Lecture 16 Flashcards

1
Q

what do group 11’s inhibit?

A

cerotenoid biosynthesis

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2
Q

groups 11,12, 13, and 27 are all…

A

carotenoid biosynthesis inhibitors

‘bleaching herbicides’

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3
Q

group 11 herbicide

A

Amitrole - no loner manufactured

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4
Q

what do group 13’s inhibit?

A

DOXP - enzyme the is key to plastid synthesis

  • bleaching and stunting
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5
Q

group 13 herbicide

A

Clomazone

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6
Q

Clomazone (13) application and uptake

A

soil applied

readily absorbed in roots

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7
Q

Clamazone (13) persistence

A

rapid degradation

can persist under extremely dry conditions

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8
Q

Clomazone (13) resistance(found or not?)

A

no host resistant biotypes reported

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9
Q

Clomazone (13) symptoms

A

seedlings emerge bur are bleached and become necrotic

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10
Q

what does group 14’s inhibit?

A

PPO

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11
Q

group 14 selectivity

A

suppression of broadleaves, some grasses

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12
Q

group 14 uptake

A

rapid uptake by leaves

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13
Q

are group 14’s subject to photodegradaton?

A

yes

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14
Q

group 14 herbicides

A

sulfentrazone - Triazolinones

Safulfenacil - Pyrimidinediones

Flumioxazin - N-Phenylpthalimides

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15
Q

group 14 MOA’s

A

inhibit heme and vhlorophyll synthesis

leads to loss of chlorophyll and carotenoids producing ‘leaky’ membranes

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16
Q

group 14 symptoms

A

appear rapidly

water soaked appearance

leaves yellowed and desiccated in 1-3 days

17
Q

group 14 selectivity

A

rapid metabolism into inactive compounds

18
Q

group 14 persistance

A

rapid breakdown (microbial)

19
Q

Triallate (15) application

A

soil applied

20
Q

does triallate affect monocots or dicots?

A

monocots only

21
Q

Triallate target site

A

fatty acid synthesis

22
Q

triallate MOA

A

inhibits cell elongation and division

23
Q

triallate symptoms

A

shoestringing in grassy weeds

failure to emerge

24
Q

do group 15’s affect seeds or shoot?

A

effect on shoot formation