Mastitis Flashcards
3 ways organisms can gain entry?
- galactogenic
- haematogenous
- percutaneous
6 ways to resist infection?
- smooth muscle sphincter
- keratin from ep
- Furtenberg’s rosette
- leukocytes
- lactoferrin (from ep, sequesters Fe that bacteria need)
- defensins and Ig
peracute presentation?
life-threatening , gangrenous , near parturition, systemic signs
Gross: swollen, painful, blue-black, blistered, oozing, cold to touch, haemorrhagic lobules, ventral abdomen oedema, toxaemia
Acute presentation?
with or without systemic signs
interstitial oedema with neutrophilic infiltration
Chronic presentation?
progressive loss of secretory ability
fibroplasia, ducts may be obstructed by polyps
can slough a quarter
hard and atrophic gland
Subclinical presentation?
detect by SCC over 200,000
no clinical signs and no milk changes
Key organisms for lactating mastitis?
- staph aureus
- strep agalactiae
- strep dysgalactiae
- step uberis
- e.coli
- mycoplasma bovis
( corynebacterium bovis, pseudomonas aeruginosa, klebsiella pneumoniae, yeasts / fungi )
dry cow organisms?
arcanobacterium pyogenes
strep dysgalactiae
peptostreptococcus indolicus
staph aureus mastitis?
contagious
subclinical to gangrenous
lives on skin - teat
very hard to treat
no vaccine
strep agalactiae mastitis?
contagious
subclinical
naturally in teat
host adapted
no vaccine
strep dysgalactiae mastitis?
contagious
acute
naturally outside teat and needs wound to invade
not host adapted
no vaccine
strep uberis mastitis?
contagious / envt
acute / subclinical
naturally in env’t and needs trauma to enter
no vaccine
e.coli mastitis?
environmental
acute
ABX resistant
killed J5 vaccine
mycoplasma bovis mastitis?
acute
haematogenous from genital / resp tract
3 way to present mastitis?
dry vs lactating
contagious vs environmental
clinical vs subclinical