Lecture 6 - Eating Disorders Flashcards

1
Q

What is PICA?

A
  • persistent eating of non-nurtitive, non-food substances
  • 1mth
  • childhood onset most common
  • in adults: MI and ID
  • prev unclear
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

What is Rumination Disorder?

A
  • repeated regurgitation of food after feeding/eating
  • 1mth
  • childhood onset most common
  • in adults: MI and ID
  • prev unclear
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

What is Avoidant/Restrictive Food Intake Disorder?

A
  • avoidance/restriction of food intake
  • failure to meet requirements for nutrition or insufficient energy intake through oral intake of food
  • weight loss, nutritional deficiency, dependence on enteral feeding/oral nutritional supplements, interfered functioning
  • childhood onset most common
  • COMORBIDITIES: anxiety, OCD, neurodevelopmental (ASD, ADHD, ID)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

What are the DSM-5 criteria for Anorexia?

A
  • restriction of energy intake leading to low body weight
  • intense fear of gaining weight/becoming fat or persistent behaviour interfering with weight gain
  • disturbance in way one’s shape/weight is experienced
  • self-evaluation unduly influenced by shape/weight
  • persistent lack of recognition of seriousness of low body weight
  • subtypes: restricting, binging/purging
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

What is the prev, onset, comorbidity and course of AN?

A
  • prev: 0.1-1% (10F:1M)
  • subthreshold more common
  • onset: late teens/early 20s (later onset becoming more common)
  • comorbidity: 60% dep, 33% anx
  • course: highly variable
  • recover
  • fluctuate + relapse
  • chronic deteriorating course (20%)
  • cross-over to BN (50%)
  • high rates of suicide
  • mortality rate 5-10%
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

What are the associated features of AN?

A

DISORDERED EATING

  • food rituals
  • food preoccupation
  • food preferences
  • cook for others, not self
  • hungry but refuse to eat

PHYSIOLOGICAL COMPLICATIONS

  • hair thin
  • low HR/BP
  • anaemia
  • kidney failure
  • bloat
  • cog feature: mood, mmeory, concentration

PSYCHOLOGICAL FEATURES

  • perfectionism
  • feelings of ineffectiveness
  • inflexible thinking
  • overly restrained emotional expression (eg. anger)
  • limited social spontaneity
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

What are the DSM-5 criteria for Bulimia?

A
  • recurrent episodes of binge eating
  • recurrent inappropriate compensatory behaviours to prevent weight gain
  • binging 1/wk for 3 mths
  • self-evaluation unduly influences by shape/weight
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

What is the prev, onset, comorbidity and course of BN?

A
  • prev: 1-1.5% (10F:1M)
  • subthreshold more common
  • onset: late-early adulthood (later than AN)
  • often occurs after an episode of dieting
  • course: chronic or intermittent
  • often lasts several years
  • remission and recurrences of binging often alternate
  • comorbidity: MDD, anxiety, substance use 33%
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

What are the associated features of BN?

A

PSYCHOLOGICAL FEATURES

  • low SE
  • guilt
  • preoccupied with food
  • recognise behaviour is abnormal

PYSIOLOGICAL COMPLICATIONS

  • anaemia
  • swollen cheeks
  • abraised knuckles
  • irregular HR
  • sore/irritated throat
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

What is Binge Eating Disorder? Prevalence?

A
  • recurrent episodes of binge eating (size, time, control)
  • 1/wk for 3mths

3+ of

  • eating rapidly
  • eating until uncomfortably full
  • eating when not physically hungry
  • eating alone (embarrassed)
  • disgusted with oneself (depression, guilt)
  • 1.6% F, 0.8% M
  • higher in obese populations
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

What are OSFED and UFED?

A

Other Specified FED:

  • atypical AN (not low weight)
  • atypical BED (lower freq/duration)
  • atypical BN (lower freq/duration)
  • night eating syndrome
  • purging disorder (without binging)

Unspecified FED:

  • behaviours cause clinical distress/impairment but don’t meet full criteria for any other disorder
  • clinician may not want to specify which criteria were met
  • may be insufficient info to make specific diagnosis
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

What is the deal with males and eating disorders?

A
  • 10% of EDs in males
  • 17% diet
  • 31% want heavier, 31% want lighter
  • body dissatisfaction increasing (15% in 1972, 34% in 1985 and 47% in 1997)
  • extreme dieting/self-induced vomiting increasing more in M than F
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

What are the treatment outcomes for EDs?

A
  • worse for AN (50%) than BN (75%)
  • better short-term from BED (25-80%) and at 1yr follow-up
  • higher remission for EDNOS short-term (<5yrs) (68%) > but at 5yrs there is little diff b/w EDNOS and BN
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

What are the prognostic factors of EDs?

A
  • AN: severity, duration, body/weight concern
  • BN: severity, psychiatric comorbidity
  • BED: early abstinence from binge eating and to interpersonal factors
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

What are the genetic risk factors for EDs?

A
  • moderate heritability
  • AN: 50-60%
  • BN: 58-83%
  • BED: 82%
  • teenage obesity: 86%
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

What are the biological risk factors for EDs?

A
  • chem imbalances in neuroendocrine system (hunger, appetite, digestion, emotions, thinking etc.)
  • 5HT and NE low in acutely ill (link b/w MDD and EDs? or result of starvation?)
  • excessive levels of cortisol in AN (and MDD) > issue with hypothalamus
  • brain structure: parietal cortex smaller > overestimate own weight, lack insight… or caused by starvation??
17
Q

What is the relationship between ED and cognition?

A
  • generally average-high intellect
  • in acutely unwell AN: mild cog deficits (exec functioning, memory, verbal and visuospatial processing) > not in recovered patients
  • deficits in cog flexibility and central coherence (AN and BN)
18
Q

What is the role of personality in EDs?

A
  • AN-R: obsessive, perfectionistic, socially inhibited, harm avoidance, emotionally restrained
  • AN-BP: impulsive, extroverted
  • AN: OCPD traits in childhood??
  • BN: impulsive, interpersonally sensitive, low SE, Cluster B + Cluster C (esp. BPD), substance use
19
Q

What are the early environmental risk factors in EDs?

A
  • AN: obstetric complications, childhood feeding/sleeping problems, high physical exercise, over-anxious parenting, OCPD traits, perfectionism, -ve SE
  • BN: obstetrics complications, dieting, childhood/parental obesity, alcoholism, pubertal timing, sexual abuse, -ve SE
  • BED: childhood obesity, family over-eating/binge-eating, high parental demands, -ve affect, parental mood, substance misuse, perfectionism, separation from parents, maternal problems with parenting
20
Q

What are the interpersonal and social risk factors in EDs?

A

INTERPERSONAL

  • trouble fam/personal rships
  • difficulty expression emotions/feelings
  • history of teasing about size/weight
  • high parental expectations + maladaptive parenting (chicken v egg?)
  • childhood feeding difficulties
  • history of trauma or abuse (60-75% BN patient have a history of physical/sexual abuse)
21
Q

What are the cultural risk factors in EDs?

A
  • media and cultural pressures
  • glorify thinness + obtaining perfect body
  • narrow definitions of beauty
  • value people on physical appearance NOT inner qualities
  • people in professions emphasizing thinness are more vulnerable (modelling, dancing, weightlifting)
22
Q

Explain the GxE interactions in EDs

A
  • lability may result from accumulation of psychosocial risk factors
  • trigger illness in genetically vulnerable adolescents
  • currently looking into epigenetics
23
Q

Explain the aetiological model of EDs

A
  • predisposing: vulnerability
  • precipitating: stressors; lead to weight loss > benefits of weight loss discovered
  • perpetuating: maintain weight loss
24
Q

What is the transdiagnostic model of EDs?

A

AN > BN 50%
BN > AN 30%

  • same core psychopathology underlies all EDs (over-evalute eat/shape/weight and their control)
  • expressed in similar attitudes/behaviour (restriction, vomit/laxatives, body checking, binging, preoccupation with eating/shape/weight)

MAINTAINING PROCESSES

  • perfectionism
  • low SE
  • mood intolerance
  • interpersonal difficulties
25
Q

What is orthorexia?

A
  • obsession with proper nutrition
  • restrictive diet + ritualised patterns of eating + rigid avoidance of foods that are ‘unhealthy’ or ‘impure’
  • may lead to: nutritional deficiencies, medical complications similar to AN, psychological complications (frustration, guilt, self-loathing, social isolation)

15 item measure

  • 13% Aus dieticians
  • 45% Turkish doctors
  • 56% Turkish performance artists
26
Q

Explain the transdiagnostic treatment model of EDs

A
  • suitable for all EDs (specific diagnosis is not relevant)
  • treatment content dictated by the presenting psychopathological features and the processes maintaining them

4 STAGES

  1. 4 weeks. Engage, educate, initial formulation
  2. Review progress so far, identify barriers to change, formal assessment of the 4 transdiagnostic processes, revise/extend formulation
  3. Dictated by formulation, modify eating disorder psychopathology, address additional processes
  4. Ensuring progress is maintained after treatment finishes