Lameness Flashcards

1
Q

Neonates (3)

A
  • Foot lesions
  • Splay leg
  • Crushing
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2
Q

Foot lesions in nenonates (6)

A
  • Born with cartilaginous ‘slippers’
  • Farrowing pen floors hard
  • Bruising of soles within 24 hours
  • Peaks at 6-8 days
  • Gone by 14 days
  • Damage to accessory digits
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3
Q

Sucking Pigs (6)

A
  • Foot lesions
  • Knee erosion
  • Joint ill
  • Trauma
  • Tenosynovitis
  • New flooring often causes more lesions during first use
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4
Q

Erosions of the sole (6)

A
  • Accompanies bruising in neonates
  • Infection can enter to cause tenosynovitis
  • Peaks at 5-7 days
  • Disappears by 14 days
  • Accessory digit lesions peak at 7-8 days
  • Only tenosynovitis remains
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5
Q

Trauma (4)

A
  • Particularly to accessory digits
  • Slip through incorrectly-sized mesh
  • Rip easily
  • Sow may stand on piglets toes
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6
Q

Joint ill (5)

A
  • From infection with streptococci such as S. suis type 1, but also environmental e.coli, staphs and streps
  • Infection gains entry through any open wound early in life, for example, via the navel, teeth clipping, tail docking or foot injury
  • In older age groups caused by Glasser’s
  • Pyrexia
  • Responsive to antimicrobial.
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7
Q

Weaners (5)

A
  • Infectious arthritis (Glasser’s disease, streptococcal arthritis, Staphylococcus hyicus, Mycoplasma hyorhinis)
  • Trauma
  • Foot lesions
  • Neurogenic lamenesses
  • Rickets
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8
Q

Weaner Infectious Arthritis (6)

A
  • H. parasuis in Glasser’s
  • S. suis type 2 or 14
  • M. hyorhinis (3-10 weeks)
  • All give rise to hot, fluid-filled joints
  • S. suis and Glasser’s associated with fever and mortality
  • Can respond to treatment
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9
Q

Growers (6)

A
  • Consequences of earlier infectious arthritis (Glasser’s disease, streptococcal arthritis)
  • Fresh infectious arthritis (Mycoplasma hyosynoviae)
  • Bursitis
  • Trauma
  • Foot lesions
  • Neurogenic lamenesses
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10
Q

Mycoplasma hyosynoviae (7)

A
  • 35-115 kg and in recently purchased breeding stock
  • Infection oronasal at 10-12 weeks
  • Afebrile in most cases
  • Affected pigs limp, shift weight
  • Clear yellowish brown joint fluid
  • Confirm by bacterial isolation or serology
  • Treat with tiamulin, lincomycin, tetracyclines and Nsaid’s
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11
Q

Adventitous Bursitis (5)

A
  • Extremely common (85% pigs)
  • Arises when pigs housed on solid floors or slats
  • Causes 0.5 kg reduction in carcase yield through trimming
  • Prevent by early bedding on soft flooring
  • Can become infected/ ruptured if housed on abrasive floor or wet fed
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12
Q

Finishers (8)

A
Governs transport for slaughter
• Osteochondrosis dissecans
• Mycoplasma hyosynoviae
• Erysipelas
• Bursitis
• Trauma
• Consequences of earlier conditions
• Foot lesions
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13
Q

Osteochondrosis

  • age
  • what (3)
  • CS (5)
  • predisposition (4)
A

• From 1 day of age

  • Incomplete ossification of cartilage at growth plates
  • Results in collapse of joint surfaces (degenerative joint disease) and epiphyseolysis
  • Flakes of cartilage in fluid from damaged joints
  • Heavy, rapidly-growing pigs
  • Elbows of some Duroc boars
  • Slow growth may reduce (gilts)
  • Effect of loose housing
CS:
• Swaying gait and knock knees
• Sudden collapse with epiphyseolysis
• May resolve by 6-9 months of age
• Slow growth may reduce (gilts)
• Effect of loose housing
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14
Q

Young Breeding Stock (6)

A

A cause of dispute over animals fit for purpose with effects on fertility

  • Osteochondrosis dissecans
  • Mycoplasma hyosynoviae
  • Erysipelas
  • Trauma
  • Foot lesions
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15
Q

Adults (6)

A
A major cause (30-35%) of culling
• Erysipelas
• Osteochondrosis
• Foot lesions
• Trauma – particularly since all sows became loose housed
• Osteitis
• Osteoarthritis
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16
Q

Lameness in Boars

  • effects (3)
  • causes (5)
  • Tx
A
  • Effect on fertility (Mating not achieved)
  • Forelimb lameness might not affect service
  • Common cause of reluctance to serve
  • Results from many of the same causes of lameness as in sows
  • Service problems with restive sows
  • Poor flooring
  • Inadequate bedding
  • Osteochondrosis of the elbow in some Durocs

• Dilemma: to cull or not to cull; to transport or not to transport?

17
Q

Sow Lameness causes (6)

A
  • Sow to sow interaction in loose housed systems
  • Floor quality
  • Absence of bedding
  • Eroded concrete especially where wet feeding
  • Soft ground outdoors gives overgrown hooves
  • widths
  • Slurry levels and cleaning
18
Q

Fighting Sows and Lameness (7)

A
  • Lameness post weaning when mixed
  • Slippery floors can lead to broken legs and the splits, interactions make it worse
  • Wounds from fighting
  • Restricted access to food
  • Protect by providing walls and feeding individually
  • Electronic sow feeders
  • Resurface floors
19
Q

Proliferative Osteitis (5)

A
• Lameness in sows
• Haemorrhage and osteophytes 
– Femoral greater trochanter 
– Humeral medial condyle
• First parity gilts after weaning
20
Q

Foot Lesions (4)

Dealing With Foot Lesions (5)

A
  • Footrot/ Bush Foot
  • Laminitis
  • Overgrowth and deformity of the claws
  • Erosive foot lesions
  • Trim feet (may require sedation/ handling facilities)
  • Treat infections (but surgical debridement may be required)
  • Remove digits (economically unviable)
  • Correct flooring
  • Foot baths
21
Q

Managing the Lame Sow (6)

A
  • Identify cause of lameness
  • Decide whether too lame to recover
  • Pen separately
  • Provide warmth and bedding, ensure water and food within reach
  • Do not serve with a boar
  • Help to stand from time to time
22
Q

Culling Policy (5)

A
  • Cull for lameness but transport a problem
  • May decide to keep until ready
  • Fractures, fistulae etc can break down during transport
  • Kill if in doubt
  • If decide to kill, kill safely if free bullet