Lecture Exam 2 Flashcards

1
Q

Response of VE to exercise

A

Increase linearly and then greater linearly at AnT
Lower VE in trained individuals
Trained individuals can go to greater work rate and higher VE

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2
Q

Reponse of VCO2 to exercise

A

Increase linearly and then greater linearly at AnT
Lower VCO2 in trained individuals
Trained VCO2 can go to greater work rate and higher VCO2

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3
Q

Response of VO2 to exercise

A

Increases linearly until plateau or decrease

Trained individuals can go to higher VO2 and work rate

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4
Q

Causes for breakaway (AnT) in VE

A
  1. Increase (breakaway) in VCO2

2. Increase lactate (Breakaway), decrease pH due to an increase of H+

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5
Q

Cause for breakaway (AnT) in VCO2

A

Breakaway in lactate production

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6
Q

Training effects on:
VE
VCO2
VO2

A

Decrease
Decrease
Increase VO2 max

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7
Q

Why does VE change with training

A

Increase ability for gase exchange:

Greater capillarisation
Larger lung volume
Greater alveolar VE due to neural adjustments
Greater blood volume and Hb levels

Decrease sensitivity for chemoreceptors to respiratory stimulators such as CO2 and Lactate in blood

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8
Q

Why does VCO2 change with training

A

Slower production of CO2 in the conversion of pyruvate to acetyle CoA and krebs cycle

Less buffering of lactic acid into CO2 and H2O

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9
Q

Why does VO2 change with training

A

Improved metabolic/biomechanical efficiency

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10
Q

Primary muscle fibers for:
< AT

> AT

A

< AT:
SO, FOG
Fat - mix oxidation
Aerobic

> AT:
FOG, FG
Mix - CHO
Increase lactic acid
Anaerobic
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11
Q

Alveolar ventilation rate = VA

VA =

A

(Vt - Vd) x f

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12
Q

Trained individuals have a greater VA because

A

Of the slow and deep breathing patterns which cause greater Vt

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13
Q

Vt depth and rate of breathing in trained athletes is

A

Slower and deeper than untrained athletes

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14
Q

PO2 and PCO2 move from…

A

Areas of high Pp to an area of lower Pp due to diffusion

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15
Q

Atmospheric air PO2 : PCO2 =

A

159 mmHg ; 0.3 mmHg

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16
Q

Alveoli PO2 : PCO2 =

A

100 mmHg : 40 mmHg

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17
Q

What is the reason for a decrease in PO2 and an increase of PCO2 from the atmospheric air and air in lungs

A

Due to dilution of atmospheric air with the residual lung gases

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18
Q

Time for gas exchange at
Rest =
Exercise =

Limiting factor?

A
  1. 75 seconds
  2. 3 - 0.4 seconds

However it is not a limiting factor because it only takes 0.3 seconds for complete gas exchange to occur

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19
Q

Diffusion of O2 happens in:

A

Lungs: Alveoli to pulmonary capillaries

Muscle tissue: muscle capillaries to muscle tissue

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20
Q

Diffusion of CO2 happens in:

A

Lungs: Pulmonary capillaries to alveoli

Muscle tissue: muscle tissue to muscle capillaries

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21
Q

PO2 is greatest when and where

A

Before the exchange

In arteries (100)

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22
Q

PCO2 is greatest when and where

A

After the exchange

In veins (46)

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23
Q

When you breathe in it causes pressure to

A

Increase

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24
Q

When you breathe out it causes pressure to

A

Decrease

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25
Q = P/R
Cardiac output = Pressure / resistance
26
What is pressure gradient indicated by
MAP (mean arterial pressure) Best indicator of driving force of circulation***
27
MAP (mean arterial pressure) =
Diastolic + 1/3 of systolic - diastolic Best indicator of driving force of circulation***
28
Blood flows from an area of
High pressure to an area of low pressure From: Left ventricle to aorta, arteries, arterioles, capillaries, venules, veins and back to right atrium
29
Pressure gradient in: 1. Arteries 2. Arterioles 3. Capillaries 4. Venules 5. Veins
1. Dramatic drop in MAP 2. Increase systemic vascular tree 3. Increase systemic vascular tree 4. Pressure gradient low 5. Pressure gradient low, contraction of skeletal muslce pushes blood back to heart
30
Factors that increase venous return:
Muscle pumping Ventilatory or respiratory pumping Vasoconstriction of veins Pressure head
31
What happens in the ventilatory/respiratory pumping
Pressure in thorasic decrease Increase in abdominal pressure beacuse of diaphragm is pulled Blood flows from abdominal to thoracic pushing blood to right atrium
32
What is vasoconstriction
Reflex constriciton of veins Drains the muscle Controlled by CNS
33
What effects peripheral resistance
Viscosity Length of circulatory pathway Vasoconstriction/Vasodilation
34
What happens to peripheral resistance when: Viscosity increases Viscosity decreases Length of circulation increases Length of circulation decreases Vasoconstriction Vasodilation
Increases Decreases Increases Decreases Increases Decreases
35
Increase in cardiac output due to
Decrease resistence and therefore a decrease viscosity and length Increase in pressure gradient during exercise (Increase SV and HR) Increase blood volume following training
36
During exercise, the %Q is distributed increases in... Decrease in...
Skeletal muscle and skin Kidneys, abdomin and other tissue
37
During exercise, the pressure decreases due to
Vasodilation of arterioles and muscle capillaries in active skeletal muscle
38
What is Fick equation
VO2 = Q x (A - VO2 difference)
39
VO2 = Q = A-VO2 difference =
Oxygen uptake rate Cardiac output Oxygen extraction
40
Exercise affects on components of ficks equation
Acute training: ``` Increases: Maximal and submaximal values due to: 1. Increase Mitochondria 2. Increase Myoglobin 3. Capillarisation 4. Oxidative enzymes and cytochrome activity ```
41
Cardiac responses from rest to submax to max workloads: ``` Q SV HR VE VO2 mm blood lactic acid Muscle blood flow ```
Q = increases Due to HR only SV = slight increase and then plateau Due to HR only HR = Same max HR in trained and untrained Untrained just reaches max heart rate at lower workload VE = Increase with greater than linear increase at AnT VO2 = Increase with constant or drop at top/max Trained individuals had less VO2 max and reaches it at heavier workload mm Blood LA = Production of lactic acid come at higher workload Muscle blood flow = increases
42
Increase EDV + Decrease ESV =
Increase SV
43
EDV is directly related to...
Ventricular volume and venous return
44
ESV is directly related to...
Contractility of myocardium and peripheral resistance
45
Pulmonary diffusion capacity increase from...
Rest to submax to max
46
What are the factors that effect Pulmonary diffusion capacity Changes in these factors from illness will...
1. Alveolar membrane - smoking 2. Interstitial fluid - not enough H20 3. Capillary membrane plasma - damage diabetes 4. Red blood cells - anemia Decrease PDC and endurance
47
Diffusion pathway (movement of O2 through tissue) can occur in...
Alveolar membrane Interstitial fluid Capillary membrane plasma Red blood cells
48
An increase in diffusion pathway occurs due to:
Capillaries open around alveoli Increase contact area Increase O2 diffusion
49
Trained people have higher PDC because:
More capillaries open around alveoli Size of alveoli is greater accounting for larger lung volumes Trained individuals have higher blood volume Trained individuals have higher hemoglobin levels
50
Factors determining oxygen in the blood
1. Ventilation rate (VE) 2. Pulmonary diffusion capacity (PDC) 3. Characteristics of diffusion pathway 4. Diffusion gradient and diffusion time - decreases with altitude 5. Altitude above 1500m 6. Characteristics of blood - RBC and Hb levels
51
For every increase of 1000m above 1500m, the VO2 decreases by...
10 %
52
Greater RBCs =
Greater Oxygen in blood
53
What is the greatest transports of oxygen (99%)
Hemoglobin
54
The PO2 determines...
The % of oxygen that is saturated with hemoglobin
55
Allosteric protein does what
Enhances O2 availability by two fold
56
Hemoglobin has binding sites for effectors that...
Can alter binding of other molecules and substrates like CO2 and H+
57
What is Cooperative within hemoglobin
When 1 oxygen attaches to hemoglobin it causes the second binds more easy and then the third and then the fourth even more easily.
58
The Hb O2 curve shifts to the right during exercise because of:
Decrease pH Increase PCO2 Increase Temp Increase DPG No effect on O2 loading on Hb Increase O2 availability to muscle tissue (unloading)
59
What does the curve reveal about the lungs and muscle tissue
Lungs: Hb is almost completely saturated with oxygen where PO2 is 100mmHG Muscle tissue: PO2 10 - 30 mmHg Hb has less affinity for O2 and therefore O2 is released from Hb so that it can diffuse into the muscle
60
During exercise, what effects and shifts occur
Shift to the right Increase oxygen availability for muscle tissue Bohr effect Haldene effect
61
What is the Bohr effect
Increase PCO2 and H+ = enhanced release of oxygen from Hb
62
What is the Haldene effect
High PO2 in alveoli enhances the release of CO2 and H+ from Hb in the lungs Enhances removal of Co2 and H+ from body
63
What is the 1st major adaptation in high altitude Hours DPG Curve Effects
After 48 hours Increase in 2-3 DPG levels ** Shifts curve to right = increases the amount of oxygen released to muscle tissues and increases endurance performing capabilities
64
What is epogen
Prescription eyrthropoietin = Increases RBC and Hb levels, oxygen carrying capacity Also increases viscosity which is not good = Heart attack
65
What are the exercise effects of EDV and ESV
During exercise: EDV increases ESV decreases Increases SV
66
Exercise effects on the heart contractility
Exercise increases contractility of heart due to increase activity of the sympathetic nerveous system and frank starling law Increase EDV puts a pre-stretch on the myocardium which results in greater force contraction ESV is decreased and SV increases
67
Endurance training increases:
Blood volume and ventricular volume
68
Strength training increases
Ventricle wall thickness
69
Difference between trained and untrained at rest: ``` Q SV HR A-V difference VE VO2 mm LA Muscle blood flow ```
``` Q = Same at rest ** SV = Higher in trained HR = Lower in trained A-V difference = Same at rest ** VE = Lower in trained but about the same at rest VO2 = Same at rest ** mm LA = Same at rest ** Muscle blood flowSame at rest ** ```
70
Difference between trained and untrained at submax and max ``` Q SV HR A-V difference VE VO2 mm LA Muscle blood flow ```
Q = Continues to increase in trained due to increase HR SV = Higher in trained due to increase blood volume and wall thickness and increase venticular volume = increase myocardial efficancy HR = lower in trained due to increase myocardial efficancy = less workload needed to maintain workload, increased vagus nerve domin A-V difference = increases at submax to increase in max VO2 due to increase Q VE = Lower in trained VO2 = Lower due to improved cap, myoglobin and enzymes mm LA = increases at higher workload due to higher lactate acid tolerance, breakaway at AnT
71
Trained individuals have greater: What type of HR do they have
``` Blood volume Capillaries Hb concentrations Mitochondria in muscle tissues Myocardial efficancy Myoglobin Oxygen enzymes Ventricular volume Vall thickness ``` Bradycardia (endurance training) = due to increase vagus control
72
Influences on cardiorespiratory responses
``` Increase cerebral cortex activity Increase Kinesthetic feedback Increased chemorecptor response Increase catecholamine release Increase Temp Increase altitude O2 Enrichment Smoking = Increases airway resistance Blood doping = Increases OCC ```
73
How does increase cerebral cortex activity have an influence on: Ventilation HR & SV Blood vessels
``` Increase HR Increase SV Increase vasodilation heart and muscles Increase Ventilation ** Increase vasoconstriction of other tissue ```
74
How does increase kinesthetic feedback have an influence: Ventilation HR & SV Blood vessels 1st onset of exercise is from...
``` Increase HR Increase SV Increase vasodilation heart and muscles Increase Ventilation ** Increase vasoconstriction of other tissue ``` Joint receptors
75
How does Increase chemoreceptor reponse have an influence: Ventilation HR & SV Blood vessels
Increase PCO2 = Increase Ventilation, HR and SV Decrease pH = Increase Ventilation, HR and SV Decrease PO2 = Increase Ventilation, HR and SV PCO2 and pH cause vasoconstriciton of other tissues PO2 causes vasodilation in heart/muscles and vasoconstriction of other tissues
76
How does increase catecholamine release have an influence: Ventilation HR & SV Blood vessels
``` Increase HR Increase SV Increase vasodilation heart and muscles Increase Ventilation ** Increase vasoconstriction of other tissue ```
77
How does increase temp have an influence: Ventilation HR & SV Blood vessels
Increase HR Increase SV Increase vasodilation heart and muscles Increase Ventilation **
78
How does increase altitude have an influence on PO2
Decrease P ATM = decrease PO2
79
How does smoking have an influence
Increases airway resistance | Increase CO = Decreases Hb that is saturated with O2
80
How does blood doping have an influence
Increase oxygen carrying capacity
81
Oxyhemoglobin is..
In relaxed state, all heme complexus exposed
82
Deoxyhemoglobin is..
oxygen forced off heme structure and goes to cells of the body during exercise
83
Oxy goes to Deoxy...
To give cells oxygen through bonding of amino acid chains
84
Oxygen carrying capacity = Females have...
Hb + RBCs Lower than males as they lose oxygen during menstral cycle
85
How much CO2 is transported in the blood
7 - 10% Is important for PCO2 and regulating cardiorespiratory responses
86
In plasma CO2 + H2O =
H2CO3 which dissocaited into HCO3 and H+ HCO3 is buffered by plasma proteins
87
In RBCs CO2 + H2O =
H2CO3 (carbonic acid) which is converted by carbonic anhydrase to form HCO3 and H+ H+ is buffered by Hb 60 - 70% carried by carbonic acid
88
How much CO2 Combines with Hb
23 - 30% = carbaminogemoglobin
89
What is barconate eliminated by
Kidneys
90
What happens in the Haldene effect
High PO2 leads to release of H+ and CO2 from hemoglobin H+ and CO2 in alveolar capillaries diffuse to alveoli where they are eliminated with expiration An increase in PO2 and PCO2 = more CO2 eliminated from body and more CO2 transported in blood
91
Lactate breakaway at AnT is due to
Increase of VCO2
92
What is lactate buffered by What are the products What does this cause
Sodium bicarbonate** Products: H2CO3 - H2O + CO2 (expired) NaLA (sodium lactate) Causes breakaway in VCO2, VE Decreases pH
93
AnT =
Anaerobic threshold
94
AnT breakaway at what Due to for each
VE, VCO2 and Lactate VE breakaway due to Increase VCO2 and increase lactic acid (Increase PCO2 and decrease pH) VCO2 breakaway due to increase lactate Lactate breakaway due to buffering systen not able to keep up with lactate production by muscle tissue after AnT
95
What type of training increase AnT
Increase VO2 max training
96
What do we use AnT to predict
``` Cardiorespiratory fitness Endurance capabilities Exercise prescription Tolerance to environmental extremes To set long term work paces to aviod lactate production ```
97
Detection of AnT
Rating of RPE Breakaways Peaking out in FeCO2 Bottoming out in FeO2
98
3 principles of exercise physiology
Max tension - actin-myosin binding Speed of contraction - size of axon and conc. myosin atpase Endurance - regeneration of ATP
99
Oxygen deficit occurs before...
Steady state
100
Oxygen debt occurs after...
Max vo2 is reduced
101
Alactacid restores
1st stage | Phosphagen that were depleted in O2 deficit
102
Lactacid is the...
2nd stage | Removal of lactate by oxidation
103
Oxygen uptake kinetics: Related to... Rate of VO2 response will...
muscle mass and training influence rate or amount of O2 deficit use
104
What is the maximal O2 deficit capacity What are the factors
4-7L O2 in a trained individual Phosphagen stores and lactic acid tolerance
105
Why is O2 debt is greater than O2 deficit
O2 uptake yielding ATP production is required as well as lactate removal Therefore O2 uptake level of metabolism is elevated by myocardium and respiratory muscles Lingering effects of hormones such as thyroxine and catecholamines Increase heat production Increase circulation and increase myocardial O2 uptake rate
106
Alactacid occurs based on...
Oxidative metabolism
107
Lactacid is where
Lactate is converted to pyruvate to enter krebs cyle in muscles Sweat/urine amino acid production, gluconeogensis
108
Rate of recovery for: Passive Active
Passive: alactacid - 50% within 30 seconds, 100% within 2 - 3mins lactacid - 50% within 25-30mins, 100% within 1-2 hours Active: Alactacid - Same Lactacid - 50% within 10-15mins, 100% within 30mins - 1 hour
109
What does active lactacid require
High rate of oxidative metabolism without lactate accumulation, just below AnT Within 7 mins of exercise Ventilation under control with heart rate at 140-160 Moderate intensity Must keep moving
110
Pacing from: Fast to slow Even pace Slow to fast
Fast to slow: Endurance - better for athletes with high % of ST muscle They have high H-LDH which clears lactate Even: Best performance time Fastest pace kept even Slow to fast: Sprinters - better for athletes with more FT muscle = FINISHING KICK Have a lower O2 deficit in first 2 mins Have more M-LDH than endurance athletes
111
What are the three training principles
Overload Progressioin Specificity
112
What is overload
Higher than normal demands
113
What is progression
Increasing workloads
114
What is specificity
Motor unit training
115
Within program design, Task analysis is broken down into
Skill Strength Metabolic
116
When doing program design, what should we consider
``` Age Fitness goals Motivation Injury/disability Equipment Time Interests Variety ```
117
What are the different program phases
Pre-season = 1-2 months prior - build specific fitness In season = Maintain Post season = maintain general fitness
118
What is periodisation / cycle training
Heavy training cycles mixed with lighter training cycles
119
Phosphagen metabolism occurs within Anaerobic glycolysis occurs within Oxidative occurs within
0-20 seconds 45 secs - 3mins 3 - 4min (3:45) - 135 mins
120
What happens at 3:45 mins (3-4min) of exercise
Where 50% of aerobic energy production and 50% anaerobic energy production
121
What are the interval training guidelines for: Phosphagen metabolism
``` Minimum % HR max - >= 95% 0-30secs 4-5sets 8-10 reps 1/3 work ratio Passive recovery ```
122
What are the interval training guidelines for: Anaerobic glycolysis
``` Minimum % HR max - >= 90% 30-60secs 4-5sets 5 reps 1/3 work ratio Active recovery ``` ``` 60-120secs 2-3 sets 5 reps 1/2 work ratio ** Active recovery ``` ``` 2-3 mins 1-2 sets 4-6reps 1/2 work ratio ** Active recovery ```
123
What are the interval training guidelines for: Oxidative
``` Minimum % HR max - >= 85% 3-5 mins 1 set 3-4 reps 1/1 work ratio Passive recovery ```
124
What are the recovery heart rates between reps and sets
Between reps: 70% of max HR Between sets: 60% of max HR
125
Predicted max heart rate is
220 - age = leg exercise 208 - age = water exercise 207 - age = arm exercise
126
Endurance training general guidelines Heart rate max =
>= 75% Max HR = FOR GENERAL FITNESS 85 - 95% Max HR = FOR COMPETITVE PREP 220 - age (+- 10 bpm)
127
% max HR =
Max HR x (% max / 100)
128
Endurance training compared to interval training
Physiologically and psychologically less demanding Generally used to develop general overall cardiorespiratory endurance (around 75% HR Max) Can be used in conjunction with interval training for compeitive prep (85 - 95% of HR max) Generally less specificity in training
129
What else can be used to know if the intensity of endurance training is greater than AnT
Breakaway in VE = hyperventilation Breakaway in lactic acid
130
ACSM recommends that the duration and frequency of endurance training is
20 - 60 mins 3 - 5 times a week
131
After 4 weeks of detraining what is loss
Lose 50% of cardiorespiratory fitness developed
132
What is the fitness classifications based on VO2 max LOW
Female: <= 29 ml/kg/min Male: <= 34 ml/kg/min ``` 60-70% of HR max 50-60% of HRR 50-60% of VO2 max RPE (fairly light to somewhat hard) Breathing is comfortable, unaware 20-30mins, 3 days a week ```
133
What is the fitness classifications based on VO2 max MODERATE
Female: 30 - 44 ml/kg/min Male: 35-49 ml/kg/min ``` 70-80% of HR max 60-75% of HRR 60-75% of VO2 max RPE is somewhat hard to hard Aware of breathing 30-45mins, 4 days a week ```
134
What is the fitness classifications based on VO2 max HIGH
Female: >= 45 ml/kg/min Male: >= 50 ml/kg/min ``` 80-90% of HR max 75-85% of HRR 75-85% of VO2 max RPE = 15-17 hard to very hard Respiratory distress 45-60 mins, 5 days a week ```
135
Power =
Work / Time (Force x distance) / Time Force x velocity
136
Strength =
Maximal force from one contraction (1 rep max)
137
Isometric =
Force = resistance No movement Can provide a maximal overload Joint angle specificity
138
Concentric =
Force > resistance Movement in direction of force vector Overload can be near maximal But then speed will be slow AT extremes in room **
139
Essentric =
Force < resistance Movement in direction of resistance vector Overload can be maximal 120% of 1 rep max
140
Isokinetic =
Force > resistance Overload can be maximal Controlled speed may be fast or slow
141
Muscular endurance = What is it not dependent on
Is a measure of work capacity under moderate to high resistance loads. It mainly depends on strength and anaerobic capabilities and is also a function of the relative load involved NOT DEPENDENT ON AEROBIC OXIDATIVE METABOLISM
142
Strength ------ Muscle endurance ------- Cardiorespiratory endurance
1 RM 2 - 3 mins > 3 - 4 mins
143
Basic training guidelines for Isometric
100% of maximum effort 5 sec/rep 5 reps/exercise
144
Basic training guidelines for Concentric
8-10 exercises 1 set 8-12 repitions 2 days per week 80 - 70% of 1 rep max
145
Basic training guidelines for Eccentric
120% of 1 RM 3 - 5 sets/exercise 6-8 reps 3-5 times a week
146
Basic training guidelines for Isokinetic
100% of max effort 3 sets 8-15 reps 2-4 days a week Training speed should be as fast as or faster than the speed of movement involved in the sports skill for which the athlete is training
147
Basic training guidelines for Circuit
Involves 6 - 15 exercises Mainly concentric 30 - 40 secs 15 - 20 secs rest 40 - 60% of 1 RM Timed circuit 30 - 40 mins Can be effective at increasing strength, muscle endurance, VO2 max and decreasing body fat
148
Basic training guidelines for Muscular endurance
15 - 20 reps up to 30 - 40 reps 2 - 3 sets 3 times a week
149
Acute muscle soreness
Acute soreness is due to ischemia as blood flow is occluded at 60 % of maximal voluntary contraction = 60% of 1 RM
150
Delayed muscle soreness
24-48 hours post workout damage of muscle or connective tissue
151
Training adaptation of Resistance training Decrease in... Increase in...
Decrease in: % BF FW Capiliary density Aerobic enzyme activity Increase in: Everything else
152
Training adaptation of Sprint training Decrease in... Increase in...
Decrease in: Body mass % BF FW Body circumferences Increase in: Everything else But... Bone mineralization and connective tissue strength and mass only if it is weight bearing
153
Training adaptation of endurance training Decrease in... Increase in...
Decrease in: ``` Body mass % BF FW Body circumferences FT fiber to ST fiber ``` Increase in: Everything else But... Bone mineralization and connective tissue strength and mass only if it is weight bearing
154
Muscle mass breakaway in ____ around the age of ____
Males | 12
155
Force produced is greater in
Males than females
156
Steroid effects of: Anabolic Androgenic
Anabolic = Increase lean tissue development and strength Androgenic = Increase masculinisation or feminisation
157
What are the side effects of exogenous intake
Liver or kidnet damage CHD sterility Closure of long bone growth Servere acne Masculinisation or femisation Increase risk of some cancers
158
Strength training helps endurance training Endurance training helps strength training
True False
159
ACSM recommendation on fitness and exercise guide lines
60 - 90% HR max 50 - 85% HRR or VO2 Max 20 - 60 mins per session 3 - 5 days per week
160
Training adaptation of Resistance training on: ``` Rest HR Rest BP Ventricular wall thickness Ventricular volume SV Myocardial efficiency Blood lipid profiles Glucose tolerance Self esteem Performance ```
``` Rest HR = Decrease Rest BP = Decrease Ventricular wall thickness = Increase Ventricular volume = SV = Increase Myocardial efficiency = Increase Blood lipid profiles = No change Glucose tolerance = Improved Self esteem = Increase Performance = Increase ```
161
Training adaptation of sprint training on: ``` Rest HR Rest BP Ventricular wall thickness Ventricular volume SV Myocardial efficiency Blood lipid profiles Glucose tolerance Self esteem Performance ```
Rest HR = Decrease Rest BP = Decrease Ventricular wall thickness = No change Ventricular volume = Increase SV = Increase Myocardial efficiency = Increase Blood lipid profiles = Improved Glucose tolerance = Improved Self esteem = Increase Performance = Increase
162
Training adaptation of endurance training on: ``` Rest HR Rest BP Ventricular wall thickness Ventricular volume SV Myocardial efficiency Blood lipid profiles Glucose tolerance Self esteem Performance ```
Rest HR = Decrease Rest BP = Decrease Ventricular wall thickness = Increase Ventricular volume = Increase SV = Increase Myocardial efficiency = Increase Blood lipid profiles = Improved Glucose tolerance = Improved Self esteem = Increase Performance = Increase
163
Every American adult should accumulate
30 mins or more of moderate intensity physical activity over the course of most days of the week
164
What is the breakaway in VE at AnT detected by
Chemoreceptors - detect CO2 increase and pH decrease
165
At submax exercise, trained individuals have a lowerer Due to Trained individuals have a _____ and _____ breath
VE ``` Capillarisation larger long volumes Greater alveolar ventilation rate Great Hb and blood volume Decrease sensitivity in chemorecptors ``` Slow and deep
166
How much O2 is carried in the plasma
1%
167
PO2 is greatest in the PCO2 is greatest in the
Capillary Muscle
168
The higher the PO2, the greater...
The saturation of Hb with oxygen
169
Hb dissociation curve describes...
How much oxygen is bound to Hb in given Partial pressure
170
Allosteric refers to...
the interaction between spatially distinct sites
171
Ventricular volume is also known as
Anatomical volume
172
0.3 g per kg of sodium bicarbonate will..
Increase lactic acid tolerance and capacity of anaerobic glycolysis
173
AnT in untrained is _____ AnT in trained is ______
50 - 60% VO2 max 70 - 80% VO2 max
174
The capacity of anaerobic energy systems*** limits...
Max O2 deficit capacity
175
Phosphagen stores are related to...
muscle mass and training status ***