Lecture 12: Integumentary System Flashcards

1
Q

What is the largest organ in the body?

A

The skin
* Constituting 15-20% of the body weight and consisting of three primary layers

The overall primary function of the skin is to protect underlying structures from external injury and farmful substances

Other functions:
* Holding the organs together
* Sensory perception
* Contributing to fluid balance
* Controlling temperature
* Absorbing ultraviolet UV radiation
* Metabolizing vitamin D
* Synthesizing epidermal lipids

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2
Q

The skin helps metabolize vitamin D. Why do we need vitamin D?

A

Contributes to strong bones

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3
Q

Skin 3 layers

A

Epidermis
Dermis
SubQ

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4
Q

Some lab values that are important for the integumetary system / would healing - what are the values
:
* Per albumin - value norms / closely realted to
* Albumin - value norms / closely related to
* Glucose
* Glycosylated hemoglobin (HbA1c)

A

Per-albumin: 20-40 mg/dL and
* Closely related to malnutirtion - ICD code (priamry diganosis)

Albumin: 3.5-5.5g/dL
* Closely related to malnutirtion - ICD code (primary diagnosis)

Glucose: 70-115 mg/dL (fasting)
* This is a pin prick

Glycosylated hemoglobin (HBA1c): 4-6%
* This is a pin prick

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5
Q

KNOW: 1 in every 4 people will consult a physician for a skin lesion

What is the primary lesion?

A

The first leasion to appear

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6
Q

5 s/s of skin disease?

A

1) Pruritus
2) Urticaria
3) rash
4) Blisters
5) Xeroderma

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7
Q

Pruritus means

A

Itching

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8
Q

Urticaria means?

A

Hives

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9
Q

Xeroderma

A

Dry skin

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10
Q

KNOW: dry skin and itching often go together

A
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11
Q

Superficial inflammation of the skin
* Clinical manifestations
* tx?

A

Dermatitis
* more of blanket statement

Clinical manigestations:
* Vesicles
* Redness, edema, oozing
* Crusting, scaling
* Itching - puritis

Topical medications are used

NOTE: someone w/ more melinin might not show obvious s/s even though its actually there

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12
Q

6 types of dermatitis (remember its a blanket term)

A

1) Atopic dermatitis
2) Contact dermatitis
3) Eczema (dry crusty skin)
4) Statis dermatittis
5) Environmental dermatoses
6) Incontinence Associated dermatitits

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13
Q

Development of areas of very dry, thin skin and sometimes shallow ulcers of the lower legs primarily as a result of venous insufficiency (often coupled w/ venous insufficiency)
* Clinical manigestations
* Tx

A

Stasis dermatisis - skin condition that occurs when blood pools in the venins of the LE due to poor cirulation (i.e., stasis)

Clinical manigestations
* Itching
* Feeling of heaviness in legs
* Brown stained skin
* Open shallow lesion - because this is in the venous system and is superficial to the arterial system

Tx:
* Reducing venous hypertension (with chronic venous insufficiency valves arent working well and the blood starts pooling in the veins - this is what cuases this)

Stasis = not moving / staying still/ static
* Blood not moving = infestation of blood not moving

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14
Q

Environment Dermatoses causes

A

1) Irritant and allergic dermatitis - think rubbing up against something that you’re allergic to or is irriatting
2) Rosacea: more common in woemn - due to hormone flucuations - think red powdery mask over face
3) Acne legions
4) Pigmentary changes
5) Photosensitivity reactions - walking out in the suddent and suddenly turning red (sometimes not all the time)
6) Systemic sclerosis - abnormality of collagen (talked about this in lungs lecture)
7) Infectious disorders - cause some skin abnoramilties
8) Cutaneous malignancy - think abnormal moles

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15
Q

Chronic facial disorder of middle-aged and older people. May be due to hormones. No known cause or factor has been identified to explain the pathogensis of this disorder

A

Rosacea

KNOW: An acneiform rosacea can occur with papules, pustules, and oily skin (basically saying it looks like acne sometimes)

harder to identify on african american - looks like acne

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16
Q

Skin damage resulting from chronic urine or feces exposure

A

Incontience-Associated Dermatitis

KNOW: Characterized by pink or red discoloration with erythema-may have skin erosion and maceration
* think of it as a diaper rash for babies
* can happen to heavier set that arent taken care of
* maceration = skin is in contact w/ too much moister = milky foggy foam around it = easier to tear
* this can also be a sign of neglect / abuse

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17
Q

What are the two bacterial skin infections?

A

1) Impetigo
2) Cellulitis

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18
Q

Cellulitis:
* What is it?
* Assocaited w/?
* s/s (3)
* Tx (1)

A

Bacerial skin infection

Associated w/ lymphodema but can occur in the absence
* major risk factor

red, swollen, hot painful skin - think s/s of infection

needs antibotics

NOTE: it moves super quick - type of dermatitis that becomes inflammaed

can happen anywhere where bacteria can get in

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19
Q

What is Impetigo?
* Where does it occur?
* is it contagious
* What does it look like?

A

Bacterial infection of skin

Occurs in superficial layers of the skin

Erythematous plaques with a yellow crust and may be itchy or painful

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20
Q

What is this?
* is it bacterial or viral

A

Warts

Viral skin infection

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21
Q

What is this?
* is it bacterial or viral?

A

Shingles

Viral skin infection

notice how its following nerve root
* more common if you had chicken pox and over age of 60
* can occur in any part of body

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22
Q

Is Herpes Zoster a bacterial or viral skin infection?

A

Viral

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23
Q

3 Fungal infections

A

1) Ringworm (Tinea Corporis)
2) Athletes Foot (Tinea PEdis) - dry itchy red on feet
3) Yeast infection (Candidiasis)

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24
Q

What is this?

A

Yeast infection in the neck

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25
Q

What is this?

A

Ringworm on scalp
* often due to transmision from animals

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26
Q

What are our 2 parasitic infections?

A

1) Scabies
2) Pediculosis (Lousiness) (lice)

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27
Q

How do most people get lice

A

Sharing hats

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28
Q

How do scabies enter body?

A

Burror into skin

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29
Q

What are our 4 kinds of wounds

A

1) Pressure Injuries (new term for pressure sores - done because they arent as perventable as we thought they were - do not have to have wound formation to be pathological)
2) Arterial Insufficiency Ulcers - little bit deeper, due to PAD
3) Venous insufficiency Ulcers - more superficial - due to chronic venous insurfficancty = venous hypertension
4) Diabetic Foot Ulcers - glucse in there breaking down NS/artical system - which provides nutritents and sensation to the foot - something irttates skin before it staarts breaking down (think small cut)

30
Q

Prolonged pressure, necrosis of tissues?
* where in the body does it foten occur
* Common sites
* Risk factors (6)

A

Pressure injuries
* when you have prolonged pressure you get ischemia in that area which leads to necrosis
* more things go into it than just lying in one position (think malnutirtion / albumin levels)

Often occur over boney areas

Common sites: Heels, lateral malleoli, tochanters, ischial tuberositites, sacrum, scapulae, ears - NPTE

Risk factors:
* Immobility
* Decreased sensation - they won’t be able to feel which will lead them to not shift = more pressure in one area
* Moisture - any type
* Friction or shearing forces - rodo pegs are little blocks that help counter act friction between layers of skin - this thins as we get older - these shearing forces act more at this point
* Decreased arterial perfusion - if arteries arent going to the area enough = trophic issues = more at risk
* Abnormal BMI - too low because of malnutrition or too high due to malnutirtion and improper absorption and storage of adipose = increased pressure

31
Q

Which stage of pressure injuries has unbroken skin and appears as a bruse
* what does it not reach at this stage

A

Stage 1
* is stage 1 if skin is unbroken
* However, you can’t really tell how deep it goes
* usualy does not reach subQ area

32
Q

Blanceable vs Non Blanchable
* What does it mean if its non blanchable?
* What stage pressure injuries is this often tested in?

A

Blanchable - if you push down on the skin it should pale and then come back to normal.

Non-Blachable - Never gets pale - just stays that bruised area
* Indicitive that its gone through deeper layers

NOTE: this is often tested in stage 1 pressure injuries

Notice the pale spot on blanchable

33
Q

What stage of pressure injury actaully breaks the skin and is in the dermis?

A

Stage 2 pressure injuries
* have not reached subQ

34
Q

Which stage of pressure injuries has reached the SubQ layer

A

Stage 3

NOTE: theres only fat that stands between the outside world and the muscle

NOTE: the white in there is bad

35
Q

Which stage of pressure injury has reached bone?
* what are they at risk for

A

Stage 4

at risk for osteomyolitis = bone infection - requires antibiotics

36
Q

Can you tell what stage a deep tissue injury is?
* Often confused w/

A

Nope, its unstageable

From the top you can’t tell how deep the wound is because the skin isnt broken
* However, its normally black

Its non-blanchable

**Often confused w/ a stage 1 compression injury
* dilinate w/ blanching test (stage 1 should be blanchable)
* Also a stage 1 is more red while this is a blacker color
* Theres so much ischemia there is tissue death, it just hasnt opened
**

37
Q

This is proper positioning to place a pt for pressure injuries
* can float heels if pt is supine (often done wrong nothing should be touching the heels)

To turn from supine they’re going to need to be in this position
* can use wedge or buildup pillows

A
38
Q

Arterial Insufficiency Ulcers:
* Caused by
* What does it feel like
* What does skin look like
* Where do wounds develop? (2)

A

caused by atherosclerosis obliterans
* Arteries are not working properly and so trophic changes happen

Deep and painful wounds
* Deeper because of the location of the arterial system
* Painful because arterial system provides blood to NS which gets painful if it doesnt have blood

Skin is cold and pale, hairlessness - due to trophic changes

Wounds develop over the lateral malleolus and toes - because of the way the arterial system runs

39
Q

Venous Insufficiency Ulcers:
* causes
* What do the wounds feel like
* Do they have a pulse?
* Symptoms (3)
* Where do wounds develop?

A

causes: anything that causes venous hypertension
* Venous thrombosis
* Varicose veins
* Otehr venous problems

Painless and superficial wounds, good peripheral pulses
* So the arteries are what cause the pulse not the veins
* This is a good way to deliniate the two

Symptoms:
* Edema present
* Hemosiderine staining (darkening of skin)
* Inverted champne bottle look (think upside champign bottle)

Wounds develop over medial side of ankle, gator region (above the medial malleolius)

40
Q

Prognosis for venous insufficiency ulcers
* if they’ve had more than 1 year what is the recurrence rate
* How much fibrin% = worse prognosis?
* Ankle brachial pressure index less than what = bad prognosis
* What tretment yeilds bad prognisis

A

recurrance rate w/ more than a year w/ the wound = 70%

Larger wounds = worse prognosis

Fibrin in >50% of wound surface = worse prognosis
* we don’t want the fibrin in there

Ankle-Brachial pressure index <0.8

Hx of venous stripping/ligation
* Going into vein and inserting shit
* However, if you develop wounds this is not the best thing

41
Q

Eschar means

A

Black, dried out, dead tissue

kind of looks like gangreen

however, its stable and the wound is closed
* good if its dry - we don’t want these wet

42
Q

Slough means

A

Liquified or wet dead tissue, yellow
* causes bad smell
* Will cover the wound bed - we actaully want to scrape this off

43
Q

UNdermining means

A

Bigger area of tissue that ends under the edge, like a shelf
* Ends of the wound curling in - well you have that tissue underneath that has the undermining in it
* Leads to tunneling
* Bad and can lead to bacterial formation and can open up other areas of the body

44
Q

Tunneling means

A

Tracts extending out from the wound

45
Q

Treatment for wounds

1) Prevention of additional ulcers
2) Decreasing pressure on wound
3) Wound management
4) Surgical intervention
5) Improving the nutritional status (think getting enough protein to speed up healing process)

A
46
Q

Skin Cancer:

The American Cancer Society estimates that skin cancers are the most prevalent form of cancer, eventually affecting nearly all Caucasian people older than 65 years old

Solar radiation causes most skin cancers, and protection from the sun during the first two decades of life significantly reduces risk of skin cancer

A
47
Q

Two Benign Lesion types

A

Seborrheic Keratosis

Nevi (Moles)
* as long as they are not changing you’re okay

48
Q

2 Premalignant Lesions

A

Actinic Keratosis (moles) - due to many years od exposure to UV light

Bowen Disease - does not always turn into a malignant lesion

49
Q

Two Malignant Nonmelanoma Carinoms?

A

1) Basal Cell Carinoma
2) Squamous cell carinomas

50
Q

Whats the most common malignant nonmelanoma carcinoma?
* Does it grow quickly?
* Does it metastize easily?

A

Basal cell carcinoma

Grows slowly, rarely metastasize

51
Q

second most common malignant nonmelanoma carcinomas
* does it metastasize?
* Does it grow quickly

A

Squamous cell carcinoma

2nd most common

Has the potential to metastasize and grows faster

52
Q

What causes Malignant Melanoma?
* where do they occur (5)
* Who gets it more
* Clinical manigestation?

A

Occurs from sun expsoure but can be found in the oral cavity, anus, vagina, esophagus and eye

Men > women

Clinical manifestation
* 70% arise from preexisting moles

53
Q

larger than a pencil errasor is problem

A
54
Q

Connective tissue malignancy that manifests as a skin or mucous membrane disorder - has 4 types
* what is this and what are the 4 types
* Risk factors
* Pathogensis
* Clinical manigestations
* How do you help it

A

Kaposi Sarcoma

Risk:
* Ummunocompromise (transplants)
* AIDS

Pathogensis: Associated w/ herpes virus

Clinical manigestations: Itching and painful lesions can impinge on nerves or organs
* dont just grow on surface but internally so they can impinge the nerve roots as well

Help by: Prevent skin breakdown and wound management
* keep it dry - don’t let the skin stab or break

55
Q

Chronic disease of skin w/ erythematous (red) plaques covered w/ a silvery scale
* Risk factors (6)
* How fast is skin turn over
* Clinical manigestations?
* Tx? (2)

A

Psoriasis - chroni, noncontagious autoimmune disease that causes raised, inflamed, red skin patches that can be itchy or painful.

Risk: - its an autoimmune disease / affected by diet that you keep (certain dies can reduce the plaques)
* Psoratic arthritis
* Rx use
* Stress
* Excessive alc use
* Smoking
* Injury to the skin

Skin turn over is 3 to 4 days versus the normal 26-28 days
* You have increased skin cells, which increases that hard plaque

Manigestations:
* Scales appear mainly in scalp, knee, elbows, and genitalia (they appear where there is hair)

Tx: Long wave UV light & ora drugs

56
Q

Lupus erythematosus
* what causes
* What organ does it affect

A

autoimmune

affects the kdineys - so it has systemic issues as well

red connective inflammatory tissue is common. Looks like rosasia - however, it has systemic pathologies

Cutaneous LupusErythematosus
* Chronic inflammatory disorder of the connective tissue
* clinical manifestations = discoid lesions

57
Q

Chronic, diffuse disease of connective tissue causing fibrosis of the skin, joints, blood vessels, and internal organins
* What causes it
* What other disease can it occur to
* What two things being expsoed to can cause this
* Other risk factor (1)
* Clinical manifestation?

A

Scleroderma (effects lungs as well)
* systemic sclerosis

Autoimmune

Can occur w/ Raynauds phenomena

Expsure to silica / chemical exposure (both can trigger that auto immune response)

Smoking can affect the severity

Clinical Manigestations:
* Cutaneous ulcerations, esophageal dusmobility
* Skin thickening, contractures, joint pain, GI complications
* Late stage = Pulmonary artery HTN, MSK pain, heart lung, and kidney involvement

58
Q

Idiopathic inflammatoy disease of the muscle (2)
* How does it progression
* What cases it
* Clinical manigestations

A

Polymyositis and Dermatomyositis

Idiopathic inflammatory disease of the muscle tissue

Progress slowly, with frequent exacerbations and remissions (much like RA)

Caused by: Unknown / autoimmune pathogensis

Clinical manigestations:
* Weakness of pelvis, neck, and pharynx
* Malaise
* Weight loss
* Fatigue and muscle wasting

its an inflammatory disorder - so you will have to dose exercise specifially to that pt to avoid an inflammatory response

59
Q

Cold injuries: - applys to our modalitties

over expsoreu to cold air or water

Frostbite

Use to be a military problem

Oathogensis and clinical manifestations

Do not rub or massage the area

Pain meds and anti-infalmmatory medications, rapid rewarming (will turn red)

A
60
Q

Burns: Exposure to any thermal, chemical, electrical or radiation source

What are:
* Zone of coagulation?
* Zone of status? (time frame)
* Zone of hypermia?

A

Zone of coaguation: cells are irreversibly damaged (can’t bring them back)

Zone of status: Injured cells may die within 24-48 hrs w/o treatment

Zone of hyperemia: site of minimal cell damage
* think just the redness after a minor burn

61
Q

What does the rule of nines tell us?

A

how much of a % of the body is burned

Burns to 50% of body would be very extreme
* used a lot in burn realm

62
Q

What layer of skin do superficial burns only affect
* Another name for these kinds of burns
* how long do they last
* are the cells fully dead
* is there blister formation?

A

Only affect the epidermis

also called first degree burns

Discomfort lasts 48 hrs - cells not fully dead

No blister formation

63
Q

Partical thickness burns go down to what layer of skin?
* another name for this kind of burn?
* Is there blister formation?

A

Goes down to the dermis

Second degree burn

Blister formation covering an extensive area

Sensitivity to cold air

64
Q

Full thickness burn reaches what layer of skin?
* Other name for it?
* are they painful?

A

reaches the SubQ

Third/Fourth degree burn

Due to prolonged exposure

Little to no pain because nerve ends are burned off

65
Q
A
66
Q

What happens in pigmentary disorders?

A

hyper/hypo pigmentation

67
Q

What do we do for blistering diseases?
* Do we pop them?

A

We do wound care for them

Don’t pop the blisters - can introduce bacteria into the wound that can cause issues

68
Q

Cutaneous Sarcoidosis
* Nonnecrotizing granulomas, inflammatory lesions
* Less common unless in wound care center

A
69
Q

What to do for skin lesions

A
70
Q

Many of these skin disorders are contagious and require careful handling by all health care progessionals to avoid spreading the infection and becoming contaminated themselves

A

Skin infections

71
Q

quiz here

A