Lecture 19– Introduction to psychiatry Flashcards

1
Q

psychiatry is based on

A

body, mind, spirit connection

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2
Q

philosophical issues: dualism

A
  • a set of views about the relationship between mind and matter, which begin with the claim that mental phenomena are in some respects non-physical.
  • (“the ghost in the machine”)
  • Rene Descartes (1641) First to clearly identify the mind with consciousness and self- awareness and to distinguish this from the brain which was the seat of intelligence
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3
Q

philosophical issues

A

Neurosis

  • “disorders of sense and motion” due to a “general affection of the nervous system”
  • Included a range of conditions (e.g. epilepsy, mania, hysteria, diabetes, etc.), with no identifiable physiological cause (e.g. fever)
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4
Q

Medical disease that were psychiatric

A
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5
Q

homosexuality and psychiatrty

A

was orignally seen as a pathological disease

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6
Q

emergent schisms

A

Overtime neurology and psychiatry were classified as different

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7
Q

neurology

A

disorders of the NS with established aetiologies, demonstratable anatomical pathology and physical symptoms e.g. Parkinsons, stroke, epilepsy, Huntingotns, brain injury

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8
Q

psychiatry

A

disorders of the mood, thought and behaviour with no or only mino physical signs with no visible pathology

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9
Q

organic vs function in psychiatry

A
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10
Q

Approaches to mental illness- 1

A
  • Identify chemical imbalances, changes in transmitters and receptors and attempt to correct with drugs (psychopharmacotherapy).
  • è Revolutionised psychiatry! Patients (mainly psychotic) in straight jackets or locked up - freed.
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11
Q

Approaches to mental illness- 2

A
  • Mental illness results from upbringing and environmental factors
  • Seek to understand, work through, find resolutions/adaptations è psychotherapy/social approaches.
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12
Q

was the rise of psychiatry..

A
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13
Q

Movement against psychiatry- anti-psychiatry

A

Some people think antipsychotics are used too much etc

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14
Q

Why do we need classification?

A
  • To enable clinicians to communicate with each other about patients
  • To understand implications of diagnosis (Sx, prognosis, treatment, etc.)
  • To facilitate research
  • & to relate research findings to everyday practice
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15
Q
A
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16
Q

Criticisms of Classification:

A
  • Categorisation denies consideration of unique personal difficulties
  • Labels deviant behaviour as an illness
  • Individuals do not fit neatly into categories (stigma)
17
Q

diagnosis in psychiatry

A
  • – ‘knowing the underlying cause’ of symptoms & signs
    • (useful, e.g. for appendicitis è understanding, Rx, etc.)
    • BUT rarely can do in psychiatry (except in organic conditions)
  • Lack of clear disease categories in psychiatry , therefore instead of diagnosis or disease we use disorder
18
Q

disorders in psychiatry

A
  • “a clinically recognisable set of symptoms or behaviour associated in most cases with distress and with interference with personal functions. Social deviance or conflict alone, without personal dysfunction, should not be included in mental disorder as defined here”
  • This means that most psychiatric disorders are
    • Not based on theoretical concepts or aetiology
    • Are based on recongisable clusters of symptoms and behaviours
19
Q

Symptoms

A

Experienced and reported by the patient

20
Q

Sign

A

Discovered by doctor during examination

21
Q

diagnostic heiarchy

A

Organic- we know what’s going on in the brain e.g. Alzheimer’s

Psychotic- delusion and hallucination

Neurotic- anxiety and mood disorders

22
Q

Psychiatric Genetic

A
  • to aid classification
  • risk estimation
  • to assist in the development of new treatments

Future of Psychiatric Genetics?

  • Problems with co-morbidities & classification (!)
  • Need new approaches: e.g. candidate gene driven (rather than disorder driven)
23
Q

psychosis (schizoprenia (genetic studies)

A
  • lifetime risk of 1% in the general population
  • risk in siblings (& DZ twins) is around 8–10%
  • risk increases as more relatives are affected
  • monozygotic (MZ) twin pairs have ~45% concordance
  • over 50% of MZ co-twins are unaffected, despite being virtually identical genetically, so non-inherited risk factors are also important
  • likely that the aetiology is multi-factorial, i.e. many genetic and environmental factors act together to influence risk, and a single risk factor is unlikely to cause the disorder on its own
24
Q

pschiatry future compared to present and past

A