Lecture 13 group 1 Flashcards

Herbicide Modes of Action

1
Q

What do Group 1 herbicides inhibit?

A

Fatty acid / lipid biosynthesis

ACCase inhibitors

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2
Q

is there high soil residual for group 1’s?

A

no - rapidly degraded in soil (microbial)

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3
Q

what tissues do group 1’s damage?

A

meristem tissue

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4
Q

do group 1’s effect broadleaves?

A

no

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5
Q

three classes of group 1 ACCase inhibitors

A

1) Aryloxyphenoxy Propionates - “fops”

2) Cyclohexanediones - “dims”

3) Phenylpyrazolin - “dens”

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6
Q

Aryloxyphenoxy Propionates “fops” properties

A

pro - herbicides with methyl, ethyl and butyl groups

Typically parent is acid so needs alcohol group to move past into plant

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7
Q

Aryloxyphenoxy Propionates - Fops, Dims or Dens?

A

Fops

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8
Q

Cyclohexanediones “dims” properties

A

photodegredate, breakdown when exposed to light

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9
Q

Cyclohexanediones - Fops, Dims or Dens?

A

Dims

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10
Q

Phenylpyrazolin “dens”

A

newest class

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11
Q

Phenylpyrazolin - Fops, Dims or Dens?

A

Dens

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12
Q

Aryloxyphenoxy Propionates (AOPP) examples

A

Clodinafop - propargyl
Fenoxaprop-p-ethyl
Fluizafop-p-butyl
Quizalofop

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13
Q

Cyclohexanediones (CD) examples

A

Clethodim
Sethoxydim
Tepraloxydim
Tralkoxydim

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14
Q

Phenylpyrazolin example

A

Pinoxaden

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15
Q

group 1 MOA and injury

A
  • inhibit enzyme Acetyl CoA Carboxylase (ACCase) -> essential for fatty acid biosynthesis for membrane production
  • loss of cell integrity
  • injury at growth point, yellowing, inhibit meristem, young leaves pull out
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16
Q

Chlorosis

A

Plant yellowing due to loss of chlorophyll

17
Q

Necrosis

A

Localized death of living tissue

18
Q

two forms of selectivity

A

1) enzyme bonding affinity

2) differential metabolism

19
Q

Enzyme binding affinity - selectivity monocots vs dicots

A

Dicots - low enzyme bonding affinity

Monocots - high enzyme bonding affinity

20
Q

differential metabolism - selectivity

A

Monocots - differential metabolism

Weeds vs. crops