lecture 5/6 Flashcards
most anterior part of cerebellum vermis
nodule
function cerebellum
receives proprioception
coordinates balance
eye hand coordination
predicts consequences of movement
T/F cerebellum is coordinator and predictor of corticol output
true
T/F cerebellum not needed for skilled manipulation of mm
false, its critical
T/F cerebellum used for skilled manipulation of mental concepts
t
cerebellum mutism
not able to speak, can’t predict
information the cerebellum gets
cortical
proprioception
vestibular
vestibular information
project to paravermis / flocculonodular lobe
position head/ body in space
orient eye movements during locomotion
proprioceptive information
ia, II fibres from mm to
Ib from golgi tendon organs
from clarkes column
terminate in anterior lobe /vermis
anterior lobe associated w
limbs
vermis associated w
limb
cortical information
cortex projects to continue nuclei
fine motor UE, hand dexterity / eye coordination
olivocerebellar fibers
afferents from olive project to entire cerebellum cortex
climbing fibres
outflow from cerebellum
via deep cerebellar nuclei
dentatorubrothalmic tract
to vestibular nuclei / olives
dentatorubrothalmic tract
from dentate to red to thalamus to cortex
archicerebellum =
vestibulocerebellum
paleocerebellum =
spinocerebellum
neocerebellum =
cerebrocerebllum
vestibulocerebellum
flocculonodular lobe and paravermis
feedback to vestibular nuclei / SC
trunk control
spinocerebellum
vermis and anterior lobe
feedback to cortex/ SC
extremities
cerebrocerebllum
posterior lobe
topographical of extremity
eye movement
speech
coordination of movement
cerebellar loops
vestibulocerebllear
spinocerebellar
cerebrocerebllar
what do cerebellar loops allow
coordinated balance smooth movement
anticipation of movement
predictions that feedback to the loop
what fine tunes ongoing movements
spinocerebllar loop
information from __ to pontine uncle cross over to
cortex
contralateral cerebellum
red nucleus
relay nucleus
T/F olives project to all of the cerebellum
true
what calculates feed forward loops
olives
what does automation of cortical output
spinocerebellar loop
midline cerebellar disease
gait difficulty
imbalance
abnormal head posture
oculomotor dysfunction
lesion to flocculonodular node
truncal ataxia
nystagmus
lateral cerebellar disease
posterior lobe function
cerebellum mutism eye / speech movement coordinate movements dysmetria dysdiokinesiea
dysmetria
over n under shoot
dysdiokinesiea
rapid agonist antagonist movements
lesions to anterior lobe
affects spinocerebllear input
gait ataxia
ethanol is toxic to what
purkinje cells
ethanol effects which lobe
anterior
is ethanol and gait ataxia reversible
usually
when is ethanol gait ataxia not reversible
chronic alcoholic
T/F cerebellum related to emotion / cognition
yes
cerebellum emotion / intellectual processing
judge facial expression
language (grammar adjustments)
neuropsychiatric manifestions
neuropsychiatric manifestions
exaggerated or diminution of repose to environment in automatic processing of emotions
T/F cerebellum effects communciton with non motor cognitive association areas
yes
interaction between cerebellum / cognitive cortical networks
interaction with cerebral executive control
interaction with default mode network
function basal ganglia
modulate voluntary motor activity
three circuits in basal ganglia
motor
associative
limbic
motor circuit
controls body and eye movements
direct/indirect pathway
associative circuit
higher level cognitive function (planning)
limbic circuit
emotional / motivational processing
activity in basal ganglia encodes for
decision to move
direction/ amplitude of movement
motor expression of emotion
efficiency of movement (procedure)
T/F thamalus under acute inhibition when we are not moving
false chronic
release inhibtion model components
direct and indirect pathway
direct pathway
release tonic inhibition of thalamus
leads to more execution of motor cortex
indirect pathway
inhibit output from thalamus
less execution of motor cortex
input to
caudate and putamens
output from
globus pallidus
what pathway facilities target orientated movements
direct
which pathway impacts potentially competing movements
indirect pathways
T/f direct and indirect pathways are simultaneous
true
T/F direct and indirect pathways do not provide fine balance for cortical output
false, they do
diseases related to lesions in basal ganglia
parkinsons
ballism
huntingtons
tourettes, OCD
parskinsons
inhibition of motor output
weaker direct pathway
decrease in movement and facial expression
ballism / huntingtons disease
excessive motor output
T/f basal ganglia related to nearo disorders
yea Tourettes and OCD
ballism
sudden uncontrolled flinging of movements of extremities
underlying cause of ballism
stroke of contralateral subthalmic nucleus
hemi ballism
loss indirect pathway
more involuntary motor output
can’t suppress the extra movements
huntingtons
degeneration of striatum
hyperkinetic movement disorder
direct n indirect pathway effected
parts of striatum
caudate and putamen
damage to striatum effects
direct n indirect pathway
hypokinesia akinesia and hypminmia loss or decrease associated with what disease
parkinson
parsons does what to inhibition
inhibits it less (so more left over)
excited the inhibition (adding even more)
t/f damage to substance nigra related to Parkinson’s
yes
how do basal ganglia interact w rest of brain
the three circuits
mask face or rich in dopaminergic neutrons what circuit
limbic
are all basal ganglia circuits working seperate or at once
at once
