Lumbosacral Disease Flashcards

1
Q

cauda equina

A

bundle of nerve roots caudal to the conus medullaris

peripheral nerves NOT spinal cord

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2
Q

conus medullaris

A

end of the spinal cord

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3
Q

do all spinal segments line up with the vertebral bodies

A

NO - spinal cord is shorter than the vertebral column

lines up from T1 to L2

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4
Q

in dogs - what vertebrae does the sacral cord begin at

A

sacral cord begins at L5

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5
Q

where do majority of nerve roots exit from the vertebrae to become spinal nerve

A

exit out of the foramen CAUDAL to the corresponding vertebrae

(ex. spinal nerve L2 exits out of the foramen between L2 and L3)

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6
Q

what are the short spinal nerves

A
  • cervical
  • thoracolumbar

just need to travel straight across because T1 to L2 lines up with corresponding vertebrae

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7
Q

what are the long spinal nerves

A
  • caudal lumbar
  • sacral
  • caudal

have to travel caudally before they can exit
(ex. spinal nerve L7 originates at L4-5 and must travel down to L7-S1 to exit)

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8
Q

where do nerve roots C1-7 exit from the vertebrae to become spinal nerves

A

exit out of the foramen CRANIAL to the corresponding vertebrae

(ex. spinal nerve C2 exits between C1 and C2)

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9
Q

is there a C8 spinal nerve and vertebrae

A

there is a C8 spinal nerve but NO C8 vertebrae

C8 spinal nerve exits between C7 and T1

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10
Q

what structure do all sacral and caudal nerve roots pass over before exiting out of the vertebral foramen

A

L7 to S1 disc space

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11
Q

what are the differences between the cauda equina and spinal cord

A

cauda equina is made of peripheral nerves NOT spinal cord

  • more robust/hardy; can be manipulated during surgery if needed
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12
Q

where does the spinal cord end/cauda equina begin in dogs and cats

A

L7 to caudal nerve roots

dogs: L6-7
cats: L7-S1

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13
Q

what are the most important peripheral nerves that arise from the cauda equina

A
  • sciatic nerve (L6 to S2)
  • pudendal nerve (S1 to S3)
  • pelvic nerve (S1 to S2)
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14
Q

cauda equina syndrome (CES)

A

group of neurologic signs caused by a lesion affecting the cauda equina

can NOT differentiate from a L7-caudal myelopathy (normal thoracic limbs, LMN signs in pelvic limbs)
- must rule out L7-Cd myelopathy

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15
Q

general clinical signs of CES and L7-Cd myelopathy

A
  1. motor
  2. bladder
  3. sensory
  4. patellar reflex
  5. apparent pain
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16
Q

motor signs CES/L7-Cd

A

LMN signs in PLs, tail, and anus
- tail: reduced carriage, wag, flaccidity
- anus: reduced/absent perineal reflex, fecal incontinence
- PLs: reduced withdrawal and gastrocnemius reflexes, plantigrade stance, reduced flexion of hock and stifle, neurogenic muscle atrophy, difficulty rising/jumping, scuffing, paraparesis

note: NOT paraplegic - hip flexors are still functional

17
Q

bladder signs CES/L7-Cd

A

unable to urinate or completely empty bladder

loss of bladder tone on palpation

dribbling urine (history of UTI)

18
Q

sensory signs CES/L7-Cd

A
  • CP deficits
  • abnormal sensation (paresthesia, hyperesthesia, hypesthesia, anesthesia of PLs and tail)
19
Q

patellar reflex signs CES/L7-Cd

A

“pseudo-hyperexaggeration”
- appears like UMN signs but is not
- actually caused by a prolonged extension phase of the reflex –> “lazy extension” (slow not exaggerated)

caused by loss of antagonism on the muscles innervated by the femoral nerve by muscles innervated by the sciatic nerve

20
Q

apparent pain of CES/L7-Cd

A
  • reluctance to jump/rise
  • pain on tail extension
  • pain on spine palpation
  • hunched/crouched position
  • pain on LS joint palpation
  • lameness

compensation for PL lameness by putting weight on TLC

21
Q

CES diagnosis

A

rule out myelopathies using:
- history, PE, NE, minimum database, CSF analysis
- dynamic rads of entire vertebral column
- CT, MRI
- electrophysiology (NM disease)
- aspirates - cytology or culture
- surgical exploration

22
Q

what are differentials for CES

A
  • degenerative (IVDD)
  • anomalous
  • infectious
  • trauma
23
Q

lumbrosacral stenosis

A

degenerative condition that occurs secondary to IVDD type II, hypertrophy/hyperplasia of ligaments, thickening of vertebral arch/facets +/- instability

24
Q

lumbosacral stenosis signalment

A

breeds: large breed (GSDs)
age: middle to older

25
Q

lumbosacral stenosis clinical signs

A

slowly progressive trouble rising

ranges from no deficits +/- pain to LMN signs with urinary and fecal incontinence

can be asymmetrical lameness

26
Q

LSS diagnostics

A
  • ortho exam
  • spinal rads
27
Q

LSS treatment and prognosis

A

tx: conservative vs surgical (dorsal laminectomy + stabilization)

prognosis: good if mild signs, poor if incontinent

28
Q

congenital malformations causing CES

A

spina bifida
myelomeningocele
sacrocaudal dysgenesis

causes compression/disease of the cauda equina

29
Q

discospondylitis

A

infection of the disc and adjacent vertebral bodies

common at lumbosacral junction

often affects multiple endplates

30
Q

causes of discospondylitis

A
  • direct (bite wound, migrating grass awn)
  • hematogenous (dental disease, endocarditis)
31
Q

discospondylitis signalment

A

breed: large
age: young but can be any

32
Q

discospondylitis clinical signs

A
  • fever
  • marked spinal hyperesthesia
  • NO neurologic deficits
33
Q

discospondylitis diagnosis

A
  1. radiographs - end plate destruction, aggressive bone lesion, +/- pathologic fractures
  2. blood and urine cultures
  3. disc space aspirates
34
Q

discospondylitis treatment and prognosis

A

tx: long term antibiotics (4-6 months) + rest

prognosis: good for bacterial, poor for fungal

35
Q

trauma to cauda equina

A

very common - CE is able to tolerate more instability and displacement than the spinal cord

treatment is surgical