Leg & Muscle Contraction Flashcards

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1
Q

What ankle ligament is commonly injured and how

A

Anterior talofibular ligament in a low ankle sprain
Due to overinversion/supination of foot
“Always tears first”

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2
Q

Most common high ankle sprain

A

Anterior inferior tibiofibular ligament

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3
Q

Signs of lumbosacral radiculopathy

A

paresthesia & weakness related to specific lumbosacral spinal nerves

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4
Q

What is the main cause of radiculopathy and how does it occur

A

Intervertebral disc (nucleus pulposus) herniates posterolaterally thro annulus fibrosus due to thin posterior longitudinal ligament

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5
Q

The nerve above/below is affected in a herniation?

A

Below herniation

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6
Q

What tests are positive in lumbosacral radiculopathy

A

Straight leg raise
Contralateral straight leg raise
Reverse straight leg raise (femoral stretch)

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7
Q

In L3-L4 herniation, which nerve root is affected & what is the clinical findings?

A

L4
weakness of knee extension & decreased patellar reflex

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8
Q

In L4-L5 herniation, which nerve root is affected and clinical findings

A

L5
Weakness in dorsiflexion & heel walking

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9
Q

L5-S1 herniation - nerve root & clinical findings

A

S1
Weakness in plantarflexion & toe walking
Decreased Achilles reflex

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10
Q

Describe the contraction velocity, fiber colour and predominant metabolism of Type I skeletal muscle fibres

A

Contraction velocity: Slow
Fiber colour: Red
Predominant metabolism: Oxidative phosphorylation (sustained contraction)
“1 slow red ox”

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11
Q

Is the number of mitochondria, myoglobin high or low in Type I and II skeletal muscle fibres

A

Type I: high
Type II: low

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12
Q

Type of training that increases type I skeletal muscle fibers

A

Endurance

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13
Q

Type of training that increases type II skeletal muscle fibers

A

Weight/resistance training, sprinting

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14
Q

Describe the contraction velocity, fiber colour and predominant metabolism of Type II skeletal muscle fibres

A

Contraction velocity: fast
Fiber colour: white
Predominant metabolism: Anaerobic glycolysis
“2 fast white antelopes”

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15
Q

What happens to myofibrils in skeletal muscle atrophy and how

A

Decreases
via removal via ubiquitin-protease system

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16
Q

What happens to myonuclei in skeletal muscle atrophy and how

A

Decreases
via selective apoptosis

17
Q

What happens to myofibrils in skeletal muscle hypertrophy and how

A

Increases
via addition of sarcomeres in parallel

18
Q

What happens to myonuclei in skeletal muscle hypertrophy and how

A

Increases
via fusion of satellite cells, which repair damaged myofibrils (absent in cardiac muscles)