L3- last of 'the tour' Flashcards
three types of junctions b/w a tooth and periodontal tissues
+ brief description
- gomphosis joint - via PDL, cementum and alveolar bone (bundle bone, cribriform plate, inner cortical plate
- Dentinocementum Junction (DCJ) - involves the hyaline layer of hopewell smith and cements the cementum to the mantle dentin
- CEJ - cementoenamel junction
- butt joint b/w cementum and enamel
- overlap of cementum onto enamel
- gap between cementum and enamel
what is cementum - general
principle function?
calcified covering over the dentin of the roots (50-200 um)
principle function = provide anchorage of tooth to alveolus through the collagen bundles of periodontal ligament SHARPEYS FIBERS
HARD AVASCULAR C.T.
- chemical structure more like bone
- mineralized outer layer - collagen fibers of periodontal ligament are inserted
T/F cementum becomes pathologically altered during Perio disease
TRUE – it can be lost following disease process and is associated with periodontal tx.
spontaneous and therapeutic regeneration of cementum has been difficult to achieve
major difference between bone and cementum
Cementum is AVASCULAR – so it NO OSTEON -LIKE STRUCTURES
- gets nutrition / vascular diffusion from PDL - not osteons
- relatively thin and limited repair /regeneration
is cementum a reservoir of minerals?
NO – therfore more static than bone
function of cementum
- attachment/ support via acellular cementum plus the hyaline layer
- adapation/ protection - during tooth movement and weating - via cellular cementum
- assist in mainting occlusal relationships - maintain width of PDL at apex
relationship between attirtion and cementum
attrition is the wear of the occlusal surfaces and with this there is continual erupting as a result which is termed ACTIVE ERUPTION – which is proportional to the amount of attrition – this process is accomplished via cementum deposits of cellular cementum at the apical ends of roots - which push tooth up
supraeruption
if no opposing force of tooth – continues to erupt via the deposition of cellular cementum at the apex of teeth with no antagonist tooth
which is less susceptible to resorption; alveolar bone or cementum?
cementum - so provides protection too
is cementum poorly mineralized?
composition?
yes - poorly mineralized.
60% inorganic (opposed to dentin which is 70% inorganic and enamel which is 95% inorganic)
bone composition
mineral
organic
water
mineral/ inorganic = 45%
organic = 30%
water = 25%
cementum vs bone
cementum contains ONLY TYPE I COLLAGEN
- more permeable
- no extensive remodeling like bone so DOES NOT TURN OVER – NOT RESORBED PHYSIOLOGICALLY
- 60% inorganic
- 27 % organic
ALVEOLAR bone
- 45 % inorganic
- 30% organic
- type I and type II collagen
resorption in cementum?
NOT PHYSIOLOGICALLY BUT IN PATHOLOGICAL CONDITIONS
- resorption due to inflammation
- resorption due to orthodontic tooth movement
cellular vs. acellular cementum
where?
turnover?
Cellular –> more towards apex and cementocytes get trapped in lacunae / in matrix as it calcifies (origin of cementocytes)
acellular –> covers coronal aspect of the root and has NO CEMENTOCYTES and after tooth errupts there is no longer any formation of this
two types of collagen in cementum
- intrinsic fibers – during initial formation and as the cementoblasts lay it down and calcification occurs – become trapped
- sharpeys fibers that get imbedded within the cementum from the PDL
- formed by fibroblasts
major cell types associated with cementum
- cementoblasts
- cementocytes
- PDL ligament fibroblasts
- odontoclasts/ cementoclasts
cementoblasts - origin and describe
from ectomesenchymal layer within the dental follicle – later on may come from undifferentiated cells in the PDL
- morphologically similar to fibroblasts - but located in close proximity to the cemental surface and extend cytoplasmic processes towards cementum
what do cementoblasts produce? how is this arranged
produce the intrinsic collagen fibers and their long axis is more/less parallel to tooth surface
(opposed to the extrinsic collagen fibers from PDL that are more/less perpendicular to tooth surface)
what secretes cementum matrix?
rate?
location?
cementoblasts do by apposition at the cemental surface
- grows slowly in thickess throughout life - but NOT REMODELED continuously (like bone)
cementocytes
how produced?
over rapid periods of cementogenesis - cementoblasts that become TRAPPED W/IN LUCUNAE – then have cytoplasmic processes extending toward cemental surface through canaliculi
- less organelles = less metabolic/ very little metabolic activity
periodontal fibroblasts considered cementum cells?
YES – b/c although not produced by them they become mineralized as they become incorporated into cementum - so they do contribute to cementogenesis
odontoclasts aka?
function?
type of cell?
aka –> cementoclasts
multi-nucleated giant cells
involved in the resorption process but are indistinguishable from osteoclasts
three relationships between cementum and enamel at the CEJ?
percentage of each?
can they co-exist?
30% = Butt joint – > do not overlap but meet up against each other
60-65% = OVERLAP (cementum overlaps enamel)
5-10% = GAP b/w the enamel and cementum - which can expose dentin on root - may have root sensativity problem
*these realtionships can co-exist on a single tooth
cementicles
describe + types?
implications
developmental anormality in which a clump of afibrillar or fibrillar cementum (or mixture) gets either attached/sessile to the cemental surface or lie free within the pdl adjacent to cementum – can mimick calculus and the attached ones may serve as a promoter of periodontal disease if exposed
imbedded ones have no clinical relevance
is cementum usually exposed?
clinical correlations
NO – usually covered by alveolar bone and gingiva
EXPOSED W/ GINGIVAL RECESSION OR PERIO POCKETS
PERMEABLE TO PLAQUE BACTERIA – so can get to the dentin subjacent to pathologically exposed cementum
can CT cells adhere to previously exposed cementum?
implication?
NO – cannot adhere to previously exposed OR toxin-containing cementum
clinical correlations to scaling and root planning relating to cementum
this process is aimed at removing claculus and bacterial deposits often removes the relatively thin layer of cementum — which leaves underlying surface with a ‘smear-layer’ and unknown findings whether or not this layer allows for new cementum formation – may be a clinically weaker junction now
new cementum formation? goal?
because cementum mediated attachment of the tooth to the PDL and bone this is a high sought out therapeutic measure - however limited repair and regeneration capacity
can alveolar bone fuse directly with cementum or dentin?
what is this called?
YES – ankylosis but this is undeserible b/c get increased resoprtion from osteoclast activity
function of the hyaline layer of cementum?
‘cements’ the cementum to the MANTLE dentin
acellular cementum primary function
attachment function
cellular secondary cementum function
ADAPTION
what makes PDL ligament ‘not’ a ligament in terms of its functions and characteristics
- rich vascular supply with array of cellular components for allowing homeostasis and repair
- hydrodynamic interactions is a key function
- vascular and lymphatic vessels to PDL supply nutrients and remove metabolic by-products
may be called PERIODONTAL MEMBRANE b/c ligament is a misnomer as a ligament is (usually) dense, avascular, band of connective tissue with limited ability to repair /heal
T/F the PDL contains the progenitor cells of the dental follicle necessary for regeneration of cementum and bone as well as PDL?
TRUE
five main key functions of the PDL?
- supportive
- nutritive
- sensory
- formative
- healing
extracellular fibers of PDL composition?
function?
composed mainly of collagens and associated molecules that CONNECT cementum with alveolar bone
cells in PDL that are responsible for maintenance and regeneration?
fibroblasts, neurovascualr elements of periodontal tissues like progenitor undifferentiated cells
how does PDL carry out adaption? via?
viscoelastic components
- designed primarily to withstand he considerable forces of mastication and/or bruxism & nocturnal grinding of teeth
how does PDL carry out attachement? via?
via tensile strengh components desginged to connect teeth to bone
how does PDL carry out sensory perception? via?
nerves – designed both to properly position the jaws during normal function and to withstanf the occlusal forces
how does PDL absorb light/moderate/ heavy forces?
light – ibtravascular fluid pushes out of blood vessels
moderate – extravascular fluids forced out of pdl space into adjacent marrow spaced
heavier– taken up by principle fibers
healing of bone grafts?
via the PDL