John's Part pg 45-52 Flashcards

1
Q

The forehead movement area is usually moved in bilateral symmetry, and it usually spared in unilateral lesions involving corticobulbar pathways….why is its movements usually spared?

A

equal innervation from each cerebral hemisphere will keep the movements going….ask me for any question on this one, i tried to make it make sense

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2
Q

what does the stapedius muscle do

A

dampens the movement of the tympanic membrane

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3
Q

what supplies parasympathetic secretory and vasodilators to the submax and subling salivary glands and to the lacrimal glands, as well as the membrane of mouth and tonngue

A

the chordatympani

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4
Q

what does parasympathetic stim of CN VII do to mucus

A

increase secretion of thin and watery mucus

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5
Q

what does sympathetic stim of CN VII do to mucus

A

causes a scant supply of thick and turbid saliva

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6
Q

what is “prosopoplegia”

A

peripheral facial paralysis

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7
Q

where could a peripheral lesion of CN VII be located?

A

pontine nuclei, facial canal, geniculate ganglion, peripheral to geniuculate gang, but central to stapedius branch, peripheral to stapedius branch, but central to chordatympani nerve, body of the parotid, in the chordatympani

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8
Q

where is the lesion of bells palsy?

A

peripheral to the geniculate ganglion

*all peripheral lesions of CN VII will have a “flaccid paralysis” of all ipsilateral face muscles distal to lesion

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9
Q

so, what can happen with central lesions

A

it will show more as “signs of weakness” bc of the bilateral innervation to forehead…
eyes are only partially involved
mouth and neck are fully involved

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10
Q

exam of CN VII

A

have them look up, examine wrinkles

have them smile and frown, puff out cheeks

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11
Q

CN VII does some special sense, right? well, what does a central lesion do?

A

taste for anterior 2/3 of tongue, a lesion will rarely cause a complete loss of taste…
lesions in cranial nerve VII must be proximal to the stylomastoid foramen to affect taste

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12
Q

what is a complete loss of taste called?

A

aquesia

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13
Q

quick…the pathway of the cochlear nerve

A

leaves and goes to cochlear nuclei, to the ipsilateral lateral lemniscus, ascend to the inferior colliculus and the medial geniculate body, they terminate on the cortex of the hescel’s gyrus

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14
Q

what is the loss of hearing

A

hypoacusis, with or without tinnitus

probably a conduction loss, receptor disease, or lesion

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15
Q

the 2 major types of hearing loss

A

conductive and sensorineural

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16
Q

sensorineural hearing loss will be caused by what

A

usually attributed to disease of organ of corti

17
Q

conductive hearing loss will be caused by what

A

auditory canal obstruction
tympanic membrane -> direct or indirect trauma
ossicles: trauma or advanced aging
accumulation of fluid in middle ear—rarely chronic ear infxns

18
Q

vestibular diseases are always accompanied by?

other symptoms?

A

vertigo

nausea, anxiety, and oscillopsia ( visual perception of rapid to and fro movements often accompanying nystagmus)

19
Q

caloric irrigation

COWS

A

Cold opposite - warm same

cold water causes nystagmus on opposite side

20
Q

cervicogenic proprioceptive disease test

A

patient, in swivel chair, grab their head and have them rotate shoulders, if reproduction of symptoms occurs, disease is suspected.

21
Q

what is schwabach’s test

A

place buzzing tuning fork in front of patients EAM, when they cannot hear it any longer then put it up next to your ear, use your own hearing to compare with the patients

22
Q

dont forget the other tests for CN VIII

A

watch tick, weber, rinne, finger rustle

23
Q

what is the malingering test

A

a loud noise from out of sight should cause the patient to blink