (Addressing maintaining factors) - Main body of treatment, involves addressing key maintaining factors (weight/shape, mood related eating behaviours)

Stage 4 (ending well) - Ensuring progress is maintained, relapse prevention

136
Q

Explain the psychological treatment of eating disorders for children/adolescents

A

Key emphasis on parental involvement/responsibility in home setting

2/3 are fully weight restoed, and 75-90% restored at 5 year followups

Phase 1: Parents in charge of weight restoration - “re feeding”

Phase 2: Parents transition control over eating back to adolescent (very therapeutic)

Phase 3: Discuss adolescent developmental issue, establish health identity

137
Q

Path to become psychiatrist

A

Fully-qualified medical doctor

Specialist training and qualifications in the diagnosis, treatment and prevention of mental illness

Specialised knowledge of neurobiological changes/causes of mental disorders

In general, treat people with a diagnosed mental disorder

Can prescribe medication for mental health conditions

Treatment takes a bio -medical approach (some also take a psychological approach)

  • Medical degree
  • 4-6 years (depending on whether you are
    coming into the degree as a university
    undergraduate or graduate
  • Internship
  • 1 year of hospital rotations/placement
  • Residency
  • 1 year of specialised hospital
    rotations/placements
  • Vocational Training
  • 5 years to complete a Fellowship of the Royal
    Australian and New Zealand College of
    Psychiatrists (FRANZCP)
138
Q

Path to become clinical psycholgist

A

Look at book - should be common sense

139
Q

What do social workers do? What are the qualifications and training involved

A
  1. Work in direct services (e.g., healthcare, welfare, housing etc.)
    * Helping people cope with problems related to social cultural issues including
    but not limited to poverty, legal issues or human rights
  2. Work in the clinical field (more similar to psychologists)
    * Diagnose and treat mental, behavioural or emotional health issues
    * Focus: environment and cultural factors (sociological approach) rather than
    psychological or personal characteristics (i.e. Family Therapy)
    * Help clients develop practical plans to improve personal well well-being and make
    referrals for services

Qualifications & Training

Bachelor of Social work (4 years)

Masters of Social Work (2 years + other 3 year Bachelor degree)

140
Q

What do counsellors do? What are the qualifications and training involved

A

Assist people to gain understanding of themselves and make changes in
their lives:

May simply provide a non-judgmental ‘listening ear’
May work in a particular area (e.g. marriage/family/school counsellors)

Qualifications & Training:
Level of training is wide and varied
Ranging from weekend correspondence course to Bachelor degree
The term ‘counsellor’ can be used without any particular qualification

To register as fully qualified:
Bachelor of Counselling (3 years)
Graduate Diploma in Counselling (2 years) + previous Bachelor degree

141
Q

What are the restricted titles

A

Psychologist is a restricted title. Can only use this if you are registered with the psychology board register

142
Q

What are some unrestricted titles

A

Counsellor, Therapist, Psychotherapist, Life
Coach, Spiritual Advisor